Expression of the glucocorticoid receptor is decreased in experimental Staphylococcus aureus sepsis.

[1]  M. Lagerquist,et al.  Estradiol ameliorates arthritis and protects against systemic bone loss in Staphylococcus aureus infection in mice , 2012, Arthritis Research & Therapy.

[2]  I. Jo,et al.  Early initiation of low-dose corticosteroid therapy in the management of septic shock: a retrospective observational study , 2012, Critical Care.

[3]  J. Ijzermans,et al.  Mild versus strong anti-inflammatory therapy during early sepsis in mice: A matter of life and death* , 2011, Critical care medicine.

[4]  D. Annane Adjunct Therapy for Sepsis: How Early? , 2010, Current infectious disease reports.

[5]  Yinglu Feng,et al.  Flow cytometry analysis of glucocorticoid receptor expression and binding in steroid-sensitive and steroid-resistant patients with systemic lupus erythematosus , 2009, Arthritis research & therapy.

[6]  S. Lamberts,et al.  Glucocorticoid receptor mRNA levels are selectively decreased in neutrophils of children with sepsis , 2009, Intensive Care Medicine.

[7]  N. Khardori Hydrocortisone Therapy for Patients with Septic Shock , 2008 .

[8]  J. Sunderram,et al.  Occurrence and outcomes of sepsis: Influence of race* , 2007, Critical care medicine.

[9]  M. Cooper,et al.  Corticosteroid insufficiency in acutely ill patients. , 2003, The New England journal of medicine.

[10]  I. Adcock,et al.  p38 Mitogen-activated protein kinase-induced glucocorticoid receptor phosphorylation reduces its activity: role in steroid-insensitive asthma. , 2002, The Journal of allergy and clinical immunology.

[11]  T. Pritts,et al.  Expression and binding activity of the glucocorticoid receptor are upregulated in septic muscle. , 2002, American journal of physiology. Regulatory, integrative and comparative physiology.

[12]  D. Annane,et al.  Effect of treatment with low doses of hydrocortisone and fludrocortisone on mortality in patients with septic shock. , 2002, JAMA.

[13]  P. Herrlich,et al.  Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptor , 2001, The EMBO journal.

[14]  A. Chow,et al.  Temporal Sequence and Kinetics of Proinflammatory and Anti-Inflammatory Cytokine Secretion Induced by Toxic Shock Syndrome Toxin 1 in Human Peripheral Blood Mononuclear Cells , 2001, Infection and Immunity.

[15]  G. Caramori,et al.  Cross‐talk between pro‐inflammatory transcription factors and glucocorticoids , 2001, Immunology and cell biology.

[16]  J. Cidlowski,et al.  Proinflammatory cytokines regulate human glucocorticoid receptor gene expression and lead to the accumulation of the dominant negative β isoform: A mechanism for the generation of glucocorticoid resistance , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[17]  W. V. Berghe,et al.  Mechanisms of anti-inflammatory action and of immunosuppression by glucocorticoids: negative interference of activated glucocorticoid receptor with transcription factors , 2000, Journal of Neuroimmunology.

[18]  C. Pariante,et al.  Printed in U.S.A. Copyright © 1999 by The Endocrine Society The Proinflammatory Cytokine, Interleukin-1�, Reduces , 2022 .

[19]  G. Piwien-Pilipuk,et al.  Inhibition of glucocorticoid receptor binding by nitric oxide. , 1999, Molecular pharmacology.

[20]  C. Pariante,et al.  Printed in U.S.A. Copyright © 1999 by The Endocrine Society The Proinflammatory Cytokine, Interleukin-1�, Reduces , 2022 .

[21]  M. Welsh,et al.  Heat shock protein 90-dependent (geldanamycin-inhibited) movement of the glucocorticoid receptor through the cytoplasm to the nucleus requires intact cytoskeleton. , 1998, Molecular endocrinology.

[22]  Zhao,et al.  The dual role of interferon‐γ in experimental Staphylococcus aureus septicaemia versus arthritis , 1998, Immunology.

[23]  A. Munck,et al.  Control by basal phosphorylation of cell cycle-dependent, hormone-induced glucocorticoid receptor hyperphosphorylation. , 1997, Molecular endocrinology.

[24]  S. Lamberts,et al.  Temperature‐induced down‐regulation of the glucocorticoid receptor in peripheral blood mononuclear leucocyte in patients with sepsis or septic shock , 1995, Clinical endocrinology.

[25]  P. Rothwell,et al.  Cortisol response to corticotropin and survival in septic shock , 1991, The Lancet.

[26]  A. Munck,et al.  Glucocorticoid receptors and actions. , 1990, The American review of respiratory disease.

[27]  A. Gwosdow,et al.  Interleukin 1 stimulation of the hypothalamic-pituitary-adrenal axis. , 1990, The American journal of physiology.

[28]  P. Nowell,et al.  Dexamethasone inhibition of interleukin 1 beta production by human monocytes. Posttranscriptional mechanisms. , 1988, The Journal of clinical investigation.

[29]  R. Evans,et al.  Cooperative and positional independent trans-activation domains of the human glucocorticoid receptor. , 1988, Cold Spring Harbor symposia on quantitative biology.