Effects of chronic administration of MK-801 upon local cerebral glucose utilisation and ligand binding to the NMDA receptor complex

[1]  M. Johnston,et al.  MK-801 pretreatment enhances N-methyl-d-aspartate-mediated brain injury and increases brain N-methyl-d-aspartate recognition site binding in rats , 1990, Neuroscience.

[2]  J. Garthwaite,et al.  Excitatory amino acid neurotoxicity and neurodegenerative disease. , 1990, Trends in pharmacological sciences.

[3]  J. Mcculloch,et al.  The effects of N-methyl-D-aspartate receptor blockade with MK-801 upon the relationship between cerebral blood flow and glucose utilisation , 1990, Brain Research.

[4]  L. Iversen,et al.  Phencyclidine, dizocilpine, and cerebrocortical neurons. , 1990, Science.

[5]  D. Manallack,et al.  Subchronic administration of MK-801 in the rat decreases cortical binding of [3H]d-AP5, suggesting down-regulation of the corticalN-methyl-d-aspartate receptors , 1989, Neuroscience.

[6]  J. Mcculloch,et al.  Journal of Cerebral Blood Flow and Metabolism Effects of Mk-801 upon Local Cerebral Glucose Utilisation in Conscious Rats and in Rats Anaesthetised with Halothane , 2022 .

[7]  J. Mcculloch,et al.  Effects of NMDA antagonists, MK-801 and CPP, upon local cerebral glucose use , 1989, Brain Research.

[8]  S. Iversen,et al.  The behavioural effects of MK-801: a comparison with antagonists acting non-competitively and competitively at the NMDA receptor. , 1989, European journal of pharmacology.

[9]  J. Olney,et al.  Pathological changes induced in cerebrocortical neurons by phencyclidine and related drugs. , 1989, Science.

[10]  J. Liebman,et al.  Behavioral tolerance and sensitization to CGS 19755, a competitive N-methyl-D-aspartate receptor antagonist. , 1988, The Journal of pharmacology and experimental therapeutics.

[11]  D. Graham,et al.  Focal Cerebral Ischaemia in the Cat: Treatment with the Glutamate Antagonist MK-801 after Induction of Ischaemia , 1988, Journal of Cerebral Blood Flow and Metabolism.

[12]  J. Mcculloch,et al.  The glutamate antagonist MK‐801 reduces focal ischemic brain damage in the rat , 1988, Annals of neurology.

[13]  W. Koek,et al.  MK-801, a proposed noncompetitive antagonist of excitatory amino acid neurotransmission, produces phencyclidine-like behavioral effects in pigeons, rats and rhesus monkeys. , 1988, The Journal of pharmacology and experimental therapeutics.

[14]  E. Wong,et al.  Quantitative autoradiography of [3H]‐MK‐801 binding sites in mammalian brain , 1988, British journal of pharmacology.

[15]  D. Graham,et al.  Protective Effect of the Glutamate Antagonist, MK-801 in Focal Cerebral Ischemia in the Cat , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[16]  E. Wong,et al.  [3H]MK‐801 Labels a Site on the N‐Methyl‐D‐Aspartate Receptor Channel Complex in Rat Brain Membranes , 1988, Journal of neurochemistry.

[17]  R. C. Collins,et al.  The functional anatomy and pathology of lithium-pilocarpine and high-dose pilocarpine seizures , 1987, Neuroscience.

[18]  R. Gill,et al.  Systemic administration of MK-801 protects against ischemia-induced hippocampal neurodegeneration in the gerbil , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[19]  E. London,et al.  Alterations in local cerebral glucose utilization induced by phencyclidine , 1987, Brain Research.

[20]  L. Iversen,et al.  The anticonvulsant MK-801 is a potent N-methyl-D-aspartate antagonist. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[21]  N. Bowery,et al.  Light microscopic autoradiographic localisation of [3H]glycine and [3H]strychnine binding sites in rat brain. , 1986, European journal of pharmacology.

[22]  C. Cotman,et al.  Distribution of N-methyl-D-aspartate-sensitive L-[3H]glutamate-binding sites in rat brain , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[23]  R. C. Collins,et al.  Focal cortical seizures cause distant thalamic lesions. , 1982, Science.

[24]  C. Pert,et al.  Chronic phencyclidine treatment decreases phencyclidine and dopamine receptors in rat brain , 1982, Pharmacology Biochemistry and Behavior.

[25]  S. Castellani,et al.  Acute and chronic phencyclidine effects on locomotor activity, stereotypy and ataxia in rats. , 1981, European journal of pharmacology.

[26]  Y. Ben-Ari,et al.  Electrographic, clinical and pathological alterations following systemic administration of kainic acid, bicuculline or pentetrazole: Metabolic mapping using the deoxyglucose method with special reference to the pathology of epilepsy , 1981, Neuroscience.

[27]  S. D. Glick,et al.  Localisation of phencyclidine-induced changes in brain energy metabolism , 1979, Nature.

[28]  M. Reivich,et al.  THE [14C]DEOXYGLUCOSE METHOD FOR THE MEASUREMENT OF LOCAL CEREBRAL GLUCOSE UTILIZATION: THEORY, PROCEDURE, AND NORMAL VALUES IN THE CONSCIOUS AND ANESTHETIZED ALBINO RAT 1 , 1977, Journal of neurochemistry.

[29]  Choi Dw Methods for antagonizing glutamate neurotoxicity. , 1990 .

[30]  P. Contreras,et al.  PCP‐induced alterations in cerebral glucose utilization in rat brain: Blockade by metaphit, a PCP‐receptor‐acylating agent , 1987, Synapse.

[31]  G. Paxinos,et al.  The Rat Brain in Stereotaxic Coordinates , 1983 .

[32]  B. Arison,et al.  Disposition and metabolism of (+)-5-methyl-10,11-dihydro-5H-dibenzo[a,d] cyclohepten-5,10-imine in rats, dogs, and monkeys. , 1983, Drug metabolism and disposition: the biological fate of chemicals.

[33]  J. Mcculloch Mapping Functional Alterations in the CNS With [14C]Deoxyglucose , 1982 .

[34]  B. Clineschmidt,et al.  Anticonvulsant activity of (+)‐5‐methyl‐10, 11‐dihydro‐5H‐dibenzo[a, d]cyclohepten‐5, 10‐imine (MK‐801), a substance with potent anticonvulsant, central sympathomimetic, and apparent anxiolytic properties , 1982 .