Amyloid Deposition Is Associated with Impaired Default Network Function in Older Persons without Dementia
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Keith A. Johnson | R. Buckner | R. Sperling | B. Hyman | T. Hedden | D. Selkoe | M. Pihlajamäki | P. LaViolette | Maija M. Pihlajamaki | J. Becker | D. Rentz | J. O’Brien | K. O'Keefe | G. Marshall
[1] M. Folstein,et al. Clinical diagnosis of Alzheimer's disease: Report of the NINCDS—ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease , 2011, Neurology.
[2] Kelly O'Keefe,et al. Evidence of Altered Posteromedial Cortical fMRI Activity in Subjects at Risk for Alzheimer Disease , 2010, Alzheimer disease and associated disorders.
[3] Cindee M. Madison,et al. Episodic memory loss is related to hippocampal-mediated beta-amyloid deposition in elderly subjects. , 2009, Brain : a journal of neurology.
[4] H. Pashler,et al. Puzzlingly High Correlations in fMRI Studies of Emotion, Personality, and Social Cognition 1 , 2009, Perspectives on psychological science : a journal of the Association for Psychological Science.
[5] M. Rugg,et al. The relationship between aging, performance, and the neural correlates of successful memory encoding. , 2009, Cerebral cortex.
[6] Keith A. Johnson,et al. Cortical Hubs Revealed by Intrinsic Functional Connectivity: Mapping, Assessment of Stability, and Relation to Alzheimer's Disease , 2009, The Journal of Neuroscience.
[7] Jeffrey A. James,et al. Frequent amyloid deposition without significant cognitive impairment among the elderly. , 2008, Archives of neurology.
[8] Arthur Konnerth,et al. Clusters of Hyperactive Neurons Near Amyloid Plaques in a Mouse Model of Alzheimer's Disease , 2008, Science.
[9] Susan M Resnick,et al. Longitudinal Cerebral Blood Flow and Amyloid Deposition: An Emerging Pattern? , 2008, Journal of Nuclear Medicine.
[10] Peter Fransson,et al. The precuneus/posterior cingulate cortex plays a pivotal role in the default mode network: Evidence from a partial correlation network analysis , 2008, NeuroImage.
[11] J. Morris,et al. The Cortical Signature of Alzheimer's Disease: Regionally Specific Cortical Thinning Relates to Symptom Severity in Very Mild to Mild AD Dementia and is Detectable in Asymptomatic Amyloid-Positive Individuals , 2008, Cerebral cortex.
[12] O. Sporns,et al. Mapping the Structural Core of Human Cerebral Cortex , 2008, PLoS biology.
[13] Zhijun Zhang,et al. Default-mode network activity distinguishes amnestic type mild cognitive impairment from healthy aging: A combined structural and resting-state functional MRI study , 2008, Neuroscience Letters.
[14] Keith A. Johnson,et al. Imaging amyloid deposition in Lewy body diseases , 2008, Neurology.
[15] L. Nyberg,et al. Altered deactivation in individuals with genetic risk for Alzheimer's disease , 2008, Neuropsychologia.
[16] Maija Pihlajamäki,et al. Impaired medial temporal repetition suppression is related to failure of parietal deactivation in Alzheimer disease. , 2008, The American journal of geriatric psychiatry : official journal of the American Association for Geriatric Psychiatry.
[17] G. Schroth,et al. Extensive cerebral calcification in a patient with systemic lupus erythematosus , 2008, Journal of Neurology, Neurosurgery, and Psychiatry.
[18] R. Sperling,et al. Age-related memory impairment associated with loss of parietal deactivation but preserved hippocampal activation , 2008, Proceedings of the National Academy of Sciences.
[19] D. Holtzman,et al. Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease , 2008, Nature.
[20] Justin L. Vincent,et al. Disruption of Large-Scale Brain Systems in Advanced Aging , 2007, Neuron.
[21] V. Calhoun,et al. Selective changes of resting-state networks in individuals at risk for Alzheimer's disease , 2007, Proceedings of the National Academy of Sciences.
[22] Paul Maruff,et al. β-amyloid imaging and memory in non-demented individuals: evidence for preclinical Alzheimer's disease , 2007 .
[23] J. Petrella,et al. Prognostic Value of Posteromedial Cortex Deactivation in Mild Cognitive Impairment , 2007, PLoS ONE.
[24] R. Sperling,et al. Hippocampal activation in adults with mild cognitive impairment predicts subsequent cognitive decline , 2007, Journal of Neurology, Neurosurgery, and Psychiatry.
[25] Anatol C. Kreitzer,et al. Aberrant Excitatory Neuronal Activity and Compensatory Remodeling of Inhibitory Hippocampal Circuits in Mouse Models of Alzheimer's Disease , 2007, Neuron.
[26] Keith A. Johnson,et al. Imaging of amyloid burden and distribution in cerebral amyloid angiopathy , 2007, Annals of neurology.
[27] Michael Erb,et al. Hippocampal activation in patients with mild cognitive impairment is necessary for successful memory encoding , 2007, Journal of Neurology, Neurosurgery & Psychiatry.
[28] B. Levine,et al. The functional neuroanatomy of autobiographical memory: A meta-analysis , 2006, Neuropsychologia.
[29] Benjamin J. Shannon,et al. Coherent spontaneous activity identifies a hippocampal-parietal memory network. , 2006, Journal of neurophysiology.
[30] Vince D. Calhoun,et al. Alterations in Memory Networks in Mild Cognitive Impairment and Alzheimer's Disease: An Independent Component Analysis , 2006, The Journal of Neuroscience.
[31] J. Morris,et al. The Uniform Data Set (UDS): Clinical and Cognitive Variables and Descriptive Data From Alzheimer Disease Centers , 2006, Alzheimer disease and associated disorders.
[32] J. Schneider,et al. Neuropathology of older persons without cognitive impairment from two community-based studies , 2006, Neurology.
[33] Felice Sun,et al. Brain imaging evidence of preclinical Alzheimer's disease in normal aging , 2006, Annals of neurology.
[34] Daniel L. Schacter,et al. Understanding metamemory: Neural correlates of the cognitive process and subjective level of confidence in recognition memory , 2006, NeuroImage.
[35] Steven Mennerick,et al. Synaptic Activity Regulates Interstitial Fluid Amyloid-β Levels In Vivo , 2005, Neuron.
[36] S. Rombouts,et al. Altered resting state networks in mild cognitive impairment and mild Alzheimer's disease: An fMRI study , 2005, Human brain mapping.
[37] Benjamin J. Shannon,et al. Parietal lobe contributions to episodic memory retrieval , 2005, Trends in Cognitive Sciences.
[38] Benjamin J. Shannon,et al. Molecular, Structural, and Functional Characterization of Alzheimer's Disease: Evidence for a Relationship between Default Activity, Amyloid, and Memory , 2005, The Journal of Neuroscience.
[39] M. Albert,et al. Increased hippocampal activation in mild cognitive impairment compared to normal aging and AD , 2005, Neurology.
[40] S. DeKosky,et al. Kinetic Modeling of Amyloid Binding in Humans using PET Imaging and Pittsburgh Compound-B , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[41] N. Schuff,et al. Pattern of cerebral hypoperfusion in Alzheimer disease and mild cognitive impairment measured with arterial spin-labeling MR imaging: initial experience. , 2005, Radiology.
[42] Gregory G. Brown,et al. fMRI evidence of compensatory mechanisms in older adults at genetic risk for Alzheimer disease , 2005, Neurology.
[43] Morris Moscovitch,et al. Characterizing spatial and temporal features of autobiographical memory retrieval networks: a partial least squares approach , 2004, NeuroImage.
[44] S. M. Daselaar,et al. When less means more: deactivations during encoding that predict subsequent memory , 2004, NeuroImage.
[45] Dominic M. Walsh,et al. Deciphering the Molecular Basis of Memory Failure in Alzheimer's Disease , 2004, Neuron.
[46] R. Petersen. Mild cognitive impairment as a diagnostic entity , 2004, Journal of internal medicine.
[47] M. Albert,et al. Medial temporal lobe function and structure in mild cognitive impairment , 2004, Annals of neurology.
[48] Mark E Wheeler,et al. Functional-anatomic correlates of remembering and knowing , 2004, NeuroImage.
[49] M. Greicius,et al. Default-mode network activity distinguishes Alzheimer's disease from healthy aging: Evidence from functional MRI , 2004, Proc. Natl. Acad. Sci. USA.
[50] W. Klunk,et al. Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound‐B , 2004, Annals of neurology.
[51] J. Morris,et al. Functional deactivations: Change with age and dementia of the Alzheimer type , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[52] Russell A. Poldrack,et al. Putting names to faces: Successful encoding of associative memories activates the anterior hippocampal formation , 2003, NeuroImage.
[53] Chester A. Mathis,et al. A lipophilic thioflavin-T derivative for positron emission tomography (PET) imaging of amyloid in brain. , 2002, Bioorganic & medicinal chemistry letters.
[54] J. Morris,et al. Current concepts in mild cognitive impairment. , 2001, Archives of neurology.
[55] M. Rugg,et al. Task-dependency of the neural correlates of episodic encoding as measured by fMRI. , 2001, Cerebral cortex.
[56] L. Freire,et al. Motion Correction Algorithms May Create Spurious Brain Activations in the Absence of Subject Motion , 2001, NeuroImage.
[57] G. Alexander,et al. Declining brain activity in cognitively normal apolipoprotein E ɛ4 heterozygotes: A foundation for using positron emission tomography to efficiently test treatments to prevent Alzheimer's disease , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[58] G L Shulman,et al. INAUGURAL ARTICLE by a Recently Elected Academy Member:A default mode of brain function , 2001 .
[59] Mark S. Cohen,et al. Patterns of brain activation in people at risk for Alzheimer's disease. , 2000, The New England journal of medicine.
[60] Keith A. Johnson,et al. Perfusion abnormalities in prodromal AD , 2000, Neurobiology of Aging.
[61] B L Holman,et al. Preclinical prediction of Alzheimer's disease using SPECT , 1998, Neurology.
[62] M. Corbetta,et al. Common Blood Flow Changes across Visual Tasks: II. Decreases in Cerebral Cortex , 1997, Journal of Cognitive Neuroscience.
[63] N. Foster,et al. Metabolic reduction in the posterior cingulate cortex in very early Alzheimer's disease , 1997, Annals of neurology.
[64] J. Price,et al. Cerebral amyloid deposition and diffuse plaques in ``normal'' aging , 1996, Neurology.
[65] S. Snyder,et al. Relative sparing of nitric oxide synthase–containing neurons in the hippocampal formation in Alzheimer's disease , 1992, Annals of neurology.
[66] Bradley T. Hyman,et al. Distribution of Alzheimer‐type pathologic changes in nondemented elderly individuals matches the pattern in Alzheimer's disease , 1992, Neurology.
[67] M. Greicius,et al. Resting-state functional connectivity reflects structural connectivity in the default mode network. , 2009, Cerebral cortex.
[68] M. Mintun,et al. Brain volume decline in aging: evidence for a relation between socioeconomic status, preclinical Alzheimer disease, and reserve. , 2008, Archives of neurology.
[69] Paul Maruff,et al. Beta-amyloid imaging and memory in non-demented individuals: evidence for preclinical Alzheimer's disease. , 2007, Brain : a journal of neurology.
[70] Yvette I. Sheline,et al. Potential antecedent marker of Alzheimer disease , 2006 .
[71] S. Love,et al. Loss of perineuronal net N-acetylgalactosamine in Alzheimer’s disease , 2006, Acta Neuropathologica.
[72] David M Holtzman,et al. Synaptic activity regulates interstitial fluid amyloid-beta levels in vivo. , 2005, Neuron.
[73] Nikos Makris,et al. Automatically parcellating the human cerebral cortex. , 2004, Cerebral cortex.
[74] J. Raaijmakers,et al. Neuroanatomical correlates of episodic encoding and retrieval in young and elderly subjects. , 2003, Brain : a journal of neurology.
[75] A. Nappi,et al. Alzheimer ' s Disease : Cell-Specific Pathology Isolates the Hippocampal Formation , 2022 .