Recombinant human Hsp70 protects against lipoteichoic acid-induced inflammation manifestations at the cellular and organismal levels
暂无分享,去创建一个
[1] E. Rozhkova,et al. Effect of extracellular recombinant human heat shock protein 70 (HSP70) on protein pattern observed after endotoxin-induced macrophage activation , 2011, Molecular Biology.
[2] T. Gustot. Multiple organ failure in sepsis: prognosis and role of systemic inflammatory response , 2011, Current opinion in critical care.
[3] Sue E. Poynter,et al. Biological activity of truncated C-terminus human heat shock protein 72. , 2011, Immunology letters.
[4] D. Remick,et al. The pathogenesis of sepsis. , 2011, Annual review of pathology.
[5] V. Karpov,et al. Exogenous mammalian extracellular HSP70 reduces endotoxin manifestations at the cellular and organism levels , 2010, Annals of the New York Academy of Sciences.
[6] Jason C. Young. Mechanisms of the Hsp70 chaperone system. , 2010, Biochemistry and cell biology = Biochimie et biologie cellulaire.
[7] B. Henderson. Integrating the cell stress response: a new view of molecular chaperones as immunological and physiological homeostatic regulators , 2010, Cell biochemistry and function.
[8] G. M. Nagaraja,et al. Chaperokine Function of Recombinant Hsp72 Produced in Insect Cells Using a Baculovirus Expression System Is Retained* , 2009, The Journal of Biological Chemistry.
[9] H. Hirasawa,et al. Blood glucose control in patients with severe sepsis and septic shock. , 2009, World journal of gastroenterology.
[10] E. Rozhkova,et al. Exogenous heat shock proteins (HSP70) significantly inhibit endotoxin-induced activation of human neutrophils , 2009, Doklady Biological Sciences.
[11] M. Tsan,et al. Heat shock proteins and immune system , 2009, Journal of leukocyte biology.
[12] A. Ferguson,et al. Gram-positive toxic shock syndromes. , 2009, The Lancet. Infectious diseases.
[13] T. Hartung,et al. Cellular trafficking of lipoteichoic acid and Toll‐like receptor 2 in relation to signaling; role of CD14 and CD36 , 2008, Journal of leukocyte biology.
[14] K. Wasan,et al. Discovery and Development of Toll-Like Receptor 4 (TLR4) Antagonists: A New Paradigm for Treating Sepsis and Other Diseases , 2008, Pharmaceutical Research.
[15] M. Yurinskaya,et al. Dynamics of the Fas-and stress-induced apoptosis of human neutrophils under the action of endotoxins , 2006, Doklady Biological Sciences.
[16] M. Fleshner,et al. Releasing signals, secretory pathways, and immune function of endogenous extracellular heat shock protein 72 , 2006, Journal of leukocyte biology.
[17] Y. Inagaki,et al. ORAL ADMINISTRATION OF GERANYLGERANYLACETONE IMPROVES SURVIVAL RATE IN A RAT ENDOTOXIN SHOCK MODEL: ADMINISTRATION TIMING AND HEAT SHOCK PROTEIN 70 INDUCTION , 2005, Shock.
[18] M. De la Fuente,et al. Role of free radicals in sepsis: antioxidant therapy. , 2005, Current pharmaceutical design.
[19] Chien-Huang Lin,et al. Lipoteichoic acid induces nuclear factor‐κB activation and nitric oxide synthase expression via phosphatidylinositol 3‐kinase, Akt, and p38 MAPK in RAW 264.7 macrophages , 2005, Immunology.
[20] D. Christiani,et al. Clinical predictors of and mortality in acute respiratory distress syndrome: Potential role of red cell transfusion* , 2005, Critical care medicine.
[21] Xuetao Cao,et al. Heat shock up‐regulates expression of Toll‐like receptor‐2 and Toll‐like receptor‐4 in human monocytes via p38 kinase signal pathway , 2005, Immunology.
[22] T. Evans,et al. Epidemiology of acute lung injury , 2005, Current opinion in critical care.
[23] A. Miguel,et al. Sepsis and SOFA score: related outcome for critically ill renal patients. , 2004, Clinical Nephrology.
[24] Chieh-fu Chen,et al. Prevention of macrophage adhesion molecule-1 (Mac-1)-dependent neutrophil firm adhesion by taxifolin through impairment of protein kinase-dependent NADPH oxidase activation and antagonism of G protein-mediated calcium influx. , 2004, Biochemical pharmacology.
[25] M. Tsan,et al. Cytokine function of heat shock proteins. , 2004, American journal of physiology. Cell physiology.
[26] T. Hartung,et al. Highly purified lipoteichoic acid activates neutrophil granulocytes and delays their spontaneous apoptosis via CD14 and TLR2 , 2004, Journal of leukocyte biology.
[27] S. Foster,et al. Peptidoglycan and Lipoteichoic Acid in Gram-Positive Bacterial Sepsis: Receptors, Signal Transduction, Biological Effects, and Synergism , 2003, Shock.
[28] L. O’Neill. Therapeutic targeting of Toll-like receptors for inflammatory and infectious diseases. , 2003, Current opinion in pharmacology.
[29] D. Mannino,et al. The epidemiology of sepsis in the United States from 1979 through 2000. , 2003, The New England journal of medicine.
[30] K. Irie,et al. Effect of Heat Stress on Lipopolysaccharide-Induced Vascular Permeability Change in Mice , 2002, Journal of Pharmacology and Experimental Therapeutics.
[31] H. Redmond,et al. Cellular Apoptosis and Organ Injury in Sepsis: A Review , 2002, Shock.
[32] X. Ding,et al. Over-expression of hsp-70 inhibits bacterial lipopolysaccharide-induced production of cytokines in human monocyte-derived macrophages. , 2001, Cytokine.
[33] S. Akira,et al. Discrimination of bacterial lipoproteins by Toll-like receptor 6. , 2001, International immunology.
[34] A. Barral,et al. A simple method for human peripheral blood monocyte isolation. , 2000, Memorias do Instituto Oswaldo Cruz.
[35] Antal‐Szalmás,et al. Evaluation of CD14 in host defence , 2000, European journal of clinical investigation.
[36] J. Vincent,et al. Has the mortality of septic shock changed with time. , 1998, Critical care medicine.
[37] R. Thieringer,et al. Role of stress-activated mitogen-activated protein kinase (p38) in beta 2-integrin-dependent neutrophil adhesion and the adhesion-dependent oxidative burst. , 1998, Journal of immunology.
[38] S. Foster,et al. Mechanism of Gram-positive Shock: Identification of Peptidoglycan and Lipoteichoic Acid Moieties Essential in the Induction of Nitric Oxide Synthase, Shock, and Multiple Organ Failure , 1998, The Journal of experimental medicine.
[39] Z. Darieva,et al. Effects of exogenous stress protein 70 on the functional properties of human promonocytes through binding to cell surface and internalization. , 1998, Cell stress & chaperones.
[40] M. Galdiero,et al. CD11a/CD18 and CD11b/18 modulation by lipoteichoic acid, N-acetyl-muramyl-alpha-alanyl-D-isoglutamine, muramic acid and protein A from Staphylococcus aureus. , 1996, FEMS immunology and medical microbiology.
[41] S. Wright,et al. Potential role of membrane internalization and vesicle fusion in adhesion of neutrophils in response to lipopolysaccharide and TNF. , 1996, Journal of immunology.
[42] C. Thiemermann,et al. Role for intracellular platelet‐activating factor in the circulatory failure in a model of Gram‐positive shock , 1995, British journal of pharmacology.
[43] J. Vincent,et al. Yearbook of Intensive Care and Emergency Medicine , 1995, Yearbook of Intensive Care and Emergency Medicine.
[44] J. Vincent,et al. Serum cytokine levels in human septic shock. Relation to multiple-system organ failure and mortality. , 1993, Chest.
[45] H. Lode,et al. Septicemia in 980 patients at a university hospital in Berlin: prospective studies during 4 selected years between 1979 and 1989. , 1992, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[46] T. Hennet,et al. Lipopolysaccharide synergizes with tumour necrosis factor-alpha in cytotoxicity assays. , 1992, Immunology.
[47] M. Werner-Washburne,et al. The translation machinery and 70 kd heat shock protein cooperate in protein synthesis , 1992, Cell.
[48] R. Bone,et al. The pathogenesis of sepsis. , 1991, Annals of internal medicine.
[49] A. Baue. Nutrition and metabolism in sepsis and multisystem organ failure. , 1991, The Surgical clinics of North America.
[50] F. Hawker. Liver Dysfunction in Critical Illness , 1991, Anaesthesia and intensive care.
[51] W. R. Mccabe,et al. Pathophysiology of bacteremia. , 1983, The American journal of medicine.
[52] V. A. Merkulov,et al. [Production of 70 kDa recombinant human heat shock protein in baculovirus expression system and assessment of its antiviral activity]. , 2011, Zhurnal mikrobiologii, epidemiologii, i immunobiologii.
[53] M. Weinrich,et al. Fibrinolytic and procoagulant activity in septic and haemorrhagic shock. , 2010, Clinical hemorheology and microcirculation.
[54] T. Evans,et al. Gram-positive and Gram-negative Sepsis: Two Disease Entities? , 2008 .
[55] M. Evgen’ev,et al. Exogenous heat shock protein 70 mediates sepsis manifestations and decreases the mortality rate in rats , 2006, Cell stress & chaperones.
[56] C. Gogos,et al. Pro- versus anti-inflammatory cytokine profile in patients with severe sepsis: a marker for prognosis and future therapeutic options. , 2000, The Journal of infectious diseases.
[57] W. Fischer,et al. On the basic structure of poly(glycerophosphate) lipoteichoic acids. , 1990, Biochemistry and cell biology = Biochimie et biologie cellulaire.
[58] A. Bøyum,et al. Separation of leukocytes from blood and bone marrow. Introduction. , 1968 .