Deficient Cutaneous Antibacterial Competence in Cutaneous T-Cell Lymphomas: Role of Th2-Mediated Biased Th17 Function
暂无分享,去创建一个
H. Volk | W. Sterry | R. Sabat | M. Beyer | M. Gonsior | H. Mitsui | J. Krueger | K. Wolk | Nicholas Gulati | M. Kadin | S. Gellrich | K. Witte | E. Witte | D. Humme
[1] Y. Tada,et al. The role of IL-32 in cutaneous T-cell lymphoma. , 2014, The Journal of investigative dermatology.
[2] M. Raftery,et al. IL-29 Is Produced by TH17 Cells and Mediates the Cutaneous Antiviral Competence in Psoriasis , 2013, Science Translational Medicine.
[3] C. Geisler,et al. Bacterial Toxins Fuel Disease Progression in Cutaneous T-Cell Lymphoma , 2013, Toxins.
[4] R. Bende,et al. A mutated B cell chronic lymphocytic leukemia subset that recognizes and responds to fungi , 2013, The Journal of experimental medicine.
[5] W. Ouyang,et al. Therapeutic opportunities of the IL-22–IL-22R1 system , 2013, Nature Reviews Drug Discovery.
[6] L. Hooper,et al. Epithelial antimicrobial defence of the skin and intestine , 2012, Nature Reviews Immunology.
[7] Y. Tada,et al. IL-22, but Not IL-17, Dominant Environment in Cutaneous T-cell Lymphoma , 2011, Clinical Cancer Research.
[8] P. Porcu,et al. Evolving Insights in the Pathogenesis and Therapy of Cutaneous T‐cell lymphoma (Mycosis Fungoides and Sezary Syndrome) , 2011, British journal of haematology.
[9] Wolfram Sterry,et al. Pathogenesis of Mycosis fungoides , 2011, Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG.
[10] C. Geisler,et al. Malignant cutaneous T-cell lymphoma cells express IL-17 utilizing the Jak3/Stat3 signaling pathway. , 2011, The Journal of investigative dermatology.
[11] H. Volk,et al. Deficiency of IL-22 Contributes to a Chronic Inflammatory Disease: Pathogenetic Mechanisms in Acne Inversa , 2011, The Journal of Immunology.
[12] R. Sabat,et al. Interleukin-28 and interleukin-29: novel regulators of skin biology. , 2010, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[13] D. Sasseville,et al. Transcriptional Profiles Predict Disease Outcome in Patients with Cutaneous T-Cell Lymphoma , 2010, Clinical Cancer Research.
[14] K. Asadullah,et al. The Th17 cytokine IL‐22 induces IL‐20 production in keratinocytes: A novel immunological cascade with potential relevance in psoriasis , 2009, European journal of immunology.
[15] S. Devesa,et al. Cutaneous lymphoma incidence patterns in the United States: a population-based study of 3884 cases. , 2009, Blood.
[16] Martin L. Moore,et al. A Functional IL-13 Receptor Is Expressed on Polarized Murine CD4+ Th17 Cells and IL-13 Signaling Attenuates Th17 Cytokine Production1 , 2009, The Journal of Immunology.
[17] H. Volk,et al. IL-22 and IL-20 are key mediators of the epidermal alterations in psoriasis while IL-17 and IFN-γ are not , 2009, Journal of Molecular Medicine.
[18] J. Carucci,et al. Low Expression of the IL-23/Th17 Pathway in Atopic Dermatitis Compared to Psoriasis1 , 2008, The Journal of Immunology.
[19] R. Bassett,et al. Prevalence and treatment of Staphylococcus aureus colonization in patients with mycosis fungoides and Sézary syndrome , 2008, The British journal of dermatology.
[20] J. Tobias,et al. Differential effects of interleukin-2 and interleukin-15 versus interleukin-21 on CD4+ cutaneous T-cell lymphoma cells. , 2008, Cancer research.
[21] C. Geisler,et al. Nonmalignant T cells stimulate growth of T-cell lymphoma cells in the presence of bacterial toxins. , 2007, Blood.
[22] K. Asadullah,et al. Interleukin (IL)‐19, IL‐20 and IL‐24 are produced by and act on keratinocytes and are distinct from classical ILs , 2006, Experimental dermatology.
[23] T. Bieber,et al. Mechanism of HBD-3 deficiency in atopic dermatitis. , 2006, Clinical immunology.
[24] L. Fouser,et al. Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides , 2006, The Journal of experimental medicine.
[25] K. Asadullah,et al. IL‐22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis , 2006, European journal of immunology.
[26] J. Segre,et al. Epidermal barrier formation and recovery in skin disorders. , 2006, The Journal of clinical investigation.
[27] R. Clark,et al. Skin-derived interleukin-7 contributes to the proliferation of lymphocytes in cutaneous T-cell lymphoma. , 2006, Blood.
[28] J. Schröder,et al. Psoriatic scales: a promising source for the isolation of human skin‐derived antimicrobial proteins , 2005, Journal of leukocyte biology.
[29] K. Asadullah,et al. Is there an interaction between interleukin-10 and interleukin-22? , 2005, Genes and Immunity.
[30] J. Schröder,et al. Antimicrobial psoriasin (S100A7) protects human skin from Escherichia coli infection , 2005, Nature Immunology.
[31] K. Asadullah,et al. IL-22 increases the innate immunity of tissues. , 2004, Immunity.
[32] J. Travers,et al. Cytokine Milieu of Atopic Dermatitis, as Compared to Psoriasis, Skin Prevents Induction of Innate Immune Response Genes 1 , 2003, The Journal of Immunology.
[33] Miller,et al. of Tissues , 2003 .
[34] Tomas Ganz,et al. Endogenous antimicrobial peptides and skin infections in atopic dermatitis. , 2002, The New England journal of medicine.
[35] M. Dhodapkar,et al. The growth of cutaneous T-cell lymphoma is stimulated by immature dendritic cells. , 2002, Blood.
[36] Christina H. Park,et al. Human beta-defensin-2 production in keratinocytes is regulated by interleukin-1, bacteria, and the state of differentiation. , 2002, The Journal of investigative dermatology.
[37] Rainer Hedrich,et al. Identification of a novel, multifunctional β-defensin (human β-defensin 3) with specific antimicrobial activity , 2001, Cell and Tissue Research.
[38] R. Dummer,et al. From inflammation to neoplasia: mycosis fungoides evolves from reactive inflammatory conditions (lymphoid infiltrates) transforming into neoplastic plaques and tumors. , 2001, Archives of dermatology.
[39] J. Greene,et al. Infectious complications of cutaneous t-cell lymphoma. , 2001, Cancer control : journal of the Moffitt Cancer Center.
[40] C. Vogelmeier,et al. Identification of a novel, multifunctional beta-defensin (human beta-defensin 3) with specific antimicrobial activity. Its interaction with plasma membranes of Xenopus oocytes and the induction of macrophage chemoattraction. , 2001, Cell and tissue research.
[41] S. Rutz,et al. Microanatomical compartments of clonal and reactive T cells in mycosis fungoides: molecular demonstration by single cell polymerase chain reaction of T cell receptor gene rearrangements. , 2000, The Journal of investigative dermatology.
[42] M. Salgaller. American Association for Cancer Research , 2000, Expert opinion on investigational drugs.
[43] S. Bogen,et al. Sezary lineage cells can be induced to proliferate via CD28-mediated costimulation. , 1997, Journal of immunology.
[44] J. Musser,et al. Association of Erythrodermic Cutaneous T-Cell Lymphoma, Superantigen-Positive Staphylococcus aureus, and Oligoclonal T-Cell Receptor Vβ Gene Expansion , 1997 .
[45] J. Musser,et al. Association of erythrodermic cutaneous T-cell lymphoma, superantigen-positive Staphylococcus aureus, and oligoclonal T-cell receptor V beta gene expansion. , 1997, Blood.
[46] Y. Tokura,et al. Cutaneous colonization with staphylococci influences the disease activity of Sézary syndrome: a potential role for bacterial superantigens , 1995, The British journal of dermatology.
[47] S. Lessin,et al. Th2 cytokine mRNA expression in skin in cutaneous T-cell lymphoma. , 1994, The Journal of investigative dermatology.
[48] P. Gaulard,et al. Intraepidermal localization of the clone in cutaneous T-cell lymphoma. , 1992, Journal of the American Academy of Dermatology.
[49] Y. Tokura,et al. Stimulation of cutaneous T-cell lymphoma cells with superantigenic staphylococcal toxins. , 1992, The Journal of investigative dermatology.
[50] P. Bunn,et al. Septicemic complications of the cutaneous T-cell lymphomas. , 1981, The American journal of medicine.
[51] I I Lelis,et al. [Atopic dermatitis]. , 1980, Vestnik dermatologii i venerologii.
[52] M. Sharratt,et al. The relationship of transepidermal water loss to skin temperature in psoriasis and eczema. , 1975, The Journal of investigative dermatology.