Pathological features of vessel co-option versus sprouting angiogenesis
暂无分享,去创建一个
P. Vermeulen | L. Dirix | S. V. van Laere | C. Verhoef | D. Grünhagen | C. Lugassy | R. Barnhill | M. Gerling | C. Fernández Moro | E. Caspani | M. Dirix | Emily Latacz
[1] R. Kerbel,et al. Vessel co-option in cancer , 2019, Nature Reviews Clinical Oncology.
[2] H. Nakshatri,et al. Flower isoforms promote competitive growth in cancer , 2019, Nature.
[3] P. Vermeulen,et al. Histopathological growth patterns of colorectal liver metastasis exhibit little heterogeneity and can be determined with a high diagnostic accuracy , 2019, Clinical & Experimental Metastasis.
[4] D. Nguyen,et al. Transcriptomic Hallmarks of Tumor Plasticity and Stromal Interactions in Brain Metastasis , 2019, Cell reports.
[5] Z. Szallasi,et al. Origin and Distribution of Connective Tissue and Pericytes Impacting Vascularization in Brain Metastases With Different Growth Patterns , 2019, Journal of neuropathology and experimental neurology.
[6] H. Moch,et al. Eight autopsy cases of melanoma brain metastases showing angiotropism and pericytic mimicry. Implications for extravascular migratory metastasis , 2019, Journal of cutaneous pathology.
[7] B. Stanger,et al. The tumor as organizer model , 2019, Science.
[8] P. Vermeulen,et al. Salvage treatment for recurrences after first resection of colorectal liver metastases: the impact of histopathological growth patterns , 2019, Clinical & Experimental Metastasis.
[9] P. Vermeulen,et al. Angiogenic desmoplastic histopathological growth pattern as a prognostic marker of good outcome in patients with colorectal liver metastases , 2019, Angiogenesis.
[10] P. van Dam,et al. Histopathological growth patterns as a candidate biomarker for immunomodulatory therapy. , 2018, Seminars in cancer biology.
[11] G. Bergers,et al. The reciprocal function and regulation of tumor vessels and immune cells offers new therapeutic opportunities in cancer. , 2018, Seminars in cancer biology.
[12] V. Servois,et al. Replacement and desmoplastic histopathological growth patterns: A pilot study of prediction of outcome in patients with uveal melanoma liver metastases , 2018, The journal of pathology. Clinical research.
[13] M. Rosenblum,et al. Pericyte-like spreading by disseminated cancer cells activates YAP and MRTF for metastatic colonization , 2018, Nature Cell Biology.
[14] B. Döme,et al. Role of (myo)fibroblasts in the development of vascular and connective tissue structure of the C38 colorectal cancer in mice , 2018, Cancer communications.
[15] B. Bozóky,et al. Growth patterns of colorectal cancer liver metastases and their impact on prognosis: a systematic review , 2018, BMJ open gastroenterology.
[16] A. Harris,et al. Non-angiogenic tumours and their influence on cancer biology , 2018, Nature Reviews Cancer.
[17] G. Yousef,et al. Elucidating mechanisms of sunitinib resistance in renal cancer: an integrated pathological-molecular analysis , 2017, Oncotarget.
[18] Yves Sucaet,et al. International consensus guidelines for scoring the histopathological growth patterns of liver metastasis , 2017, British Journal of Cancer.
[19] H. Augustin,et al. Organotypic vasculature: From descriptive heterogeneity to functional pathophysiology , 2017, Science.
[20] J. Larkin,et al. Vessel co‐option is common in human lung metastases and mediates resistance to anti‐angiogenic therapy in preclinical lung metastasis models , 2016, The Journal of pathology.
[21] G. G. Van den Eynden,et al. Vessel co-option mediates resistance to anti-angiogenic therapy in liver metastases , 2016, Nature Medicine.
[22] T. Rodríguez,et al. Cell Competition and Its Role in the Regulation of Cell Fitness from Development to Cancer. , 2016, Developmental cell.
[23] H. Kleinman,et al. Imaging of Angiotropism/Vascular Co-Option in a Murine Model of Brain Melanoma: Implications for Melanoma Progression along Extravascular Pathways , 2016, Scientific Reports.
[24] G. Tiegs,et al. Modulation of liver tolerance by conventional and nonconventional antigen-presenting cells and regulatory immune cells , 2016, Cellular & Molecular Immunology.
[25] A. Harris,et al. Why some tumours trigger neovascularisation and others don’t: the story thus far , 2016, Chinese journal of cancer.
[26] R. Jain,et al. Investigation of the Lack of Angiogenesis in the Formation of Lymph Node Metastases. , 2015, Journal of the National Cancer Institute.
[27] A. Cole,et al. Hepatic progenitor cells of biliary origin with liver repopulation capacity , 2015, Nature Cell Biology.
[28] J. Tímár,et al. Mechanism of tumour vascularization in experimental lung metastases , 2015, The Journal of pathology.
[29] S. Martinez,et al. Glioblastoma: A Pathogenic Crosstalk between Tumor Cells and Pericytes , 2014, PloS one.
[30] T. Mikkelsen,et al. Mechanisms of Glioma Formation: Iterative Perivascular Glioma Growth and Invasion Leads to Tumor Progression, VEGF-Independent Vascularization, and Resistance to Antiangiogenic Therapy12 , 2014, Neoplasia.
[31] M. Kris,et al. Serpins Promote Cancer Cell Survival and Vascular Co-Option in Brain Metastasis , 2014, Cell.
[32] Frank Winkler,et al. Invasion patterns in brain metastases of solid cancers. , 2013, Neuro-oncology.
[33] H. Kleinman,et al. Could pericytic mimicry represent another type of melanoma cell plasticity with embryonic properties? , 2013, Pigment cell & melanoma research.
[34] R. Jain. Normalizing tumor microenvironment to treat cancer: bench to bedside to biomarkers. , 2013, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[35] J. Pichler,et al. Extent of peritumoral brain edema correlates with prognosis, tumoral growth pattern, HIF1a expression and angiogenic activity in patients with single brain metastases , 2012, Clinical and Experimental Metastasis.
[36] J. A. van der Laak,et al. Effects of Dual Targeting of Tumor Cells and Stroma in Human Glioblastoma Xenografts with a Tyrosine Kinase Inhibitor against c-MET and VEGFR2 , 2013, PloS one.
[37] S. Vandenberg,et al. Role of connexins in metastatic breast cancer and melanoma brain colonization , 2013, Journal of Cell Science.
[38] L. Ellis,et al. Endothelial cells promote the colorectal cancer stem cell phenotype through a soluble form of Jagged-1. , 2013, Cancer cell.
[39] Csaba Bödör,et al. Structural analysis of oval‐cell–mediated liver regeneration in rats , 2012, Hepatology.
[40] Z. Dong,et al. Claudin-2 Promotes Breast Cancer Liver Metastasis by Facilitating Tumor Cell Interactions with Hepatocytes , 2012, Molecular and Cellular Biology.
[41] B. Döme,et al. Lack of Angiogenesis in Experimental Brain Metastases , 2011, Journal of neuropathology and experimental neurology.
[42] F. Pépin,et al. Claudin-2 is selectively enriched in and promotes the formation of breast cancer liver metastases through engagement of integrin complexes , 2011, Oncogene.
[43] Matthias Hermes,et al. Prediction and validation of cell alignment along microvessels as order principle to restore tissue architecture in liver regeneration , 2010, Proceedings of the National Academy of Sciences.
[44] N. Sibson,et al. The Vascular Basement Membrane as “Soil” in Brain Metastasis , 2009, PloS one.
[45] P Hahnfeldt,et al. Migration rules: tumours are conglomerates of self-metastases , 2009, British Journal of Cancer.
[46] Masahiro Inoue,et al. Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis. , 2009, Cancer cell.
[47] E. van Marck,et al. Different growth patterns of non-small cell lung cancer represent distinct biologic subtypes. , 2008, The Annals of thoracic surgery.
[48] E. Crivellato,et al. Contribution of endothelial cells to organogenesis: a modern reappraisal of an old Aristotelian concept , 2007, Journal of anatomy.
[49] P. V. Van Schil,et al. Distinct angiogenic and non‐angiogenic growth patterns of lung metastases from renal cell carcinoma , 2007, Histopathology.
[50] D. L. Le Couteur,et al. T lymphocytes interact with hepatocytes through fenestrations in murine liver sinusoidal endothelial cells , 2006, Hepatology.
[51] K. Gatter,et al. Is nonangiogenesis a novel pathway for cancer progression? A study using 3-dimensional tumour reconstructions , 2006, British Journal of Cancer.
[52] M. Oertel,et al. Cell competition leads to a high level of normal liver reconstitution by transplanted fetal liver stem/progenitor cells. , 2006, Gastroenterology.
[53] K. Allison,et al. Radiographically occult, diffuse intrasinusoidal hepatic metastases from primary breast carcinomas: a clinicopathologic study of 3 autopsy cases. , 2004, Archives of pathology & laboratory medicine.
[54] C. Lugassy,et al. Angiotropic malignant melanoma and extravascular migratory metastasis: description of 36 cases with emphasis on a new mechanism of tumour spread , 2004, Pathology.
[55] Pieter Wesseling,et al. Antiangiogenic Therapy of Cerebral Melanoma Metastases Results in Sustained Tumor Progression via Vessel Co-Option , 2004, Clinical Cancer Research.
[56] J. Weyler,et al. Prognostic value of nonangiogenic and angiogenic growth patterns in non-small-cell lung cancer , 2004, British Journal of Cancer.
[57] E. van Marck,et al. Breast adenocarcinoma liver metastases, in contrast to colorectal cancer liver metastases, display a non-angiogenic growth pattern that preserves the stroma and lacks hypoxia , 2004, British Journal of Cancer.
[58] C. Degott,et al. Angiogenesis and extracellular matrix remodelling in bronchioloalveolar carcinomas: distinctive patterns in mucinous and non‐mucinous tumours , 2004, Histopathology.
[59] C. Colpaert,et al. Cutaneous breast cancer deposits show distinct growth patterns with different degrees of angiogenesis, hypoxia and fibrin deposition , 2003, Histopathology.
[60] A. Nicholson,et al. Vascular phenotype in angiogenic and non-angiogenic lung non-small cell carcinomas , 2002, British Journal of Cancer.
[61] P. Wesseling,et al. Vascular endothelial growth factor-A(165) induces progression of melanoma brain metastases without induction of sprouting angiogenesis. , 2002, Cancer research.
[62] E. van Marck,et al. Lack of angiogenesis in lymph node metastases of carcinomas is growth pattern‐dependent , 2002, Histopathology.
[63] E. van Marck,et al. Liver metastases from colorectal adenocarcinomas grow in three patterns with different angiogenesis and desmoplasia , 2001, The Journal of pathology.
[64] J. Rossant,et al. Liver Organogenesis Promoted by Endothelial Cells Prior to Vascular Function , 2001, Science.
[65] H. Gröne,et al. Constitutive activation of hypoxia-inducible genes related to overexpression of hypoxia-inducible factor-1alpha in clear cell renal carcinomas. , 2001, Cancer research.
[66] R. Price,et al. Evidence for novel non-angiogenic pathway in breast-cancer metastasis , 2000, The Lancet.
[67] G. Yancopoulos,et al. Vessel cooption, regression, and growth in tumors mediated by angiopoietins and VEGF. , 1999, Science.
[68] M Buyse,et al. Non-small-cell lung carcinoma tumor growth without morphological evidence of neo-angiogenesis. , 1997, The American journal of pathology.
[69] S. Love,et al. ‘Revertant’ DCIS in human axillary breast carcinoma metastases , 1997, The Journal of pathology.
[70] M. Buyse,et al. Immunocytochemical markers in stage I lung cancer: relevance to prognosis. , 1997, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[71] T. Terada,et al. Histologic growth patterns of metastatic carcinomas of the liver. , 1996, Japanese journal of clinical oncology.
[72] M. Kage,et al. Pathomorphologic characteristics of small hepatocellular carcinoma: A special reference to small hepatocellular carcinoma with indistinct margins , 1995, Hepatology.
[73] J. Risteli,et al. Immunohistochemical Evidence that Lung Carcinomas Grow on Alveolar Basement Membranes , 1990, The American journal of surgical pathology.
[74] Dr. Johannes Erichsen. Zwei Fälle von Carcinosis acuta miliaris , 1861, Archiv für pathologische Anatomie und Physiologie und für klinische Medicin.
[75] Tracy T Batchelor,et al. Glioblastoma recurrence after cediranib therapy in patients: lack of "rebound" revascularization as mode of escape. , 2011, Cancer research.
[76] W. Leenders,et al. Vessel co-option: how tumors obtain blood supply in the absence of sprouting angiogenesis. , 2002, Endothelium : journal of endothelial cell research.