c-Jun N terminal kinase modulates NOX-4 derived ROS production and myofibroblasts differentiation in human breast stromal cells
暂无分享,去创建一个
R. Arancibia | N. Tobar | Jorge Martínez | Patricio C. Smith | Nicolás Méndez | M. Toyos | Carla Urra
[1] R. Arancibia,et al. c-Jun N terminal kinase modulates NOX-4 derived ROS production and myofibroblasts differentiation in human breast stromal cells , 2014, BMC Cancer.
[2] R. Samarakoon,et al. TGF-β signaling in tissue fibrosis: redox controls, target genes and therapeutic opportunities. , 2013, Cellular signalling.
[3] R. Ginnan,et al. NADPH oxidase 4 is required for interleukin-1β-mediated activation of protein kinase Cδ and downstream activation of c-jun N-terminal kinase signaling in smooth muscle. , 2013, Free radical biology & medicine.
[4] J. Eisenbart,et al. Mitochondrial Reactive Oxygen Species Regulate Transforming Growth Factor-β Signaling , 2012, The Journal of Biological Chemistry.
[5] P. Chiarugi,et al. Oxidative Stress, Tumor Microenvironment, and Metabolic Reprogramming: A Diabolic Liaison , 2012, International journal of cell biology.
[6] Yong-mei Yang,et al. Activation of JNK Signaling Mediates Connective Tissue Growth Factor Expression and Scar Formation in Corneal Wound Healing , 2012, PloS one.
[7] G. Schett,et al. Jun N-terminal kinase as a potential molecular target for prevention and treatment of dermal fibrosis , 2012, Annals of the rheumatic diseases.
[8] L. Bubendorf,et al. ROS signaling by NOX4 drives fibroblast-to-myofibroblast differentiation in the diseased prostatic stroma. , 2011, Molecular endocrinology.
[9] M. Landström,et al. Non-Smad signaling pathways , 2011, Cell and Tissue Research.
[10] J. Guerrero,et al. NOX4-dependent ROS production by stromal mammary cells modulates epithelial MCF-7 cell migration , 2010, British Journal of Cancer.
[11] H. Forman,et al. Oxidative Modification of Nuclear Mitogen-activated Protein Kinase Phosphatase 1 Is Involved in Transforming Growth Factor β1-induced Expression of Plasminogen Activator Inhibitor 1 in Fibroblasts* , 2010, The Journal of Biological Chemistry.
[12] M. Shimoda,et al. Carcinoma-associated fibroblasts are a rate-limiting determinant for tumour progression , 2010, Seminars in cell & developmental biology.
[13] P. C. Smith,et al. Triclosan inhibits tumor necrosis factor-alpha-stimulated urokinase production in human gingival fibroblasts. , 2009, Journal of periodontal research.
[14] M. Bissell,et al. Breast cancer by proxy: can the microenvironment be both the cause and consequence? , 2009, Trends in molecular medicine.
[15] Olivier De Wever,et al. Stromal myofibroblasts are drivers of invasive cancer growth , 2008, International journal of cancer.
[16] M. Ushio-Fukai,et al. Reactive oxygen species and angiogenesis: NADPH oxidase as target for cancer therapy. , 2008, Cancer letters.
[17] D. Radisky,et al. Matrix metalloproteinase-induced fibrosis and malignancy in breast and lung. , 2008, Proceedings of the American Thoracic Society.
[18] D. Sorescu,et al. NAD(P)H Oxidase 4 Mediates Transforming Growth Factor-β1–Induced Differentiation of Cardiac Fibroblasts Into Myofibroblasts , 2005, Circulation research.
[19] E. Cukierman,et al. Stromagenesis: the changing face of fibroblastic microenvironments during tumor progression. , 2005, Seminars in cancer biology.
[20] C. Heldin,et al. Non-Smad TGF-β signals , 2005, Journal of Cell Science.
[21] P. Storz. Reactive oxygen species in tumor progression. , 2005, Frontiers in bioscience : a journal and virtual library.
[22] Z. Werb,et al. The fibroblastic coconspirator in cancer progression. , 2005, Cold Spring Harbor symposia on quantitative biology.
[23] N. Fusenig,et al. Friends or foes — bipolar effects of the tumour stroma in cancer , 2004, Nature Reviews Cancer.
[24] T. Cotter,et al. JNK Regulates HIPK3 Expression and Promotes Resistance to Fas-mediated Apoptosis in DU 145 Prostate Carcinoma Cells* , 2004, Journal of Biological Chemistry.
[25] R. Weinberg,et al. Reconstruction of functionally normal and malignant human breast tissues in mice. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[26] S. Hirohashi,et al. Fibrotic focus in infiltrating ductal carcinoma of the breast: A significant histopathological prognostic parameter for predicting the long-term survival of the patients , 1998, Breast Cancer Research and Treatment.
[27] Mina J Bissell,et al. The organizing principle: microenvironmental influences in the normal and malignant breast. , 2002, Differentiation; research in biological diversity.
[28] B. Hinz,et al. Myofibroblasts and mechano-regulation of connective tissue remodelling , 2002, Nature Reviews Molecular Cell Biology.
[29] W. Dröge. Free radicals in the physiological control of cell function. , 2002, Physiological reviews.
[30] A. Levitzki,et al. Enhanced ROS Production in Oncogenically Transformed Cells Potentiates c-Jun N-Terminal Kinase and p38 Mitogen-Activated Protein Kinase Activation and Sensitization to Genotoxic Stress , 2001, Molecular and Cellular Biology.
[31] R. Weinberg,et al. Heterotypic signaling between epithelial tumor cells and fibroblasts in carcinoma formation. , 2001, Experimental cell research.
[32] Leila Mohammadi,et al. BMC Cancer , 2001 .
[33] J. Massagué,et al. Controlling TGF-β signaling , 2000, Genes & Development.
[34] H. Moses,et al. Interdependent SMAD and JNK Signaling in Transforming Growth Factor-β-mediated Transcription* , 1999, The Journal of Biological Chemistry.
[35] M. Trush,et al. Diphenyleneiodonium, an NAD(P)H oxidase inhibitor, also potently inhibits mitochondrial reactive oxygen species production. , 1998, Biochemical and biophysical research communications.
[36] Jeffrey L. Wrana,et al. TGFβ signals through a heteromeric protein kinase receptor complex , 1992, Cell.