Carnitine palmitoyltransferase 1C promotes cell survival and tumor growth under conditions of metabolic stress.
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B. Faubert | Russell G. Jones | S. Berger | T. Mak | M. Tsao | Ping Huang | C. Thompson | K. Tsuchihara | K. Kannan | R. Hill | A. Wakeham | A. Elia | W. Bakker | J. Silvester | P. Reilly | J. Growney | K. Zaugg | Yi Yao | R. Kiarash | J. Mason | S. Sawyer | B. Fuerth | T. Kalliomäki | Xunyi Luo | V. Nadeem | D. Bungard | Sireesha Yalavarthi | Y. Moolani | Annick You Ten | M. Robinson | G. Pan | Vincent Nadeem | Tuula M Kalliomäki | C. Thompson | C. Thompson
[1] David Carling,et al. Signaling Kinase AMPK Activates Stress-Promoted Transcription via Histone H2B Phosphorylation , 2010, Science.
[2] K. Struhl,et al. A transcriptional signature and common gene networks link cancer with lipid metabolism and diverse human diseases. , 2010, Cancer cell.
[3] D. Nomura,et al. Monoacylglycerol Lipase Regulates a Fatty Acid Network that Promotes Cancer Pathogenesis , 2010, Cell.
[4] Sam W. Lee,et al. GAMT, a p53‐Inducible Modulator of Apoptosis, Is Critical for the Adaptive Response to Nutrient Stress , 2009, Molecular cell.
[5] G. Shulman,et al. Brain‐specific carnitine palmitoyl‐transferase‐1c: role in CNS fatty acid metabolism, food intake, and body weight , 2008, Journal of neurochemistry.
[6] B. Turk,et al. AMPK phosphorylation of raptor mediates a metabolic checkpoint. , 2008, Molecular cell.
[7] N. Casals,et al. CPT1c Is Localized in Endoplasmic Reticulum of Neurons and Has Carnitine Palmitoyltransferase Activity* , 2008, Journal of Biological Chemistry.
[8] J. Menéndez,et al. Fatty acid synthase and the lipogenic phenotype in cancer pathogenesis , 2007, Nature Reviews Cancer.
[9] B. Viollet,et al. Systemic treatment with the antidiabetic drug metformin selectively impairs p53-deficient tumor cell growth. , 2007, Cancer research.
[10] T. Mak,et al. Metabolic Targeting as an Anticancer Strategy: Dawn of a New Era? , 2007, Science's STKE.
[11] R. Shaw,et al. Glucose metabolism and cancer. , 2006, Current opinion in cell biology.
[12] Siobhan McCormack,et al. Rapamycin synergizes with the epidermal growth factor receptor inhibitor erlotinib in non–small-cell lung, pancreatic, colon, and breast tumors , 2006, Molecular Cancer Therapeutics.
[13] Y. Liu,et al. Fatty acid oxidation is a dominant bioenergetic pathway in prostate cancer , 2006, Prostate Cancer and Prostatic Diseases.
[14] H. Pelicano,et al. Glycolysis inhibition for anticancer treatment , 2006, Oncogene.
[15] B. Viollet,et al. 5′-AMP-Activated Protein Kinase (AMPK) Is Induced by Low-Oxygen and Glucose Deprivation Conditions Found in Solid-Tumor Microenvironments , 2006, Molecular and Cellular Biology.
[16] J. Swinnen,et al. Increased lipogenesis in cancer cells: new players, novel targets , 2006, Current opinion in clinical nutrition and metabolic care.
[17] T. Shimokawa,et al. The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[18] C. Thompson,et al. Akt-dependent transformation: there is more to growth than just surviving , 2005, Oncogene.
[19] Russell G. Jones,et al. AMP-activated protein kinase induces a p53-dependent metabolic checkpoint. , 2005, Molecular cell.
[20] R. Gillies,et al. Why do cancers have high aerobic glycolysis? , 2004, Nature Reviews Cancer.
[21] R. Ramsay,et al. Carnitine acyltransferases and their influence on CoA pools in health and disease. , 2004, Molecular aspects of medicine.
[22] J. Brown,et al. Exploiting tumour hypoxia in cancer treatment , 2004, Nature Reviews Cancer.
[23] Nigel Price,et al. A novel brain-expressed protein related to carnitine palmitoyltransferase I. , 2002, Genomics.
[24] S. Lowe,et al. Generation and Characterization of Smac/DIABLO-Deficient Mice , 2002, Molecular and Cellular Biology.
[25] R. Cardiff,et al. Mammary Disease Mice Model Premalignant Polyoma Middle-T Transgenic Updated Version , 2001 .
[26] R. Gascoyne,et al. Interleukin 13 and interleukin 13 receptor are frequently expressed by Hodgkin and Reed-Sternberg cells of Hodgkin lymphoma. , 2001, Blood.
[27] Yong-Yeon Cho,et al. Fatty acid induced glioma cell growth is mediated by the acyl-CoA synthetase 5 gene located on chromosome 10q25.1-q25.2, a region frequently deleted in malignant gliomas , 2000, Oncogene.
[28] Christian A. Rees,et al. Molecular portraits of human breast tumours , 2000, Nature.
[29] C. Hoppel,et al. Fatty acid import into mitochondria. , 2000, Biochimica et biophysica acta.
[30] K. Kinzler,et al. Requirement for p53 and p21 to sustain G2 arrest after DNA damage. , 1998, Science.
[31] A. Joyner,et al. A mouse model of Greig cephalo–polysyndactyly syndrome: the extra–toesJ mutation contains an intragenic deletion of the Gli3 gene , 1993, Nature Genetics.
[32] R. Cardiff,et al. Induction of mammary tumors by expression of polyomavirus middle T oncogene: a transgenic mouse model for metastatic disease , 1992, Molecular and cellular biology.
[33] J. Sambrook,et al. Molecular Cloning: A Laboratory Manual , 2001 .