Familial medullary thyroid carcinoma and multiple endocrine neoplasia type 2B map to the same region of chromosome 10 as multiple endocrine neoplasia type 2A.

[1]  T. Lehner,et al.  Genetic homogeneity between acute and chronic forms of spinal muscular atrophy , 1990, Nature.

[2]  F. Giancotti,et al.  Elevated levels of the α 5 β 1 fibronectin receptor suppress the transformed phenotype of Chinese hamster ovary cells , 1990, Cell.

[3]  K. Kidd,et al.  The genetic defect in multiple endocrine neoplasia type 2A maps next to the centromere of chromosome 10. , 1990, American journal of human genetics.

[4]  C. Larsson,et al.  Localization of the MEN1 gene to a small region within chromosome 11q13 by deletion mapping in tumors. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[5]  D. Housman,et al.  Complete physical map of the WAGR region of 11p13 localizes a candidate Wilms' tumor gene , 1990, Cell.

[6]  K. Davies,et al.  Very mild muscular dystrophy associated with the deletion of 46% of dystrophin , 1990, Nature.

[7]  P. Green,et al.  A genetic linkage map of 32 loci on human chromosome 10. , 1989, Genomics.

[8]  L. Tsui,et al.  Erratum: Identification of the Cystic Fibrosis Gene: Genetic Analysis , 1989, Science.

[9]  C. Mathew,et al.  Linked markers flanking the gene for multiple endocrine neoplasia type 2A. , 1989, Genomics.

[10]  J. Herman,et al.  Low incidence of loss of chromosome 10 in sporadic and hereditary human medullary thyroid carcinoma. , 1989, Cancer research.

[11]  C. Mathew,et al.  Development of multiple endocrine neoplasia type 2A does not involve substantial deletions of chromosome 10. , 1989, Genomics.

[12]  P. Goodfellow,et al.  Assignment of the gene encoding the beta‐subunit of the human fibronectin receptor (β‐FNR) to chromosome 10p11.2 , 1989, Annals of human genetics.

[13]  Jackson Ce,et al.  Genetic mechanisms of neoplasia in MEN 2. , 1989 .

[14]  D. Easton,et al.  The clinical and screening age-at-onset distribution for the MEN-2 syndrome. , 1989, American journal of human genetics.

[15]  Y. Nakamura,et al.  An extended genetic linkage map of markers for human chromosome 10. , 1988, Genomics.

[16]  Y. Nakamura,et al.  Genetic alterations during colorectal-tumor development. , 1988, The New England journal of medicine.

[17]  H. Willard,et al.  Chromosome-specific alpha satellite DNA: isolation and mapping of a polymorphic alphoid repeat from human chromosome 10. , 1988, Genomics.

[18]  C. Larsson,et al.  Multiple endocrine neoplasia type 1 gene maps to chromosome 11 and is lost in insulinoma , 1988, Nature.

[19]  Shirley A. Miller,et al.  A simple salting out procedure for extracting DNA from human nucleated cells. , 1988, Nucleic acids research.

[20]  M. Daly,et al.  A genetic linkage map of the human genome , 1987, Cell.

[21]  K. Kidd,et al.  Assignment of multiple endocrine neoplasia type 2A to chromosome 10 by linkage , 1987, Nature.

[22]  C. Mathew,et al.  Deletion of genes on chromosome 1 in endocrine neoplasia , 1987, Nature.

[23]  M. Koenig,et al.  Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals , 1987, Cell.

[24]  J. J. A. Holden,et al.  A polymorphic locus, D10S5, at 10q21.1 , 1987, Nucleic Acids Res..

[25]  Y. Li,et al.  Bgl II RFLP recognized by a human IRBP cDNA localized to chromosome 10. , 1987, Nucleic acids research.

[26]  C. Mathew,et al.  Exclusion of linkage of loci on chromosome 19 with multiple endocrine neoplasia, type 2. , 1987, Cytogenetics and cell genetics.

[27]  C. Mathew,et al.  A linked genetic marker for multiple endocrine neoplasia type 2A on chromosome 10 , 1987, Nature.

[28]  Stephen H. Friend,et al.  A human DNA segment with properties of the gene that predisposes to retinoblastoma and osteosarcoma , 1986, Nature.

[29]  S. Baylin,et al.  Familial medullary thyroid carcinoma without associated endocrinopathies: A distinct clinical entity , 1986, The British journal of surgery.

[30]  N. Copeland,et al.  Loss of heterozygosity in three embryonal tumours suggests a common pathogenetic mechanism , 1985, Nature.

[31]  J. Ott,et al.  Multilocus linkage analysis in humans: detection of linkage and estimation of recombination. , 1985, American journal of human genetics.

[32]  A. Feinberg,et al.  A technique for radiolabeling DNA restriction endonuclease fragments to high specific activity. , 1983, Analytical biochemistry.

[33]  A. Tashjian,et al.  Age-related probability of development of hereditary medullary thyroid carcinoma. , 1982, The Journal of pediatrics.

[34]  A. Knudson,et al.  Mutation and childhood cancer: a probabilistic model for the incidence of retinoblastoma. , 1975, Proceedings of the National Academy of Sciences of the United States of America.

[35]  J. F. Hennessy,et al.  A comparison of pentagastrin injection and calcium infusion as provocative agents for the detection of medullary carcinoma of the thyroid. , 1974, The Journal of clinical endocrinology and metabolism.

[36]  M. Block.  Familial medullary carcinoma of the thyroid. , 1967, GP.

[37]  A. Pearse.  Common cytochemical and ultrastructural characteristics of cells producing polypeptide hormones (the APUD series) and their relevance to thyroid and ultimobranchial C cells and calcitonin , 1968, Proceedings of the Royal Society of London. Series B. Biological Sciences.

[38]  J. Hazard,et al.  Medullary (solid) carcinoma of the thyroid; a clinicopathologic entity. , 1959, The Journal of clinical endocrinology and metabolism.