Injection of pre-psoriatic skin with CD4+ T cells induces psoriasis.

[1]  B. Nickoloff The immunologic and genetic basis of psoriasis. , 1999, Archives of dermatology.

[2]  S. Porcelli,et al.  Response of murine and normal human skin to injection of allogeneic blood-derived psoriatic immunocytes: detection of T cells expressing receptors typically present on natural killer cells, including CD94, CD158, and CD161. , 1999, Archives of dermatology.

[3]  O. Mandelboim,et al.  The Transmembrane Sequence of Human Histocompatibility Leukocyte Antigen (HLA)-C as a Determinant in Inhibition of a Subset of Natural Killer Cells , 1999, The Journal of experimental medicine.

[4]  B. Nickoloff Pathogenesis and immunointervention strategies for psoriasis , 1998 .

[5]  James T. Elder,et al.  Linkage analysis of human leukocyte antigen (HLA) markers in familial psoriasis: strong disequilibrium effects provide evidence for a major determinant in the HLA-B/-C region. , 1998, American journal of human genetics.

[6]  B. Nickoloff,et al.  Superantigens, autoantigens, and pathogenic T cells in psoriasis. , 1998, The Journal of investigative dermatology.

[7]  D. Anthony,et al.  T cells reactive to a single immunodominant self-restricted allopeptide induce skin graft rejection in mice. , 1998, The Journal of clinical investigation.

[8]  S. Bauer,et al.  Recognition of stress-induced MHC molecules by intestinal epithelial gammadelta T cells. , 1998, Science.

[9]  A. Hayday,et al.  Conservation of T cell receptor conformation in epidermal gammadelta cells with disrupted primary Vgamma gene usage. , 1998, Science.

[10]  R. Steinman,et al.  Intraepidermal lymphocytes in psoriatic lesions are activated GMP‐17(TIA‐1)+CD8+CD3+ CTLs as determined by phenotypic analysis , 1998, Journal of cutaneous pathology.

[11]  P. Stastny,et al.  MICA, a new polymorphic HLA-related antigen, is expressed mainly by keratinocytes, endothelial cells, and monocytes , 1997, Immunogenetics.

[12]  Y. Ullmann,et al.  T-lymphocyte dependence of psoriatic pathology in human psoriatic skin grafted to SCID mice. , 1997, The Journal of investigative dermatology.

[13]  J. Browne,et al.  Identification of a major susceptibility locus on chromosome 6p and evidence for further disease loci revealed by a two stage genome-wide search in psoriasis. , 1997, Human molecular genetics.

[14]  M. Detmar,et al.  Animal models of psoriasis , 1997, Nature Medicine.

[15]  R. Biassoni,et al.  Major histocompatibility complex class I‐specific receptors on human natural killer and T lymphocytes , 1997, Immunological reviews.

[16]  C. Janeway,et al.  Innate immunity: impact on the adaptive immune response. , 1997, Current opinion in immunology.

[17]  B. Nickoloff,et al.  Dermal injection of immunocytes induces psoriasis. , 1996, The Journal of clinical investigation.

[18]  John A Todd,et al.  Genetic Analysis of Autoimmune Disease , 1996, Cell.

[19]  T. Zollner,et al.  Pulling the trigger on psoriasis , 1996, Nature.

[20]  Eric O Long,et al.  Inhibitory MHC class I receptors on NK and T cells: a standard nomenclature. , 1996, Immunology today.

[21]  A. Gottlieb,et al.  Successful ultraviolet B treatment of psoriasis is accompanied by a reversal of keratinocyte pathology and by selective depletion of intraepidermal T cells , 1995, The Journal of experimental medicine.

[22]  D. Carlo,et al.  CD8+ T‐Cells in Psoriatic Lesions Preferentially Use T‐Cell Receptors Vβ3 and/or Vβ13.1 Genes , 1995 .

[23]  L. Lanier,et al.  Development of human T and natural killer cells. , 1995, Blood.

[24]  James G. Krueger,et al.  Response of psoriasis to a lymphocyte-selective toxin (DAB389IL-2) suggests a primary immune, but not keratinocyte, pathogenic basis , 1995, Nature Genetics.

[25]  J. Watson,et al.  Analysis of the intra-epithelial lymphocyte compartment in SCID mice that received co-isogenic CD4+ T cells. Evidence that mature post-thymic CD4+ T cells can be induced to express CD8 alpha in vivo. , 1995, Journal of immunology.

[26]  M. Burdick,et al.  Severe combined immunodeficiency mouse and human psoriatic skin chimeras. Validation of a new animal model. , 1995, The American journal of pathology.

[27]  I. Jónsdóttir,et al.  Psoriasis: a T-cell-mediated autoimmune disease induced by streptococcal superantigens? , 1995, Immunology today.

[28]  L. Steinman,et al.  Escape from “horror autotoxicus”: Pathogenesis and treatment of autoimmune disease , 1995, Cell.

[29]  W. Havran,et al.  Modulation of epithelial cell growth by intraepithelial gamma delta T cells. , 1994, Science.

[30]  D. Carlo,et al.  CD8+ T cells in psoriatic lesions preferentially use T-cell receptor V beta 3 and/or V beta 13.1 genes. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[31]  D. Geraghty,et al.  A second lineage of mammalian major histocompatibility complex class I genes. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[32]  H. Auchincloss,et al.  Indirect recognition by helper cells can induce donor-specific cytotoxic T lymphocytes in vivo , 1994, The Journal of experimental medicine.

[33]  James T. Elder,et al.  The genetics of psoriasis. , 1994, Journal of the American Academy of Dermatology.

[34]  D. Chaplin,et al.  Identification in the HLA class I region of a gene expressed late in keratinocyte differentiation. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[35]  S. Akazaki,et al.  Specific nucleotide sequence of HLA-C is strongly associated with psoriasis vulgaris. , 1991, The Journal of investigative dermatology.

[36]  B. Nickoloff,et al.  The cytokine network in psoriasis. , 1991, Archives of dermatology.

[37]  K. L. Johnson,et al.  Enrichment and function of urushiol (poison ivy)-specific T lymphocytes in lesions of allergic contact dermatitis to urushiol. , 1990, Journal of immunology.

[38]  A. Gottlieb,et al.  HLA region genes and immune activation in the pathogenesis of psoriasis. , 1990, Archives of dermatology.

[39]  R. Tigelaar,et al.  Roles of CD4+ and CD8+ T cells in murine contact sensitivity revealed by in vivo monoclonal antibody depletion. , 1990, Journal of immunology.

[40]  G. Trinchieri,et al.  Biology of Natural Killer Cells , 1989, Advances in Immunology.

[41]  B. Goldstein Psoriasis: Proceedings of the Fourth International Symposium , 1988 .

[42]  E. Engleman,et al.  Evidence for an association between CD8 molecules and the T cell receptor complex on cytotoxic T cells. , 1987, Journal of immunology.

[43]  P. Biberfeld,et al.  Subpopulations of mononuclear cells in microscopic lesions of psoriatic patients. Selective accumulation of suppressor/cytotoxic T cells in epidermis during the evolution of the lesion. , 1984, The Journal of investigative dermatology.

[44]  M. Viander,et al.  Basic and interferon-augmented natural killer (NK) cell activity in psoriasis. , 1983, Acta dermato-venereologica.

[45]  B. Dallapiccola,et al.  Searching for psoriasis susceptibility genes in Italy: genome scan and evidence for a new locus on chromosome 1. , 1999, The Journal of investigative dermatology.

[46]  A. Balsari,et al.  Flow cytometric analysis of activation markers on stimulated T cells and their correlation with cell proliferation. , 1997, Cytometry.

[47]  R. S. Rogers Genetics of psoriasis. , 1975, Minnesota medicine.