Multiple sclerosis: a study of CXCL10 and CXCR3 co-localization in the inflamed central nervous system
暂无分享,去创建一个
R. Ravid | R. Ransohoff | H. Lassmann | R. Strieter | P. Kivisäkk | C. Trebst | D. Olsen | F. Sellebjerg | T. Sørensen | Karen L. Klaege | A. Majmudar
[1] H. Hartung,et al. Chemokines and chemokine receptors in inflammatory demyelinating neuropathies: a central role for IP-10. , 2002, Brain : a journal of neurology.
[2] R. Ransohoff,et al. Investigating chemokines and chemokine receptors in patients with multiple sclerosis: opportunities and challenges. , 2001, Archives of neurology.
[3] C. V. Jensen,et al. Chemokines CXCL10 and CCL2: differential involvement in intrathecal inflammation in multiple sclerosis , 2001, European journal of neurology.
[4] R. Ransohoff,et al. CCR1+/CCR5+ mononuclear phagocytes accumulate in the central nervous system of patients with multiple sclerosis. , 2001, The American journal of pathology.
[5] H. Keirstead,et al. Neutralization of the Chemokine CXCL10 Reduces Inflammatory Cell Invasion and Demyelination and Improves Neurological Function in a Viral Model of Multiple Sclerosis1 , 2001, The Journal of Immunology.
[6] J. Flier,et al. Differential expression of CXCR3 targeting chemokines CXCL10, CXCL9, and CXCL11 in different types of skin inflammation , 2001, The Journal of pathology.
[7] A. Compston,et al. Recommended diagnostic criteria for multiple sclerosis: Guidelines from the international panel on the diagnosis of multiple sclerosis , 2001, Annals of neurology.
[8] S. Kunkel,et al. EncephalomyelitisDuring Experimental Autoimmune Accumulation in the Central Nervous System T Cell + Control of Encephalitogenic CD4 -Inducible Protein-10) g CXCL10 (IFN-and , 2001 .
[9] H. Lassmann,et al. Migratory activity and functional changes of green fluorescent effector cells before and during experimental autoimmune encephalomyelitis. , 2001, Immunity.
[10] R. Bergamaschi,et al. Serum and CSF levels of MCP-1 and IP-10 in multiple sclerosis patients with acute and stable disease and undergoing immunomodulatory therapies , 2001, Journal of Neuroimmunology.
[11] Y. Itoyama,et al. Chemokine receptor expression on T cells in blood and cerebrospinal fluid at relapse and remission of multiple sclerosis: imbalance of Th1/Th2-associated chemokine signaling , 2001, Journal of Neuroimmunology.
[12] T. Hamilton,et al. Expression of Mig (Monokine Induced by Interferon-γ) Is Important in T Lymphocyte Recruitment and Host Defense Following Viral Infection of the Central Nervous System1 , 2001, Journal of Immunology.
[13] M. Serio,et al. Cell cycle-dependent expression of CXC chemokine receptor 3 by endothelial cells mediates angiostatic activity. , 2001, The Journal of clinical investigation.
[14] C. D. DE GROOT,et al. The role of chemokines and chemokine receptors in CNS inflammation. , 2001 .
[15] L. Trentin,et al. CXC chemokines IP-10 and mig expression and direct migration of pulmonary CD8+/CXCR3+ T cells in the lungs of patients with HIV infection and T-cell alveolitis. , 2000, American journal of respiratory and critical care medicine.
[16] S. Majumder,et al. Chemokines and chemokine receptors in inflammation of the nervous system: manifold roles and exquisite regulation , 2000, Immunological reviews.
[17] J. Parisi,et al. Heterogeneity of multiple sclerosis lesions: Implications for the pathogenesis of demyelination , 2000, Annals of neurology.
[18] R. Ransohoff,et al. Chemokine receptor antagonism as a new therapy for multiple sclerosis , 2000, Expert opinion on investigational drugs.
[19] J. Newcombe,et al. Expression of the interferon‐γ‐inducible chemokines IP‐10 and Mig and their receptor, CXCR3, in multiple sclerosis lesions , 2000, Neuropathology and applied neurobiology.
[20] K. Matsushima,et al. International union of pharmacology. XXII. Nomenclature for chemokine receptors. , 2000, Pharmacological reviews.
[21] A. Zlotnik,et al. Chemokines: a new classification system and their role in immunity. , 2000, Immunity.
[22] J. Noseworthy. Progress in determining the causes and treatment of multiple sclerosis , 1999, Nature.
[23] H. Weiner,et al. CCR5(+) and CXCR3(+) T cells are increased in multiple sclerosis and their ligands MIP-1alpha and IP-10 are expressed in demyelinating brain lesions. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[24] R. Rabin,et al. Chemokine receptor responses on T cells are achieved through regulation of both receptor expression and signaling. , 1999, Journal of immunology.
[25] Jakob S. Jensen,et al. Expression of specific chemokines and chemokine receptors in the central nervous system of multiple sclerosis patients. , 1999, The Journal of clinical investigation.
[26] M. Chilosi,et al. Involvement of the IP-10 chemokine in sarcoid granulomatous reactions. , 1998, Journal of immunology.
[27] James G. Boyd,et al. Interferon–inducible T Cell Alpha Chemoattractant (I-TAC): A Novel Non-ELR CXC Chemokine with Potent Activity on Activated T Cells through Selective High Affinity Binding to CXCR3 , 1998, The Journal of experimental medicine.
[28] C. Mackay,et al. The chemokine receptors CXCR3 and CCR5 mark subsets of T cells associated with certain inflammatory reactions. , 1998, The Journal of clinical investigation.
[29] A. Luster,et al. Chemokines--chemotactic cytokines that mediate inflammation. , 1998, The New England journal of medicine.
[30] T. Sørensen,et al. Etiology and pathogenesis of multiple sclerosis. , 1998, Seminars in neurology.
[31] R. Ransohoff,et al. Synchronous synthesis of alpha- and beta-chemokines by cells of diverse lineage in the central nervous system of mice with relapses of chronic experimental autoimmune encephalomyelitis. , 1997, The American journal of pathology.
[32] Simon A. Jones,et al. Chemokine receptor specific for IP10 and mig: structure, function, and expression in activated T-lymphocytes , 1996, The Journal of experimental medicine.
[33] M. Christiansen,et al. Qualitative assessment of intrathecal IgG synthesis by isoelectric focusing and immunodetection: interlaboratory reproducibility and interobserver agreement. , 1996, Scandinavian journal of clinical and laboratory investigation.
[34] R. Ransohoff,et al. In situ hybridization analysis of glial fibrillary acidic protein mRNA reveals evidence of biphasic astrocyte activation during acute experimental autoimmune encephalomyelitis. , 1996, The American journal of pathology.
[35] Hans Lassmann,et al. Monocyte/macrophage differentiation in early multiple sclerosis lesions , 1995, Annals of neurology.
[36] P. Gray,et al. Chemokine expression in murine experimental allergic encephalomyelitis , 1995, Journal of Neuroimmunology.
[37] W. Brück,et al. Oligodendrocytes in the early course of multiple sclerosis , 1994, Annals of neurology.
[38] R. Ransohoff,et al. Astrocyte expression of mRNA encoding cytokines IP‐10 and JE/MCP‐1 in experimental autoimmune encephalomyelitis , 1993, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[39] F. Da. Demyelinating optic neuritis: clinical features and differential diagnosis. , 1991 .
[40] W. Hickey,et al. T‐lymphocyte entry into the central nervous system , 1991, Journal of neuroscience research.
[41] B. Engelhardt,et al. Interaction of T Lymphocytes with Cerebral Endothelial Cells in vitro , 1991, Brain pathology.
[42] A. A. Eisen,et al. MRI in the diagnosis of MS , 1988, Neurology.