Ultrafine (nanometre) particle mediated lung injury
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W. MacNee | K. Donaldson | X. Li | W. Macnee
[1] K. Donaldson,et al. Differences in the extent of inflammation caused by intratracheal exposure to three ultrafine metals: role of free radicals. , 1998, Journal of toxicology and environmental health. Part A.
[2] D. Costa,et al. Soluble transition metals mediate residual oil fly ash induced acute lung injury. , 1997, Journal of toxicology and environmental health.
[3] W. MacNee,et al. Pro-inflammatory effect of particulate air pollution (PM10) In vivo and In vitro , 1997 .
[4] David M. Brown,et al. Surface Free Radical Activity of PM10 and Ultrafine Titanium Dioxide: A Unifying Factor in Their Toxicity? , 1997 .
[5] W. MacNee,et al. Adverse health effects of PM10 particles: involvement of iron in generation of hydroxyl radical. , 1996, Occupational and environmental medicine.
[6] P. S. Gilmour,et al. Free radical activity and pro-inflammatory effects of particulate air pollution (PM10) in vivo and in vitro. , 1996, Thorax.
[7] W. MacNee,et al. Systemic oxidative stress in asthma, COPD, and smokers. , 1996, American journal of respiratory and critical care medicine.
[8] J. Finkelstein,et al. Characterization of the early pulmonary inflammatory response associated with PTFE fume exposure. , 1996, Toxicology and applied pharmacology.
[9] G. Oberdörster,et al. Significance of particle parameters in the evaluation of exposure-dose-response relationships of inhaled particles , 1996 .
[10] J. Paulauskis,et al. Regulation of Macrophage Inflammatory Protein-1 mRNA by Oxidative Stress (*) , 1996, The Journal of Biological Chemistry.
[11] W. Pepelko,et al. Pulmonary inflammatory, chemokine, and mutagenic responses in rats after subchronic inhalation of carbon black. , 1996, Toxicology and applied pharmacology.
[12] L. J. King,et al. Physiologically Based Pharmacokinetics of Uptake by Inhalation of a Series of 1,1,1-Trihaloethanes: Correlation with Various Physicochemical Parameters , 1996 .
[13] J. Mauderly,et al. Particle overload in the rat lung and lung cancer : implications for human risk assessment : proceedings of a conference held at the Massachusetts Institute of Technology on March 29 and 30, 1995 , 1996 .
[14] J. Carter,et al. Characterizing mutagenesis in the hprt gene of rat alveolar epithelial cells. , 1995, Experimental lung research.
[15] M. Luster,et al. Iron and reactive oxygen species in the asbestos-induced tumor necrosis factor-alpha response from alveolar macrophages. , 1995, American journal of respiratory cell and molecular biology.
[16] W. MacNee,et al. Particulate air pollution and acute health effects , 1995, The Lancet.
[17] Wolfgang Koch,et al. Chronic Inhalation Exposure of Wistar Rats and two Different Strains of Mice to Diesel Engine Exhaust, Carbon Black, and Titanium Dioxide , 1995 .
[18] M Volante,et al. Physicochemical properties of crystalline silica dusts and their possible implication in various biological responses. , 1995, Scandinavian journal of work, environment & health.
[19] B. Weiss,et al. Association of particulate air pollution and acute mortality: involvement of ultrafine particles? , 1995, Inhalation toxicology.
[20] B. Lehnert,et al. Correlation Between Particle Size, in Vivo Particle Persistence, and Lung Injury , 1994 .
[21] H. Pahl,et al. Regulation of the transcription factors NF-kappa B and AP-1 by redox changes. , 1994, Chemico-biological interactions.
[22] P. Borm,et al. Increased expression of manganese-containing superoxide dismutase in rat lungs after inhalation of inflammatory and fibrogenic minerals. , 1994, Free Radical Biology & Medicine.
[23] P. Baeuerle,et al. Function and activation of NF-kappa B in the immune system. , 1994, Annual review of immunology.
[24] G. Oberdörster,et al. Pulmonary retention of ultrafine and fine particles in rats. , 1992, American journal of respiratory cell and molecular biology.
[25] S. Weitzman,et al. The role of free radicals in asbestos-induced diseases. , 1992, Free radical biology & medicine.
[26] P. Morrow,et al. Volumetric loading of alveolar macrophages (AM): a possible basis for diminished AM-mediated particle clearance. , 1992, Experimental lung research.
[27] J. Ferin,et al. Increased pulmonary toxicity of ultrafine particles? II. Lung lavage studies , 1990 .
[28] Robert Gelein,et al. Increased pulmonary toxicity of ultrafine particles? I. Particle clearance, translocation, morphology , 1990 .
[29] K. Driscoll,et al. Differential effects of mineral dusts on the in vitro activation of alveolar macrophage eicosanoid and cytokine release. , 1990, Toxicology in vitro : an international journal published in association with BIBRA.
[30] K. Donaldson,et al. Inflammation generating potential of long and short fibre amosite asbestos samples. , 1989, British journal of industrial medicine.
[31] N. Dalal,et al. Detection of reactive free radicals in fresh coal mine dust and their implication for pulmonary injury. , 1989, The Annals of occupational hygiene.
[32] Xianglin Shi,et al. Generation of free radicals from freshly fractured silica dust. Potential role in acute silica-induced lung injury. , 1988, The American review of respiratory disease.
[33] P. Morrow,et al. Possible mechanisms to explain dust overloading of the lungs. , 1988, Fundamental and applied toxicology : official journal of the Society of Toxicology.
[34] J Addison,et al. The pathogenicity of long versus short fibre samples of amosite asbestos administered to rats by inhalation and intraperitoneal injection. , 1986, British journal of experimental pathology.
[35] A. C. Allison,et al. AN EXAMINATION OF THE CYTOTOXIC EFFECTS OF SILICA ON MACROPHAGES , 1966, The Journal of experimental medicine.