Effects of chronic administration of MK-801 upon local cerebral glucose utilisation and ligand binding to the NMDA receptor complex
暂无分享,去创建一个
[1] M. Johnston,et al. MK-801 pretreatment enhances N-methyl-d-aspartate-mediated brain injury and increases brain N-methyl-d-aspartate recognition site binding in rats , 1990, Neuroscience.
[2] J. Garthwaite,et al. Excitatory amino acid neurotoxicity and neurodegenerative disease. , 1990, Trends in pharmacological sciences.
[3] J. Mcculloch,et al. The effects of N-methyl-D-aspartate receptor blockade with MK-801 upon the relationship between cerebral blood flow and glucose utilisation , 1990, Brain Research.
[4] L. Iversen,et al. Phencyclidine, dizocilpine, and cerebrocortical neurons. , 1990, Science.
[5] D. Manallack,et al. Subchronic administration of MK-801 in the rat decreases cortical binding of [3H]d-AP5, suggesting down-regulation of the corticalN-methyl-d-aspartate receptors , 1989, Neuroscience.
[6] J. Mcculloch,et al. Journal of Cerebral Blood Flow and Metabolism Effects of Mk-801 upon Local Cerebral Glucose Utilisation in Conscious Rats and in Rats Anaesthetised with Halothane , 2022 .
[7] J. Mcculloch,et al. Effects of NMDA antagonists, MK-801 and CPP, upon local cerebral glucose use , 1989, Brain Research.
[8] S. Iversen,et al. The behavioural effects of MK-801: a comparison with antagonists acting non-competitively and competitively at the NMDA receptor. , 1989, European journal of pharmacology.
[9] J. Olney,et al. Pathological changes induced in cerebrocortical neurons by phencyclidine and related drugs. , 1989, Science.
[10] J. Liebman,et al. Behavioral tolerance and sensitization to CGS 19755, a competitive N-methyl-D-aspartate receptor antagonist. , 1988, The Journal of pharmacology and experimental therapeutics.
[11] D. Graham,et al. Focal Cerebral Ischaemia in the Cat: Treatment with the Glutamate Antagonist MK-801 after Induction of Ischaemia , 1988, Journal of Cerebral Blood Flow and Metabolism.
[12] J. Mcculloch,et al. The glutamate antagonist MK‐801 reduces focal ischemic brain damage in the rat , 1988, Annals of neurology.
[13] W. Koek,et al. MK-801, a proposed noncompetitive antagonist of excitatory amino acid neurotransmission, produces phencyclidine-like behavioral effects in pigeons, rats and rhesus monkeys. , 1988, The Journal of pharmacology and experimental therapeutics.
[14] E. Wong,et al. Quantitative autoradiography of [3H]‐MK‐801 binding sites in mammalian brain , 1988, British journal of pharmacology.
[15] D. Graham,et al. Protective Effect of the Glutamate Antagonist, MK-801 in Focal Cerebral Ischemia in the Cat , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[16] E. Wong,et al. [3H]MK‐801 Labels a Site on the N‐Methyl‐D‐Aspartate Receptor Channel Complex in Rat Brain Membranes , 1988, Journal of neurochemistry.
[17] R. C. Collins,et al. The functional anatomy and pathology of lithium-pilocarpine and high-dose pilocarpine seizures , 1987, Neuroscience.
[18] R. Gill,et al. Systemic administration of MK-801 protects against ischemia-induced hippocampal neurodegeneration in the gerbil , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] E. London,et al. Alterations in local cerebral glucose utilization induced by phencyclidine , 1987, Brain Research.
[20] L. Iversen,et al. The anticonvulsant MK-801 is a potent N-methyl-D-aspartate antagonist. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[21] N. Bowery,et al. Light microscopic autoradiographic localisation of [3H]glycine and [3H]strychnine binding sites in rat brain. , 1986, European journal of pharmacology.
[22] C. Cotman,et al. Distribution of N-methyl-D-aspartate-sensitive L-[3H]glutamate-binding sites in rat brain , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[23] R. C. Collins,et al. Focal cortical seizures cause distant thalamic lesions. , 1982, Science.
[24] C. Pert,et al. Chronic phencyclidine treatment decreases phencyclidine and dopamine receptors in rat brain , 1982, Pharmacology Biochemistry and Behavior.
[25] S. Castellani,et al. Acute and chronic phencyclidine effects on locomotor activity, stereotypy and ataxia in rats. , 1981, European journal of pharmacology.
[26] Y. Ben-Ari,et al. Electrographic, clinical and pathological alterations following systemic administration of kainic acid, bicuculline or pentetrazole: Metabolic mapping using the deoxyglucose method with special reference to the pathology of epilepsy , 1981, Neuroscience.
[27] S. D. Glick,et al. Localisation of phencyclidine-induced changes in brain energy metabolism , 1979, Nature.
[28] M. Reivich,et al. THE [14C]DEOXYGLUCOSE METHOD FOR THE MEASUREMENT OF LOCAL CEREBRAL GLUCOSE UTILIZATION: THEORY, PROCEDURE, AND NORMAL VALUES IN THE CONSCIOUS AND ANESTHETIZED ALBINO RAT 1 , 1977, Journal of neurochemistry.
[29] Choi Dw. Methods for antagonizing glutamate neurotoxicity. , 1990 .
[30] P. Contreras,et al. PCP‐induced alterations in cerebral glucose utilization in rat brain: Blockade by metaphit, a PCP‐receptor‐acylating agent , 1987, Synapse.
[31] G. Paxinos,et al. The Rat Brain in Stereotaxic Coordinates , 1983 .
[32] B. Arison,et al. Disposition and metabolism of (+)-5-methyl-10,11-dihydro-5H-dibenzo[a,d] cyclohepten-5,10-imine in rats, dogs, and monkeys. , 1983, Drug metabolism and disposition: the biological fate of chemicals.
[33] J. Mcculloch. Mapping Functional Alterations in the CNS With [14C]Deoxyglucose , 1982 .
[34] B. Clineschmidt,et al. Anticonvulsant activity of (+)‐5‐methyl‐10, 11‐dihydro‐5H‐dibenzo[a, d]cyclohepten‐5, 10‐imine (MK‐801), a substance with potent anticonvulsant, central sympathomimetic, and apparent anxiolytic properties , 1982 .