Mitochondrial implication in apoptosis. Towards an endosymbiont hypothesis of apoptosis evolution
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[1] HenryFliss,et al. Apoptosis in Ischemic and Reperfused Rat Myocardium , 1996 .
[2] G. Kroemer,et al. Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo , 1995, The Journal of experimental medicine.
[3] G. Kroemer,et al. Redox regulation of apoptosis: Impact of thiol oxidation status on mitochondrial function , 1997, European journal of immunology.
[4] G. Cohen,et al. An interleukin‐1β‐converting enzyme‐like protease is a common mediator of apoptosis in thymocytes , 1995, FEBS letters.
[5] Xiaodong Wang,et al. Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome c , 1996, Cell.
[6] M. Raff,et al. Programmed cell death and Bcl-2 protection in very low oxygen , 1995, Nature.
[7] John Calvin Reed,et al. Interactions among members of the Bcl-2 protein family analyzed with a yeast two-hybrid system , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[8] J. Greenberg,et al. Programmed cell death: a way of life for plants. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[9] H. D. del Portillo,et al. Heat shock induction of apoptosis in promastigotes of the unicellular organism Leishmania (Leishmania) amazonensis , 1996, Journal of cellular physiology.
[10] M. Raff,et al. Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death , 1996, The Journal of cell biology.
[11] G. Kroemer,et al. Inhibitors of permeability transition interfere with the disruption of the mitochondrial transmembrane potential during apoptosis , 1996, FEBS letters.
[12] D. Newmeyer,et al. Cell-free apoptosis in Xenopus egg extracts: Inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondria , 1994, Cell.
[13] P. O’Farrell,et al. Spatial programming of gene expression in early Drosophila embryogenesis. , 1986, Annual review of cell biology.
[14] P. Henkart. Apoptosis: O death, where is thy sting? , 1995, Journal of immunology.
[15] S. Snyder,et al. Mitochondrial benzodiazepine receptor linked to inner membrane ion channels by nanomolar actions of ligands. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[16] H. Winkler,et al. Cloning and expression of the Rickettsia prowazekii ADP/ATP translocator in Escherichia coli. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[17] J. Xiang,et al. BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[18] M. Zoratti,et al. Modulation of the mitochondrial megachannel by divalent cations and protons. , 1992, The Journal of biological chemistry.
[19] M W Gray,et al. Origin and evolution of mitochondrial DNA. , 1989, Annual review of cell biology.
[20] Z. Oltvai,et al. Checkpoints of dueling dimers foil death wishes , 1994, Cell.
[21] S. Nagata,et al. No requirement of reactive oxygen intermediates in Fas‐mediated apoptosis , 1994, FEBS letters.
[22] M. Zoratti,et al. The high-conductance channel of porin-less yeast mitochondria. , 1995, Biochimica et Biophysica Acta.
[23] R. Stroud,et al. Ion-channel-forming colicins. , 1998, Current opinion in structural biology.
[24] S. Lipton,et al. Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function , 1995, Neuron.
[25] K Kusano,et al. Selfish behavior of restriction-modification systems , 1995, Science.
[26] J. Coyle,et al. Kainate induces apoptosis in neurons , 1996, Neuroscience.
[27] S. Snyder,et al. Isolation of the mitochondrial benzodiazepine receptor: association with the voltage-dependent anion channel and the adenine nucleotide carrier. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[28] G. Kroemer,et al. Bcl-2 inhibits the mitochondrial release of an apoptogenic protease , 1996, The Journal of experimental medicine.
[29] C. Franceschi,et al. Protective effect of N-acetylcysteine in tumor necrosis factor-alpha-induced apoptosis in U937 cells: the role of mitochondria. , 1995, Experimental cell research.
[30] H. Ikeda,et al. DNA fragmentation of human infarcted myocardial cells demonstrated by the nick end labeling method and DNA agarose gel electrophoresis. , 1995, The American journal of pathology.
[31] Y. Tsujimoto,et al. Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitors. , 1996, Oncogene.
[32] S. Korsmeyer,et al. Bax-Deficient Mice with Lymphoid Hyperplasia and Male Germ Cell Death , 1995, Science.
[33] I. Reynolds,et al. Mitochondrial Depolarization in Glutamate-Stimulated Neurons: An Early Signal Specific to Excitotoxin Exposure , 1996, The Journal of Neuroscience.
[34] M. Raff,et al. Constitutive expression of the machinery for programmed cell death , 1996, The Journal of cell biology.
[35] D. Bredesen,et al. Bcl-2 inhibition of neural death: decreased generation of reactive oxygen species. , 1993, Science.
[36] P. Petit,et al. Commitment to apoptosis is associated with changes in mitochondrial biogenesis and activity in cell lines conditionally immortalized with simian virus 40. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[37] M. Runswick,et al. The mitochondrial transport protein superfamily , 1993, Journal of bioenergetics and biomembranes.
[38] G. Kroemer,et al. Apoptosis-associated derangement of mitochondrial function in cells lacking mitochondrial DNA. , 1996, Cancer research.
[39] D. Green,et al. The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.
[40] P. Bernardi,et al. Interactions of Cyclophilin with the Mitochondrial Inner Membrane and Regulation of the Permeability Transition Pore, a Cyclosporin A-sensitive Channel (*) , 1996, The Journal of Biological Chemistry.
[41] P. Petit,et al. Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosis , 1995, The Journal of cell biology.
[42] Dean P. Jones,et al. Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria Blocked , 1997, Science.
[43] C. Mannella,et al. Perspectives on the mitochondrial multiple conductance channel , 1996, Journal of bioenergetics and biomembranes.
[44] K. Schulze-Osthoff,et al. Cell nucleus and DNA fragmentation are not required for apoptosis , 1994, The Journal of cell biology.
[45] J. Lemasters,et al. The mitochondrial permeability transition: a new pathophysiological mechanism for Reye's syndrome and toxic liver injury. , 1996, The Journal of pharmacology and experimental therapeutics.
[46] M H Saier,et al. The mitochondrial carrier family of transport proteins: structural, functional, and evolutionary relationships. , 1993, Critical reviews in biochemistry and molecular biology.
[47] G. Evan,et al. Inhibition of Ced-3/ICE-related Proteases Does Not Prevent Cell Death Induced by Oncogenes, DNA Damage, or the Bcl-2 Homologue Bak , 1997, The Journal of cell biology.
[48] C. Zwizinski,et al. Release of mitochondrial matrix proteins through a Ca2+-requiring, cyclosporin-sensitive pathway. , 1989, Biochemical and biophysical research communications.
[49] J. Mazat,et al. Microtubule‐active drugs suppress the closure of the permeability transition pore in tumour mitochondria , 1996, FEBS letters.
[50] M Ptashne,et al. How the lambda repressor and cro work. , 1980, Cell.
[51] G. Kroemer,et al. Mitochondrial perturbations define lymphocytes undergoing apoptotic depletion in vivo , 1995, European journal of immunology.
[52] G. Kroemer. The pharmacology of T cell apoptosis. , 1995, Advances in immunology.
[53] I. Wertz,et al. Diverse molecular provocation of programmed cell death. , 1996, Trends in biochemical sciences.
[54] R. Gottlieb,et al. Loss of Function of Cytochrome c in Jurkat Cells Undergoing Fas-mediated Apoptosis* , 1996, The Journal of Biological Chemistry.
[55] G. Kroemer,et al. Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death , 1995, The Journal of experimental medicine.
[56] R. Benz,et al. Modulation of Neisseria Porin (PorB) by Cytosolic ATP/GTP of Target Cells: Parallels between Pathogen Accommodation and Mitochondrial Endosymbiosis , 1996, Cell.
[57] R. Meadows,et al. X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death , 1996, Nature.
[58] C. Thompson,et al. bcl-XL is the major bcl-x mRNA form expressed during murine development and its product localizes to mitochondria. , 1994, Development.
[59] D. Ellis,et al. Apoptosis in procyclic Trypanosoma brucei rhodesiense in vitro. , 1996, Cell death and differentiation.
[60] K. Orita,et al. Activation of apoptosis during the reperfusion phase after rat liver ischemia. , 1996, Transplantation proceedings.
[61] H. Horvitz,et al. C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2 , 1994, Cell.
[62] P. Henkart. ICE family proteases: mediators of all apoptotic cell death? , 1996, Immunity.
[63] C. Franceschi,et al. Mitochondria are selective targets for the protective effects of heat shock against oxidative injury. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[64] Jean-Claude Martinou,et al. Overexpression of BCL-2 in transgenic mice protects neurons from naturally occurring cell death and experimental ischemia , 1994, Neuron.
[65] Seamus J. Martin,et al. Protease activation during apoptosis: Death by a thousand cuts? , 1995, Cell.
[66] H. Ginsburg,et al. Metabolic interconnection between the human malarial parasite Plasmodium falciparum and its host erythrocyte. Regulation of ATP levels by means of an adenylate translocator and adenylate kinase. , 1989, The Journal of biological chemistry.
[67] G. Kroemer,et al. Mitochondrial control of nuclear apoptosis , 1996, The Journal of experimental medicine.
[68] G. Kroemer,et al. Bcl-2 and Bcl-XL antagonize the mitochondrial dysfunction preceding nuclear apoptosis induced by chemotherapeutic agents. , 1997, Cancer research.
[69] Patrick R. Griffin,et al. Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis , 1995, Nature.
[70] S. Mpoke,et al. DNA digestion and chromatin condensation during nuclear death in Tetrahymena. , 1996, Experimental cell research.
[71] M. Zoratti,et al. The mitochondrial permeability transition. , 1995, Biochimica et biophysica acta.
[72] L. Cheng,et al. Programmed cell death by default in embryonic cells, fibroblasts, and cancer cells. , 1995, Molecular biology of the cell.
[73] John Calvin Reed,et al. Structure-function comparisons of the proapoptotic protein Bax in yeast and mammalian cells , 1996, Molecular and cellular biology.
[74] G. Kroemer,et al. Chloromethyl-X-Rosamine is an aldehyde-fixable potential-sensitive fluorochrome for the detection of early apoptosis. , 1996, Cytometry.
[75] W. Greenhalf,et al. Role of mitochondria and C‐terminal membrane anchor of Bcl‐2 in Bax induced growth arrest and mortality in Saccharomyces cerevisiae , 1996, FEBS letters.
[76] P. Nicotera,et al. Energy supply and the shape of death in neurons and lymphoid cells , 1997, Cell Death and Differentiation.
[77] E. Wang,et al. Bcl-2 potentiates the maximal calcium uptake capacity of neural cell mitochondria. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[78] Y. Tsujimoto,et al. Prevention of hypoxia-induced cell death by Bcl-2 and Bcl-xL , 1995, Nature.
[79] M. Colombini,et al. ATP Flux Is Controlled by a Voltage-gated Channel from the Mitochondrial Outer Membrane* , 1996, The Journal of Biological Chemistry.
[80] G. Cohen,et al. Benzyloxycarbonyl-Val-Ala-Asp (OMe) fluoromethylketone (Z-VAD.FMK) inhibits apoptosis by blocking the processing of CPP32. , 1996, The Biochemical journal.
[81] J. Cohen,et al. Programmed cell death in the immune system. , 1991, Advances in immunology.
[82] T. Deckwerth,et al. Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor , 1993, The Journal of cell biology.
[83] Y. Tsujimoto. Apoptosis and necrosis: Intracellular ATP level as a determinant for cell death modes , 1997, Cell Death and Differentiation.
[84] A. F. Schinder,et al. Mitochondrial Dysfunction Is a Primary Event in Glutamate Neurotoxicity , 1996, The Journal of Neuroscience.
[85] D. Häder,et al. Evidence for UV-B-induced DNA degradation in Euglena gracilis mediated by activation of metal-dependent nucleases. , 1995, Journal of photochemistry and photobiology. B, Biology.
[86] M. Yarmolinsky,et al. Programmed cell death in bacterial populations , 1995, Science.
[87] S. Novgorodov,et al. The Peptide Mastoparan Is a Potent Facilitator of the Mitochondrial Permeability Transition (*) , 1995, The Journal of Biological Chemistry.
[88] John Calvin Reed,et al. Investigation of the subcellular distribution of the bcl-2 oncoprotein: residence in the nuclear envelope, endoplasmic reticulum, and outer mitochondrial membranes. , 1993, Cancer research.
[89] D. Andrews,et al. Bcl‐2 mutants with restricted subcellular location reveal spatially distinct pathways for apoptosis in different cell types. , 1996, The EMBO journal.
[90] P. Golstein,et al. The target cell nucleus is not required for cell-mediated granzyme- or Fas-based cytotoxicity , 1995, The Journal of experimental medicine.
[91] G. Kroemer,et al. Mitochondrial control of apoptosis. , 1997, Immunology today.
[92] John Calvin Reed,et al. Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA , 1993, Nature.
[93] Alan M. Jones,et al. Logjam at the Styx: Programmed cell death in plants , 1996 .
[94] G. Kroemer,et al. Mitochondrial permeability transition triggers lymphocyte apoptosis. , 1996, Journal of immunology.
[95] L Margulis,et al. Symbiotic theory of the origin of eukaryotic organelles; criteria for proof. , 1975, Symposia of the Society for Experimental Biology.
[96] J. Davoust,et al. Programmed cell death in Dictyostelium. , 1994, Journal of cell science.
[97] B. Chernyak,et al. Modulation of the Mitochondrial Permeability Transition Pore by Pyridine Nucleotides and Dithiol Oxidation at Two Separate Sites (*) , 1996, The Journal of Biological Chemistry.
[98] M. Raff,et al. Programmed cell death and Bcl‐2 protection in the absence of a nucleus. , 1994, The EMBO journal.
[99] John Calvin Reed,et al. Structure-function analysis of the Bcl-2 oncoprotein. Addition of a heterologous transmembrane domain to portions of the Bcl-2 beta protein restores function as a regulator of cell survival. , 1993, The Journal of biological chemistry.
[100] J. Hoek,et al. Protoporphyrin IX, an endogenous ligand of the peripheral benzodiazepine receptor, potentiates induction of the mitochondrial permeability transition and the killing of cultured hepatocytes by rotenone. , 1994, The Journal of biological chemistry.
[101] G. Schulz,et al. Porins: general to specific, native to engineered passive pores. , 1996, Current opinion in structural biology.
[102] A. Wyllie,et al. Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics , 1972, British Journal of Cancer.
[103] E. Lam,et al. In Situ Detection of nDNA Fragmentation during the Differentiation of Tracheary Elements in Higher Plants , 1995, Plant physiology.
[104] Z. Oltvai,et al. Role of membrane anchor domain of Bcl-2 in suppression of apoptosis caused by E1B-defective adenovirus. , 1994, The Journal of biological chemistry.
[105] N. Rawlings,et al. Evolutionary families of peptidases. , 1993, The Biochemical journal.
[106] G. Cohen,et al. An ICE‐like protease is a common mediator of apoptosis induced by diverse stimuli in human monocytic THP.1 cells , 1995, FEBS letters.
[107] K. Kinnally,et al. A mitochondrial signal peptide from Neurospora crassa increases the permeability of isolated rat liver mitochondria. , 1996, Archives of biochemistry and biophysics.
[108] G. Kroemer,et al. Glutathione depletion is an early and calcium elevation is a late event of thymocyte apoptosis. , 1997, Journal of immunology.
[109] L. Benítez-Bribiesca. [Apoptosis in the pathogenesis and treatment of disease]. , 1995, Gaceta medica de Mexico.
[110] R. B. Jensen,et al. Programmed cell death in bacteria: proteic plasmid stabilization systems , 1995, Molecular microbiology.