Ultrafine (nanometre) particle mediated lung injury

[1]  K. Donaldson,et al.  Differences in the extent of inflammation caused by intratracheal exposure to three ultrafine metals: role of free radicals. , 1998, Journal of toxicology and environmental health. Part A.

[2]  D. Costa,et al.  Soluble transition metals mediate residual oil fly ash induced acute lung injury. , 1997, Journal of toxicology and environmental health.

[3]  W. MacNee,et al.  Pro-inflammatory effect of particulate air pollution (PM10) In vivo and In vitro , 1997 .

[4]  David M. Brown,et al.  Surface Free Radical Activity of PM10 and Ultrafine Titanium Dioxide: A Unifying Factor in Their Toxicity? , 1997 .

[5]  W. MacNee,et al.  Adverse health effects of PM10 particles: involvement of iron in generation of hydroxyl radical. , 1996, Occupational and environmental medicine.

[6]  P. S. Gilmour,et al.  Free radical activity and pro-inflammatory effects of particulate air pollution (PM10) in vivo and in vitro. , 1996, Thorax.

[7]  W. MacNee,et al.  Systemic oxidative stress in asthma, COPD, and smokers. , 1996, American journal of respiratory and critical care medicine.

[8]  J. Finkelstein,et al.  Characterization of the early pulmonary inflammatory response associated with PTFE fume exposure. , 1996, Toxicology and applied pharmacology.

[9]  G. Oberdörster,et al.  Significance of particle parameters in the evaluation of exposure-dose-response relationships of inhaled particles , 1996 .

[10]  J. Paulauskis,et al.  Regulation of Macrophage Inflammatory Protein-1 mRNA by Oxidative Stress (*) , 1996, The Journal of Biological Chemistry.

[11]  W. Pepelko,et al.  Pulmonary inflammatory, chemokine, and mutagenic responses in rats after subchronic inhalation of carbon black. , 1996, Toxicology and applied pharmacology.

[12]  L. J. King,et al.  Physiologically Based Pharmacokinetics of Uptake by Inhalation of a Series of 1,1,1-Trihaloethanes: Correlation with Various Physicochemical Parameters , 1996 .

[13]  J. Mauderly,et al.  Particle overload in the rat lung and lung cancer : implications for human risk assessment : proceedings of a conference held at the Massachusetts Institute of Technology on March 29 and 30, 1995 , 1996 .

[14]  J. Carter,et al.  Characterizing mutagenesis in the hprt gene of rat alveolar epithelial cells. , 1995, Experimental lung research.

[15]  M. Luster,et al.  Iron and reactive oxygen species in the asbestos-induced tumor necrosis factor-alpha response from alveolar macrophages. , 1995, American journal of respiratory cell and molecular biology.

[16]  W. MacNee,et al.  Particulate air pollution and acute health effects , 1995, The Lancet.

[17]  Wolfgang Koch,et al.  Chronic Inhalation Exposure of Wistar Rats and two Different Strains of Mice to Diesel Engine Exhaust, Carbon Black, and Titanium Dioxide , 1995 .

[18]  M Volante,et al.  Physicochemical properties of crystalline silica dusts and their possible implication in various biological responses. , 1995, Scandinavian journal of work, environment & health.

[19]  B. Weiss,et al.  Association of particulate air pollution and acute mortality: involvement of ultrafine particles? , 1995, Inhalation toxicology.

[20]  B. Lehnert,et al.  Correlation Between Particle Size, in Vivo Particle Persistence, and Lung Injury , 1994 .

[21]  H. Pahl,et al.  Regulation of the transcription factors NF-kappa B and AP-1 by redox changes. , 1994, Chemico-biological interactions.

[22]  P. Borm,et al.  Increased expression of manganese-containing superoxide dismutase in rat lungs after inhalation of inflammatory and fibrogenic minerals. , 1994, Free Radical Biology & Medicine.

[23]  P. Baeuerle,et al.  Function and activation of NF-kappa B in the immune system. , 1994, Annual review of immunology.

[24]  G. Oberdörster,et al.  Pulmonary retention of ultrafine and fine particles in rats. , 1992, American journal of respiratory cell and molecular biology.

[25]  S. Weitzman,et al.  The role of free radicals in asbestos-induced diseases. , 1992, Free radical biology & medicine.

[26]  P. Morrow,et al.  Volumetric loading of alveolar macrophages (AM): a possible basis for diminished AM-mediated particle clearance. , 1992, Experimental lung research.

[27]  J. Ferin,et al.  Increased pulmonary toxicity of ultrafine particles? II. Lung lavage studies , 1990 .

[28]  Robert Gelein,et al.  Increased pulmonary toxicity of ultrafine particles? I. Particle clearance, translocation, morphology , 1990 .

[29]  K. Driscoll,et al.  Differential effects of mineral dusts on the in vitro activation of alveolar macrophage eicosanoid and cytokine release. , 1990, Toxicology in vitro : an international journal published in association with BIBRA.

[30]  K. Donaldson,et al.  Inflammation generating potential of long and short fibre amosite asbestos samples. , 1989, British journal of industrial medicine.

[31]  N. Dalal,et al.  Detection of reactive free radicals in fresh coal mine dust and their implication for pulmonary injury. , 1989, The Annals of occupational hygiene.

[32]  Xianglin Shi,et al.  Generation of free radicals from freshly fractured silica dust. Potential role in acute silica-induced lung injury. , 1988, The American review of respiratory disease.

[33]  P. Morrow,et al.  Possible mechanisms to explain dust overloading of the lungs. , 1988, Fundamental and applied toxicology : official journal of the Society of Toxicology.

[34]  J Addison,et al.  The pathogenicity of long versus short fibre samples of amosite asbestos administered to rats by inhalation and intraperitoneal injection. , 1986, British journal of experimental pathology.

[35]  A. C. Allison,et al.  AN EXAMINATION OF THE CYTOTOXIC EFFECTS OF SILICA ON MACROPHAGES , 1966, The Journal of experimental medicine.