Requirement for CD44 in activated T cell extravasation into an inflammatory site.
暂无分享,去创建一个
[1] E. Puré,et al. CD44 is necessary for optimal contact allergic responses but is not required for normal leukocyte extravasation , 1993, The Journal of experimental medicine.
[2] L. Picker,et al. CD44 and its ligand hyaluronate mediate rolling under physiologic flow: a novel lymphocyte-endothelial cell primary adhesion pathway , 1996, The Journal of experimental medicine.
[3] B. Haynes,et al. Measurement of an adhesion molecule as an indicator of inflammatory disease activity. Up-regulation of the receptor for hyaluronate (CD44) in rheumatoid arthritis. , 2010, Arthritis and rheumatism.
[4] P. Marrack,et al. Superantigens: bacterial and viral proteins that manipulate the immune system. , 1993, Annual review of cell biology.
[5] J. Sprent. T and B memory cells , 1994, Cell.
[6] S. Jalkanen,et al. A lymphoid cell surface glycoprotein involved in endothelial cell recognition and lymphocyte homing in man , 1986, European journal of immunology.
[7] S. Kubik,et al. CLS 2210, Hyaluronidase and the Cardiac Lymphatic System , 1985, Angiology.
[8] P. Kincade,et al. CD44 and its interaction with extracellular matrix. , 1993, Advances in immunology.
[9] P. Marrack,et al. Profound deletion of mature T cells in vivo by chronic exposure to exogenous superantigen. , 1993, Journal of immunology.
[10] L. Picker,et al. Lymphocyte Homing and Homeostasis , 1996, Science.
[11] P. Libby,et al. Favorable effects of hyaluronidase on electrocardiographic evidence of necrosis in patients with acute myocardial infarction. , 1977, The New England journal of medicine.
[12] T. Issekutz. Inhibition of in vivo lymphocyte migration to inflammation and homing to lymphoid tissues by the TA-2 monoclonal antibody. A likely role for VLA-4 in vivo. , 1991, Journal of immunology.
[13] H. Degrendele,et al. CD44 activation and associated primary adhesion is inducible via T cell receptor stimulation. , 1997, Journal of immunology.
[14] R. Bräuer,et al. Therapeutic effects of antibodies against adhesion molecules in murine collagen type II-induced arthritis. , 1995, Autoimmunity.
[15] J. Sprent,et al. Antigen-induced selective recruitment of circulating lymphocytes. , 1971, Cellular immunology.
[16] M. Glimcher,et al. Receptor-Ligand Interaction Between CD44 and Osteopontin (Eta-1) , 1996, Science.
[17] K. Miyake,et al. Hyaluronate can function as a cell adhesion molecule and CD44 participates in hyaluronate recognition , 1990, The Journal of experimental medicine.
[18] T. Springer. Traffic signals for lymphocyte recirculation and leukocyte emigration: The multistep paradigm , 1994, Cell.
[19] C. Mackay,et al. T-cell memory: the connection between function, phenotype and migration pathways. , 1991, Immunology today.
[20] R N Cahill,et al. The effects of antigen on the migration of recirculating lymphocytes through single lymph nodes , 1976, The Journal of experimental medicine.
[21] T. Herrmann,et al. In vivo responses of CD4+ and CD8+ cells to bacterial superantigens , 1992, European journal of immunology.
[22] Elizabeth Simpson,et al. A rapid method for the isolation of functional thymus‐derived murine lymphocytes , 1973, European journal of immunology.
[23] D. McGregor,et al. THE MEDIATOR OF CELLULAR IMMUNITY , 1971, The Journal of experimental medicine.
[24] T. Glant,et al. Anti-CD44 treatment abrogates tissue aedema and leukocyte infiltration in murine arthrtis , 1995, Nature Medicine.
[25] J. R. de los Toyos,et al. Lymphocyte recognition of high endothelium: antibodies to distinct epitopes of an 85-95-kD glycoprotein antigen differentially inhibit lymphocyte binding to lymph node, mucosal, or synovial endothelial cells , 1987, The Journal of cell biology.
[26] K. O. Wik,et al. Preparation and properties of fluorescein-labelled hyaluronate. , 1975, Carbohydrate research.
[27] L. McIntire,et al. P-selectin mediates neutrophil rolling on histamine-stimulated endothelial cells. , 1993, Biophysical journal.