Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1
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Paul T. Schumacker | D. James Surmeier | D. Surmeier | J. Surmeier | J. N. Guzman | E. Ilijić | P. Schumacker | J. Kondapalli | D. Wokosin | Javier Sanchez-Padilla | David Wokosin | J. Sánchez-Padilla | Jaime N. Guzman | Ema Ilijic | Jyothisri Kondapalli | J. N. Guzmán | J. Sánchez-Padilla | Jyothisri Kondapalli | Jaime N. Guzmán | Javier Sánchez-Padilla
[1] Mark S. Cohen,et al. Patterns of brain activation in people at risk for Alzheimer's disease. , 2000, The New England journal of medicine.
[2] R. K. Hutson,et al. Abnormal connectivity in the posterior cingulate and hippocampus in early Alzheimer's disease and mild cognitive impairment , 2008, Alzheimer's & Dementia.
[3] Patrizia Rizzu,et al. Mutations in the DJ-1 Gene Associated with Autosomal Recessive Early-Onset Parkinsonism , 2002, Science.
[4] C. Almeida,et al. Oligomerization of Alzheimer's β-Amyloid within Processes and Synapses of Cultured Neurons and Brain , 2004, The Journal of Neuroscience.
[5] Shaomin Li,et al. Soluble Oligomers of Amyloid β Protein Facilitate Hippocampal Long-Term Depression by Disrupting Neuronal Glutamate Uptake , 2009, Neuron.
[6] Jürgen Götz,et al. Primary support cultures of hippocampal and substantia nigra neurons , 2008, Nature Protocols.
[7] R. Kesner,et al. Differential contributions of dorsal hippocampal subregions to memory acquisition and retrieval in contextual fear‐conditioning , 2004, Hippocampus.
[8] José Luis de la Pompa,et al. Differential Requirement for Caspase 9 in Apoptotic Pathways In Vivo , 1998, Cell.
[9] P. Greengard,et al. Beta-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses , 2005, Neurobiology of Disease.
[10] Tamas L. Horvath,et al. Uncoupling Protein-2 Is Critical for Nigral Dopamine Cell Survival in a Mouse Model of Parkinson's Disease , 2005, The Journal of Neuroscience.
[11] K. Johnson. An Update. , 1984, Journal of food protection.
[12] Mark F Bear,et al. NMDA Induces Long-Term Synaptic Depression and Dephosphorylation of the GluR1 Subunit of AMPA Receptors in Hippocampus , 1998, Neuron.
[13] D. James Surmeier,et al. Robust Pacemaking in Substantia Nigra Dopaminergic Neurons , 2009, The Journal of Neuroscience.
[14] J. Morrison,et al. Life and death of neurons in the aging brain. , 1997, Science.
[15] C. Reutelingsperger,et al. Annexin V-affinity assay: a review on an apoptosis detection system based on phosphatidylserine exposure. , 1998, Cytometry.
[16] M. Elstner,et al. Dopaminergic midbrain neurons are the prime target for mitochondrial DNA deletions , 2008, Journal of Neurology.
[17] J. Bourne,et al. Balancing structure and function at hippocampal dendritic spines. , 2008, Annual review of neuroscience.
[18] G. Cole,et al. Rapid annexin-V labeling in synaptosomes , 2004, Neurochemistry International.
[19] A. Schapira. Mitochondria in the aetiology and pathogenesis of Parkinson's disease , 2008, The Lancet Neurology.
[20] David Smith,et al. Involvement of Caspases in Proteolytic Cleavage of Alzheimer’s Amyloid-β Precursor Protein and Amyloidogenic Aβ Peptide Formation , 1999, Cell.
[21] T. Gasser,et al. DJ-1 and prevention of oxidative stress in Parkinson's disease and other age-related disorders. , 2009, Free radical biology & medicine.
[22] B. Hyman,et al. APPSW Transgenic Mice Develop Age‐related Aβ Deposits and Neuropil Abnormalities, but no Neuronal Loss in CA1 , 1997, Journal of neuropathology and experimental neurology.
[23] Thomas D. Schmittgen,et al. Analyzing real-time PCR data by the comparative CT method , 2008, Nature Protocols.
[24] D. Bers,et al. Oxygen-bridged Dinuclear Ruthenium Amine Complex Specifically Inhibits Ca2+ Uptake into Mitochondria in Vitroand in Situ in Single Cardiac Myocytes* , 1998, The Journal of Biological Chemistry.
[25] J. Olsen,et al. L‐type calcium channel blockers and Parkinson disease in Denmark , 2009, Annals of neurology.
[26] Zayd M. Khaliq,et al. Pacemaking in Dopaminergic Ventral Tegmental Area Neurons: Depolarizing Drive from Background and Voltage-Dependent Sodium Conductances , 2010, The Journal of Neuroscience.
[27] Mark A. Wilson,et al. The Parkinson's disease protein DJ-1 is neuroprotective due to cysteine-sulfinic acid-driven mitochondrial localization , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[28] George A. Carlson,et al. The Relationship between Aβ and Memory in the Tg2576 Mouse Model of Alzheimer's Disease , 2002, The Journal of Neuroscience.
[29] P. Balaban,et al. Caspase‐like activity is essential for long‐term synaptic plasticity in the terrestrial snail Helix , 2006, The European journal of neuroscience.
[30] D. Ferrari,et al. Apoptosis signaling by death receptors. , 1998, European journal of biochemistry.
[31] S. Younkin,et al. Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic Mice , 1996, Science.
[32] Guiquan Chen,et al. A learning deficit related to age and β-amyloid plaques in a mouse model of Alzheimer's disease , 2000, Nature.
[33] D. Bennett,et al. Caspase-3 is enriched in postsynaptic densities and increased in Alzheimer's disease. , 2008, The American journal of pathology.
[34] M. Sheng,et al. Caspase-3 Activation via Mitochondria Is Required for Long-Term Depression and AMPA Receptor Internalization , 2010, Cell.
[35] T. Bliss,et al. Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice , 1999, Nature Neuroscience.
[36] E. Pérez-Payá,et al. A Nanoconjugate Apaf-1 Inhibitor Protects Mesothelial Cells from Cytokine-Induced Injury , 2009, PloS one.
[37] A. Bookout,et al. High‐Throughput Real‐Time Quantitative Reverse Transcription PCR , 2005, Current protocols in molecular biology.
[38] B. Lowell,et al. The mitochondrial uncoupling-protein homologues , 2005, Nature Reviews Molecular Cell Biology.
[39] Thomas Arendt,et al. Synaptic degeneration in Alzheimer’s disease , 2009, Acta Neuropathologica.
[40] L. Kováč. Bioenergetics , 2008 .
[41] F. Schmitt,et al. Synaptic alterations in CA1 in mild Alzheimer disease and mild cognitive impairment , 2007, Neurology.
[42] D. Nicholls. Oxidative Stress and Energy Crises in Neuronal Dysfunction , 2008, Annals of the New York Academy of Sciences.
[43] Mark Bowlby,et al. Early-onset behavioral and synaptic deficits in a mouse model of Alzheimer's disease. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[44] Mark R. Cookson. DJ‐1, PINK1, and their effects on mitochondrial pathways , 2010, Movement disorders : official journal of the Movement Disorder Society.
[45] Wade Morishita,et al. Generation of Silent Synapses by Acute In Vivo Expression of CaMKIV and CREB , 2005, Neuron.
[46] Keisuke Kuida,et al. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice , 1996, Nature.
[47] Keisuke Kuida,et al. Reduced Apoptosis and Cytochrome c–Mediated Caspase Activation in Mice Lacking Caspase 9 , 1998, Cell.
[48] P. Bernardi,et al. The mitochondrial permeability transition pore and its involvement in cell death and in disease pathogenesis , 2007, Apoptosis.
[49] M. Mattson,et al. Evidence for Synaptic Apoptosis , 1998, Experimental Neurology.
[50] A. Bookout,et al. High‐Throughput Real‐Time Quantitative Reverse Transcription PCR , 2005 .
[51] T. Joh,et al. Neuroprotection and Neuronal Differentiation Studies Using Substantia Nigra Dopaminergic Cells Derived from Transgenic Mouse Embryos , 1999, The Journal of Neuroscience.
[52] R. Tsien,et al. Imaging Dynamic Redox Changes in Mammalian Cells with Green Fluorescent Protein Indicators* , 2004, Journal of Biological Chemistry.
[53] B. Lowell,et al. Genipin inhibits UCP2-mediated proton leak and acutely reverses obesity- and high glucose-induced beta cell dysfunction in isolated pancreatic islets. , 2006, Cell metabolism.
[54] C. Tanner,et al. USE OF ANTIHYPERTENSIVES AND THE RISK OF PARKINSON DISEASE , 2009, Neurology.
[55] J. Penney,et al. The functional anatomy of disorders of the basal ganglia , 1995, Trends in Neurosciences.
[56] Mikio Shoji,et al. Age-Dependent Changes in Brain, CSF, and Plasma Amyloid β Protein in the Tg2576 Transgenic Mouse Model of Alzheimer's Disease , 2001, The Journal of Neuroscience.
[57] S. Papa,et al. Reactive oxygen species, mitochondria, apoptosis and aging , 2004, Molecular and Cellular Biochemistry.
[58] D. Clayton,et al. Dynamic Role of Postsynaptic Caspase-3 and BIRC4 in Zebra Finch Song-Response Habituation , 2006, Neuron.
[59] D. Green,et al. The Pathophysiology of Mitochondrial Cell Death , 2004, Science.
[60] J. Stuart,et al. Superoxide activates mitochondrial uncoupling proteins , 2002, Nature.
[61] Joseph E LeDoux,et al. Differential contribution of amygdala and hippocampus to cued and contextual fear conditioning. , 1992, Behavioral neuroscience.
[62] K. Jellinger,et al. Activation of caspase-3 in single neurons and autophagic granules of granulovacuolar degeneration in Alzheimer's disease. Evidence for apoptotic cell death. , 1999, The American journal of pathology.
[63] Robert W. Taylor,et al. High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease , 2006, Nature Genetics.
[64] D. James Surmeier,et al. ‘Rejuvenation’ protects neurons in mouse models of Parkinson’s disease , 2007, Nature.
[65] T. Lanz,et al. Dendritic spine loss in the hippocampus of young PDAPP and Tg2576 mice and its prevention by the ApoE2 genotype , 2003, Neurobiology of Disease.
[66] L M Loew,et al. Membrane potential can be determined in individual cells from the nernstian distribution of cationic dyes. , 1988, Biophysical journal.
[67] R. Malenka,et al. AMPA receptor trafficking and synaptic plasticity. , 2002, Annual review of neuroscience.
[68] R. Malinow,et al. AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss , 2006, Neuron.
[69] G. Rotilio,et al. Calcineurin Activity Is Regulated Both by Redox Compounds and by Mutant Familial Amyotrophic Lateral Sclerosis‐Superoxide Dismutase , 2000, Journal of neurochemistry.
[70] Laura C. Greaves,et al. Mitochondrial DNA Mutations and Aging , 2007, Annals of the New York Academy of Sciences.
[71] M. Eisenberg,et al. Calcium channel blockers: an update. , 2004, The American journal of medicine.
[72] A. J. Lambert,et al. Mitochondrial superoxide and aging: uncoupling-protein activity and superoxide production. , 2004, Biochemical Society symposium.
[73] D. Surmeier,et al. MEF-2 regulates activity-dependent spine loss in striatopallidal medium spiny neurons , 2010, Molecular and Cellular Neuroscience.
[74] Ralph A. Nixon,et al. Aβ peptide immunization reduces behavioural impairment and plaques in a model of Alzheimer's disease , 2000, Nature.
[75] B. Bean,et al. Roles of Subthreshold Calcium Current and Sodium Current in Spontaneous Firing of Mouse Midbrain Dopamine Neurons , 2007, The Journal of Neuroscience.
[76] Francesco Cecconi,et al. Apaf1 (CED-4 Homolog) Regulates Programmed Cell Death in Mammalian Development , 1998, Cell.