Functional Role of Monocytes and Macrophages for the Inflammatory Response in Acute Liver Injury
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[1] S. Gordon,et al. Monocyte heterogeneity and innate immunity. , 2003, Immunity.
[2] K. Okita,et al. Protection against acetaminophen-induced liver injury in vivo by an iron chelator, deferoxamine. , 1995, Scandinavian journal of gastroenterology.
[3] G. Plitas,et al. Conventional DCs reduce liver ischemia/reperfusion injury in mice via IL-10 secretion. , 2010, The Journal of clinical investigation.
[4] T. Standiford,et al. MCP-1 protects mice in lethal endotoxemia. , 1997, The Journal of clinical investigation.
[5] F. Geissmann,et al. Blood monocytes: development, heterogeneity, and relationship with dendritic cells. , 2009, Annual review of immunology.
[6] J. Dranoff,et al. Secretion of MCP-1/CCL2 by bile duct epithelia induces myofibroblastic transdifferentiation of portal fibroblasts. , 2006, American journal of physiology. Gastrointestinal and liver physiology.
[7] D. Petersen,et al. Activation of Kupffer cells during the course of carbon tetrachloride-induced liver injury and fibrosis in rats. , 2001, Experimental and molecular pathology.
[8] G. Schmitz,et al. Peripheral blood mononuclear phagocyte subpopulations as cellular markers in hypercholesterolemia. , 1996, Arteriosclerosis, thrombosis, and vascular biology.
[9] P. Libby,et al. Identification of Splenic Reservoir Monocytes and Their Deployment to Inflammatory Sites , 2009, Science.
[10] V. Fadok,et al. Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF. , 1998, The Journal of clinical investigation.
[11] K. Schäkel,et al. The CD16+ (FcγRIII+) Subset of Human Monocytes Preferentially Becomes Migratory Dendritic Cells in a Model Tissue Setting , 2002, The Journal of experimental medicine.
[12] E. Pamer,et al. Additive Roles for MCP-1 and MCP-3 in CCR2-Mediated Recruitment of Inflammatory Monocytes during Listeria monocytogenes Infection1 , 2008, The Journal of Immunology.
[13] J. Yamate,et al. Participation of Functionally Different Macrophage Populations and Monocyte Chemoattractant Protein-1 in Early Stages of Thioacetamide-induced Rat Hepatic Injury , 2009, Toxicologic pathology.
[14] Steffen Jung,et al. Monocytes give rise to mucosal, but not splenic, conventional dendritic cells , 2007, The Journal of experimental medicine.
[15] F. Geissmann,et al. Blood monocytes: distinct subsets, how they relate to dendritic cells, and their possible roles in the regulation of T‐cell responses , 2008, Immunology and cell biology.
[16] S. Kunkel,et al. Exaggerated hepatic injury due to acetaminophen challenge in mice lacking C-C chemokine receptor 2. , 2000, The American journal of pathology.
[17] R. Crofton,et al. The origin, kinetics, and characteristics of the kupffer cells in the normal steady state , 1978, The Journal of experimental medicine.
[18] S. Miyagawa,et al. Expression of Fas Ligand by Hepatic Macrophages in Patients with Fulminant Hepatic Failure , 2005, The American Journal of Gastroenterology.
[19] L. Moldawer,et al. Lipopolysaccharide and D-galactosamine-induced hepatic injury is mediated by TNF-alpha and not by Fas ligand. , 2000, American journal of physiology. Regulatory, integrative and comparative physiology.
[20] C. Smith,et al. Mechanisms of neutrophil‐induced parenchymal cell injury , 1997, Journal of leukocyte biology.
[21] D. Laskin,et al. Potential role of activated macrophages in acetaminophen hepatotoxicity. II. Mechanism of macrophage accumulation and activation. , 1986, Toxicology and applied pharmacology.
[22] J. Prinz,et al. The CD14+CD16+ monocytes in erysipelas are expanded and show reduced cytokine production , 2002, European journal of immunology.
[23] F. Tacke,et al. Infiltrating monocytes versus resident kupffer cells: Do alternatively activated macrophages need to be targeted alternatively? , 2011, Hepatology.
[24] Michael P Holt,et al. Exacerbation of acetaminophen-induced disturbances of liver sinusoidal endothelial cells in the absence of Kupffer cells in mice. , 2010, Toxicology letters.
[25] L. Rénia,et al. Dual role of CCR2 in the constitution and the resolution of liver fibrosis in mice. , 2009, The American journal of pathology.
[26] S. Milani,et al. Expression of monocyte chemotactic protein-1 precedes monocyte recruitment in a rat model of acute liver injury, and is modulated by vitamin E. , 1999, Journal of investigative medicine : the official publication of the American Federation for Clinical Research.
[27] F. Finkelman,et al. Local Macrophage Proliferation, Rather than Recruitment from the Blood, Is a Signature of TH2 Inflammation , 2011, Science.
[28] D. Mosser,et al. Monocyte subpopulations and their differentiation patterns during infection , 2007, Journal of leukocyte biology.
[29] T. Luedde,et al. Interleukin-8 Is Activated in Patients with Chronic Liver Diseases and Associated with Hepatic Macrophage Accumulation in Human Liver Fibrosis , 2011, PloS one.
[30] C. Weston,et al. AUTOIMMUNE, CHOLESTATIC AND BILIARY DISEASE CX3CR1 and Vascular Adhesion Protein-1-Dependent Recruitment of CD16 Monocytes Across Human Liver Sinusoidal Endothelium , 2010 .
[31] R. Weissleder,et al. Angiotensin-Converting Enzyme Inhibition Prevents the Release of Monocytes From Their Splenic Reservoir in Mice With Myocardial Infarction , 2010, Circulation research.
[32] E. Botsoglou,et al. The protective effect of alpha-tocopherol and GdCl3 against hepatic ischemia/reperfusion injury. , 2006, Surgery today.
[33] Zhigang Tian,et al. Liver: An organ with predominant innate immunity , 2007, Hepatology.
[34] H. Morselt,et al. Heterogeneity in secretory responses of rat liver macrophages of different size. , 2008, Liver.
[35] Peter Winter,et al. SuperSAGE evidence for CD14++CD16+ monocytes as a third monocyte subset. , 2011, Blood.
[36] R. de Waal Malefyt,et al. Interleukin-10 down-regulates MHC class II alphabeta peptide complexes at the plasma membrane of monocytes by affecting arrival and recycling. , 1997, Immunity.
[37] Rachel J. Johnson,et al. Hepatic Mrp4 induction following acetaminophen exposure is dependent on Kupffer cell function. , 2008, American journal of physiology. Gastrointestinal and liver physiology.
[38] I. Weissman,et al. A clonogenic common myeloid progenitor that gives rise to all myeloid lineages , 2000, Nature.
[39] Y. Sasaguri,et al. Suppression of macrophage infiltration inhibits activation of hepatic stellate cells and liver fibrogenesis in rats. , 2005, Gastroenterology.
[40] F. Tacke,et al. Chemokine-directed immune cell infiltration in acute and chronic liver disease , 2008, Expert review of gastroenterology & hepatology.
[41] D. Laskin,et al. Role of CCR2 in macrophage migration into the liver during acetaminophen‐induced hepatotoxicity in the mouse , 2002, Hepatology.
[42] F. Tacke,et al. Migratory fate and differentiation of blood monocyte subsets. , 2006, Immunobiology.
[43] G. Joos,et al. Chemokine Receptor CCR2 but Not CCR5 or CCR6 Mediates the Increase in Pulmonary Dendritic Cells during Allergic Airway Inflammation1 , 2007, The Journal of Immunology.
[44] K. Hara,et al. Effect of gadolinium chloride treatment on concanavalin A-induced cytokine mRNA expression in mouse liver. , 1998, Japanese journal of pharmacology.
[45] N. Van Rooijen,et al. Subpopulations of Mouse Blood Monocytes Differ in Maturation Stage and Inflammatory Response1 , 2004, The Journal of Immunology.
[46] Laura P James,et al. Acetaminophen-induced hepatotoxicity. , 2003, Drug metabolism and disposition: the biological fate of chemicals.
[47] T. Hibi,et al. CCR9+ macrophages are required for acute liver inflammation in mouse models of hepatitis. , 2012, Gastroenterology.
[48] L. Sibley,et al. Gr1(+) inflammatory monocytes are required for mucosal resistance to the pathogen Toxoplasma gondii. , 2008, Immunity.
[49] William M. Lee,et al. Soluble CD163 from activated macrophages predicts mortality in acute liver failure. , 2007, Journal of hepatology.
[50] John Savill,et al. Requirements for Apoptotic Cell Contact in Regulation of Macrophage Responses1 , 2006, The Journal of Immunology.
[51] S. Wigmore,et al. Heme oxygenase system in hepatic ischemia-reperfusion injury. , 2010, World journal of gastroenterology.
[52] C. Gandhi,et al. Inhibition of Kupffer cell‐mediated early proinflammatory response with carbon monoxide in transplant‐induced hepatic ischemia/reperfusion injury in rats , 2008, Hepatology.
[53] S. Milani,et al. Up-regulated expression of fractalkine and its receptor CX3CR1 during liver injury in humans. , 2002, Journal of hepatology.
[54] E. Schmidt,et al. Evidence for Kupffer cell migration along liver sinusoids, from high-resolution in vivo microscopy. , 1992, The American journal of physiology.
[55] R. Weissleder,et al. From Their Splenic Reservoir in Mice With Myocardial Infarction Angiotensin-Converting Enzyme Inhibition Prevents the Release of Monocytes , 2010 .
[56] Michael P Holt,et al. Identification and characterization of infiltrating macrophages in acetaminophen‐induced liver injury , 2008, Journal of leukocyte biology.
[57] T. Okanoue,et al. Activated Kupffer cells play an important role in intra-hepatic Th1-associated necro-inflammation in Concanavalin A-induced hepatic injury in mice. , 2003, Hepatology research : the official journal of the Japan Society of Hepatology.
[58] F. Balkwill,et al. Tumour necrosis factor. , 1989, British medical bulletin.
[59] Haiyang Xie,et al. Kupffer cells contribute to concanavalin A-induced hepatic injury through a Th1 but not Th17 type response-dependent pathway in mice. , 2011, Hepatobiliary & pancreatic diseases international : HBPD INT.
[60] C. Mackay,et al. Monocyte chemotactic protein‐1, ‐2, and ‐3 are distinctively expressed in portal tracts and granulomata in primary biliary cirrhosis: implications for pathogenesis , 2001, The Journal of pathology.
[61] D. de Silva. Tumour necrosis factor , 1990, The Ceylon medical journal.
[62] T. Thalhammer,et al. MCP-1 and MIP3-alpha serum levels in acute liver failure and molecular adsorbent recirculating system (MARS) treatment: A pilot study , 2009, Scandinavian journal of gastroenterology.
[63] D. Laskin,et al. Distinct roles of tumor necrosis factor-alpha and nitric oxide in acute liver injury induced by carbon tetrachloride in mice. , 2001, Toxicology and applied pharmacology.
[64] J. Vinel,et al. Reactive oxygen intermediates and eicosanoid production by Kupffer cells and infiltrated macrophages in acute and chronic liver injury induced in rats by CCl4 , 2000, Inflammation Research.
[65] A. Seifalian,et al. Liver ischemia/reperfusion injury: Processes in inflammatory networks—A review , 2010, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[66] G. Kolios,et al. Role of Kupffer cells in the pathogenesis of liver disease. , 2006, World journal of gastroenterology.
[67] G. Gores,et al. Cellular and molecular mechanisms of liver injury. , 2008, Gastroenterology.
[68] F. Ginhoux,et al. Hepatic recruitment of the inflammatory Gr1+ monocyte subset upon liver injury promotes hepatic fibrosis , 2009, Hepatology.
[69] David Julius,et al. Cellular and Molecular Mechanisms of Pain , 2009, Cell.
[70] Christian Trautwein,et al. Inflammatory Pathways in Liver Homeostasis and Liver Injury , 2009, Clinical reviews in allergy & immunology.
[71] E. O’Neill,et al. T cell activation-associated hepatic injury: mediation by tumor necrosis factors and protection by interleukin 6 , 1994, The Journal of experimental medicine.
[72] N. Pumford,et al. Pretreatment of mice with macrophage inactivators decreases acetaminophen hepatotoxicity and the formation of reactive oxygen and nitrogen species , 1999, Hepatology.
[73] J. Van Damme,et al. Monocyte chemotactic protein-3. , 2002, European cytokine network.
[74] Markus G. Manz,et al. Development of Monocytes, Macrophages, and Dendritic Cells , 2010, Science.
[75] P. Hayes,et al. Animal models of fulminant hepatic failure: A critical evaluation , 2000, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[76] G. Alexander,et al. Tumour necrosis factor production in fulminant hepatic failure: relation to aetiology and superimposed microbial infection , 1990, Clinical and experimental immunology.
[77] T. Walsh,et al. Pro-inflammatory cytokine release and mediation of the acute phase protein response in fulminant hepatic failure , 1998, Intensive Care Medicine.
[78] B. Passlick,et al. The novel subset of CD14+/CD16+ blood monocytes is expanded in sepsis patients. , 1993, Blood.
[79] M. Czaja,et al. Monocyte chemoattractant protein 1 (MCP‐1) expression occurs in toxic rat liver injury and human liver disease , 1994, Journal of leukocyte biology.
[80] K. Migita,et al. Liver architecture, cell function, and disease , 2009, Seminars in Immunopathology.
[81] T. Luedde,et al. Pharmacological inhibition of the chemokine CCL2 (MCP-1) diminishes liver macrophage infiltration and steatohepatitis in chronic hepatic injury , 2011, Gut.
[82] A. J. Valente,et al. Cultured human liver fat-storing cells produce monocyte chemotactic protein-1. Regulation by proinflammatory cytokines. , 1993, The Journal of clinical investigation.
[83] H. Kiwada,et al. Repopulation of murine Kupffer cells after intravenous administration of liposome-encapsulated dichloromethylene diphosphonate. , 1996, American Journal of Pathology.
[84] D. Brenner,et al. CX3CL1‐CX3CR1 interaction prevents carbon tetrachloride‐induced liver inflammation and fibrosis in mice , 2010, Hepatology.
[85] Steffen Jung,et al. Blood monocytes consist of two principal subsets with distinct migratory properties. , 2003, Immunity.
[86] H. Farrar,et al. Cytokines and Toxicity in Acetaminophen Overdose , 2005, Journal of clinical pharmacology.
[87] R. Pierce,et al. Kupffer cell heterogeneity: functional properties of bone marrow derived and sessile hepatic macrophages. , 2007, Blood.
[88] Gerken,et al. IL‐10 down‐regulates T cell activation by antigen‐presenting liver sinusoidal endothelial cells through decreased antigen uptake via the mannose receptor and lowered surface expression of accessory molecules , 1998, Clinical and experimental immunology.
[89] L. Williams,et al. Functional overlap but differential expression of CSF‐1 and IL‐34 in their CSF‐1 receptor‐mediated regulation of myeloid cells , 2010, Journal of leukocyte biology.
[90] T. Manabe,et al. FasL expression in hepatic antigen-presenting cells and phagocytosis of apoptotic T cells by FasL+ Kupffer cells are indicators of rejection activity in human liver allografts. , 2007, The American journal of pathology.
[91] A. Cumano,et al. Monitoring of Blood Vessels and Tissues by a Population of Monocytes with Patrolling Behavior , 2007, Science.
[92] Xiaojing Ma,et al. Interleukin-10 expression in macrophages during phagocytosis of apoptotic cells is mediated by homeodomain proteins Pbx1 and Prep-1. , 2007, Immunity.
[93] T. Luedde,et al. Functional Contribution of Elevated Circulating and Hepatic Non-Classical CD14+CD16+ Monocytes to Inflammation and Human Liver Fibrosis , 2010, PloS one.
[94] T. Hohl,et al. Monocyte-mediated defense against microbial pathogens. , 2008, Annual review of immunology.
[95] S. Blankenberg,et al. CD14+CD16+ monocytes in coronary artery disease and their relationship to serum TNF-alpha levels. , 2004, Thrombosis and haemostasis.
[96] C. Takiya,et al. Proinflammatory Clearance of Apoptotic Neutrophils Induces an IL-12lowIL-10high Regulatory Phenotype in Macrophages , 2010, The Journal of Immunology.
[97] J. Morales-González,et al. Effect of Gadolinium Chloride on Liver Regeneration Following Thioacetamide-Induced Necrosis in Rats , 2010, International journal of molecular sciences.
[98] D. Dunn,et al. Anti‐Endotoxin Monoclonal Antibodies are Protective against Hepatic Ischemia/Reperfusion Injury in Steatotic Mice , 2004, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.
[99] C. Trautwein,et al. Antagonism of the chemokine Ccl5 ameliorates experimental liver fibrosis in mice. , 2010, The Journal of clinical investigation.
[100] Steven D. Cohen,et al. Selective protein arylation and acetaminophen-induced hepatotoxicity. , 1997, Drug metabolism reviews.
[101] F. Tacke,et al. Modification of chemokine pathways and immune cell infiltration as a novel therapeutic approach in liver inflammation and fibrosis. , 2011, Inflammation & allergy drug targets.
[102] A. Yachie,et al. Selective expansion of CD16highCCR2– subpopulation of circulating monocytes with preferential production of haem oxygenase (HO)‐1 in response to acute inflammation , 2005, Clinical and experimental immunology.
[103] J. González‐Gallego,et al. An overview of animal models for investigating the pathogenesis and therapeutic strategies in acute hepatic failure. , 2009, World journal of gastroenterology.
[104] Guido Gerken,et al. Local control of the immune response in the liver , 2000, Immunological reviews.
[105] S. Friedman,et al. Interleukin-6 protects hepatocytes from CCl4-mediated necrosis and apoptosis in mice by reducing MMP-2 expression. , 2005, Journal of hepatology.
[106] Xuetao Cao,et al. Apoptotic cells attenuate fulminant hepatitis by priming Kupffer cells to produce interleukin‐10 through membrane‐bound TGF‐β , 2011, Hepatology.
[107] S. Curbishley,et al. Human hepatic sinusoidal endothelial cells can be distinguished by expression of phenotypic markers related to their specialised functions in vivo. , 2006, World journal of gastroenterology.
[108] Takashi Yamamoto,et al. SERIES : Hepatic sinusoidal cells in liver physiology and pathology , 2004 .
[109] Y. Zhai,et al. Liver Ischemia and Reperfusion Injury: New Insights into Mechanisms of Innate—Adaptive Immune‐Mediated Tissue Inflammation , 2011, American Journal of Transplantation.
[110] T. Papadopoulos,et al. IL‐10, regulatory T cells, and Kupffer cells mediate tolerance in concanavalin A–induced liver injury in mice , 2007, Hepatology.
[111] T. Hohl,et al. Monocyte Trafficking to Hepatic Sites of Bacterial Infection Is Chemokine Independent and Directed by Focal Intercellular Adhesion Molecule-1 Expression , 2010, The Journal of Immunology.
[112] G. Gores,et al. Kupffer cell engulfment of apoptotic bodies stimulates death ligand and cytokine expression , 2003, Hepatology.
[113] M. Hussain,et al. Reduced monocyte HLA‐DR expression: A novel biomarker of disease severity and outcome in acetaminophen‐induced acute liver failure , 2006, Hepatology.
[114] E. Kawasaki,et al. Molecular cloning of a complementary DNA encoding human macrophage-specific colony-stimulating factor (CSF-1). , 1985, Science.
[115] J. Brady,et al. Protective role of Kupffer cells in acetaminophen-induced hepatic injury in mice. , 2002, Chemical research in toxicology.
[116] F. Tacke,et al. Monocytes and macrophages as cellular targets in liver fibrosis. , 2009, Inflammation & allergy drug targets.
[117] D. Knook,et al. Relation between localization and function of rat liver Kupffer cells. , 1982, Laboratory investigation; a journal of technical methods and pathology.
[118] F. Ribeiro-Gomes,et al. Macrophage Interactions with Neutrophils Regulate Leishmania major Infection1 , 2004, The Journal of Immunology.
[119] Makoto Naito,et al. Development, differentiation, and maturation of Kupffer cells , 1998, Microscopy research and technique.
[120] S. Gordon,et al. Monocyte and macrophage heterogeneity , 2005, Nature Reviews Immunology.
[121] K. Böker,et al. Akutes Leberversagen , 2001, Der Internist.
[122] T. Papadopoulos,et al. Importance of Kupffer cells for T-cell-dependent liver injury in mice. , 2000, The American journal of pathology.
[123] H. Fujii,et al. Glycyrrhizin Prevents Liver Injury by Inhibition of High-Mobility Group Box 1 Production by Kupffer Cells after Ischemia-Reperfusion in Rats , 2011, Journal of Pharmacology and Experimental Therapeutics.
[124] B. Rehermann,et al. The liver as an immunological organ , 2004 .
[125] D. Vergani,et al. The importance of immune dysfunction in determining outcome in acute liver failure. , 2008, Journal of hepatology.
[126] P. Libby,et al. The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions , 2007, The Journal of experimental medicine.
[127] J. Scherberich,et al. Expanded CD14+ CD16+Monocyte Subpopulation in Patients with Acute and Chronic Infections Undergoing Hemodialysis , 1998, Infection and Immunity.
[128] Ana Cumano,et al. A Clonogenic Bone Marrow Progenitor Specific for Macrophages and Dendritic Cells , 2006, Science.
[129] Werner Müller,et al. Interleukin 6/gp130‐dependent pathways are protective during chronic liver diseases , 2003, Hepatology.
[130] T. Espevik,et al. The Proinflammatory CD14+CD16+DR++ Monocytes Are a Major Source of TNF1 , 2002, The Journal of Immunology.
[131] M. Hussain,et al. Source and characterization of hepatic macrophages in acetaminophen‐induced acute liver failure in humans , 2012, Hepatology.
[132] D. Jollow,et al. Acetaminophen structure-toxicity studies: in vivo covalent binding of a nonhepatotoxic analog, 3-hydroxyacetanilide. , 1990, Toxicology and applied pharmacology.
[133] C. Hulstaert,et al. Heterogeneity of rat liver and spleen macrophages in gadolinium chloride–induced elimination and repopulation , 1992, Journal of leukocyte biology.
[134] K. Węglarczyk,et al. Antitumor response of CD14+/CD16+ monocyte subpopulation. , 2004, Experimental hematology.
[135] F. Tacke,et al. Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques. , 2007, The Journal of clinical investigation.
[136] M. Cascales,et al. Depletion of Kupffer cell function by gadolinium chloride attenuates thioacetamide-induced hepatotoxicity. Expression of metallothionein and HSP70. , 2003, Biochemical pharmacology.
[137] R. Kedl,et al. Mechanism of T cell tolerance induction by murine hepatic Kupffer cells , 2008, Hepatology.
[138] F. Ginhoux,et al. Immature monocytes acquire antigens from other cells in the bone marrow and present them to T cells after maturing in the periphery , 2006, The Journal of experimental medicine.
[139] A. Vercelli,et al. Prevention of severe toxic liver injury and oxidative stress in MCP-1-deficient mice. , 2007, Journal of hepatology.
[140] E. Pamer,et al. Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2 , 2006, Nature Immunology.
[141] K. Pennypacker,et al. Acetaminophen-induced hepatotoxicity is associated with early changes in NF-kB and NF-IL6 DNA binding activity. , 1995, Journal of inflammation.
[142] D. Giakoustidis,et al. Blockade of Kupffer cells by gadolinium chloride reduces lipid peroxidation and protects liver from ischemia/reperfusion injury. , 2003, Hepato-gastroenterology.
[143] S. Waltz,et al. Ron receptor regulates Kupffer cell‐dependent cytokine production and hepatocyte survival following endotoxin exposure in mice , 2011, Hepatology.
[144] E. Botsoglou,et al. The Protective Effect of α-Tocopherol and GdCl3 Against Hepatic Ischemia/Reperfusion Injury , 2005, Surgery Today.
[145] R. Schwabe,et al. TLR4 enhances TGF-beta signaling and hepatic fibrosis. , 2007, Nature medicine.
[146] F. Lammert,et al. Patients with acute on chronic liver failure display "sepsis-like" immune paralysis. , 2005, Journal of hepatology.
[147] D. Vergani,et al. Role of monocytes and macrophages in experimental and human acute liver failure. , 2010, World journal of gastroenterology.
[148] G. Tiegs,et al. Tolerance Induction in Response to Liver Inflammation , 2010, Digestive Diseases.
[149] L. Williams,et al. Discovery of a Cytokine and Its Receptor by Functional Screening of the Extracellular Proteome , 2008, Science.
[150] Maylee Hsu,et al. Monocyte chemoattractant protein-1 derived from biliary innate immunity contributes to hepatic fibrogenesis , 2011, Journal of Clinical Pathology.
[151] T. Okanoue,et al. Involvement of Kupffer Cells in the Interaction Between Neutrophils and Sinusoidal Endothelial Cells in Rats , 2002, Shock.
[152] Y. Vaishnav,et al. Evidence that acetaminophen and N-hydroxyacetaminophen form a common arylating intermediate, N-acetyl-p-benzoquinoneimine. , 1980, Molecular pharmacology.
[153] Silvano Sozzani,et al. Nomenclature of monocytes and dendritic cells in blood. , 2010, Blood.
[154] J. Cofer,et al. The influence of preservation injury on rejection in the hepatic transplant recipient. , 1990, Transplantation.
[155] N. Horiike,et al. Soluble CD163 in patients with liver diseases: very high levels of soluble CD163 in patients with fulminant hepatic failure , 2004, Journal of Gastroenterology.
[156] F. Geissmann,et al. The transcription factor NR4A1 (Nur77) controls bone marrow differentiation and the survival of Ly6C− monocytes , 2011, Nature Immunology.
[157] William M. Lee,et al. Results of a Prospective Study of Acute Liver Failure at 17 Tertiary Care Centers in the United States , 2002, Annals of Internal Medicine.
[158] J. Bijlsma,et al. Fcgamma receptor expression levels on monocytes are elevated in rheumatoid arthritis patients with high erythrocyte sedimentation rate who do not use anti-rheumatic drugs. , 2003, Rheumatology.
[159] Clinton S. Robbins,et al. The multiple roles of monocyte subsets in steady state and inflammation , 2010, Cellular and Molecular Life Sciences.
[160] S. Forbes,et al. Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair. , 2005, The Journal of clinical investigation.
[161] S. Milani,et al. Increased expression of monocyte chemotactic protein-1 during active hepatic fibrogenesis: correlation with monocyte infiltration. , 1998, The American journal of pathology.
[162] B. Rollins,et al. MCP‐1‐dependent signaling in CCR2−/− aortic smooth muscle cells , 2004, Journal of leukocyte biology.
[163] J. Casanova,et al. Human CD14dim Monocytes Patrol and Sense Nucleic Acids and Viruses via TLR7 and TLR8 Receptors , 2010, Immunity.
[164] M. Ernst,et al. Identification of a novel dendritic cell‐like subset of CD64+ / CD16+ blood monocytes , 2001, European journal of immunology.
[165] Wing-Cheong Wong,et al. Gene expression profiling reveals the defining features of the classical, intermediate, and nonclassical human monocyte subsets. , 2011, Blood.
[166] K. Messmer,et al. Liver ischemia and reperfusion induces a systemic inflammatory response through Kupffer cell activation. , 1996, Shock.
[167] C. Hogaboam,et al. C-C Chemokine Ligand 2/Monocyte Chemoattractant Protein-1 Directly Inhibits NKT Cell IL-4 Production and Is Hepatoprotective in T Cell-Mediated Hepatitis in the Mouse1 , 2003, The Journal of Immunology.
[168] A. Billiau,et al. Bimodal role of endogenous interleukin‐6 in concanavalin A‐induced hepatitis in mice , 2000, Journal of leukocyte biology.
[169] H. Jaeschke. Role of Reactive Oxygen Species in Hepatic Ischemia-Reperfusion Injury and Preconditioning , 2003, Journal of investigative surgery : the official journal of the Academy of Surgical Research.
[170] Remo Guidieri. Res , 1995, RES: Anthropology and Aesthetics.
[171] A. Canbay,et al. Functional role of monocytes and macrophages for the inflammatory response in acute liver injury , 2012 .
[172] C. Herra,et al. Increased expression of Fc gamma receptors on neutrophils and monocytes may reflect ongoing bacterial infection. , 1996, Journal of medical microbiology.
[173] Claudio Lottaz,et al. Comparison of gene expression profiles between human and mouse monocyte subsets. , 2010, Blood.
[174] S. Zahler,et al. A novel technique for selective NF-κB inhibition in Kupffer cells: contrary effects in fulminant hepatitis and ischaemia–reperfusion , 2009, Gut.
[175] L. Pohl,et al. Protection against acetaminophen‐induced liver injury and lethality by interleukin 10: Role of inducible nitric oxide synthase , 2002, Hepatology.
[176] T. Luedde,et al. The fractalkine receptor CX3CR1 protects against liver fibrosis by controlling differentiation and survival of infiltrating hepatic monocytes , 2010, Hepatology.
[177] E. Schmidt,et al. Intermittence of blood flow in liver sinusoids, studied by high-resolution in vivo microscopy. , 1995, The American journal of physiology.
[178] R. Schnellmann,et al. Murine Kupffer Cells Are Protective in Total Hepatic Ischemia/Reperfusion Injury with Bowel Congestion through IL-10 , 2010, The Journal of Immunology.
[179] S. Zahler,et al. Reciprocal Activation Between CD4+ T Cells and Kupffer Cells During Hepatic Ischemia-Reperfusion , 2008, Transplantation.
[180] Modulation of macrophage functioning abrogates the acute hepatotoxicity of acetaminophen , 1995, Hepatology.
[181] A. Gerbes,et al. Role of Kupffer cells in host defense and liver disease , 2006, Liver international : official journal of the International Association for the Study of the Liver.
[182] M. Manns,et al. Acute liver failure: a life-threatening disease. , 2011, Deutsches Arzteblatt international.
[183] H. Makino,et al. CD14+,CD16+ blood monocytes and joint inflammation in rheumatoid arthritis. , 2002, Arthritis and rheumatism.