Pathways and control of ketone body metabolism: on the fringe of lipid biochemistry.
暂无分享,去创建一个
[1] A. Munnich,et al. The fasting test in paediatrics: Application to the diagnosis of pathological hypo- and hyperketotic states , 1990, European Journal of Pediatrics.
[2] C. Scriver,et al. The Metabolic and Molecular Bases of Inherited Disease, 8th Edition 2001 , 2001, Journal of Inherited Metabolic Disease.
[3] W. Fox,et al. Fetal and Neonatal Physiology , 2003 .
[4] F. Kraemer,et al. Hormone-sensitive Lipase: Control of Intracellular Tri-(di-)acylglycerol and Cholesteryl Ester Hydrolysis Structural and Biochemical Properties Thematic Review , 2022 .
[5] W. Winder,et al. Phosphorylation-activity relationships of AMPK and acetyl-CoA carboxylase in muscle. , 2002, Journal of applied physiology.
[6] R. Zechner,et al. Hormone-sensitive Lipase Deficiency in Mice Causes Diglyceride Accumulation in Adipose Tissue, Muscle, and Testis* , 2002, The Journal of Biological Chemistry.
[7] S. Patel,et al. Characterization of the Functional Interaction of Adipocyte Lipid-binding Protein with Hormone-sensitive Lipase* , 2001, The Journal of Biological Chemistry.
[8] F. Murad,et al. Diabetes-associated nitration of tyrosine and inactivation of succinyl-CoA:3-oxoacid CoA-transferase. , 2001, American journal of physiology. Heart and circulatory physiology.
[9] R. Roth,et al. Stimulation of Lipolysis and Hormone-sensitive Lipase via the Extracellular Signal-regulated Kinase Pathway* , 2001, The Journal of Biological Chemistry.
[10] F. Murad,et al. Nitration of succinyl-CoA:3-oxoacid CoA-transferase in rats after endotoxin administration , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[11] C. Deng,et al. Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[12] Martin M. Matzuk,et al. Continuous Fatty Acid Oxidation and Reduced Fat Storage in Mice Lacking Acetyl-CoA Carboxylase 2 , 2001, Science.
[13] F. Hegardt,et al. Pig liver carnitine palmitoyltransferase I, with low Km for carnitine and high sensitivity to malonyl-CoA inhibition, is a natural chimera of rat liver and muscle enzymes. , 2001, Biochemistry.
[14] A. Takahashi. [Hormone sensitive lipase]. , 2001, Nihon rinsho. Japanese journal of clinical medicine.
[15] N. Laurin,et al. The adipose tissue phenotype of hormone-sensitive lipase deficiency in mice. , 2001, Obesity research.
[16] C. Holm,et al. Molecular mechanisms regulating hormone-sensitive lipase and lipolysis. , 2003, Annual review of nutrition.
[17] D. Gorenstein,et al. Absence of perilipin results in leanness and reverses obesity in Leprdb/db mice , 2000, Nature Genetics.
[18] M. Prentki,et al. Characterization of rat liver malonyl-CoA decarboxylase and the study of its role in regulating fatty acid metabolism. , 2000, The Biochemical journal.
[19] M. Prentki,et al. Activation of Malonyl-CoA Decarboxylase in Rat Skeletal Muscle by Contraction and the AMP-activated Protein Kinase Activator 5-Aminoimidazole-4-carboxamide-1-β-d-ribofuranoside* , 2000, The Journal of Biological Chemistry.
[20] H. Miziorko,et al. 3-Hydroxy-3-methylglutaryl-CoA Synthase , 2000, The Journal of Biological Chemistry.
[21] H. Zhu,et al. The first 28 N-terminal amino acid residues of human heart muscle carnitine palmitoyltransferase I are essential for malonyl CoA sensitivity and high-affinity binding. , 2000, Biochemistry.
[22] N. Yahagi,et al. Targeted disruption of hormone-sensitive lipase results in male sterility and adipocyte hypertrophy, but not in obesity. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[23] N. Price,et al. Evidence that carnitine palmitoyltransferase I (CPT I) is expressed in microsomes and peroxisomes of rat liver , 1999, FEBS letters.
[24] Demetrios Vavvas,et al. Malonyl-CoA, fuel sensing, and insulin resistance. , 1999, American journal of physiology. Endocrinology and metabolism.
[25] J. Cechetto,et al. A mitochondrial ketogenic enzyme regulates its gene expression by association with the nuclear hormone receptor PPARα , 1998, The EMBO journal.
[26] G. Lopaschuk,et al. Characterization of cardiac malonyl-CoA decarboxylase and its putative role in regulating fatty acid oxidation. , 1998, American journal of physiology. Heart and circulatory physiology.
[27] E. Saggerson,et al. Malonyl-CoA and the regulation of fatty acid oxidation in soleus muscle. , 1998, The Biochemical journal.
[28] V. Zammit,et al. Enrichment of carnitine palmitoyltransferases I and II in the contact sites of rat liver mitochondria. , 1998, The Biochemical journal.
[29] K. Bhakoo,et al. Molecular cloning of rat mitochondrial 3-hydroxy-3-methylglutaryl-CoA lyase and detection of the corresponding mRNA and of those encoding the remaining enzymes comprising the ketogenic 3-hydroxy-3-methylglutaryl-CoA cycle in central nervous system of suckling rat. , 1998, The Biochemical journal.
[30] C. Wernstedt,et al. Identification of Novel Phosphorylation Sites in Hormone-sensitive Lipase That Are Phosphorylated in Response to Isoproterenol and Govern Activation Properties in Vitro * , 1998, The Journal of Biological Chemistry.
[31] C. Hoppel,et al. Genetic disorders of carnitine metabolism and their nutritional management. , 1998, Annual review of nutrition.
[32] D. Hardie,et al. AICA riboside increases AMP-activated protein kinase, fatty acid oxidation, and glucose uptake in rat muscle. , 1997, American journal of physiology. Endocrinology and metabolism.
[33] U. Hellman,et al. cDNA Cloning, Tissue Distribution, and Identification of the Catalytic Triad of Monoglyceride Lipase , 1997, The Journal of Biological Chemistry.
[34] G. Mitchell,et al. Enzymes of Ketone Body Utilization in Human Tissues: Protein and Messenger RNA Levels of Succinyl-Coenzyme A (CoA):3-Ketoacid CoA Transferase and Mitochondrial and Cytosolic Acetoacetyl-CoA Thiolases , 1997, Pediatric Research.
[35] V. Zammit,et al. Topology of carnitine palmitoyltransferase I in the mitochondrial outer membrane. , 1997, The Biochemical journal.
[36] F. Hegardt,et al. The effect of fasting/refeeding and insulin treatment on the expression of the regulatory genes of ketogenesis in intestine and liver of suckling rats. , 1997, Archives of biochemistry and biophysics.
[37] E. Degerman,et al. Structure, Localization, and Regulation of cGMP-inhibited Phosphodiesterase (PDE3)* , 1997, The Journal of Biological Chemistry.
[38] J. McGarry,et al. The mitochondrial carnitine palmitoyltransferase system. From concept to molecular analysis. , 1997, European journal of biochemistry.
[39] V. Zammit,et al. Regulation of mitochondrial outer-membrane carnitine palmitoyltransferase (CPT I): role of membrane-topology. , 1997, Advances in enzyme regulation.
[40] V. Zammit,et al. Evidence that both the acyl-CoA- and malonyl-CoA binding sites of mitochondrial overt carnitine palmitoyltransferase (CPT I) are exposed on the cytosolic face of the outer membrane. , 1996, Biochemical Society transactions.
[41] F. Hegardt. Regulation of mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase gene expression in liver and intestine from the rat. , 1995, Biochemical Society transactions.
[42] G. Lopaschuk,et al. High Rates of Fatty Acid Oxidation during Reperfusion of Ischemic Hearts Are Associated with a Decrease in Malonyl-CoA Levels Due to an Increase in 5′-AMP-activated Protein Kinase Inhibition of Acetyl-CoA Carboxylase (*) , 1995, The Journal of Biological Chemistry.
[43] H. Miziorko,et al. 3-Hydroxy-3-methylglutaryl-CoA lyase is present in mouse and human liver peroxisomes. , 1994, The Journal of biological chemistry.
[44] D. Labuda,et al. Human mitochondrial HMG CoA synthase: liver cDNA and partial genomic cloning, chromosome mapping to 1p12-p13, and possible role in vertebrate evolution. , 1994, Genomics.
[45] N. Casals,et al. Regulation of mitochondrial 3-hydroxy-3-methylglutaryl-coenzyme A synthase protein by starvation, fat feeding, and diabetes. , 1993, Archives of biochemistry and biophysics.
[46] J. Girard,et al. Developmental changes in mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase gene expression in rat liver, intestine and kidney. , 1993, The Biochemical journal.
[47] A. Greenberg,et al. Mechanism of hormone-stimulated lipolysis in adipocytes: translocation of hormone-sensitive lipase to the lipid storage droplet. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[48] G. Gil-Gómez,et al. Regulation of the expression of the mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase gene. Its role in the control of ketogenesis. , 1992, The Biochemical journal.
[49] B. Kemp,et al. Insulin activation of acetyl-CoA carboxylase accompanied by inhibition of the 5'-AMP-activated protein kinase. , 1992, The Journal of biological chemistry.
[50] M. Vranic,et al. Fuel Homeostasis and the Nervous System , 2012, Advances in Experimental Medicine and Biology.
[51] H. Brunengraber,et al. Nonhomogeneous labeling of liver mitochondrial acetyl-CoA. , 1991, The Journal of biological chemistry.
[52] W. Pardridge. Blood-brain barrier transport of glucose, free fatty acids, and ketone bodies. , 1991, Advances in experimental medicine and biology.
[53] D. Williamson,et al. An explanation for ketogenesis by the intestine of the suckling rat: the presence of an active hydroxymethylglutaryl-coenzyme A pathway. , 1990, Biology of the neonate.
[54] F. Féry,et al. Ketone body production and disposal: effects of fasting, diabetes, and exercise. , 1989, Diabetes/metabolism reviews.
[55] R. Colbran,et al. Phosphorylation of bovine hormone-sensitive lipase by the AMP-activated protein kinase. A possible antilipolytic mechanism. , 1989, European journal of biochemistry.
[56] B. Landau,et al. Pseudoketogenesis in the perfused rat heart. , 1988, The Journal of biological chemistry.
[57] P. K. Tubbs,et al. Succinylation and inactivation of 3-hydroxy-3-methylglutaryl-CoA synthase by succinyl-CoA and its possible relevance to the control of ketogenesis. , 1985, The Biochemical journal.
[58] P. K. Tubbs,et al. 3-Hydroxy-3-methylglutaryl-coenzyme A synthase from ox liver. Purification, molecular and catalytic properties. , 1985, The Biochemical journal.
[59] I. Cobden,et al. 初期食道静脈りゅう治療におけるテルイプレシン(グリプレシン)とバソプレシンの比較試験 , 1982 .
[60] J. McGarry,et al. Regulation of hepatic fatty acid oxidation and ketone body production. , 1980, Annual review of biochemistry.
[61] M. Lane,et al. 3-Hydroxy-3-methylgutaryl-CoA synthase. Participation of acetyl-S-enzyme and enzyme-S-hydroxymethylgutaryl-SCoA intermediates in the reaction. , 1977, The Journal of biological chemistry.
[62] A. Garber,et al. Hepatic ketogenesis and gluconeogenesis in humans. , 1974, The Journal of clinical investigation.
[63] A. Fenselau,et al. Ketone body usage by mammals. Acetoacetate substrate inhibition of coa transferase from various rat tissues , 1974 .
[64] K. Alberti,et al. Treatment of Diabetic Coma with Continuous Low-dose Infusion of Insulin , 1974, British medical journal.
[65] W. Bortz,et al. Ketone-body production and oxidation in fasting obese humans. , 1974, The Journal of clinical investigation.
[66] W. Bartley,et al. Factors involved in the control of the activity of enzymes of hepatic ketogenesis during development of the rat , 1974 .
[67] J. Flatt. On the Maximal Possible Rate of Ketogenesis , 1972, Diabetes.
[68] D. Williamson,et al. Enzymes of ketone-body utilisation in human brain. , 1971, Lancet.
[69] H. Krebs,et al. Ketone-body utilization by adult and suckling rat brain in vivo. , 1971, The Biochemical journal.
[70] H. Krebs,et al. Activities of enzymes involved in acetoacetate utilization in adult mammalian tissues. , 1971, The Biochemical journal.
[71] H. Krebs,et al. The fuel of respiration of rat kidney cortex. , 1969, The Biochemical journal.
[72] P. Felig,et al. Liver and kidney metabolism during prolonged starvation. , 1969, The Journal of clinical investigation.
[73] H. Krebs,et al. Activity and intracellular distribution of enzymes of ketone-body metabolism in rat liver. , 1968, The Biochemical journal.
[74] G F Cahill,et al. Brain metabolism during fasting. , 1967, The Journal of clinical investigation.
[75] D. Kipnis,et al. Hormone-fuel interrelationships during fasting. , 1966, The Journal of clinical investigation.
[76] H. Krebs,et al. Acetoacetate as fuel of respiration in the perfused rat heart. , 1961, The Biochemical journal.
[77] A. N. Wick,et al. THE EFFECT OF CONCENTRATION ON THE RATE OF UTILIZATION OF β-HYDROXYBUTYRIC ACID BY THE RABBIT , 1941 .