Variation of extrachromosomal circular DNA in cancer cell lines

[1]  Howard Y. Chang,et al.  The evolutionary dynamics of extrachromosomal DNA in human cancers , 2022, Nature Genetics.

[2]  Howard Y. Chang,et al.  Extrachromosomal DNA in the cancerous transformation of Barrett’s oesophagus , 2022, bioRxiv.

[3]  M. Mohiyuddin,et al.  Targeted removal of mitochondrial DNA from mouse and human extrachromosomal circular DNA with CRISPR-Cas9 , 2022, Computational and structural biotechnology journal.

[4]  V. Bafna,et al.  Extrachromosomal DNA in Cancer. , 2022, Annual review of genomics and human genetics.

[5]  B. Regenberg,et al.  A unifying model for extrachromosomal circular DNA load in eukaryotic cells. , 2022, Seminars in cell & developmental biology.

[6]  Howard Y. Chang,et al.  Targeted profiling of human extrachromosomal DNA by CRISPR-CATCH , 2021, bioRxiv.

[7]  Kevin C. Johnson,et al.  Live-cell imaging shows uneven segregation of extrachromosomal DNA elements and transcriptionally active extrachromosomal DNA hubs in cancer. , 2021, Cancer discovery.

[8]  Howard Y. Chang,et al.  EcDNA hubs drive cooperative intermolecular oncogene expression , 2020, bioRxiv.

[9]  Howard Y. Chang,et al.  Extrachromosomal DNA is associated with oncogene amplification and poor outcome across multiple cancers , 2020, Nature Genetics.

[10]  J. Houseley,et al.  Replicative aging is associated with loss of genetic heterogeneity from extrachromosomal circular DNA in Saccharomyces cerevisiae , 2020, bioRxiv.

[11]  Richard C. Sallari,et al.  Functional Enhancers Shape Extrachromosomal Oncogene Amplifications , 2019, Cell.

[12]  Howard Y. Chang,et al.  Circular ecDNA promotes accessible chromatin and high oncogene expression , 2019, Nature.

[13]  Anders Krogh,et al.  Sensitive detection of circular DNAs at single-nucleotide resolution using guided realignment of partially aligned reads , 2019, BMC Bioinformatics.

[14]  V. Bafna,et al.  Exploring the landscape of focal amplifications in cancer using AmpliconArchitect , 2019, Nature Communications.

[15]  Joshua M. Dempster,et al.  Genetic and transcriptional evolution alters cancer cell line drug response , 2018, Nature.

[16]  G. Church,et al.  CRISPR-C: circularization of genes and chromosome by CRISPR in human cells , 2018, bioRxiv.

[17]  B. Wiedenmann,et al.  Octreotide Does Not Inhibit Proliferation in Five Neuroendocrine Tumor Cell Lines , 2018, Front. Endocrinol..

[18]  Michael P Snyder,et al.  Circular DNA elements of chromosomal origin are common in healthy human somatic tissue , 2018, Nature Communications.

[19]  Jiming Jiang,et al.  Extrachromosomal circular DNA-based amplification and transmission of herbicide resistance in crop weed Amaranthus palmeri , 2018, Proceedings of the National Academy of Sciences.

[20]  Lisa K Chopin,et al.  Whole-Genome Sequence of the Metastatic PC3 and LNCaP Human Prostate Cancer Cell Lines , 2017, G3: Genes, Genomes, Genetics.

[21]  Jessica M. Rusert,et al.  Extrachromosomal oncogene amplification drives tumor evolution and genetic heterogeneity , 2017, Nature.

[22]  Eric Talevich,et al.  CNVkit: Genome-Wide Copy Number Detection and Visualization from Targeted DNA Sequencing , 2016, PLoS Comput. Biol..

[23]  David Botstein,et al.  Extrachromosomal circular DNA is common in yeast , 2015, Proceedings of the National Academy of Sciences.

[24]  S. Nelson,et al.  Targeted Therapy Resistance Mediated by Dynamic Regulation of Extrachromosomal Mutant EGFR DNA , 2014, Science.

[25]  Ryan M. Layer,et al.  Extrachromosomal MicroDNAs and Chromosomal Microdeletions in Normal Tissues , 2012, Science.

[26]  A. Levan,et al.  Have double minutes functioning centromeres? , 2009, Hereditas.

[27]  N. Shimizu,et al.  Nonselective DNA damage induced by a replication inhibitor results in the selective elimination of extrachromosomal double minutes from human cancer cells , 2007, Genes, chromosomes & cancer.

[28]  F. Apiou,et al.  Molecular structure of double-minute chromosomes bearing amplified copies of the epidermal growth factor receptor gene in gliomas. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[29]  Douglas Miller,et al.  Formation of extrachromosomal circular DNA in HeLa cells by nonhomologous recombination , 1994, Nucleic Acids Res..

[30]  R. J. Meador,et al.  Double minutes are frequently found in ovarian carcinomas. , 1993, Cancer genetics and cytogenetics.

[31]  M. Quintanilla,et al.  A role for the E-cadherin cell-cell adhesion molecule during tumor progression of mouse epidermal carcinogenesis , 1991, The Journal of cell biology.

[32]  J. Gaubatz.  Extrachromosomal circular DNAs and genomic sequence plasticity in eukaryotic cells. , 1990, Mutation research.

[33]  G. Wahl,et al.  The importance of circular DNA in mammalian gene amplification. , 1989, Cancer research.

[34]  A. Balmain,et al.  v-ras genes from harvey and BALB murine sarcoma viruses can act as initiators of two-stage mouse skin carcinogenesis , 1986, Cell.

[35]  J. Lechner,et al.  Establishment and characterization of a human prostatic carcinoma cell line (PC-3). , 1979, Investigative urology.

[36]  A. Yunis,et al.  Purification and characterization of a plasminogen activator secreted by cultured human pancreatic carcinoma cells. , 1977, Biochemistry.

[37]  H. D. Møller,et al.  Circle-Seq: Isolation and Sequencing of Chromosome-Derived Circular DNA Elements in Cells. , 2020, Methods in molecular biology.

[38]  J. Horoszewicz,et al.  Human pancreatic adenocarcinoma: In vitro and in vivo morphology of a new tumor line established from ascites , 2007, In Vitro Cellular & Developmental Biology - Plant.

[39]  N. Fusenig,et al.  Phenotypic and cytogenetic characteristics of different stages during spontaneous transformation of mouse keratinocytes in vitro. , 1985, Carcinogenesis; a comprehensive survey.