Transcription factor Zbtb38 downregulates the expression of anti-inflammatory IL1r2 in mouse model of rheumatoid arthritis.
暂无分享,去创建一个
T. Glant | T. Rauch | Gyula Hoffmann | V. Tubak | D. Tóth | T. Ocskó | Vilmos Tubak
[1] H. Heng,et al. Transient and stable vector transfection: Pitfalls, off-target effects, artifacts. , 2017, Mutation research.
[2] L. Poellinger,et al. A Molecular Mechanism To Switch the Aryl Hydrocarbon Receptor from a Transcription Factor to an E3 Ubiquitin Ligase , 2017, Molecular and Cellular Biology.
[3] Tommy W. Terooatea,et al. Cell-specific Kaiso (ZBTB33) Regulation of Cell Cycle through Cyclin D1 and Cyclin E1* , 2016, The Journal of Biological Chemistry.
[4] Richard D Emes,et al. Genome-wide profiling in treatment-naive early rheumatoid arthritis reveals DNA methylome changes in T and B lymphocytes. , 2016, Epigenomics.
[5] K. Deane. Rheumatoid arthritis: Autoantibodies, citrullinated histones and initiation of synovitis , 2015, Nature Reviews Rheumatology.
[6] C. Gabay,et al. The interleukin (IL)-1 cytokine family--Balance between agonists and antagonists in inflammatory diseases. , 2015, Cytokine.
[7] Seong‐Su Han,et al. SUMOylation-disrupting WAS mutation converts WASp from a transcriptional activator to a repressor of NF-κB response genes in T cells. , 2015, Blood.
[8] O. Elemento,et al. DNA Methylation Dynamics of Germinal Center B Cells Are Mediated by AID. , 2015, Cell reports.
[9] Ronald P. Schuyler,et al. Whole-genome fingerprint of the DNA methylome during human B cell differentiation , 2015, Nature Genetics.
[10] Y. Iwakura,et al. IL-1 Receptor Type 2 Suppresses Collagen-Induced Arthritis by Inhibiting IL-1 Signal on Macrophages , 2015, The Journal of Immunology.
[11] G. Dressler,et al. The Groucho-associated Phosphatase PPM1B Displaces Pax Transactivation Domain Interacting Protein (PTIP) to Switch the Transcription Factor Pax2 from a Transcriptional Activator to a Repressor* , 2015, The Journal of Biological Chemistry.
[12] K. Bock. Homeostatic control of xeno- and endobiotics in the drug-metabolizing enzyme system. , 2014, Biochemical pharmacology.
[13] B. Stockinger,et al. The aryl hydrocarbon receptor: multitasking in the immune system. , 2014, Annual review of immunology.
[14] C. Garlanda,et al. Negative regulatory receptors of the IL-1 family. , 2013, Seminars in immunology.
[15] Kyster K. Nanan,et al. The POZ-ZF Transcription Factor Kaiso (ZBTB33) Induces Inflammation and Progenitor Cell Differentiation in the Murine Intestine , 2013, PloS one.
[16] Gyula Hoffmann,et al. Differentially expressed epigenome modifiers, including aurora kinases A and B, in immune cells in rheumatoid arthritis in humans and mouse models. , 2013, Arthritis and rheumatism.
[17] P. Farnham,et al. ZBTB33 binds unmethylated regions of the genome associated with actively expressed genes , 2013, Epigenetics & Chromatin.
[18] E. Park,et al. Interferon Gamma Suppresses Collagen-Induced Arthritis by Regulation of Th17 through the Induction of Indoleamine-2,3-Deoxygenase , 2013, PloS one.
[19] T. Tedder,et al. IL-10-producing regulatory B cells (B10 cells) in autoimmune disease , 2013, Arthritis Research & Therapy.
[20] G. Firestein,et al. Regulation of DNA Methylation in Rheumatoid Arthritis Synoviocytes , 2013, The Journal of Immunology.
[21] Wei Wang,et al. DNA methylome signature in rheumatoid arthritis , 2012, Annals of the rheumatic diseases.
[22] Sung-Uk Lee,et al. POK/ZBTB proteins: an emerging family of proteins that regulate lymphoid development and function , 2012, Immunological reviews.
[23] Georg Schett,et al. The pathogenesis of rheumatoid arthritis. , 2011, The New England journal of medicine.
[24] Chuan He,et al. Tet Proteins Can Convert 5-Methylcytosine to 5-Formylcytosine and 5-Carboxylcytosine , 2011, Science.
[25] T. Glant,et al. Proteoglycan-induced arthritis and recombinant human proteoglycan aggrecan G1 domain-induced arthritis in BALB/c mice resembling two subtypes of rheumatoid arthritis. , 2011, Arthritis and rheumatism.
[26] T. Rauch,et al. DNA methylation profiling using the methylated-CpG island recovery assay (MIRA). , 2010, Methods.
[27] J. O'dell,et al. State-of-the-art: rheumatoid arthritis , 2010, Annals of the rheumatic diseases.
[28] Xi Liu,et al. Structural insights into the assembly and activation of IL-1β with its receptors , 2010, Nature Immunology.
[29] P. Defossez,et al. Sequence-specific recognition of methylated DNA by human zinc-finger proteins , 2010, Nucleic acids research.
[30] C. Gabay,et al. IL-1 pathways in inflammation and human diseases , 2010, Nature Reviews Rheumatology.
[31] Guillaume J. Filion,et al. A Family of Human Zinc Finger Proteins That Bind Methylated DNA and Repress Transcription , 2006, Molecular and Cellular Biology.
[32] Robert Brown,et al. DNA methyltransferase inhibitors and the development of epigenetic cancer therapies. , 2005, Journal of the National Cancer Institute.
[33] T. Rauch,et al. Methylated-CpG island recovery assay: a new technique for the rapid detection of methylated-CpG islands in cancer , 2005, Laboratory Investigation.
[34] H. Joller-jemelka,et al. Glucose-induced β cell production of IL-1β contributes to glucotoxicity in human pancreatic islets. , 2002, The Journal of clinical investigation.
[35] A. Garcia-Gonzalez,et al. Interleukin 1beta (IL-1beta), IL-10, tumor necrosis factor-alpha, and cellular proliferation index in peripheral blood mononuclear cells in patients with ankylosing spondylitis. , 2002, The Journal of rheumatology.
[36] V. Bardwell,et al. The BCL-6 POZ domain and other POZ domains interact with the co-repressors N-CoR and SMRT , 1998, Oncogene.
[37] M. Privalsky,et al. Components of the SMRT Corepressor Complex Exhibit Distinctive Interactions with the POZ Domain Oncoproteins PLZF, PLZF-RARα, and BCL-6* , 1998, The Journal of Biological Chemistry.
[38] T. Glant,et al. Progressive polyarthritis induced in BALB/c mice by aggrecan from normal and osteoarthritic human cartilage. , 1998, Arthritis and rheumatism.
[39] P. Young,et al. Soluble type II interleukin 1 (IL-1) receptor binds and blocks processing of IL-1 beta precursor and loses affinity for IL-1 receptor antagonist. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[40] J. Dayer,et al. Inhibition of the production and effects of interleukins‐1 and tumor necrosis factor α in rheumatoid arthritis , 1995 .
[41] S. Dower,et al. Binding of IL-1 alpha, IL-1 beta, and IL-1 receptor antagonist by soluble IL-1 receptors and levels of soluble IL-1 receptors in synovial fluids. , 1994, Journal of immunology.
[42] G. Privé,et al. The BTB domain, found primarily in zinc finger proteins, defines an evolutionarily conserved family that includes several developmentally regulated genes in Drosophila. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[43] A. Silman,et al. Twin concordance rates for rheumatoid arthritis: results from a nationwide study. , 1993, British journal of rheumatology.
[44] S Hanash,et al. Evidence for impaired T cell DNA methylation in systemic lupus erythematosus and rheumatoid arthritis. , 1990, Arthritis and rheumatism.
[45] D H Sachs,et al. Establishment and characterization of BALB/c lymphoma lines with B cell properties. , 1979, Journal of immunology.
[46] Chris Cotsapas,et al. Immune-mediated disease genetics: the shared basis of pathogenesis. , 2013, Trends in immunology.
[47] T. Glant,et al. Proteoglycan aggrecan-induced arthritis: a murine autoimmune model of rheumatoid arthritis. , 2004, Methods in molecular medicine.
[48] H. Joller-jemelka,et al. Glucose-induced beta cell production of IL-1beta contributes to glucotoxicity in human pancreatic islets. , 2002, The Journal of clinical investigation.