Improved tolerance of acute severe hypoxic stress in chronic hypoxic diaphragm is nitric oxide-dependent

[1]  T. Best,et al.  Role of nitric oxide in muscle regeneration following eccentric muscle contractions in rat skeletal muscle , 2013, The Journal of Physiological Sciences.

[2]  J. Gamboa,et al.  Muscle endurance and mitochondrial function after chronic normobaric hypoxia: contrast of respiratory and limb muscles , 2012, Pflügers Archiv - European Journal of Physiology.

[3]  Shakir Ali,et al.  Chronic hypobaric hypoxia mediated skeletal muscle atrophy: role of ubiquitin–proteasome pathway and calpains , 2012, Molecular and Cellular Biochemistry.

[4]  T. L. Dutka,et al.  Modulation of contractile apparatus Ca2+ sensitivity and disruption of excitation–contraction coupling by S‐nitrosoglutathione in rat muscle fibres , 2011, The Journal of physiology.

[5]  G. Lamb,et al.  Differential effects of peroxynitrite on contractile protein properties in fast- and slow-twitch skeletal muscle fibers of rat. , 2011, Journal of applied physiology.

[6]  K. O'Halloran,et al.  Chronic hypoxia increases rat diaphragm muscle endurance and sodium–potassium ATPase pump content , 2010, European Respiratory Journal.

[7]  I. Kanno,et al.  Contribution of nitric oxide to cerebral blood flow regulation under hypoxia in rats , 2010, The Journal of Physiological Sciences.

[8]  J. Siamwala,et al.  eNOS phosphorylation in health and disease. , 2010, Biochimie.

[9]  A. Kavazis,et al.  Nitric oxide and AMPK cooperatively regulate PGC‐1α in skeletal muscle cells , 2010, The Journal of physiology.

[10]  D. Jore,et al.  Advances in biomolecular and medicinal chemistry. , 2010, Biochimie.

[11]  M. Slevin,et al.  Changes in contractile properties of skinned single rat soleus and diaphragm fibres after chronic hypoxia , 2010, Pflügers Archiv - European Journal of Physiology.

[12]  J. Gamboa,et al.  Mitochondrial content and distribution changes specific to mouse diaphragm after chronic normobaric hypoxia. , 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

[13]  V. Lira,et al.  Endothelial nitric oxide synthase is involved in calcium-induced Akt signaling in mouse skeletal muscle. , 2009, Nitric oxide : biology and chemistry.

[14]  S. Hussain,et al.  Oxidised proteins and superoxide anion production in the diaphragm of severe COPD patients , 2009, European Respiratory Journal.

[15]  G. Gemes,et al.  Nitric oxide activates ATP-sensitive potassium channels in mammalian sensory neurons: action by direct S-nitrosylation , 2009, Molecular pain.

[16]  C. Ottenheijm,et al.  Diaphragm adaptations in patients with COPD , 2008, Respiratory research.

[17]  A. Ascensão,et al.  Vitamin E prevents hypobaric hypoxia-induced mitochondrial dysfunction in skeletal muscle. , 2007, Clinical science.

[18]  Y. Lecarpentier,et al.  Oxidative stress of myosin contributes to skeletal muscle dysfunction in rats with chronic heart failure. , 2007, American journal of physiology. Heart and circulatory physiology.

[19]  Freya Q. Schafer,et al.  Nitric oxide as a cellular antioxidant: a little goes a long way. , 2006, Free radical biology & medicine.

[20]  A. Ascensão,et al.  Acute and severe hypobaric hypoxia increases oxidative stress and impairs mitochondrial function in mouse skeletal muscle. , 2005, Journal of applied physiology.

[21]  V. Jacquemond,et al.  Nitric oxide synthase inhibition affects sarcoplasmic reticulum Ca2+ release in skeletal muscle fibres from mouse , 2005, The Journal of physiology.

[22]  C. Ottenheijm,et al.  Diaphragm dysfunction in chronic obstructive pulmonary disease. , 2005, American journal of respiratory and critical care medicine.

[23]  S. Hussain,et al.  Oxidative stress and respiratory muscle dysfunction in severe chronic obstructive pulmonary disease. , 2005, American journal of respiratory and critical care medicine.

[24]  J. Viña,et al.  Hypoxia-induced dysfunction of rat diaphragm: role of peroxynitrite. , 2005, American journal of physiology. Lung cellular and molecular physiology.

[25]  Y. Jammes,et al.  Matched adaptations of electrophysiological, physiological, and histological properties of skeletal muscles in response to chronic hypoxia , 2005, Pflügers Archiv.

[26]  B. Allard,et al.  Control of intracellular calcium in the presence of nitric oxide donors in isolated skeletal muscle fibres from mouse , 2004, The Journal of physiology.

[27]  Y. Fukuchi,et al.  Hypoxia and hypercapnia affect contractile and histological properties of rat diaphragm and hind limb muscles. , 2004, Pathophysiology : the official journal of the International Society for Pathophysiology.

[28]  C. Gregory,et al.  Human diaphragm remodeling associated with chronic obstructive pulmonary disease: clinical implications. , 2003, American journal of respiratory and critical care medicine.

[29]  A. Bradford,et al.  Effects of chronic hypobaric hypoxia on contractile properties of rat sternohyoid and diaphragm muscles , 2003, Clinical and experimental pharmacology & physiology.

[30]  T. Vanden Hoek,et al.  ROS and NO trigger early preconditioning: relationship to mitochondrial KATP channel. , 2003, American journal of physiology. Heart and circulatory physiology.

[31]  T. Dawson,et al.  Critical role for nitric oxide signaling in cardiac and neuronal ischemic preconditioning and tolerance. , 2001, The Journal of pharmacology and experimental therapeutics.

[32]  C. Colton,et al.  Mechanisms of the antioxidant effects of nitric oxide. , 2001, Antioxidants & redox signaling.

[33]  M. Mori,et al.  Regulation of diaphragmatic nitric oxide synthase expression during hypobaric hypoxia. , 2000, American journal of physiology. Lung cellular and molecular physiology.

[34]  J. Stamler,et al.  The Skeletal Muscle Calcium Release Channel Coupled O2 Sensor and NO Signaling Functions , 2000, Cell.

[35]  J. Lawler,et al.  Interaction of nitric oxide and reactive oxygen species on rat diaphragm contractility. , 2000, Acta physiologica Scandinavica.

[36]  E. Marbán,et al.  Activation of mitochondrial ATP-dependent potassium channels by nitric oxide. , 2000, Circulation.

[37]  H. Westerblad,et al.  Contractile response to low peroxide concentrations: myofibrillar calcium sensitivity as a likely target for redox‐modulation of skeletal muscle function , 2000 .

[38]  M. Mador,et al.  Diaphragmatic fatigue and high-intensity exercise in patients with chronic obstructive pulmonary disease. , 2000, American journal of respiratory and critical care medicine.

[39]  K. Sugimachi,et al.  Amelioration of liver injury by ischaemic preconditioning , 1998, The British journal of surgery.

[40]  D. Allen,et al.  Effect of nitric oxide on single skeletal muscle fibres from the mouse , 1998, The Journal of physiology.

[41]  S. Hamilton,et al.  Nitric Oxide Protects the Skeletal Muscle Ca2+Release Channel from Oxidation Induced Activation* , 1997, The Journal of Biological Chemistry.

[42]  D. Rousseau,et al.  Nitric Oxide Binding to the Heme of Neuronal Nitric-oxide Synthase Links Its Activity to Changes in Oxygen Tension* , 1996, The Journal of Biological Chemistry.

[43]  M. Polkey,et al.  Diaphragm strength in chronic obstructive pulmonary disease. , 1996, American journal of respiratory and critical care medicine.

[44]  A. Zahradníková,et al.  Inhibition of the skeletal muscle ryanodine receptor calcium release channel by nitric oxide , 1996, FEBS letters.

[45]  K. Kogure,et al.  Protection of rat hippocampus against ischemic neuronal damage by pretreatment with sublethal ischemia , 1992, Brain Research.

[46]  R. Jennings,et al.  Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. , 1986, Circulation.