Flavivirus infection up-regulates the expression of class I and class II major histocompatibility antigens on and enhances T cell recognition of astrocytes in vitro

[1]  W. L. Benedict,et al.  Multiple Sclerosis , 2007, Journal - Michigan State Medical Society.

[2]  A. Hill,et al.  Quantitative Considerations of T‐Cell Activation and Self Tolerance , 1987, Immunological reviews.

[3]  M. Rodriguez,et al.  Immune response gene products (Ia antigens) on glial and endothelial cells in virus-induced demyelination. , 1987, Journal of immunology.

[4]  C. Hughes,et al.  Antigen presentation in brain: MHC induction on brain endothelium and astrocytes compared. , 1987, Immunology.

[5]  S. Reingold,et al.  Viral etiology of multiple sclerosis: where does the truth lie? , 1986, Trends in Neurosciences.

[6]  P. A. Peterson,et al.  Adenoviruses of subgenera B, C, D, and E modulate cell-surface expression of major histocompatibility complex class I antigens. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[7]  D. Silberberg,et al.  Coronavirus infection induces H-2 antigen expression on oligodendrocytes and astrocytes. , 1986, Science.

[8]  V. ter meulen,et al.  Viral particles induce Ia antigen expression on astrocytes , 1986, Nature.

[9]  N. King,et al.  Relationship between surface H-2 concentration, size of different target cells, and lysis by cytotoxic T cells. , 1986, Cellular immunology.

[10]  G. Wong,et al.  Interferon-γ induces the expression of H-2 and Ia antigens on brain cells , 1985, Journal of Neuroimmunology.

[11]  R. Anderson,et al.  Effect of herpes simplex virus types 1 and 2 on surface expression of class I major histocompatibility complex antigens on infected cells , 1985, Journal of virology.

[12]  D. Faller,et al.  Retrovirus-induced changes in major histocompatibility complex antigen expression influence susceptibility to lysis by cytotoxic T lymphocytes. , 1985, Journal of immunology.

[13]  E. Appella,et al.  Expression of histocompatibility antigens H-2K, -D, and -L is reduced in adenovirus-12-transformed mouse cells and is restored by interferon gamma. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[14]  H. Macdonald,et al.  Clonal analysis of cytolytic T lymphocyte-mediated lysis of target cells with inducible antigen expression: correlation between antigen density and requirement for Lyt-2/3 function. , 1985, Journal of immunology.

[15]  W. Fierz,et al.  Astrocytes as antigen-presenting cells. I. Induction of Ia antigen expression on astrocytes by T cells via immune interferon and its effect on antigen presentation. , 1985, Journal of immunology.

[16]  R. Ashman,et al.  The cytotoxic response to murine cytomegalovirus. II. In vitro requirements for generation of cytotoxic T cells. , 1985, The Journal of general virology.

[17]  R. Ashman,et al.  The cytotoxic response to murine cytomegalovirus. I. Parameters in vivo. , 1985, The Journal of general virology.

[18]  C. Janeway,et al.  Quantitative variation in la antigen expression plays a central role in immune regulation. , 1984, Immunology today.

[19]  M. Esiri,et al.  Immunohistological analysis of T lymphocyte subsets in the central nervous system in chronic progressive multiple sclerosis , 1983, Journal of the Neurological Sciences.

[20]  C. Goridis,et al.  Expression of Ia antigens by cultured astrocytes treated with gamma-interferon , 1983, Neuroscience Letters.

[21]  T. Onodera,et al.  Multiple organ-reactive monoclonal autoantibodies , 1983, Nature.

[22]  T. Onodera,et al.  Virus-induced autoimmunity: monoclonal antibodies that react with endocrine tissues. , 1983, Science.

[23]  H. Rodt,et al.  Immunohistochemical identification of T-lymphocytes in the central nervous system of patients with multiple sclerosis and subacute sclerosing panencephalitis , 1982, Journal of Neuroimmunology.

[24]  P. Marrack,et al.  Antigen-inducible, H-2-restricted, interleukin-2-producing T cell hybridomas. Lack of independent antigen and H-2 recognition , 1981, The Journal of experimental medicine.

[25]  B. Bloom,et al.  Decreased expression of H-2 antigens following acute measles virus infection. , 1981, Cellular immunology.

[26]  K. Lafferty,et al.  An improved assay for interleukin 2 (lymphocyte growth factor) produced by mitogen-activated lymphocytes. , 1980, The Australian journal of experimental biology and medical science.

[27]  K. McCarthy,et al.  Preparation of separate astroglial and oligodendroglial cell cultures from rat cerebral tissue , 1980, The Journal of cell biology.

[28]  P. Morris,et al.  Distribution and quantitation of HLA-ABC and DR (Ia) antigens on human kidney and other tissues. , 1980, Transplantation.

[29]  K. Ozato,et al.  Hybridoma cell lines secreting monoclonal antibodies to mouse H-2 and Ia antigens. , 1980, Journal of immunology.

[30]  W. Tourtellotte,et al.  Multiple sclerosis , 1978, Neurology.

[31]  A. Bellamy,et al.  Clones of cytotoxic lymphocytes can recognise uninfected cells in a primary response against influenza virus , 1978, Nature.

[32]  G. Ada,et al.  Cytotoxic T cells specific for influenza virus-infected target cells. , 1977, Immunology.

[33]  J. Goding.  Conjugation of antibodies with fluorochromes: modifications to the standard methods. , 1976, Journal of immunological methods.

[34]  H. Lipton,et al.  Theiler's virus-induced demyelination: prevention by immunosuppression. , 1976, Science.

[35]  K. Pfizenmaier,et al.  Temporary presence of self-reactive cytotoxic T lymphocytes during murine lymphocytic choriomeningitis , 1975, Nature.

[36]  I. Marshall,et al.  Adaptation studies with Ross River virus: laboratory mice and cell cultures. , 1975, The Journal of general virology.

[37]  R. Blanden,et al.  Cell‐mediated cytotoxicity against ectromelia virus‐infected target cells. III. Role of the H‐2 gene complex , 1975, European journal of immunology.

[38]  L. Reed,et al.  A SIMPLE METHOD OF ESTIMATING FIFTY PER CENT ENDPOINTS , 1938 .

[39]  T. Honjo,et al.  Interleukin 4 (IgG1 induction factor): a multifunctional lymphokine acting also on T cells , 1987, European journal of immunology.

[40]  M. Blackman,et al.  Infection of cultured murine brain cells by Semliki Forest virus: effects of interferon-alpha beta on viral replication, viral antigen display, major histocompatibility complex antigen display and lysis by cytotoxic T lymphocytes. , 1987, The Journal of general virology.

[41]  T. Monath.  Pathobiology of the Flaviviruses , 1986 .

[42]  R. Schwartz.  Immune response (Ir) genes of the murine major histocompatibility complex. , 1986, Advances in immunology.

[43]  W. Fierz,et al.  T lymphocyte autoimmunity in experimental autoimmune encephalomyelitis. , 1985, Concepts in immunopathology.

[44]  G. Wong,et al.  Inducible expression of H–2 and Ia antigens on brain cells , 1984, Nature.

[45]  E. De Clercq.  Interferon induction by polynucleotides, modified polynucleotides, and polycarboxylates. , 1981, Methods in enzymology.

[46]  E. Clercq.  Interferon induction by polynucleotides, modified polynucleotides, and polycarboxylates. , 1981 .

[47]  R M Zinkernagel,et al.  MHC-restricted cytotoxic T cells: studies on the biological role of polymorphic major transplantation antigens determining T-cell restriction-specificity, function, and responsiveness. , 1979, Advances in immunology.

[48]  L. Herzenberg,et al.  Properties of monoclonal antibodies to mouse Ig allotypes, H-2, and Ia antigens. , 1978, Current topics in microbiology and immunology.