Association of paraoxonase activity and coronary blood flow.

[1]  H. Gullu,et al.  Coronary flow reserve is impaired in patients with slow coronary flow. , 2007, Atherosclerosis.

[2]  O. Erel,et al.  Paraoxonase and arylesterase activities in coronary artery disease , 2006, European journal of clinical investigation.

[3]  M. Nieminen,et al.  Association of paraoxonase-1 activity and concentration with angiographic severity and extent of coronary artery disease. , 2006, Journal of the American College of Cardiology.

[4]  H. Tanriverdi,et al.  Cigarette smoking induced oxidative stress may impair endothelial function and coronary blood flow in angiographically normal coronary arteries. , 2006, Circulation journal : official journal of the Japanese Circulation Society.

[5]  Chii‐Ming Lee,et al.  High-density lipoprotein antagonizes oxidized low-density lipoprotein by suppressing oxygen free-radical formation and preserving nitric oxide bioactivity. , 2005, Atherosclerosis.

[6]  K. Sunagawa,et al.  Vasospastic angina and microvascular angina are differentially influenced by PON1 A632G polymorphism in the Japanese. , 2005, Circulation journal : official journal of the Japanese Circulation Society.

[7]  H. Gullu,et al.  Plasma nitric oxide level and its role in slow coronary flow phenomenon. , 2005, International heart journal.

[8]  E. Mannarino,et al.  Paraoxonase-1 activity modulates endothelial function in patients with peripheral arterial disease. , 2005, Atherosclerosis.

[9]  E. Boerwinkle,et al.  Q192R polymorphism of the paraoxanase 1 gene and its association with serum lipoprotein variables and carotid artery intima-media thickness in young adults from a biracial community. The Bogalusa Heart Study. , 2004, Atherosclerosis.

[10]  J. Danesh,et al.  Four paraoxonase gene polymorphisms in 11 212 cases of coronary heart disease and 12 786 controls: meta-analysis of 43 studies , 2004, The Lancet.

[11]  O. Erel,et al.  A novel automated method to measure total antioxidant response against potent free radical reactions. , 2004, Clinical biochemistry.

[12]  J. Steghens,et al.  Plasma lipid hydroperoxides measurement by an automated xylenol orange method. , 2004, Analytical biochemistry.

[13]  T. Lehtimäki,et al.  Smoking-dependent association between paraoxonase 1 M/L55 genotype and coronary atherosclerosis in males: an autopsy study. , 2003, Atherosclerosis.

[14]  M. Akkuş,et al.  Diffuse intimal thickening of coronary arteries in slow coronary flow. , 2003, Japanese heart journal.

[15]  N. Ermis,et al.  Vascular endothelial function in patients with slow coronary flow , 2003, Coronary artery disease.

[16]  A. Tward,et al.  Decreased Atherosclerotic Lesion Formation in Human Serum Paraoxonase Transgenic Mice , 2002, Circulation.

[17]  P. Durrington,et al.  Paraoxonase Status in Coronary Heart Disease: Are Activity and Concentration More Important Than Genotype? , 2001, Arteriosclerosis, thrombosis, and vascular biology.

[18]  T. Lehtimäki,et al.  Paraoxonase producing PON1 gene M/L55 polymorphism is related to autopsy-verified artery-wall atherosclerosis. , 2001, Atherosclerosis.

[19]  C. Franceschi,et al.  The BB-paraoxonase genotype is associated with impaired brachial reactivity after acute hypertriglyceridemia in healthy subjects. , 2001, The Journal of clinical endocrinology and metabolism.

[20]  H. Ogawa,et al.  Paraoxonase gene Gln192Arg (Q192R) polymorphism is associated with coronary artery spasm , 2001, Human Genetics.

[21]  P. Amouyel,et al.  Paraoxonase polymorphism (Gln192Arg) as a determinant of the response of human coronary arteries to serotonin. , 2000, Circulation.

[22]  I. Leviev,et al.  Promoter polymorphisms of human paraoxonase PON1 gene and serum paraoxonase activities and concentrations. , 2000, Arteriosclerosis, thrombosis, and vascular biology.

[23]  J. Kastelein,et al.  Paraoxonase gene polymorphisms are associated with carotid arterial wall thickness in subjects with familial hypercholesterolemia. , 1999, Atherosclerosis.

[24]  A. Abacı,et al.  Effect of potential confounding factors on the thrombolysis in myocardial infarction (TIMI) trial frame count and its reproducibility. , 1999, Circulation.

[25]  D. Shih,et al.  Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis , 1998, Nature.

[26]  B. La Du,et al.  Paraoxonase inhibits high-density lipoprotein oxidation and preserves its functions. A possible peroxidative role for paraoxonase. , 1998, The Journal of clinical investigation.

[27]  E. Braunwald,et al.  TIMI frame count: a quantitative method of assessing coronary artery flow. , 1996, Circulation.

[28]  P. Barter,et al.  High density lipoproteins and coronary heart disease. , 1996, Atherosclerosis.

[29]  J. Berliner,et al.  Protective effect of high density lipoprotein associated paraoxonase. Inhibition of the biological activity of minimally oxidized low density lipoprotein. , 1995, The Journal of clinical investigation.

[30]  R. Erbel,et al.  Intravascular ultrasound imaging of angiographically normal coronary arteries: a prospective study in vivo. , 1994, British heart journal.

[31]  M. Lavallée,et al.  Contribution of nitric oxide to dilation of resistance coronary vessels in conscious dogs. , 1992, The American journal of physiology.

[32]  R. Busse,et al.  Endothelium-dependent modulation of vascular tone and platelet function. , 1990, European heart journal.

[33]  T. Carew.  Role of biologically modified low-density lipoprotein in atherosclerosis. , 1989, The American journal of cardiology.

[34]  M. Gotsman,et al.  Histologic evidence for small-vessel coronary artery disease in patients with angina pectoris and patent large coronary arteries. , 1986, Circulation.

[35]  R. Cannon,et al.  Hemodynamic principles in the control of coronary blood flow. , 1985, The American journal of cardiology.

[36]  B. La Du,et al.  The human serum paraoxonase/arylesterase polymorphism. , 1983, American journal of human genetics.

[37]  W. Roberts,et al.  Quantitation of coronary arterial narrowing at necropsy in sudden coronary death: analysis of 31 patients and comparison with 25 control subjects. , 1979, The American journal of cardiology.

[38]  R Gorlin,et al.  Problems in echocardiographic volume determinations: echocardiographic-angiographic correlations in the presence of absence of asynergy. , 1976, The American journal of cardiology.