Perinatal Asphyxia Induced Neuronal Loss by Apoptosis in the Neonatal Rat Striatum: A Combined TUNEL and Stereological Study
暂无分享,去创建一个
[1] J. Shaffer. Multiple Hypothesis Testing , 1995 .
[2] D. Ferriero,et al. Delayed Neurodegeneration in Neonatal Rat Thalamus after Hypoxia–Ischemia Is Apoptosis , 2001, The Journal of Neuroscience.
[3] M. Johnston,et al. Apoptosis Has a Prolonged Role in the Neurodegeneration after Hypoxic Ischemia in the Newborn Rat , 2000, The Journal of Neuroscience.
[4] C. Schmitz,et al. Perinatal asphyxia results in changes in presynaptic bouton number in striatum and cerebral cortex—a stereological and behavioral analysis , 2000, Journal of Chemical Neuroanatomy.
[5] K. Blomgren,et al. Involvement of Caspase-3 in Cell Death after Hypoxia–Ischemia Declines during Brain Maturation , 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[6] A. Brambrink,et al. Neuronal Death in Newborn Striatum after Hypoxia-Ischemia Is Necrosis and Evolves with Oxidative Stress , 2000, Neurobiology of Disease.
[7] C. Schmitz. Towards the use of state-of-the-art stereology in experimental gerontology , 2000, Experimental Gerontology.
[8] Yves Garnier,et al. Pathophysiology of perinatal brain damage , 1999, Brain Research Reviews.
[9] W. Hatton,et al. Analysis of cell death in the trochlear nucleus of the chick embryo: Calibration of the optical disector counting method reveals systematic bias , 1999, The Journal of comparative neurology.
[10] G. Sutherland,et al. Electron Microscopic Evidence against Apoptosis as the Mechanism of Neuronal Death in Global Ischemia , 1999, The Journal of Neuroscience.
[11] H. Heinsen,et al. Nerve cell loss in the thalamic mediodorsal nucleus in Huntington’s disease , 1999, Acta Neuropathologica.
[12] C. Schmitz,et al. Neuron loss in the mouse hippocampus following prenatal injection of tritiated thymidine or saline , 1999, International Journal of Developmental Neuroscience.
[13] B. Huppertz,et al. The apoptosis cascade — morphological and immunohistochemical methods for its visualization , 1999, Anatomy and Embryology.
[14] H. Hoeger,et al. Histological changes and neurotransmitter levels three months following perinatal asphyxia in the rat. , 1999, Life sciences.
[15] R. Murray,et al. Prenatal and perinatal risk factors for schizophrenia, affective psychosis, and reactive psychosis of early onset: case-control study. , 1999, BMJ.
[16] M. Delivoria-Papadopoulos,et al. Cellular mechanisms of hypoxic injury in the developing brain , 1999, Brain Research Bulletin.
[17] H. Mehmet,et al. Oxidative Metabolism, Apoptosis and Perinatal Brain Injury , 1999, Brain pathology.
[18] B. Siesjö,et al. Is the Cell Death Pathway Triggered by the Mitochondrion or the Endoplasmic Reticulum? , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[19] G. Cole,et al. Apoptosis in a neonatal rat model of cerebral hypoxia-ischemia. , 1998, Stroke.
[20] E M Glaser,et al. The coefficient of error of optical fractionator population size estimates: a computer simulation comparing three estimators , 1998, Journal of microscopy.
[21] C. Schmitz. Variation of fractionator estimates and its prediction , 1998, Anatomy and Embryology.
[22] C. Vaillancourt,et al. Caesarean section birth with general anesthesia increases dopamine‐mediated behavior in the adult rat , 1998, Neuroreport.
[23] J. Towfighi,et al. Temporal evolution of neuronal changes in cerebral hypoxia– ischemia in developing rats: a quantitative light microscopic study , 1998 .
[24] J. Weiner,et al. Programmed cell death is a universal feature of embryonic and postnatal neuroproliferative regions throughout the central nervous system , 1998, The Journal of comparative neurology.
[25] S. Nagata,et al. correction: A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD , 1998, Nature.
[26] J. Vente,et al. Hypothermia during or after severe perinatal asphyxia prevents increase in cyclic GMP-related nitric oxide levels in the newborn rat striatum , 1998, Brain Research.
[27] M. Delivoria-Papadopoulos,et al. Mechanisms of cerebral injury in perinatal asphyxia and strategies for prevention. , 1998, The Journal of pediatrics.
[28] S. Nagata,et al. A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD , 1998, Nature.
[29] M. Goiny,et al. Short- and long-term effects of perinatal asphyxia on monoamine, amino acid and glycolysis product levels measured in the basal ganglia of the rat. , 1997, Brain research. Developmental brain research.
[30] V E Koliatsos,et al. Deafferentation Causes Apoptosis in Cortical Sensory Neurons in the Adult Rat , 1997, The Journal of Neuroscience.
[31] R W Guillery,et al. Quantification without pontification: Choosing a method for counting objects in sectioned tissues , 1997, The Journal of comparative neurology.
[32] P. Nicotera,et al. The shape of cell death. , 1997, Biochemical and biophysical research communications.
[33] G. Lubec,et al. Delayed neuronal death following perinatal asphyxia in rat , 1997, Experimental Brain Research.
[34] C. Portera-Cailliau,et al. Non‐NMDA and NMDA receptor‐mediated excitotoxic neuronal deaths in adult brain are morphologically distinct: Further evidence for an apoptosis‐necrosis continuum , 1997, The Journal of comparative neurology.
[35] C. Portera-Cailliau,et al. Excitotoxic neuronal death in the immature brain is an apoptosis‐necrosis morphological continuum , 1997, The Journal of comparative neurology.
[36] B. Siesjö,et al. Calcium-related damage in ischemia. , 1996, Life sciences.
[37] M. Molinari,et al. Development of GABA and calcium binding proteins immunoreactivity in the rat hippocampus following neonatal anoxia , 1996, Neuroscience Letters.
[38] C. Nyakas,et al. Hypoxia and brain development , 1996, Progress in Neurobiology.
[39] B. Siesjö,et al. Mechanisms of secondary brain injury. , 1996, European journal of anaesthesiology.
[40] Y. Ben-Ari,et al. A cautionary note on the use of the TUNEL stain to determine apoptosis , 1995, Neuroreport.
[41] R. Burke,et al. Apoptosis in substantia nigra following developmental hypoxic-ischemic injury , 1995, Neuroscience.
[42] J. Gross,et al. Nicotine treatment counteracts perinatal asphyxia-induced changes in the mesostriatal/limbic dopamine systems and in motor behaviour in the four-week-old male rat , 1995, Neuroscience.
[43] Y. Itoyama,et al. Ischemic delayed neuronal death. A mitochondrial hypothesis. , 1995, Stroke.
[44] M E Greenberg,et al. Calcium signaling in neurons: molecular mechanisms and cellular consequences. , 1995, Science.
[45] P. Gluckman,et al. Repeated asphyxia causes loss of striatal projection neurons in the fetal sheep brain , 1995, Neuroscience.
[46] P. Gluckman,et al. Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal loss. , 1995, Brain research. Molecular brain research.
[47] U. Ungerstedt,et al. Long-term effects of perinatal asphyxia on basal ganglia neurotransmitter systems studied with microdialysis in rat , 1994, Neuroscience Letters.
[48] B. Barres,et al. Programmed cell death and the control of cell survival: lessons from the nervous system. , 1993, Science.
[49] D. Vaux. Toward an understanding of the molecular mechanisms of physiological cell death. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[50] S. Ben‐Sasson,et al. Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation , 1992, The Journal of cell biology.
[51] M. Raff,et al. Social controls on cell survival and cell death , 1992, Nature.
[52] L. David Tomei,et al. Apoptosis: The Molecular Basis of Cell Death , 1991 .
[53] Michael V. Johnston,et al. Physiological and pathophysiological roles of excitatory amino acids during central nervous system development , 1990, Brain Research Reviews.
[54] R. Vannucci. Experimental Biology of Cerebral Hypoxia-Ischemia: Relation to Perinatal Brain Damage , 1990, Pediatric Research.
[55] R. Burke,et al. Quantitative morphological analysis of striatal cholinergic neurons in perinatal asphyxia , 1990, Annals of neurology.
[56] F. Meyer. Calcium, neuronal hyperexcitability and ischemic injury , 1989, Brain Research Reviews.
[57] M. Johnston,et al. Neurotoxicity ofN-methyl-d-aspartate is markedly enhanced in developing rat central nervous system , 1988, Brain Research.
[58] H. Gundersen. Stereology of arbitrary particles * , 1986, Journal of microscopy.
[59] M. Johnston. Neurotransmitter alterations in a model of perinatal hypoxic‐ischemic brain injury , 1983, Annals of neurology.
[60] M. Raichle. The pathophysiology of brain ischemia , 1983, Annals of neurology.
[61] F. Cockburn,et al. Neurological Aspects of Perinatal Asphyxia , 1974, Developmental medicine and child neurology.
[62] A. Wyllie,et al. Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics , 1972, British Journal of Cancer.
[63] P. Clarke,et al. Developmental cell death: morphological diversity and multiple mechanisms , 2004, Anatomy and Embryology.
[64] M. Lipowska,et al. Postnatal development of the rat striatum--a study using in situ DNA end labeling technique. , 1998, Acta neurobiologiae experimentalis.
[65] F. Capani,et al. Long term changes in NADPH-diaphorase reactivity in striatal and cortical neurons following experimental perinatal asphyxia: neuroprotective effects of hypothermia. , 1997, The International journal of neuroscience.
[66] G. Majno,et al. Apoptosis, oncosis, and necrosis. An overview of cell death. , 1995, The American journal of pathology.
[67] J. Kerr. Definition and incidence of apoptosis : A historical perspective , 1991 .
[68] R. Oppenheim. Cell death during development of the nervous system. , 1991, Annual review of neuroscience.
[69] A. Wyllie,et al. Cell death: the significance of apoptosis. , 1980, International review of cytology.