Role of epoxyeicosatrienoic acids in protecting the myocardium following ischemia/reperfusion injury.
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[1] J. Falck,et al. Role of Soluble Epoxide Hydrolase in Postischemic Recovery of Heart Contractile Function , 2006, Circulation research.
[2] J. Falck,et al. Epoxyeicosatrienoic acids in cardioprotection: ischemic versus reperfusion injury. , 2006, American journal of physiology. Heart and circulatory physiology.
[3] P. Wolf,et al. Heart disease and stroke statistics--2006 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee. , 2006, Circulation.
[4] J. Falck,et al. Effects of selective inhibition of cytochrome P-450 omega-hydroxylases and ischemic preconditioning in myocardial protection. , 2006, American journal of physiology. Heart and circulatory physiology.
[5] M. Fornage,et al. Genetic variation in soluble epoxide hydrolase (EPHX2) and risk of coronary heart disease: The Atherosclerosis Risk in Communities (ARIC) study. , 2006, Human molecular genetics.
[6] M. Fornage,et al. The soluble epoxide hydrolase gene harbors sequence variation associated with susceptibility to and protection from incident ischemic stroke. , 2005, Human molecular genetics.
[7] R. Hui,et al. Arachidonic Acid Epoxygenase Metabolites Stimulate Endothelial Cell Growth and Angiogenesis via Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase/Akt Signaling Pathways , 2005, Journal of Pharmacology and Experimental Therapeutics.
[8] J. Daut,et al. K(ATP) channels and preconditioning: a re-examination of the role of mitochondrial K(ATP) channels and an overview of alternative mechanisms. , 2005, Journal of molecular and cellular cardiology.
[9] R. Cannon. Mechanisms, management and future directions for reperfusion injury after acute myocardial infarction , 2005, Nature Clinical Practice Cardiovascular Medicine.
[10] H. Nakaya,et al. Mitochondrial Ca2+-Activated K+ Channels in Cardiac Myocytes: A Mechanism of the Cardioprotective Effect and Modulation by Protein Kinase A , 2005, Circulation.
[11] D. Kroetz,et al. REGULATION AND INHIBITION OF ARACHIDONIC ACID ω-HYDROXYLASES AND 20-HETE FORMATION , 2005 .
[12] D. Kroetz,et al. Regulation and inhibition of arachidonic acid omega-hydroxylases and 20-HETE formation. , 2005, Annual review of pharmacology and toxicology.
[13] B. Hammock,et al. Epoxide hydrolases: mechanisms, inhibitor designs, and biological roles. , 2005, Annual review of pharmacology and toxicology.
[14] B. Hammock,et al. Effect of soluble epoxide hydrolase inhibition on epoxyeicosatrienoic acid metabolism in human blood vessels. , 2004, American journal of physiology. Heart and circulatory physiology.
[15] J. Falck,et al. Cytochrome P450 omega-hydroxylase inhibition reduces infarct size during reperfusion via the sarcolemmal KATP channel. , 2004, Journal of molecular and cellular cardiology.
[16] J. Falck,et al. Enhancement of Cardiac L-Type Ca2+ Currents in Transgenic Mice with Cardiac-Specific Overexpression of CYP2J2 , 2004, Molecular Pharmacology.
[17] Xiaoying Wang,et al. Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice. , 2004, American journal of physiology. Heart and circulatory physiology.
[18] J. Falck,et al. Inhibition of Cytochrome P450&ohgr;-Hydroxylase: A Novel Endogenous Cardioprotective Pathway , 2004, Circulation research.
[19] K. Lindpaintner,et al. Risk of Coronary Artery Disease Associated With Polymorphism of the Cytochrome P450 Epoxygenase CYP2J2 , 2004, Circulation.
[20] J. Foley,et al. Enhanced Postischemic Functional Recovery in CYP2J2 Transgenic Hearts Involves Mitochondrial ATP-Sensitive K+ Channels and p42/p44 MAPK Pathway , 2004, Circulation research.
[21] A. Halestrap. Mitochondrial permeability: Dual role for the ADP/ATP translocator? , 2004, Nature.
[22] Hiromu Tanimoto,et al. Experimental psychology: Event timing turns punishment to reward , 2004, Nature.
[23] E. Marbán,et al. Multiprotein complex containing succinate dehydrogenase confers mitochondrial ATP-sensitive K+ channel activity. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[24] R. Bolli,et al. Myocardial Protection at a Crossroads: The Need for Translation Into Clinical Therapy , 2004, Circulation research.
[25] I. Fleming. Cytochrome P450 epoxygenases as EDHF synthase(s). , 2004, Pharmacological research.
[26] E. Olson,et al. Glycogen synthase kinase-3β mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore , 2004 .
[27] DerekHausenloy,et al. Transient Mitochondrial Permeability Transition Pore Opening Mediates Preconditioning-Induced Protection , 2004 .
[28] M. Duchen,et al. Transient Mitochondrial Permeability Transition Pore Opening Mediates Preconditioning-Induced Protection , 2004, Circulation.
[29] B. O’Rourke,et al. Evidence for Mitochondrial K Channels and Their Role in Cardioprotection , 2004 .
[30] S. Javadov,et al. Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection. , 2004, Cardiovascular research.
[31] M. Yeager,et al. Reduction of ischemia and reperfusion-induced myocardial damage by cytochrome P450 inhibitors. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[32] M. Fornage,et al. Polymorphism of the Soluble Epoxide Hydrolase Is Associated With Coronary Artery Calcification in African-American Subjects: The Coronary Artery Risk Development In Young Adults (CARDIA) Study , 2004, Circulation.
[33] E. Murphy,et al. Primary and secondary signaling pathways in early preconditioning that converge on the mitochondria to produce cardioprotection. , 2004, Circulation research.
[34] Xiang Fang,et al. Epoxyeicosatrienoic acids (EETs): metabolism and biochemical function. , 2004, Progress in lipid research.
[35] E. Olson,et al. Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore. , 2004, The Journal of clinical investigation.
[36] M. Duchen,et al. Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury. , 2003, Cardiovascular research.
[37] U. de Faire,et al. Allelic variants of cytochromes P450 2C modify the risk for acute myocardial infarction. , 2003, Pharmacogenetics.
[38] D. Zeldin,et al. Up-Regulation of Endothelial Nitric-Oxide Synthase by Endothelium-Derived Hyperpolarizing Factor Involves Mitogen-Activated Protein Kinase and Protein Kinase C Signaling Pathways , 2003, Journal of Pharmacology and Experimental Therapeutics.
[39] J. Downey,et al. Preconditioning the myocardium: from cellular physiology to clinical cardiology. , 2003, Physiological reviews.
[40] G. Gross,et al. KATP channels and myocardial preconditioning: an update. , 2003, American journal of physiology. Heart and circulatory physiology.
[41] Peipei Ping,et al. Role of the mitochondrial permeability transition in myocardial disease. , 2003, Circulation research.
[42] D. Manuel,et al. Burden of cardiovascular disease in Canada. , 2003, The Canadian journal of cardiology.
[43] J. E. Maxwell,et al. Polymorphisms in human soluble epoxide hydrolase. , 2003, Molecular pharmacology.
[44] P. Austin,et al. Outcomes of acute myocardial infarction in Canada. , 2003, The Canadian journal of cardiology.
[45] J. Tu,et al. Impact of an acute myocardial infarction report card in Ontario, Canada. , 2003, International journal for quality in health care : journal of the International Society for Quality in Health Care.
[46] D. Manuel,et al. Cause-deleted health-adjusted life expectancy of Canadians with selected chronic conditions. , 2003, Chronic diseases in Canada.
[47] Yongge Liu,et al. Cytoprotective Role of Ca2+- Activated K+ Channels in the Cardiac Inner Mitochondrial Membrane , 2002, Science.
[48] T. Lu,et al. Stereospecific activation of cardiac ATP-sensitive K(+) channels by epoxyeicosatrienoic acids: a structural determinant study. , 2002, Molecular pharmacology.
[49] V. Calderone. Large-conductance, ca(2+)-activated k(+) channels: function, pharmacology and drugs. , 2002, Current medicinal chemistry.
[50] A. Wickenden. K(+) channels as therapeutic drug targets. , 2002, Pharmacology & therapeutics.
[51] Peipei Ping,et al. Expression of Activated PKC Epsilon (PKC ϵ) Protects the Ischemic Heart, without Attenuating Ischemic H+ Production , 2002 .
[52] R. Roman,et al. P-450 metabolites of arachidonic acid in the control of cardiovascular function. , 2002, Physiological reviews.
[53] P. Ping,et al. Expression of activated PKC epsilon (PKC epsilon) protects the ischemic heart, without attenuating ischemic H(+) production. , 2002, Journal of molecular and cellular cardiology.
[54] T. Lu,et al. Activation of ATP‐sensitive K+ channels by epoxyeicosatrienoic acids in rat cardiac ventricular myocytes , 2001, The Journal of physiology.
[55] D. Zeldin. Epoxygenase Pathways of Arachidonic Acid Metabolism* , 2001, The Journal of Biological Chemistry.
[56] J. Falck,et al. Overexpression of cytochrome P450 CYP2J2 protects against hypoxia-reoxygenation injury in cultured bovine aortic endothelial cells. , 2001, Molecular pharmacology.
[57] K. Node,et al. Activation of Gαs Mediates Induction of Tissue-type Plasminogen Activator Gene Transcription by Epoxyeicosatrienoic Acids* , 2001, The Journal of Biological Chemistry.
[58] D. Thompson,et al. Pathways of Epoxyeicosatrienoic Acid Metabolism in Endothelial Cells , 2001, The Journal of Biological Chemistry.
[59] R. Busse,et al. Endothelium-Derived Hyperpolarizing Factor Synthase (Cytochrome P450 2C9) Is a Functionally Significant Source of Reactive Oxygen Species in Coronary Arteries , 2001, Circulation research.
[60] D. Harder,et al. Dual regulation of the cerebral microvasculature by epoxyeicosatrienoic acids. , 2001, Trends in cardiovascular medicine.
[61] R. Roman,et al. Renal And Cardiovascular Actions Of 20‐Hydroxyeicosatetraenoic Acid And Epoxyeicosatrienoic Acids , 2000, Clinical and experimental pharmacology & physiology.
[62] M. Stern,et al. Protective effects of low and high doses of cyclosporin A against reoxygenation injury in isolated rat cardiomyocytes are associated with differential effects on mitochondrial calcium levels. , 2000, Cell calcium.
[63] Michael V. Cohen,et al. Protein Kinase C- ξ is Responsible for the Protection of Preconditioning in Rabbit Cardiomyocytes , 1999 .
[64] R. Busse,et al. Cytochrome P450 2C is an EDHF synthase in coronary arteries , 1999, Nature.
[65] K. Ley,et al. Anti-inflammatory properties of cytochrome P450 epoxygenase-derived eicosanoids. , 1999, Science.
[66] T. Lu,et al. Effects of epoxyeicosatrienoic acids on the cardiac sodium channels in isolated rat ventricular myocytes , 1999, The Journal of physiology.
[67] M Crompton,et al. The mitochondrial permeability transition pore and its role in cell death. , 1999, The Biochemical journal.
[68] A. Szewczyk,et al. Mitochondrial ATP‐Dependent Potassium Channels: Viable Candidate Effectors of Ischemic Preconditioning a , 1999, Annals of the New York Academy of Sciences.
[69] Yongge Liu,et al. Pharmacological and histochemical distinctions between molecularly defined sarcolemmal KATP channels and native cardiac mitochondrial KATP channels. , 1999, Molecular pharmacology.
[70] R. Fryer,et al. Sarcolemmal versus mitochondrial ATP-sensitive K+ channels and myocardial preconditioning. , 1999, Circulation research.
[71] J. Borecký,et al. Ca2+-activated K channel of the BK-type in the inner mitochondrial membrane of a human glioma cell line. , 1999, Biochemical and biophysical research communications.
[72] W. Campbell,et al. Endothelium-derived hyperpolarizing factors and vascular cytochrome P450 metabolites of arachidonic acid in the regulation of tone. , 1999, Circulation research.
[73] J. Downey,et al. Protein kinase C-epsilon is responsible for the protection of preconditioning in rabbit cardiomyocytes. , 1999, Journal of molecular and cellular cardiology.
[74] Yongge Liu,et al. Mitochondrial ATP-dependent potassium channels: novel effectors of cardioprotection? , 1998, Circulation.
[75] L. Huang,et al. Cytochrome P450: a novel system modulating Ca2+ channels and contraction in mammalian heart cells , 1998, The Journal of physiology.
[76] J. Phillis,et al. Role of phospholipase A2 in the release of free fatty acids during ischemia-reperfusion in the rat cerebral cortex , 1997, Neuroscience Letters.
[77] K. Tomer,et al. Molecular Cloning, Expression, and Functional Significance of a Cytochrome P450 Highly Expressed in Rat Heart Myocytes* , 1997, The Journal of Biological Chemistry.
[78] J. Zhang,et al. Economic burden of illness in Canada, 1993. Executive summary and recommendations. , 1997, Chronic diseases in Canada.
[79] P. Pratt,et al. Identification of epoxyeicosatrienoic acids as endothelium-derived hyperpolarizing factors. , 1996, Circulation research.
[80] K. Tomer,et al. Molecular Cloning and Expression of CYP2J2, a Human Cytochrome P450 Arachidonic Acid Epoxygenase Highly Expressed in Heart (*) , 1996, The Journal of Biological Chemistry.
[81] R. DuBois,et al. Molecular cloning, expression and characterization of an endogenous human cytochrome P450 arachidonic acid epoxygenase isoform. , 1995, Archives of biochemistry and biophysics.
[82] A. Halestrap,et al. Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts. , 1993, Journal of molecular and cellular cardiology.
[83] M. Karmazyn,et al. Effects of epoxyeicosatrienoic acids on isolated hearts and ventricular myocytes. , 1993, The American journal of physiology.
[84] D. Zeldin,et al. Regio- and enantiofacial selectivity of epoxyeicosatrienoic acid hydration by cytosolic epoxide hydrolase. , 1993, The Journal of biological chemistry.
[85] M. Nelson,et al. Regulation of arterial tone by activation of calcium-dependent potassium channels. , 1992, Science.
[86] M. Crompton,et al. Inhibition of anoxia-induced injury in heart myocytes by cyclosporin A. , 1991, Journal of molecular and cellular cardiology.