Extracellular sulfatase-2 is overexpressed in rheumatoid arthritis and mediates the TNF-α-induced inflammatory activation of synovial fibroblasts
暂无分享,去创建一个
Anil Kumar Singh | S. Khuder | E. Schwarz | D. Fox | Sanghoon Han | Salahuddin Ahmed | H. M. Kenney | Paul M. Panipinto | Cynthia S. Crowson | Ruby J Siegel | Farheen S. Shaikh | Judy Vinh | Basil S Khuder | Farheen. Shaikh | C. Crowson | Farheen Shaikh | Ruby J. Siegel | Basil S. Khuder
[1] J. Hanas,et al. OKN-007 Alters Protein Expression Profiles in High-Grade Gliomas: Mass Spectral Analysis of Blood Sera , 2022, Brain sciences.
[2] H. Kim,et al. Sulfatase 1 and sulfatase 2 as novel regulators of macrophage antigen presentation and phagocytosis , 2021, Yeungnam University journal of medicine.
[3] M. Mercogliano,et al. Harnessing Tumor Necrosis Factor Alpha to Achieve Effective Cancer Immunotherapy , 2021, Cancers.
[4] J. Zaia,et al. Expression of the Extracellular Sulfatase SULF2 Affects Survival of Head and Neck Squamous Cell Carcinoma Patients , 2021, Frontiers in Oncology.
[5] Anil Kumar Singh,et al. Regulation of Synovial Inflammation and Tissue Destruction by Guanylate Binding Protein 5 in Synovial Fibroblasts From Patients With Rheumatoid Arthritis and Rats With Adjuvant‐Induced Arthritis , 2020, Arthritis & rheumatology.
[6] E. Gout,et al. HS and Inflammation: A Potential Playground for the Sulfs? , 2020, Frontiers in Immunology.
[7] N. Shaik,et al. Dissecting the Role of NF-κb Protein Family and Its Regulators in Rheumatoid Arthritis Using Weighted Gene Co-Expression Network , 2019, Front. Genet..
[8] E. Schwarz,et al. Selective Sexual Dimorphisms in Musculoskeletal and Cardiopulmonary Pathologic Manifestations and Mortality Incidence in the Tumor Necrosis Factor–Transgenic Mouse Model of Rheumatoid Arthritis , 2019, Arthritis & rheumatology.
[9] Alireza Hadj Khodabakhshi,et al. Metascape provides a biologist-oriented resource for the analysis of systems-level datasets , 2019, Nature Communications.
[10] H. Lortat-Jacob,et al. The “in and out” of glucosamine 6-O-sulfation: the 6th sense of heparan sulfate , 2016, Glycoconjugate Journal.
[11] D. Fox,et al. Synovial cellular and molecular markers in rheumatoid arthritis , 2017, Seminars in Immunopathology.
[12] F. Allain,et al. Tumour-necrosis factor-α induces heparan sulfate 6-O-endosulfatase 1 (Sulf-1) expression in fibroblasts. , 2016, The international journal of biochemistry & cell biology.
[13] B. Kallakury,et al. Expression of the extracellular sulfatase SULF2 is associated with squamous cell carcinoma of the head and neck , 2016, Oncotarget.
[14] David S. Wishart,et al. Heatmapper: web-enabled heat mapping for all , 2016, Nucleic Acids Res..
[15] J. Phillips,et al. SULF2, a heparan sulfate endosulfatase, is present in the blood of healthy individuals and increases in cirrhosis. , 2015, Clinica chimica acta; international journal of clinical chemistry.
[16] D. Pennington,et al. Cytokines and chemokines: At the crossroads of cell signalling and inflammatory disease. , 2014, Biochimica et biophysica acta.
[17] C. Libert,et al. Regulation and dysregulation of tumor necrosis factor receptor-1. , 2014, Cytokine & growth factor reviews.
[18] Abd A. Alhasan,et al. Role of 6-O-Sulfated Heparan Sulfate in Chronic Renal Fibrosis* , 2014, The Journal of Biological Chemistry.
[19] C. Moser,et al. The human sulfatase 2 inhibitor 2,4‐disulfonylphenyl‐tert‐butylnitrone (OKN‐007) has an antitumor effect in hepatocellular carcinoma mediated via suppression of TGFB1/SMAD2 and Hedgehog/GLI1 signaling , 2013, Genes, chromosomes & cancer.
[20] Davis J. McCarthy,et al. Differential expression analysis of multifactor RNA-Seq experiments with respect to biological variation , 2012, Nucleic acids research.
[21] S. Rosen,et al. Sulf-2: an extracellular modulator of cell signaling and a cancer target candidate , 2010, Expert opinion on therapeutic targets.
[22] M. Robinson,et al. A scaling normalization method for differential expression analysis of RNA-seq data , 2010, Genome Biology.
[23] G. Firestein,et al. Fibroblast‐like synoviocytes: key effector cells in rheumatoid arthritis , 2010, Immunological reviews.
[24] M. Mayhew. Tumor Necrosis Factor Inhibitors , 2009 .
[25] D. D’Lima,et al. Expression of novel extracellular sulfatases Sulf-1 and Sulf-2 in normal and osteoarthritic articular cartilage , 2008, Arthritis research & therapy.
[26] S. Mundlos,et al. Redundant function of the heparan sulfate 6‐O‐endosulfatases Sulf1 and Sulf2 during skeletal development , 2008, Developmental dynamics : an official publication of the American Association of Anatomists.
[27] J. Bradley,et al. TNF‐mediated inflammatory disease , 2008, The Journal of pathology.
[28] C. Van Waes,et al. Role of activated nuclear factor‐κB in the pathogenesis and therapy of squamous cell carcinoma of the head and neck , 2007 .
[29] A. Koch,et al. Regulation of interleukin-1β–induced chemokine production and matrix metalloproteinase 2 activation by epigallocatechin-3-gallate in rheumatoid arthritis synovial fibroblasts , 2006 .
[30] Z. Werb,et al. HSulf-2, an extracellular endoglucosamine-6-sulfatase, selectively mobilizes heparin-bound growth factors and chemokines: effects on VEGF, FGF-1, and SDF-1 , 2006, BMC Biochemistry.
[31] M. Kaler,et al. Release of full-length 55-kDa TNF receptor 1 in exosome-like vesicles: a mechanism for generation of soluble cytokine receptors. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[32] E. Schwarz,et al. The TNF-α transgenic mouse model of inflammatory arthritis , 2003, Springer Seminars in Immunopathology.
[33] Andrew G. D. Bean,et al. TNF Regulates Chemokine Induction Essential for Cell Recruitment, Granuloma Formation, and Clearance of Mycobacterial Infection1 , 2002, The Journal of Immunology.
[34] R. Locksley,et al. The TNF and TNF Receptor Superfamilies Integrating Mammalian Biology , 2001, Cell.
[35] G. Adolf,et al. Tumor necrosis factor (TNF)-dependent shedding of the p55 TNF receptor in a baboon model of bacteremia , 1995, Infection and immunity.
[36] M. Slevin,et al. Infusion of tumor necrosis factor (TNF) causes an increase in circulating TNF-binding protein in humans. , 1990, Cytokine.
[37] W. Greene,et al. Tumor necrosis factor alpha induces proteins that bind specifically to kappa B-like enhancer elements and regulate interleukin 2 receptor alpha-chain gene expression in primary human T lymphocytes. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[38] N. Zaffaroni,et al. Targeting Heparan Sulfate Proteoglycans and their Modifying Enzymes to Enhance Anticancer Chemotherapy Efficacy and Overcome Drug Resistance. , 2017, Current medicinal chemistry.
[39] C. Farquharson,et al. Expression of Sulf1 and Sulf2 in cartilage, bone and endochondral fracture healing , 2015, Histochemistry and Cell Biology.
[40] T. Jenssen,et al. Heparan sulfate expression is affected by inflammatory stimuli in primary human endothelial cells , 2011, Glycoconjugate Journal.
[41] A. Koch,et al. Regulation of interleukin-1beta-induced chemokine production and matrix metalloproteinase 2 activation by epigallocatechin-3-gallate in rheumatoid arthritis synovial fibroblasts. , 2006, Arthritis and rheumatism.
[42] E. Schwarz,et al. The TNF-alpha transgenic mouse model of inflammatory arthritis. , 2003, Springer seminars in immunopathology.
[43] M. Feldmann,et al. Anti-TNF alpha therapy of rheumatoid arthritis: what have we learned? , 2001, Annual review of immunology.
[44] L. Crofford,et al. Responsiveness of human T lymphocytes to bacterial superantigens presented by cultured rheumatoid arthritis synoviocytes. , 1996, Arthritis and rheumatism.