Neuroprotective effects of the free radical scavenger Edaravone (MCI-186) in mice permanent focal brain ischemia
暂无分享,去创建一个
S. Kuroda | M. Iwai | M. Horiuchi | H. Shichinohe | Y. Iwasaki | T. Ishikawa | H. Yasuda
[1] R. Chen,et al. Possible Inhibition of Focal Cerebral Ischemia by Angiotensin II Type 2 Receptor Stimulation , 2004, Circulation.
[2] Mamoru Tamura,et al. In vivo tracking of bone marrow stromal cells transplanted into mice cerebral infarct by fluorescence optical imaging. , 2004, Brain research. Brain research protocols.
[3] S. Kuroda,et al. FK506 reduces infarct volume due to permanent focal cerebral ischemia by maintaining BAD turnover and inhibiting cytochrome c release , 2004, Brain Research.
[4] H. Utsumi,et al. Application of in vivo ESR spectroscopy to measurement of cerebrovascular ROS generation in stroke. , 2003 .
[5] E. Wei,et al. Neuroprotective effect of ONO-1078, a leukotriene receptor antagonist, on transient global cerebral ischemia in rats. , 2003, Acta pharmacologica Sinica.
[6] V. Vallyathan,et al. Hydroxyl radical formation is greater in striatal core than in penumbra in a rat model of ischemic stroke , 2003, Journal of neuroscience research.
[7] D. DeLong,et al. Effect of a Novel Free Radical Scavenger, Edaravone (MCI-186), on Acute Brain Infarction , 2003, Cerebrovascular Diseases.
[8] L. Iacovitti,et al. Antioxidant compounds protect dopamine neurons from death due to oxidative stress in vitro , 2002, Brain Research.
[9] J. Wilcox,et al. Upregulation of Nox‐Based NAD(P)H Oxidases in Restenosis After Carotid Injury , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[10] P. Chan. Reactive Oxygen Radicals in Signaling and Damage in the Ischemic Brain , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[11] A. Majid,et al. Differences in Vulnerability to Permanent Focal Cerebral Ischemia Among 3 Common Mouse Strains , 2000, Stroke.
[12] K. Fung,et al. MCI-186: further histochemical and biochemical evidence of neuroprotection. , 2000, Life sciences.
[13] M. Moskowitz,et al. Pathobiology of ischaemic stroke: an integrated view , 1999, Trends in Neurosciences.
[14] F. Barone,et al. Inflammatory Mediators and Stroke: New Opportunities for Novel Therapeutics , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[15] M. Niwa,et al. Involvement of free radicals in cerebral vascular reperfusion injury evaluated in a transient focal cerebral ischemia model of rat. , 1999, Free radical biology & medicine.
[16] Y. Ikeda,et al. Neuroprotective effects depend on the model of focal ischemia following middle cerebral artery occlusion. , 1998, European journal of pharmacology.
[17] B. Davidson,et al. Superoxide production in vascular smooth muscle contributes to oxidative stress and impaired relaxation in atherosclerosis. , 1998, Circulation research.
[18] M. Nakashima,et al. Inhibitory effect of MCI-186, a free radical scavenger, on cerebral ischemia following rat middle cerebral artery occlusion. , 1998, General pharmacology.
[19] Toshiaki Watanabe,et al. Delayed neuronal death prevented by inhibition of increased hydroxyl radical formation in a transient cerebral ischemia , 1997, Brain Research.
[20] T. Watanabe,et al. Effects of a novel free radical scavenger, MCl-186, on ischemic brain damage in the rat distal middle cerebral artery occlusion model. , 1997, The Journal of pharmacology and experimental therapeutics.
[21] R. Busto,et al. Journal of Cerebral Blood Flow and Metabolism Simultaneous Measurement of Salicylate Hydroxylation and Glutamate Release in the Penumbral Cortex following Transient Middle Cerebral Artery Occlusion in Rats , 2022 .
[22] K. Kogure,et al. Role of Neutrophils in Radical Production during Ischemia and Reperfusion of the Rat Brain: Effect of Neutrophil Depletion on Extracellular Ascorbyl Radical Formation , 1995, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[23] T. Watanabe,et al. Protective effects of MCI-186 on cerebral ischemia: possible involvement of free radical scavenging and antioxidant actions. , 1994, The Journal of pharmacology and experimental therapeutics.
[24] Barry Halliwell,et al. Reactive Oxygen Species and the Central Nervous System , 1992, Journal of neurochemistry.
[25] B. Siesjö. Pathophysiology and treatment of focal cerebral ischemia. Part II: Mechanisms of damage and treatment. , 1992, Journal of neurosurgery.
[26] R A Swanson,et al. A Semiautomated Method for Measuring Brain Infarct Volume , 1990, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[27] T. Watanabe,et al. Effect of MCI-186 on brain edema in rats. , 1989, Stroke.
[28] I. Morita,et al. Preventive effect of MCI-186 on 15-HPETE induced vascular endothelial cell injury in vitro. , 1988, Prostaglandins, leukotrienes, and essential fatty acids.
[29] K. Abe,et al. Strong attenuation of ischemic and postischemic brain edema in rats by a novel free radical scavenger. , 1988, Stroke.
[30] L. Pitts,et al. Evaluation of 2,3,5-triphenyltetrazolium chloride as a stain for detection and quantification of experimental cerebral infarction in rats. , 1986, Stroke.
[31] L. Pitts,et al. Rat middle cerebral artery occlusion: evaluation of the model and development of a neurologic examination. , 1986, Stroke.
[32] S. Kuroda,et al. Reperfusion damage following focal ischemia: pathophysiology and therapeutic windows. , 1997, Clinical neuroscience.
[33] R. Floyd,et al. Free radical damage to protein and DNA: Mechanisms involved and relevant observations on brain undergoing oxidative stress , 1992, Annals of neurology.
[34] C. Agardh,et al. Free radicals and brain damage. , 1989, Cerebrovascular and brain metabolism reviews.