Heme oxygenase-1 gene promoter polymorphism is associated with abdominal aortic aneurysm.

[1]  O. Wagner,et al.  Heme Oxygenase-1 Genotype is a Vascular Anti-inflammatory Factor following Balloon Angioplasty , 2002, Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists.

[2]  O. Wagner,et al.  Heme Oxygenase-1 Gene Promoter Microsatellite Polymorphism is Associated with Restenosis after Percutaneous Transluminal Angioplasty , 2001, Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists.

[3]  M. Suematsu,et al.  Increased Expression of Heme Oxygenase-1 and Bilirubin Accumulation in Foam Cells of Rabbit Atherosclerotic Lesions , 2001, Arteriosclerosis, thrombosis, and vascular biology.

[4]  D. Rader,et al.  Genetic susceptibility to atherosclerosis: insights from mice. , 2000, Circulation research.

[5]  A. Choi,et al.  Carbon monoxide has anti-inflammatory effects involving the mitogen-activated protein kinase pathway , 2000, Nature Medicine.

[6]  M. Suematsu,et al.  Protective roles of endogenous carbon monoxide in neointimal development elicited by arterial injury. , 2000, American journal of physiology. Heart and circulatory physiology.

[7]  P. Libby,et al.  Plasma concentrations of interleukin-6 and abdominal aortic diameter among subjects without aortic dilatation. , 1999, Arteriosclerosis, thrombosis, and vascular biology.

[8]  G. Assmann,et al.  Strategies for the assessment of genetic coronary artery disease risk. , 1999, Current opinion in lipidology.

[9]  G. Sicard,et al.  Indomethacin inhibits expansion of experimental aortic aneurysms via inhibition of the cox2 isoform of cyclooxygenase. , 1999, Journal of vascular surgery.

[10]  G. Mann,et al.  Heme oxygenase-carbon monoxide signalling pathway in atherosclerosis: anti-atherogenic actions of bilirubin and carbon monoxide? , 1999, Cardiovascular research.

[11]  L. Golub,et al.  Pharmacologic suppression of experimental abdominal aortic aneurysms: acomparison of doxycycline and four chemically modified tetracyclines. , 1998, Journal of vascular surgery.

[12]  B. Baxter,et al.  Matrix metalloproteinase-2 production and its binding to the matrix are increased in abdominal aortic aneurysms. , 1998, Arteriosclerosis, thrombosis, and vascular biology.

[13]  B. Baxter,et al.  FORMATION of aneurysms. , 1998, Lancet.

[14]  R. Sormunen,et al.  Chronic inflammation and elastin degradation in abdominal aortic aneurysm disease: an immunohistochemical and electron microscopic study. , 1998, European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery.

[15]  Tzong-Shyuan Lee,et al.  Expression of heme oxygenase-1 in atherosclerotic lesions. , 1998, The American journal of pathology.

[16]  S. Kourembanas,et al.  Carbon Monoxide Controls the Proliferation of Hypoxic Vascular Smooth Muscle Cells* , 1997, The Journal of Biological Chemistry.

[17]  T. Rasmussen,et al.  New insights into inflammatory abdominal aortic aneurysms. , 1997, European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery.

[18]  M. Leinonen,et al.  Elevated circulating levels of inflammatory cytokines in patients with abdominal aortic aneurysm. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[19]  A. Lusis,et al.  Induction of heme oxygenase-1 inhibits the monocyte transmigration induced by mildly oxidized LDL. , 1997, The Journal of clinical investigation.

[20]  Y. Itoyama,et al.  Microsatellite polymorphism in the human heme oxygenase-1 gene promoter and its application in association studies with Alzheimer and Parkinson disease , 1997, Human Genetics.

[21]  B. Baxter,et al.  Pathogenesis of Abdominal Aortic Aneurysm: An Update and Look toward the Future , 1997, Cardiovascular surgery.

[22]  K. Takahashi,et al.  Regulation of human heme oxygenase-1 gene expression under thermal stress. , 1996, Blood.

[23]  K. Fung,et al.  Antioxidation of human low density lipoprotein by unconjugated and conjugated bilirubins. , 1996, Biochemical pharmacology.

[24]  S. Kourembanas,et al.  Endothelial cell expression of vasoconstrictors and growth factors is regulated by smooth muscle cell-derived carbon monoxide. , 1995, The Journal of clinical investigation.

[25]  P. Edwards,et al.  Atherosclerosis: basic mechanisms. Oxidation, inflammation, and genetics. , 1995, Circulation.

[26]  R. Stocker,et al.  Free and albumin-bound bilirubin are efficient co-antioxidants for alpha-tocopherol, inhibiting plasma and low density lipoprotein lipid peroxidation. , 1994, The Journal of biological chemistry.

[27]  R. Ross The pathogenesis of atherosclerosis: a perspective for the 1990s , 1993, Nature.

[28]  M. David Tilson,et al.  Suggested standards for reporting on arterial aneurysms , 1991 .

[29]  L. Naylor,et al.  d(TG)n.d(CA)n sequences upstream of the rat prolactin gene form Z-DNA and inhibit gene transcription. , 1990, Nucleic acids research.

[30]  A. Sterpetti,et al.  Inflammatory aneurysms of the abdominal aorta: incidence, pathologic, and etiologic considerations. , 1989, Journal of vascular surgery.

[31]  M. Maines Heme oxygenase: function, multiplicity, regulatory mechanisms, and clinical applications , 1988, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[32]  P. Pairolero,et al.  Inflammatory abdominal aortic aneurysms: a thirty-year review. , 1985, Journal of vascular surgery.

[33]  H. Marver,et al.  Microsomal heme oxygenase. Characterization of the enzyme. , 1969, The Journal of biological chemistry.

[34]  S. Shibahara,et al.  Microsatellite polymorphism in the heme oxygenase-1 gene promoter is associated with susceptibility to emphysema. , 2000, American journal of human genetics.

[35]  D. Carey,et al.  The matrix metalloproteinase inhibitor BB-94 limits expansion of experimental abdominal aortic aneurysms. , 1999, Journal of vascular surgery.

[36]  Y. Niida,et al.  Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency. , 1999, The Journal of clinical investigation.

[37]  D. Willis,et al.  Heme oxygenase: A novel target for the modulation of inflammatory response , 1996, Nature Medicine.