A case of lysogenic conversion: modification of cell phenotype by constitutive expression of the Mu gem operon.

[1]  Phillip A. Sharp,et al.  6 – Regulation of Transcription , 1991 .

[2]  V. Orlando,et al.  Synchronous division induced in Escherichia coli K12 by phage Mu: analysis of DNA topology and gene expression during the cell cycle. , 1991, Research in microbiology.

[3]  D. Fruci,et al.  Global changes in gene expression in Escherichia coli K12 induced by bacteriophage Mu Gem protein. , 1991, Research in microbiology.

[4]  G. Di Lallo,et al.  The bacteriophage Mu gem gene: a positive regulator of the C operon required for normal levels of late transcription. , 1990, Virology.

[5]  K. Mathee,et al.  Identification of a positive regulator of the Mu middle operon , 1990, Journal of bacteriology.

[6]  Jon Beckwith,et al.  A bacterial virulence determinant encoded by lysogenic coliphage λ , 1990 .

[7]  I. Ruberti,et al.  Mu gem3 as a tool to investigate the influence of chromosome supercoiling on gene expression in Escherichia coli K12 , 1990 .

[8]  A. M. Pedrini,et al.  The expression of the DNA ligase gene of Escherichia coli is stimulated by relaxation of chromosomal supercoiling , 1989, Molecular microbiology.

[9]  H. Krause,et al.  Supercoiling and integration host factor change the DNA conformation and alter the flow of convergent transcription in phage Mu. , 1989, The Journal of biological chemistry.

[10]  D. Galas Bacterial insertion sequences. , 1989 .

[11]  P. Manning,et al.  Genetics of Vibrio cholerae and its bacteriophages. , 1987, Microbiological reviews.

[12]  P. Monaci,et al.  Analysis of the killing effect of Mu ligts mutants on host cells , 1987 .

[13]  D. Coleman,et al.  Cloning and expression in Escherichia coli and Staphylococcus aureus of the beta-lysin determinant from Staphylococcus aureus: evidence that bacteriophage conversion of beta-lysin activity is caused by insertional inactivation of the beta-lysin determinant. , 1986, Microbial pathogenesis.

[14]  W. Ross,et al.  Mutants of Escherichia coli defective for replicative transposition of bacteriophage Mu , 1986, Journal of bacteriology.

[15]  P. Schlievert,et al.  Bacteriophage involvement in group A streptococcal pyrogenic exotoxin A production , 1986, Journal of bacteriology.

[16]  L. Paolozzi,et al.  General method for the isolation of conditional lethal mutants in any required region of the virus genome: its application to the 'semi-essential' region of phage Mu. , 1986, Journal of General Microbiology.

[17]  K. Mizuuchi,et al.  A defined system for the DNA strand-transfer reaction at the initiation of bacteriophage Mu transposition: protein and DNA substrate requirements. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[18]  J. Gralla,et al.  Supercoiling response of the lac ps promoter in vitro. , 1985, Journal of molecular biology.

[19]  R. Plasterk,et al.  Genetic switches by DNA inversions in prokaryotes. , 1984, Biochimica et biophysica acta.

[20]  A. M. Pedrini,et al.  Escherichia coli K-12 gyrB gene product is involved in the lethal effect of the ligts2 mutant of bacteriophage Mu , 1984, Journal of bacteriology.

[21]  T. Bickle,et al.  Bacteriophage survival: multiple mechanisms for avoiding the deoxyribonucleic acid restriction systems of their hosts. , 1983, Microbiological reviews.

[22]  A. Toussaint,et al.  CHAPTER 3 – Phage Mu: Transposition as a Life-Style , 1983 .

[23]  A. M. Pedrini,et al.  The topoisomerase activity of T4 amG39 mutant is restored in Mu lysogens , 1982, FEBS letters.

[24]  L. McKane,et al.  Phage-host interactions and the production of type A streptococcal exotoxin in group A streptococci , 1981, Infection and immunity.

[25]  R. Reed Transposon-mediated site-specific recombination: A defined in vitro system , 1981, Cell.

[26]  A. Campbell Evolutionary significance of accessory DNA elements in bacteria. , 1981, Annual review of microbiology.

[27]  L. Paolozzi,et al.  Two classes of Mu lig mutants: the thermosensitives for integration and replication and the hyperproducers for ligase. , 1980, Nucleic acids research.

[28]  L. Paolozzi,et al.  Restoration of ligase activity in E. coli K12 lig ts7 strain by bacteriophage Mu and cloning of a DNA fragment harbouring the Mu 'lig' gene. , 1980, Nucleic Acids Research.

[29]  P. Schlievert,et al.  Transfer of group A streptococcal pyrogenic exotoxin production to nontoxigenic strains of lysogenic conversion , 1980, Infection and immunity.

[30]  B. Sanzey,et al.  Modulation of gene expression by drugs affecting deoxyribonucleic acid gyrase , 1979, Journal of bacteriology.

[31]  D. Zipser,et al.  Inversion of the G DNA segment of phage Mu controls phage infectivity , 1978, Nature.

[32]  N. Symonds,et al.  Role of the G segment in the growth of phage Mu , 1978, Nature.

[33]  M. Howe Invertible DNA in phage Mu , 1978, Nature.

[34]  L. Lin,et al.  Reproductive fitness of P1, P2, and Mu lysogens of Escherichia coli , 1977, Journal of virology.

[35]  K. Mizuuchi,et al.  Restriction assay for integrative recombination of bacteriophage lambda DNA in vitro: requirement for closed circular DNA substrate. , 1976, Proceedings of the National Academy of Sciences of the United States of America.

[36]  A. Toussaint The DNA modification function of temperate phage Mu-1. , 1976, Virology.

[37]  R. Kudrna,et al.  λ Lysogens of E. coli reproduce more rapidly than non-lysogens , 1975, Nature.

[38]  L. Barksdale,et al.  Persisting bacteriophage infections, lysogeny, and phage conversions. , 1974, Annual review of microbiology.

[39]  K. Inoue,et al.  Conversion of toxigenicity in Clostridium botulinum type C. , 1970, Japanese journal of microbiology.

[40]  B. Zegers,et al.  Lysogenic Conversion of Staphylococci to Loss of β-Toxin. , 1965 .

[41]  L. Barksdale I. : Lysogenic Conversions in Bacteria. , 1959, Bacteriological reviews.

[42]  A. M. Pappenheimer,et al.  PHAGE-HOST RELATIONSHIPS IN NONTOXIGENIC AND TOXIGENIC DIPHTHERIA BACILLI , 1954, Journal of bacteriology.

[43]  V. J. Freeman,et al.  FURTHER OBSERVATIONS ON THE CHANGE TO VIRULENCE OF BACTERIOPHAGE-INFECTED AVIRULENT STRAINS OF CORYNEBACTERIUM DIPHTHERIAE , 1952, Journal of bacteriology.

[44]  V. J. Freeman,et al.  STUDIES ON THE VIRULENCE OF BACTERIOPHAGE-INFECTED STRAINS OF CORYNEBACTERIUM DIPHTHERIAE , 1951, Journal of bacteriology.