Live to die another day: novel insights may explain the pathophysiology behind smoker’s paradox in SARS-CoV-2 infection
暂无分享,去创建一个
[1] M. Mahmoudzadeh,et al. Effect of Nicotine on Immune System Function , 2022, Advanced pharmaceutical bulletin.
[2] Haide Chen,et al. Socializing with Smoker and Social Smoking Behavior among Chinese Male Smokers with Low Nicotine Dependence: The Mediating Roles of Belief of Smoking Rationalization and Smoker Identity , 2022, International journal of environmental research and public health.
[3] K. Papadopoulos,et al. A protective erythropoietin evolutionary landscape, NLRP3 inflammasome regulation, and multisystem inflammatory syndrome in children , 2022, Human Cell.
[4] M. Gissler,et al. New theory about the pathophysiology of preeclampsia derived from the paradox of positive effects of maternal smoking , 2022, Journal of hypertension.
[5] G. Vist,et al. Polycyclic aromatic hydrocarbons (PAHs) may explain the paradoxical effects of cigarette use on preeclampsia (PE). , 2022, Toxicology.
[6] K. Papadopoulos,et al. Too hard to die: Exercise training mediates specific and immediate SARS-CoV-2 protection , 2022, World journal of virology.
[7] Tobacco and nicotine use , 2022, Nature Reviews Disease Primers.
[8] Megan E. Piper,et al. Tobacco and nicotine use , 2022, Nature Reviews Disease Primers.
[9] N. Nalivaeva,et al. Angiotensin-converting enzyme 2 (ACE2): Two decades of revelations and re-evaluation , 2022, Peptides.
[10] G. Citerio,et al. Nitric oxide: Clinical applications in critically ill patients. , 2022, Nitric oxide : biology and chemistry.
[11] D. Ivashchenko,et al. Allele С (rs5186) of at1r is associated with the severity of COVID-19 in the Ukrainian population , 2022, Infection, Genetics and Evolution.
[12] Yuval Arbel,et al. Can smoking prevalence explain COVID-19 indicators (cases, mortality, and recovery)? A comparative study in OECD countries , 2020, Environmental Science and Pollution Research.
[13] K. Papadopoulos,et al. Genetic polymorphisms affecting nitric oxide and β-cytokine pathways may contribute to increased COVID-19 mortality in schizophrenia , 2021, Asian Journal of Psychiatry.
[14] P. Purohit,et al. MicroRNAs based regulation of cytokine regulating immune expressed genes and their transcription factors in COVID-19 , 2021, Meta Gene.
[15] F. Quintana,et al. AHR signaling is induced by infection with coronaviruses , 2021, Nature Communications.
[16] Y. Tesfaigzi,et al. Paradoxical effects of cigarette smoke and COPD on SARS-CoV-2 infection and disease , 2021, BMC Pulmonary Medicine.
[17] Jun Kobayashi. Lifestyle-mediated nitric oxide boost to prevent SARS-CoV-2 infection: A perspective , 2021, Nitric Oxide.
[18] D. Ojcius,et al. Cigarette Smoke Stimulates SARS-CoV-2 Internalization by Activating AhR and Increasing ACE2 Expression in Human Gingival Epithelial Cells , 2021, International journal of molecular sciences.
[19] Y. Kuo,et al. Smoking and risk of COVID-19 hospitalization , 2021, Respiratory Medicine.
[20] F. Bianchi,et al. Association Between Smoking and SARS-CoV-2 Infection: Cross-sectional Study of the EPICOVID19 Internet-Based Survey , 2021, JMIR Public Health and Surveillance.
[21] M. Sayadi,et al. The Impact of Smoking on Clinical Outcomes after Percutaneous Coronary Intervention in Women Compared to Men , 2021, Journal of interventional cardiology.
[22] H. Bono,et al. Inhibiting SARS-CoV-2 infection in vitro by suppressing its receptor, angiotensin-converting enzyme 2, via aryl-hydrocarbon receptor signal , 2021, Scientific Reports.
[23] Kaifeng Xu,et al. Degradation of SARS-CoV-2 receptor ACE2 by the E3 ubiquitin ligase Skp2 in lung epithelial cells , 2021, Frontiers of Medicine.
[24] E. Lazartigues,et al. Angiotensin Type 1 Receptor-Dependent Internalization of SARS-CoV-2 by Angiotensin-Converting Enzyme 2 , 2021, Hypertension.
[25] B. Dautzenberg,et al. Impact of Tobacco Smoking on the Risk of COVID-19: A Large Scale Retrospective Cohort Study , 2021, Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco.
[26] S. Ristić,et al. Association between the ACE-I/D polymorphism and nicotine dependence amongst patients with lung cancer. , 2020, Biomedical reports.
[27] C. Tascini,et al. The Paradox of the Low Prevalence of Current Smokers Among Covid-19 Patients Hospitalized in Non-Intensive Care Wards: Results From an Italian Multicenter Case-Control Study , 2020, Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco.
[28] J. Lennerstrand,et al. Mitigation of the replication of SARS-CoV-2 by nitric oxide in vitro , 2020, Redox Biology.
[29] Urvish K. Patel,et al. Is there a smoker’s paradox in COVID-19? , 2020, BMJ Evidence-Based Medicine.
[30] G. H. Oliveira-Paula,et al. Clinically relevant endothelial nitric oxide synthase polymorphisms and their impact on drug response , 2020, Expert opinion on drug metabolism & toxicology.
[31] V. Lagente,et al. Alteration of immunophenotype of human macrophages and monocytes after exposure to cigarette smoke , 2020, Scientific Reports.
[32] Zhiyong Wu,et al. Elevation of plasma angiotensin II level is a potential pathogenesis for the critically ill COVID-19 patients , 2020, Critical Care.
[33] K. Farsalinos,et al. Current smoking, former smoking, and adverse outcome among hospitalized COVID-19 patients: a systematic review and meta-analysis , 2020, Therapeutic advances in chronic disease.
[34] P. Carmeliet,et al. COVID-19: the vasculature unleashed , 2020, Nature Reviews Immunology.
[35] J. Tardif,et al. Impact of smoking on cardiovascular outcomes in patients with stable coronary artery disease. , 2020, European journal of preventive cardiology.
[36] O. Perski,et al. The association of smoking status with SARS-CoV-2 infection, hospitalisation and mortality from COVID-19: A living rapid evidence review (version 5) , 2020, Qeios.
[37] Holger Moch,et al. Endothelial cell infection and endotheliitis in COVID-19 , 2020, The Lancet.
[38] C. Vardavas,et al. COVID-19 and smoking: A systematic review of the evidence , 2020, Tobacco induced diseases.
[39] A. Alkhedaide. Tobacco smoking causes secondary polycythemia and a mild leukocytosis among heavy smokers in Taif City in Saudi Arabia , 2019, Saudi journal of biological sciences.
[40] F. Torres-Juárez,et al. Nicotine modulates molecules of the innate immune response in epithelial cells and macrophages during infection with M. tuberculosis , 2020, Clinical and experimental immunology.
[41] H. Ko,et al. The effect of outdoor smoking ban: Evidence from Korea. , 2019, Health economics.
[42] G. Joos,et al. The role of miR-155 in cigarette smoke-induced pulmonary inflammation and COPD , 2019, Mucosal Immunology.
[43] Guangbiao Zhou. Tobacco, air pollution, environmental carcinogenesis, and thoughts on conquering strategies of lung cancer , 2019, Cancer biology & medicine.
[44] A. Budaj,et al. Smoking and cardiovascular diseases - is there more paradox than expected? , 2019, Polish archives of internal medicine.
[45] Zhe Hong,et al. Combined effects of cigarette smoking, alcohol drinking and eNOS Glu298Asp polymorphism on blood pressure in Chinese male hypertensive subjects , 2019, Tobacco induced diseases.
[46] Ramotowski,et al. Smoking and cardiovascular diseases: paradox greater than expected? , 2019 .
[47] V. Kutala,et al. Impact of eNOS 27-bp VNTR (4b/a) gene polymorphism with the risk of Systemic Lupus Erythematosus in south Indian subjects. , 2018, Gene.
[48] W. Feleszko,et al. Tobacco Smoke Induces and Alters Immune Responses in the Lung Triggering Inflammation, Allergy, Asthma and Other Lung Diseases: A Mechanistic Review , 2018, International journal of environmental research and public health.
[49] Lyanne M. Kieneker,et al. Active Smoking and Hematocrit and Fasting Circulating Erythropoietin Concentrations in the General Population , 2018, Mayo Clinic proceedings.
[50] S. Ristić,et al. The insertion/deletion polymorphism in the angiotensin-converting enzyme gene and nicotine dependence in schizophrenia patients , 2017, Journal of Neural Transmission.
[51] M. Obeidat,et al. Total particulate matter concentration skews cigarette smoke's gene expression profile , 2016, ERJ Open Research.
[52] J. Tanus-Santos,et al. Endothelial nitric oxide synthase: From biochemistry and gene structure to clinical implications of NOS3 polymorphisms. , 2016, Gene.
[53] R. Narain,et al. Gene therapies in clinical trials , 2016 .
[54] P. Bundhun,et al. Impact of Modifiable Cardiovascular Risk Factors on Mortality After Percutaneous Coronary Intervention , 2015, Medicine.
[55] Yingzi Zhao,et al. Vascular nitric oxide: Beyond eNOS. , 2015, Journal of pharmacological sciences.
[56] D. Henderson,et al. Pathophysiological mechanisms of increased cardiometabolic risk in people with schizophrenia and other severe mental illnesses. , 2015, The lancet. Psychiatry.
[57] E. Fernández,et al. Secondhand smoke in outdoor settings: smokers’ consumption, non-smokers’ perceptions, and attitudes towards smoke-free legislation in Spain , 2015, BMJ Open.
[58] Charles E. Vejnar,et al. Repression of Arginase-2 Expression in Dendritic Cells by MicroRNA-155 Is Critical for Promoting T Cell Proliferation , 2014, The Journal of Immunology.
[59] T. Nolin,et al. The influence of smoking status on the pharmacokinetics and pharmacodynamics of clopidogrel and prasugrel: the PARADOX study. , 2013, Journal of the American College of Cardiology.
[60] M. Jeong,et al. ‘Smoker's paradox’ in young patients with acute myocardial infarction , 2012, Clinical and experimental pharmacology & physiology.
[61] S. Shyue,et al. β Common receptor integrates the erythropoietin signaling in activation of endothelial nitric oxide synthase , 2011, Journal of cellular physiology.
[62] T. Länne,et al. The association between circulating angiotensin-converting enzyme and cardiovascular risk in the elderly: a cross-sectional study , 2011, Journal of the renin-angiotensin-aldosterone system : JRAAS.
[63] Office on Smoking. How Tobacco Smoke Causes Disease: The Biology and Behavioral Basis for Smoking-Attributable Disease: A Report of the Surgeon General , 2010 .
[64] A. Mirazimi,et al. Dual effect of nitric oxide on SARS-CoV replication: Viral RNA production and palmitoylation of the S protein are affected , 2009, Virology.
[65] Ronald G. Crystal,et al. Smoking-Dependent Reprogramming of Alveolar Macrophage Polarization: Implication for Pathogenesis of Chronic Obstructive Pulmonary Disease1 , 2009, The Journal of Immunology.
[66] I. Faraoni,et al. miR-155 gene: a typical multifunctional microRNA. , 2009, Biochimica et biophysica acta.
[67] A. Patzak,et al. Endothelial nitric oxide synthase is predominantly involved in angiotensin II modulation of renal vascular resistance and norepinephrine release. , 2008, American journal of physiology. Regulatory, integrative and comparative physiology.
[68] U. Förstermann,et al. Endothelial Nitric Oxide Synthase in Vascular Disease: From Marvel to Menace , 2006, Circulation.
[69] P. Maes,et al. Inhibition of SARS-coronavirus infection in vitro by S-nitroso-N-acetylpenicillamine, a nitric oxide donor compound☆ , 2004, International Journal of Infectious Diseases.
[70] Chen Yan,et al. Functional Interplay Between Angiotensin II and Nitric Oxide: Cyclic GMP as a Key Mediator , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[71] D. Dudley,et al. Haplotype-Specific Effects on Endothelial NO Synthase Promoter Efficiency: Modifiable by Cigarette Smoking , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[72] H. Lee,et al. Gene Polymorphisms of Endothelial Nitric Oxide Synthase and Angiotensin-converting Enzyme in Patients with Lung Cancer , 2000, Lung.
[73] Michael Pittilo. Cigarette smoking, endothelial injury and cardiovascular disease , 2000, International journal of experimental pathology.
[74] G. Murrell,et al. Genotype dependent and cigarette specific effects on endothelial nitric oxide synthase gene expression and enzyme activity , 2000, FEBS letters.
[75] B. Mayer,et al. Tetrahydrobiopterin improves endothelium-dependent vasodilation in chronic smokers : evidence for a dysfunctional nitric oxide synthase. , 2000, Circulation research.
[76] J. Ayres,et al. Acute inhalation of cigarette smoke increases lower respiratory tract nitric oxide concentrations , 1998, Thorax.
[77] C. Rångemark,et al. Smoke-derived nitric oxide and vascular prostacyclin are unable to counteract the platelet effect of increased thromboxane formation in healthy female smokers. , 1996, Clinical physiology.
[78] R. Lassila,et al. Enhanced activation of the renin‐angiotensin‐aldosterone system in chronic cigarette smokers: A study of monozygotic twin pairs discordant for smoking , 1988, Clinical pharmacology and therapeutics.
[79] T. Higenbottam,et al. Nitric oxide yields of contemporary UK, US and French cigarettes. , 1987, International journal of epidemiology.