Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock
暂无分享,去创建一个
Yan Zeng | Zhengyu He | Xiangrui Wang | Shuzhen Chen | Guangxun Meng | W. Strober | Bing Sun | Shuhui Sun | Jing Li | Yan Wang | Bo Huang | Chang Chen | Kairui Mao | Xiaodong Wu | Mingkuan Chen | Yu Hu | Yonglei Ma | Yan Zeng
[1] V. Dixit,et al. Phosphorylation of NLRC4 is critical for inflammasome activation , 2012, Nature.
[2] Bino John,et al. miRNA-939 regulates human inducible nitric oxide synthase posttranscriptional gene expression in human hepatocytes , 2012, Proceedings of the National Academy of Sciences.
[3] C. Dinarello,et al. The Rate of Interleukin-1β Secretion in Different Myeloid Cells Varies with the Extent of Redox Response to Toll-like Receptor Triggering* , 2011, The Journal of Biological Chemistry.
[4] F. Liew,et al. Regulation of type 17 helper T-cell function by nitric oxide during inflammation , 2011, Proceedings of the National Academy of Sciences.
[5] Denis Gris,et al. Fatty acid–induced NLRP3-ASC inflammasome activation interferes with insulin signaling , 2011, Nature Immunology.
[6] S. Ryter,et al. Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome. , 2011, Nature immunology.
[7] J. Stamler,et al. The SNO-proteome: causation and classifications. , 2011, Current opinion in chemical biology.
[8] J. Tschopp,et al. A role for mitochondria in NLRP3 inflammasome activation , 2011, Nature.
[9] Yan Zeng,et al. Tripartite-Motif Protein 30 Negatively Regulates NLRP3 Inflammasome Activation by Modulating Reactive Oxygen Species Production , 2010, The Journal of Immunology.
[10] V. Dixit,et al. Redundant roles for inflammasome receptors NLRP3 and NLRC4 in host defense against Salmonella , 2010, The Journal of experimental medicine.
[11] Egil Lien,et al. NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals , 2010, Nature.
[12] T. McMahon,et al. Transport rather than diffusion-dependent route for nitric oxide gas activity in alveolar epithelium. , 2010, Free radical biology & medicine.
[13] M. Kastan,et al. The NLRP3 inflammasome protects against loss of epithelial integrity and mortality during experimental colitis. , 2010, Immunity.
[14] D. Kastner,et al. Autoinflammatory Disease Reloaded: A Clinical Perspective , 2010, Cell.
[15] E. Alnemri,et al. Anti-inflammatory Compounds Parthenolide and Bay 11-7082 Are Direct Inhibitors of the Inflammasome* , 2010, The Journal of Biological Chemistry.
[16] J. Tschopp,et al. The Inflammasomes , 2010, Cell.
[17] Y. Wang,et al. Dec2 Promotes Th2 Cell Differentiation by Enhancing IL-2R Signaling1 , 2009, The Journal of Immunology.
[18] E. Alnemri,et al. Cutting Edge: NF-κB Activating Pattern Recognition and Cytokine Receptors License NLRP3 Inflammasome Activation by Regulating NLRP3 Expression1 , 2009, The Journal of Immunology.
[19] Fuping Zhang,et al. A mutation in the Nlrp3 gene causing inflammasome hyperactivation potentiates Th17 cell-dominant immune responses. , 2009, Immunity.
[20] J. Tschopp,et al. Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence , 2009, Nature.
[21] J. Ting,et al. The NLRP3 inflammasome mediates in vivo innate immunity to influenza A virus through recognition of viral RNA. , 2009, Immunity.
[22] Akiko Iwasaki,et al. Inflammasome recognition of influenza virus is essential for adaptive immune responses , 2009, The Journal of experimental medicine.
[23] Richard A. Flavell,et al. Crucial role for the Nalp3 inflammasome in the immunostimulatory properties of aluminium adjuvants , 2008, Nature.
[24] J. Tschopp,et al. Innate Immune Activation Through Nalp3 Inflammasome Sensing of Asbestos and Silica , 2008, Science.
[25] F. Liew,et al. Nitric oxide induces CD4+CD25+ Foxp3− regulatory T cells from CD4+CD25− T cells via p53, IL-2, and OX40 , 2007, Proceedings of the National Academy of Sciences.
[26] A. Cauwels. Nitric oxide in shock. , 2007, Kidney international.
[27] E. Alnemri,et al. The pyroptosome: a supramolecular assembly of ASC dimers mediating inflammatory cell death via caspase-1 activation , 2007, Cell Death and Differentiation.
[28] F. Martinon,et al. Gout-associated uric acid crystals activate the NALP3 inflammasome , 2006, Nature.
[29] V. Dixit,et al. Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf , 2004, Nature.
[30] J. Bakker,et al. Multiple-center, randomized, placebo-controlled, double-blind study of the nitric oxide synthase inhibitor 546C88: Effect on survival in patients with septic shock* , 2004, Critical care medicine.
[31] A. Rawlingson. Nitric oxide, inflammation and acute burn injury. , 2003, Burns : journal of the International Society for Burn Injuries.
[32] R. Cross,et al. Nitric Oxide in Inflammatory Bowel Disease , 2003, Inflammatory bowel diseases.
[33] Christian Bogdan,et al. Nitric oxide and the immune response , 2001, Nature Immunology.
[34] Santiago Lamas,et al. Nitrosylation The Prototypic Redox-Based Signaling Mechanism , 2001, Cell.
[35] C. Cooper,et al. Nitric oxide synthases: structure, function and inhibition. , 2001, The Biochemical journal.
[36] C. Bogdan,et al. The role of nitric oxide in innate immunity , 2000, Immunological reviews.
[37] C. Tribble,et al. Protective roles of nitric oxide and testosterone in endotoxemia: evidence from NOS-2-deficient mice. , 1998, The American journal of physiology.
[38] S. Abramson,et al. The role of nitric oxide in inflammation and immunity. , 1998, Arthritis and rheumatism.
[39] T. Billiar,et al. Nitric oxide prevents IL-1beta and IFN-gamma-inducing factor (IL-18) release from macrophages by inhibiting caspase-1 (IL-1beta-converting enzyme). , 1998, Journal of immunology.
[40] T. Billiar,et al. Nitric oxide reversibly inhibits seven members of the caspase family via S-nitrosylation. , 1997, Biochemical and biophysical research communications.
[41] C. Nathan,et al. Nitric oxide and macrophage function. , 1997, Annual review of immunology.
[42] C. Szabó,et al. BENEFICIAL VERSUS DETRIMENTAL EFFECTS OF NITRIC OXIDE SYNTHASE INHIBITORS IN CIRCULATORY SHOCK LESSONS LEARNED FROM EXPERIMENTAL AND CLINICAL STUDIES , 1997, Shock.
[43] O. Smithies,et al. Mice lacking inducible nitric oxide synthase are not resistant to lipopolysaccharide-induced death. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[44] J. Weinberg,et al. Human mononuclear phagocyte inducible nitric oxide synthase (iNOS): analysis of iNOS mRNA, iNOS protein, biopterin, and nitric oxide production by blood monocytes and peritoneal macrophages. , 1995, Blood.
[45] I. Charles,et al. Altered immune responses in mice lacking inducible nitric oxide synthase , 1995, Nature.
[46] C. Nathan,et al. Altered responses to bacterial infection and endotoxic shock in mice lacking inducible nitric oxide synthase , 1995, Cell.
[47] S Moncada,et al. Nitric oxide synthases in mammals. , 1994, The Biochemical journal.
[48] S. Moncada,et al. The L-arginine-nitric oxide pathway. , 1993, The New England journal of medicine.
[49] F. Murad,et al. Nitric oxide activates guanylate cyclase and increases guanosine 3':5'-cyclic monophosphate levels in various tissue preparations. , 1977, Proceedings of the National Academy of Sciences of the United States of America.