Uric acid: a potent molecular contributor to pluripotent stem cell cardiac differentiation via mesoderm specification
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James J. H. Chong | F. Lan | Wen-jing Lu | Yujie Zeng | Anila Khalique | Taoyan Liu | Hua He | Jingyi Wang | Fusheng Han | Bing-bing Ke | Zhihong Zhao
[1] Zack Z Wang,et al. Epithelial–mesenchymal transition (EMT): A biological process in the development, stem cell differentiation, and tumorigenesis , 2017, Journal of cellular physiology.
[2] D. Elliott,et al. Biomarkers of Human Pluripotent Stem Cell-Derived Cardiac Lineages. , 2017, Trends in molecular medicine.
[3] Berto J. Bouma,et al. Changing Landscape of Congenital Heart Disease , 2017, Circulation research.
[4] P. Barker,et al. Ultrasound Examination of the Fetal Heart. , 2017, Obstetrical & gynecological survey.
[5] T. Benedek,et al. Genetics of Congenital Heart Disease: Past and Present , 2016, Biochemical Genetics.
[6] Pang Wei Koh,et al. Mapping the Pairwise Choices Leading from Pluripotency to Human Bone, Heart, and Other Mesoderm Cell Types , 2016, Cell.
[7] Pedro Madrigal,et al. Initiation of stem cell differentiation involves cell cycle-dependent regulation of developmental genes by Cyclin D , 2016, Genes & development.
[8] H. Ng,et al. Deterministic Restriction on Pluripotent State Dissolution by Cell-Cycle Pathways , 2015, Cell.
[9] D. Medici,et al. Signaling mechanisms of the epithelial-mesenchymal transition , 2014, Science Signaling.
[10] B. Cui,et al. Chemically Defined and Small Molecule-Based Generation of Human Cardiomyocytes , 2014, Nature methods.
[11] D. Kavanagh,et al. Mesenchymal Stem Cell Priming: Fine-tuning Adhesion and Function , 2014, Stem Cell Reviews and Reports.
[12] D. Kavanagh,et al. Mesenchymal Stem Cell Priming: Fine-tuning Adhesion and Function , 2014, Stem Cell Reviews and Reports.
[13] Samy Lamouille,et al. Molecular mechanisms of epithelial–mesenchymal transition , 2014, Nature Reviews Molecular Cell Biology.
[14] Aliccia Bollig-Fischer,et al. Systems analysis reveals a transcriptional reversal of the mesenchymal phenotype induced by SNAIL-inhibitor GN-25 , 2013, BMC Systems Biology.
[15] N. Chen,et al. Proteasome inhibitor inhibits proliferation and induces apoptosis in renal interstitial fibroblasts , 2013, Pharmacological reports : PR.
[16] E. Abdelalim. Molecular Mechanisms Controlling the Cell Cycle in Embryonic Stem Cells , 2013, Stem Cell Reviews and Reports.
[17] M. J. Kim,et al. Uric acid-induced phenotypic transition of renal tubular cells as a novel mechanism of chronic kidney disease. , 2013, American journal of physiology. Renal physiology.
[18] J. Draper,et al. Lengthened G1 phase indicates differentiation status in human embryonic stem cells. , 2013, Stem cells and development.
[19] K. McBride,et al. Cardiac teratogenicity in mouse maternal phenylketonuria: defining phenotype parameters and genetic background influences. , 2012, Molecular genetics and metabolism.
[20] Liu Wang,et al. Ascorbic acid enhances the cardiac differentiation of induced pluripotent stem cells through promoting the proliferation of cardiac progenitor cells , 2011, Cell Research.
[21] S. Gilboa,et al. Maternal Smoking and Congenital Heart Defects in the Baltimore-Washington Infant Study , 2011, Pediatrics.
[22] A. Zwinderman,et al. The changing epidemiology of congenital heart disease , 2011, Nature Reviews Cardiology.
[23] Jason L Salemi,et al. Mortality Resulting From Congenital Heart Disease Among Children and Adults in the United States, 1999 to 2006 , 2010, Circulation.
[24] M. Loane,et al. The prevalence of congenital anomalies in Europe. , 2010, Advances in experimental medicine and biology.
[25] R. Huang,et al. Epithelial-Mesenchymal Transitions in Development and Disease , 2009, Cell.
[26] Raghu Kalluri,et al. The basics of epithelial-mesenchymal transition. , 2009, The Journal of clinical investigation.
[27] Richard J. Johnson,et al. Uric Acid: The Oxidant-Antioxidant Paradox , 2008, Nucleosides, nucleotides & nucleic acids.
[28] V. Dulskienė,et al. Selected environmental risk factors and congenital heart defects. , 2008, Medicina.
[29] Jeffrey A. Feinstein,et al. Noninherited Risk Factors and Congenital Cardiovascular Defects: Current Knowledge , 2007, Pediatrics.
[30] Jeffrey A. Feinstein,et al. Noninherited Risk Factors and Congenital Cardiovascular Defects: Current Knowledge: A Scientific Statement From the American Heart Association Council on Cardiovascular Disease in the Young , 2007, Circulation.
[31] S. Dalton,et al. Cell cycle control of embryonic stem cells , 2007, Stem Cell Reviews.
[32] E. Olson,et al. A Common Progenitor at the Heart of Development , 2006, Cell.
[33] S. Weiss,et al. Wnt-dependent Regulation of the E-cadherin Repressor Snail* , 2005, Journal of Biological Chemistry.
[34] D. Noonan,et al. NPDC-1, a Novel Regulator of Neuronal Proliferation, Is Degraded by the Ubiquitin/Proteasome System through a PEST Degradation Motif* , 2004, Journal of Biological Chemistry.
[35] J. Massagué,et al. Epithelial-Mesenchymal Transitions Twist in Development and Metastasis , 2004, Cell.
[36] T. Özçelik,et al. Uric acid as radical scavenger and antioxidant in the heart , 1989, Pflügers Archiv.
[37] Richard T. Lee,et al. Ascorbic Acid Enhances Differentiation of Embryonic Stem Cells Into Cardiac Myocytes , 2003, Circulation.
[38] A. Nikolov,et al. [Maternal insulin-dependent diabetes and congenital malformations in the newborn]. , 2003, Akusherstvo i ginekologiia.
[39] Austin G Smith,et al. Signalling, cell cycle and pluripotency in embryonic stem cells. , 2002, Trends in cell biology.
[40] P. R. Yew,et al. Ubiquitin‐mediated proteolysis of vertebrate G1‐ and S‐phase regulators , 2001, Journal of cellular physiology.
[41] Francisco Portillo,et al. The transcription factor Snail controls epithelial–mesenchymal transitions by repressing E-cadherin expression , 2000, Nature Cell Biology.
[42] A. G. Herreros,et al. The transcription factor Snail is a repressor of E-cadherin gene expression in epithelial tumour cells , 2000, Nature Cell Biology.
[43] Daniel Levy,et al. Serum Uric Acid and Risk for Cardiovascular Disease and Death: The Framingham Heart Study , 1999, Annals of Internal Medicine.
[44] B. Ames,et al. Uric acid provides an antioxidant defense in humans against oxidant- and radical-caused aging and cancer: a hypothesis. , 1981, Proceedings of the National Academy of Sciences of the United States of America.