Diacylglycerol Kinase alpha is Involved in the Vitamin E-Induced Amelioration of Diabetic Nephropathy in Mice

[1]  J. B. Lopes de Faria,et al.  The use of green tea polyphenols for treating residual albuminuria in diabetic nephropathy: A double-blind randomised clinical trial , 2016, Scientific Reports.

[2]  C. Mohan,et al.  Loss of diacylglycerol kinase epsilon in mice causes endothelial distress and impairs glomerular Cox-2 and PGE2 production. , 2016, American journal of physiology. Renal physiology.

[3]  H. Ashida,et al.  Epigallocatechin-3-gallate activates diacylglycerol kinase alpha via a 67 kDa laminin receptor: A possibility of galloylated catechins as functional food to prevent and/or improve diabetic renal dysfunctions , 2015 .

[4]  J. Norman,et al.  The Diacylglycerol Kinase α/Atypical PKC/β1 Integrin Pathway in SDF-1α Mammary Carcinoma Invasiveness , 2014, PloS one.

[5]  Yosuke Kobayashi,et al.  Depression of type I diacylglycerol kinases in pancreatic β-cells from male mice results in impaired insulin secretion. , 2013, Endocrinology.

[6]  John Hwa,et al.  Recessive mutations in DGKE cause atypical hemolytic-uremic syndrome , 2013, Nature Genetics.

[7]  A. Bakkaloğlu,et al.  DGKE variants cause a glomerular microangiopathy that mimics membranoproliferative GN. , 2013, Journal of the American Society of Nephrology : JASN.

[8]  J. Norman,et al.  Diacylglycerol kinase α controls RCP-dependent integrin trafficking to promote invasive migration , 2012, The Journal of cell biology.

[9]  M. Schiffer,et al.  PKCα Mediates β-Arrestin2-dependent Nephrin Endocytosis in Hyperglycemia* , 2011, The Journal of Biological Chemistry.

[10]  H. Haller,et al.  Podocytic PKC-Alpha Is Regulated in Murine and Human Diabetes and Mediates Nephrin Endocytosis , 2010, PloS one.

[11]  H. Haller,et al.  PKC-alpha Modulates TGF-β Signaling and Impairs Podocyte Survival , 2009, Cellular Physiology and Biochemistry.

[12]  K. Asanuma,et al.  Actin up: regulation of podocyte structure and function by components of the actin cytoskeleton. , 2007, Trends in cell biology.

[13]  G. Boysen,et al.  Tandem mass spectrometry measurements of creatinine in mouse plasma and urine for determining glomerular filtration rate. , 2007, Kidney international.

[14]  I. Mérida,et al.  Importance of chroman ring and tyrosine phosphorylation in the subtype‐specific translocation and activation of diacylglycerol kinase α by d‐α‐tocopherol , 2005 .

[15]  M. Haneda,et al.  Effects of antioxidants in diabetes-induced oxidative stress in the glomeruli of diabetic rats. , 2003, Journal of the American Society of Nephrology : JASN.

[16]  Keiko Yamada,et al.  Diacylglycerol kinases: emerging downstream regulators in cell signaling systems. , 2002, Journal of biochemistry.

[17]  M. Nagata,et al.  High glucose induced VEGF expression via PKC and ERK in glomerular podocytes. , 2002, Biochemical and biophysical research communications.

[18]  Jie Chen,et al.  Phosphatidic Acid-Mediated Mitogenic Activation of mTOR Signaling , 2001, Science.

[19]  R. Tilton,et al.  Antibodies against Vascular Endothelial Growth Factor Improve Early Renal Dysfunction in Experimental Diabetes Materials and Methods Laboratory Animals , 2022 .

[20]  W. V. van Blitterswijk,et al.  Properties and functions of diacylglycerol kinases. , 2000, Cellular signalling.

[21]  I. Mérida,et al.  Type Iα phosphatidylinositol 4‐phosphate 5‐kinase is a putative target for increased intracellular phosphatidic acid , 2000, FEBS letters.

[22]  K. Sharma,et al.  Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[23]  D. Kerjaschki,et al.  Nephrin localizes at the podocyte filtration slit area and is characteristically spliced in the human kidney. , 1999, American Journal of Pathology.

[24]  P. S. St. John,et al.  Nephrin localizes to the slit pore of the glomerular epithelial cell. , 1999, Kidney international.

[25]  K. Tryggvason,et al.  Nephrin is specifically located at the slit diaphragm of glomerular podocytes. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[26]  M. Topham,et al.  Mammalian Diacylglycerol Kinases, a Family of Lipid Kinases with Signaling Functions* , 1999, The Journal of Biological Chemistry.

[27]  T. Miyata,et al.  Immunohistochemical evidence for an increased oxidative stress and carbonyl modification of proteins in diabetic glomerular lesions. , 1999, Journal of the American Society of Nephrology : JASN.

[28]  G. King,et al.  Protein kinase C activation and the development of diabetic complications. , 1998, Diabetes.

[29]  B. Gallacher,et al.  Glucose-Induced Protein Kinase C Activation Regulates Vascular Permeability Factor mRNA Expression and Peptide Production by Human Vascular Smooth Muscle Cells In Vitro , 1997, Diabetes.

[30]  G. King,et al.  Prevention of glomerular dysfunction in diabetic rats by treatment with d-alpha-tocopherol. , 1997, Journal of the American Society of Nephrology : JASN.

[31]  T. Meyer,et al.  Podocyte loss and progressive glomerular injury in type II diabetes. , 1997, The Journal of clinical investigation.

[32]  W. Arendshorst,et al.  Role of protein kinase C in angiotensin II-induced renal vasoconstriction in genetically hypertensive rats. , 1996, The American journal of physiology.

[33]  W. Kriz,et al.  Structure and function of podocytes: an update , 1995, Anatomy and Embryology.

[34]  S. Bursell,et al.  Correlation of diacylglycerol level and protein kinase C activity in rat retina to retinal circulation. , 1993, The American journal of physiology.

[35]  G. King,et al.  Preferential elevation of protein kinase C isoform beta II and diacylglycerol levels in the aorta and heart of diabetic rats: differential reversibility to glycemic control by islet cell transplantation. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[36]  S. Adler.  Characterization of glomerular epithelial cell matrix receptors. , 1992, The American journal of pathology.

[37]  C. Davidson,et al.  Increase in Diacylglycerol Mass in Isolated Glomeruli by Glucose From De Novo Synthesis of Glycerolipids , 1990, Diabetes.

[38]  P. Hillemanns,et al.  A beta 1-integrin receptor for fibronectin in human kidney glomeruli. , 1989, The American journal of pathology.

[39]  R. Larkins,et al.  Pancreatic islets synthesize phospholipids de novo from glucose via acyl-dihydroxyacetone phosphate. , 1985, Biochemical and biophysical research communications.

[40]  C. Berne.  The metabolism of lipids in mouse pancreatic islets. The biosynthesis of triacylglycerols and phospholipids. , 1975, The Biochemical journal.

[41]  K. Johnson.  An Update. , 1984, Journal of food protection.