Degradation of interleukin 8 by the serine protease MucD of Pseudomonas aeruginosa

[1]  S. Akira,et al.  Pathogen Recognition by the Innate Immune System , 2011, International reviews of immunology.

[2]  S. Minagawa,et al.  Translocation of Pseudomonas aeruginosa from the Intestinal Tract Is Mediated by the Binding of ExoS to an Na,K-ATPase Regulator, FXYD3 , 2010, Infection and Immunity.

[3]  M. Bernardini,et al.  Pseudomonas aeruginosa Exploits Lipid A and Muropeptides Modification as a Strategy to Lower Innate Immunity during Cystic Fibrosis Lung Infection , 2009, PloS one.

[4]  L. Touqui,et al.  Pseudomonas aeruginosa LPS or Flagellin Are Sufficient to Activate TLR-Dependent Signaling in Murine Alveolar Macrophages and Airway Epithelial Cells , 2009, PloS one.

[5]  A. Hauser.  The type III secretion system of Pseudomonas aeruginosa: infection by injection , 2009, Nature Reviews Microbiology.

[6]  Zhu Fu,et al.  Pseudomonas aeruginosa Inhibition of Flagellin-Activated NF-κB and Interleukin-8 by Human Airway Epithelial Cells , 2009, Infection and Immunity.

[7]  R. Sauer,et al.  Control of Pseudomonas aeruginosa AlgW protease cleavage of MucA by peptide signals and MucB , 2009, Molecular microbiology.

[8]  V. Nizet,et al.  The IL-8 protease SpyCEP/ScpC of group A Streptococcus promotes resistance to neutrophil killing. , 2008, Cell host & microbe.

[9]  F. Sutterwala,et al.  Immune recognition of Pseudomonas aeruginosa mediated by the IPAF/NLRC4 inflammasome , 2007, The Journal of experimental medicine.

[10]  Amnon Peled,et al.  A streptococcal protease that degrades CXC chemokines and impairs bacterial clearance from infected tissues , 2006, The EMBO journal.

[11]  V. Deretic,et al.  Nonclassical Pathway of Pseudomonas aeruginosa DNA-Induced Interleukin-8 Secretion in Cystic Fibrosis Airway Epithelial Cells , 2006, Infection and Immunity.

[12]  D. Ohman,et al.  Independent Regulation of MucD, an HtrA-Like Protease in Pseudomonas aeruginosa, and the Role of Its Proteolytic Motif in Alginate Gene Regulation , 2006, Journal of bacteriology.

[13]  R. Edwards,et al.  Specific C-terminal cleavage and inactivation of interleukin-8 by invasive disease isolates of Streptococcus pyogenes. , 2005, The Journal of infectious diseases.

[14]  R. Pickersgill,et al.  Shigella effector IpaH9.8 binds to a splicing factor U2AF(35) to modulate host immune responses. , 2005, Biochemical and biophysical research communications.

[15]  J. Wiener-Kronish,et al.  Single-Nucleotide-Polymorphism Mapping of the Pseudomonas aeruginosa Type III Secretion Toxins for Development of a Diagnostic Multiplex PCR System , 2003, Journal of Clinical Microbiology.

[16]  R. Mrsny,et al.  Cdc42 and Rac1 Regulate Late Events in Salmonella typhimurium-induced Interleukin-8 Secretion from Polarized Epithelial Cells* , 2002, The Journal of Biological Chemistry.

[17]  Anna L Jansson,et al.  Exoenzyme S shows selective ADP-ribosylation and GTPase-activating protein (GAP) activities towards small GTPases in vivo. , 2002, The Biochemical journal.

[18]  A. Aitken,et al.  A nonphosphorylated 14-3-3 binding motif on exoenzyme S that is functional in vivo. , 2002, European journal of biochemistry.

[19]  R. Hancock,et al.  Multidrug Efflux Systems Play an Important Role in the Invasiveness of Pseudomonas aeruginosa , 2002, The Journal of experimental medicine.

[20]  J. da Silva,et al.  Comparison of the exoS Gene and Protein Expression in Soil and Clinical Isolates of Pseudomonas aeruginosa , 2001, Infection and Immunity.

[21]  A. Wittinghofer,et al.  Structure of the ExoS GTPase activating domain , 2001, FEBS letters.

[22]  K. Aktories,et al.  The N-terminal Domain of Pseudomonas aeruginosaExoenzyme S Is a GTPase-activating Protein for Rho GTPases* , 1999, The Journal of Biological Chemistry.

[23]  J. Galán,et al.  Type III secretion machines: bacterial devices for protein delivery into host cells. , 1999, Science.

[24]  Tetsuya Matsumoto,et al.  Effect of Interleukin-10 on Gut-Derived Sepsis Caused by Pseudomonas aeruginosa in Mice , 1998, Antimicrobial Agents and Chemotherapy.

[25]  K. Tateda,et al.  Adverse effects of tumour necrosis factor in cyclophosphamide-treated mice subjected to gut-derived Pseudomonas aeruginosa sepsis. , 1997, Cytokine.

[26]  V. Deretic,et al.  Two distinct loci affecting conversion to mucoidy in Pseudomonas aeruginosa in cystic fibrosis encode homologs of the serine protease HtrA , 1996, Journal of bacteriology.

[27]  M. Kaku,et al.  Mortality rates amongst mice with endogenous septicaemia caused by Pseudomonas aeruginosa isolates from various clinical sources. , 1993, Journal of medical microbiology.

[28]  J. Coburn,et al.  ADP-ribosylation of p21ras and related proteins by Pseudomonas aeruginosa exoenzyme S , 1991, Infection and immunity.

[29]  R. Wyatt,et al.  Several GTP-binding proteins, including p21c-H-ras, are preferred substrates of Pseudomonas aeruginosa exoenzyme S. , 1989, The Journal of biological chemistry.

[30]  C. Janeway,et al.  Innate immune recognition. , 2002, Annual review of immunology.