From Agouti to Pomc—100 years of fat blonde mice

[1]  L. Yaswen,et al.  Obesity in the mouse model of pro-opiomelanocortin deficiency responds to peripheral melanocortin , 1999, Nature Medicine.

[2]  J. Wikberg,et al.  Differential influence of a selective melanocortin MC4 receptor antagonist (HS014) on melanocortin-induced behavioral effects in rats. , 1998, European journal of pharmacology.

[3]  R. Adan,et al.  Melanocortins and cardiovascular regulation. , 1998, European journal of pharmacology.

[4]  K. Clément,et al.  A frameshift mutation in human MC4R is associated with a dominant form of obesity , 1998, Nature Genetics.

[5]  A. Grüters,et al.  Severe early-onset obesity, adrenal insufficiency and red hair pigmentation caused by POMC mutations in humans , 1998, Nature Genetics.

[6]  G. Barsh,et al.  Interaction of Agouti protein with the melanocortin 1 receptor in vitro and in vivo. , 1998, Genes & development.

[7]  Ximena Opitz-Araya,et al.  Exocrine Gland Dysfunction in MC5-R-Deficient Mice: Evidence for Coordinated Regulation of Exocrine Gland Function by Melanocortin Peptides , 1997, Cell.

[8]  J. Wilkinson,et al.  The role of the agouti gene in the yellow obese syndrome. , 1997, The Journal of nutrition.

[9]  R. Cone,et al.  Targeted Disruption of the Melanocortin-4 Receptor Results in Obesity in Mice , 1997, Cell.

[10]  J. B. Tatro Receptor biology of the melanocortins, a family of neuroimmunomodulatory peptides. , 1996, Neuroimmunomodulation.

[11]  A. Aguzzi,et al.  Targeted disruption of the glucocorticoid receptor gene blocks adrenergic chromaffin cell development and severely retards lung maturation. , 1995, Genes & development.

[12]  L. Muglia,et al.  Corticotropin-releasing hormone deficiency reveals major fetal but not adult glucocorticoid need , 1995, Nature.

[13]  D. I. Våge,et al.  The melanocortin receptors: agonists, antagonists, and the hormonal control of pigmentation. , 1996, Recent progress in hormone research.

[14]  J. Toppari,et al.  Adrenocorticotropin receptor gene mutations in familial glucocorticoid deficiency: relationships with clinical features in four families. , 1995, The Journal of clinical endocrinology and metabolism.