COMT genotype predicts cortical-limbic D1 receptor availability measured with [11C]NNC112 and PET
暂无分享,去创建一个
B. Kolachana | D. Weinberger | M. Laruelle | A. Abi-Dargham | M. Slifstein | E. Simpson | P. Tabares | B. Cheng | M. Duvall | W. Frankle | D. Weinberger | Eleanor H. Simpson
[1] S S Stensaas,et al. Autoradiographic Evidence of [3H]SCH 23390 Binding Site; in Human Prefrontal Cortex (Brodmann's Area 9) , 1987, Journal of neurochemistry.
[2] G. Vauquelin,et al. Evidence for a widespread dopaminergic innervation of the human cerebral neocortex , 1989, Neuroscience Letters.
[3] P. Goldman-Rakic,et al. Autoradiographic comparison of D1-specific binding of [3H]SCH39166 and SCH23390 in the primate cerebral cortex , 1990, Brain Research.
[4] J. Mazziotta,et al. MRI‐PET Registration with Automated Algorithm , 1993, Journal of computer assisted tomography.
[5] Christer Halldin,et al. Distribution of D1- and D2-Dopamine Receptors, and Dopamine and Its Metabolites in the Human Brain , 1994, Neuropsychopharmacology.
[6] D. Law-Tho,et al. Dopamine modulation of synaptic transmission in rat prefrontal cortex: an in vitro electrophysiological study , 1994, Neuroscience Research.
[7] R. Weinshilboum,et al. Human catechol-O-methyltransferase pharmacogenetics: description of a functional polymorphism and its potential application to neuropsychiatric disorders. , 1996, Pharmacogenetics.
[8] CR Yang,et al. Dopamine D1 receptor actions in layers V-VI rat prefrontal cortex neurons in vitro: modulation of dendritic-somatic signal integration , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[9] M. Owen,et al. No evidence for allelic association between schizophrenia and a polymorphism determining high or low catechol O-methyltransferase activity. , 1996, The American journal of psychiatry.
[10] R. Murray,et al. Preferential transmission of the high activity allele of COMT in schizophrenia , 1996, Psychiatric genetics.
[11] D. Pfaff,et al. Catechol-O-methyltransferase-deficient mice exhibit sexually dimorphic changes in catecholamine levels and behavior. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[12] Allan I. Levey,et al. Dopamine Axon Varicosities in the Prelimbic Division of the Rat Prefrontal Cortex Exhibit Sparse Immunoreactivity for the Dopamine Transporter , 1998, The Journal of Neuroscience.
[13] T. Terao,et al. Association study of a functional catechol-O-methyltransferase gene polymorphism in Japanese schizophrenics , 1998, Neuroscience Letters.
[14] J Sandell,et al. Carbon-11-NNC 112: a radioligand for PET examination of striatal and neocortical D1-dopamine receptors. , 1998, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[15] J. Seamans,et al. Developing a Neuronal Model for the Pathophysiology of Schizophrenia Based on the Nature of Electrophysiological Actions of Dopamine in the Prefrontal Cortex , 1999, Neuropsychopharmacology.
[16] G. Hemmings,et al. Lack of evidence for association between the COMT locus and schizophrenia. , 1999, Psychiatric genetics.
[17] R. Roth,et al. Altered frontal cortical dopaminergic transmission in monkeys after subchronic phencyclidine exposure: involvement in frontostriatal cognitive deficits , 1999, Neuroscience.
[18] Christer Halldin,et al. Measurement of Striatal and Extrastriatal Dopamine D1 Receptor Binding Potential With [11C]NNC 112 in Humans: Validation and Reproducibility , 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[19] R. Straub,et al. Effect of COMT Val108/158 Met genotype on frontal lobe function and risk for schizophrenia , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[20] D. Durstewitz,et al. Bidirectional Dopamine Modulation of GABAergic Inhibition in Prefrontal Cortical Pyramidal Neurons , 2001, The Journal of Neuroscience.
[21] H. Herken,et al. Catechol‐O ‐methyltransferase gene polymorphism in schizophrenia: evidence for association between symptomatology and prognosis , 2001, Psychiatric genetics.
[22] A. Sampson,et al. Dopamine transporter immunoreactivity in monkey cerebral cortex: Regional, laminar, and ultrastructural localization , 2001, The Journal of comparative neurology.
[23] Osama Mawlawi,et al. Imaging Human Mesolimbic Dopamine Transmission with Positron Emission Tomography: I. Accuracy and Precision of D2 Receptor Parameter Measurements in Ventral Striatum , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[24] J. Mallet,et al. No evidence for linkage between COMT and schizophrenia in a French population , 2001, Psychiatry Research.
[25] R. V. Van Heertum,et al. Prefrontal Dopamine D1 Receptors and Working Memory in Schizophrenia , 2002, The Journal of Neuroscience.
[26] Marc Laruelle,et al. Dopamine Depletion and In Vivo Binding of PET D1 Receptor Radioligands: Implications for Imaging Studies in Schizophrenia , 2003, Neuropsychopharmacology.
[27] R. Coppola,et al. Executive subprocesses in working memory: relationship to catechol-O-methyltransferase Val158Met genotype and schizophrenia. , 2003, Archives of general psychiatry.
[28] Makoto Tsunoda,et al. High-performance liquid chromatography-fluorescent assay of catechol-O-methyltransferase activity in rat brain , 2003, Analytical and bioanalytical chemistry.
[29] B. Lipska,et al. Catechol O-methyltransferase mRNA expression in human and rat brain: evidence for a role in cortical neuronal function , 2003, Neuroscience.
[30] Paul J. Harrison,et al. Catechol-O-Methyltransferase Inhibition Improves Set-Shifting Performance and Elevates Stimulated Dopamine Release in the Rat Prefrontal Cortex , 2004, The Journal of Neuroscience.
[31] M. Egan,et al. Functional analysis of genetic variation in catechol-O-methyltransferase (COMT): effects on mRNA, protein, and enzyme activity in postmortem human brain. , 2004, American journal of human genetics.
[32] M. Egan,et al. Effect of Catechol-O-Methyltransferase val158met Genotype on Attentional Control , 2005, The Journal of Neuroscience.
[33] Hideo Tsukada,et al. Chronic NMDA Antagonism Impairs Working Memory, Decreases Extracellular Dopamine, and Increases D1 Receptor Binding in Prefrontal Cortex of Conscious Monkeys , 2005, Neuropsychopharmacology.
[34] Mark Slifstein,et al. Altered prefrontal dopaminergic function in chronic recreational ketamine users. , 2005, The American journal of psychiatry.
[35] Lin He,et al. Catechol-O-methyltransferase gene Val/Met functional polymorphism and risk of schizophrenia: A large-scale association study plus meta-analysis , 2005, Biological Psychiatry.
[36] Thomas E. Nichols,et al. Impact of complex genetic variation in COMT on human brain function , 2006, Molecular Psychiatry.
[37] M. Laruelle,et al. [11C]NNC 112 Selectivity for Dopamine D1 and Serotonin 5-HT2A Receptors: A PET Study in Healthy Human Subjects , 2007, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[38] D. Weinberger,et al. COMT Val158Met polymorphism predicts negative symptoms response to treatment with olanzapine in schizophrenia , 2007, Schizophrenia Research.
[39] R. P. Maguire,et al. Consensus Nomenclature for in vivo Imaging of Reversibly Binding Radioligands , 2007, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[40] Margit Burmeister,et al. SNPs on Chips: The Hidden Genetic Code in Expression Arrays , 2007, Biological Psychiatry.
[41] P. Buckley,et al. Catechol O-methyltransferase Val158Met Genotype and Neural Mechanisms Related to Affective Arousal and Regulation , 2008 .