Modulation of LSD1 phosphorylation by CK2/WIP1 regulates RNF168-dependent 53BP1 recruitment in response to DNA damage
暂无分享,去创建一个
Ji Liao | Jing Wang | Xingzhi Xu | Xingzhi Xu | Hailong Wang | Bin Peng | Yuan Hu | Hongli Zhao | Wenya Hou | Hongchang Zhao | Hongchang Zhao | Wenya Hou | J. Liao | Jing Wang | Bin Peng | Hailong Wang | Yuan Hu | Hongli Zhao
[1] Facundo D. Batista,et al. RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection , 2013, Molecular cell.
[2] Chi Wang,et al. The deubiquitinase USP28 stabilizes LSD1 and confers stem-cell-like traits to breast cancer cells. , 2013, Cell reports.
[3] K. Zhao,et al. PTIP Promotes Chromatin Changes Critical for Immunoglobulin Class Switch Recombination , 2010, Science.
[4] F. Forneris,et al. Human Histone Demethylase LSD1 Reads the Histone Code* , 2005, Journal of Biological Chemistry.
[5] J. Ellenberg,et al. RNF168 Binds and Amplifies Ubiquitin Conjugates on Damaged Chromosomes to Allow Accumulation of Repair Proteins , 2009, Cell.
[6] S. Gygi,et al. The histone demethylase LSD1/KDM1A promotes the DNA damage response , 2013, The Journal of cell biology.
[7] S. Ogawa,et al. The genomic landscape of nasopharyngeal carcinoma , 2014, Nature Genetics.
[8] M. Nussenzweig,et al. Regulation of DNA end joining, resection, and immunoglobulin class switch recombination by 53BP1. , 2011, Molecular cell.
[9] S. Jackson,et al. Human HDAC1 and HDAC2 function in the DNA-damage response to promote DNA nonhomologous end-joining , 2010, Nature Structural &Molecular Biology.
[10] Jie Cheng,et al. High expression of the histone demethylase LSD1 associates with cancer cell proliferation and unfavorable prognosis in tongue cancer. , 2015, Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology.
[11] Stéphanie Panier,et al. RNF168 ubiquitylates 53BP1 and controls its response to DNA double-strand breaks , 2013, Proceedings of the National Academy of Sciences.
[12] F. Lan,et al. Regulation of LSD1 histone demethylase activity by its associated factors. , 2005, Molecular cell.
[13] M. Fiscella,et al. Wip1, a novel human protein phosphatase that is induced in response to ionizing radiation in a p53-dependent manner. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[14] Yang Shi,et al. Histone Demethylation Mediated by the Nuclear Amine Oxidase Homolog LSD1 , 2004, Cell.
[15] D. Seldin,et al. Globozoospermia in mice lacking the casein kinase II alpha' catalytic subunit. , 1999, Nature genetics.
[16] E. Krebs,et al. Isolation and characterization of human cDNA clones encoding the alpha and the alpha' subunits of casein kinase II. , 1990, Biochemistry.
[17] Edward S. Miller,et al. RIDDLE immunodeficiency syndrome is linked to defects in 53BP1-mediated DNA damage signaling , 2007, Proceedings of the National Academy of Sciences.
[18] D. Chan,et al. The Protein Kinase CK2 Facilitates Repair of Chromosomal DNA Single-Strand Breaks , 2004, Cell.
[19] Makoto Nakanishi,et al. Response to DNA damage: why do we need to focus on protein phosphatases? , 2013, Front. Oncol..
[20] Stéphanie Panier,et al. Double-strand break repair: 53BP1 comes into focus , 2013, Nature Reviews Molecular Cell Biology.
[21] Liewei Wang,et al. MMSET regulates histone H4K20 methylation and 53BP1 accumulation at DNA damage sites , 2010, Nature.
[22] Benjamin P. C. Chen,et al. Protein kinase CK2 localizes to sites of DNA double-strand break regulating the cellular response to DNA damage , 2012, BMC Molecular Biology.
[23] David J. Chen,et al. PTIP associates with Artemis to dictate DNA repair pathway choice , 2014, Genes & development.
[24] A. Jeyasekharan,et al. LSD1 Overexpression Is Associated with Poor Prognosis in Basal-Like Breast Cancer, and Sensitivity to PARP Inhibition , 2015, PloS one.
[25] D. Stern,et al. Chk2 Activation and Phosphorylation-Dependent Oligomerization , 2002, Molecular and Cellular Biology.
[26] A. El-Osta,et al. Evaluation of the spatial distribution of gammaH2AX following ionizing radiation. , 2010, Journal of visualized experiments : JoVE.
[27] Xin Wang,et al. PTIP promotes DNA double‐strand break repair through homologous recombination , 2010, Genes to cells : devoted to molecular & cellular mechanisms.
[28] Kuniyoshi Iwabuchi,et al. Protein kinase CK2 is required for the recruitment of 53BP1 to sites of DNA double-strand break induced by radiomimetic drugs. , 2014, Cancer letters.
[29] Edward S. Miller,et al. The RIDDLE Syndrome Protein Mediates a Ubiquitin-Dependent Signaling Cascade at Sites of DNA Damage , 2009, Cell.
[30] Xingzhi Xu,et al. LSD1 is required for chromosome segregation during mitosis. , 2010, European journal of cell biology.
[31] Sebastian A. Wagner,et al. A Proteome-wide, Quantitative Survey of In Vivo Ubiquitylation Sites Reveals Widespread Regulatory Roles* , 2011, Molecular & Cellular Proteomics.
[32] Frédérick A. Mallette,et al. RNF8‐ and RNF168‐dependent degradation of KDM4A/JMJD2A triggers 53BP1 recruitment to DNA damage sites , 2012, The EMBO journal.
[33] Yong Chen,et al. Crystal structure of human histone lysine-specific demethylase 1 (LSD1) , 2006, Proceedings of the National Academy of Sciences.
[34] S. Elledge,et al. The DNA damage response: making it safe to play with knives. , 2010, Molecular cell.
[35] L. Donehower,et al. PPM1D dephosphorylates Chk1 and p53 and abrogates cell cycle checkpoints. , 2005, Genes & development.
[36] Michel C. Nussenzweig,et al. Rif1 Prevents Resection of DNA Breaks and Promotes Immunoglobulin Class Switching , 2013, Science.
[37] C. Mizzen,et al. Histone H4 deacetylation facilitates 53BP1 DNA damage signaling and double-strand break repair. , 2013, Journal of molecular cell biology.
[38] Wen-guang Wu,et al. Overexpression of lysine specific demethylase 1 predicts worse prognosis in primary hepatocellular carcinoma patients. , 2012, World journal of gastroenterology.
[39] Min Zhu,et al. HERC2/USP20 coordinates CHK1 activation by modulating CLASPIN stability , 2014, Nucleic acids research.
[40] R. Medema,et al. Wip1 phosphatase is associated with chromatin and dephosphorylates γH2AX to promote checkpoint inhibition , 2010, Oncogene.
[41] P. Glazer,et al. Silencing of the DNA mismatch repair gene MLH1 induced by hypoxic stress in a pathway dependent on the histone demethylase LSD1. , 2014, Cell reports.
[42] Steven J. M. Jones,et al. Comprehensive molecular characterization of human colon and rectal cancer , 2012, Nature.
[43] Wim Vermeulen,et al. RNF168 Ubiquitinates K13-15 on H2A/H2AX to Drive DNA Damage Signaling , 2012, Cell.
[44] Georges Mer,et al. Structural Basis for the Methylation State-Specific Recognition of Histone H4-K20 by 53BP1 and Crb2 in DNA Repair , 2006, Cell.
[45] Xingzhi Xu,et al. NFBD1/KIAA0170 Is a Chromatin-associated Protein Involved in DNA Damage Signaling Pathways* , 2003, The Journal of Biological Chemistry.
[46] N. Batada,et al. SET8 methyltransferase activity during the DNA double-strand break response is required for recruitment of 53BP1 , 2014, EMBO reports.
[47] N. Mosammaparast,et al. Crosstalk between ubiquitin and other post-translational modifications on chromatin during double-strand break repair. , 2014, Trends in cell biology.
[48] M. Montenarh. Protein kinase CK2 and angiogenesis. , 2014, Advances in clinical and experimental medicine : official organ Wroclaw Medical University.
[49] M. Montenarh. Protein Kinase CK 2 and Angiogenesis , 2014 .
[50] G. Hong,et al. Nucleic Acids Research , 2015, Nucleic Acids Research.
[51] Yolanda F. Darlington,et al. Wild-type p53-induced Phosphatase 1 Dephosphorylates Histone Variant γ-H2AX and Suppresses DNA Double Strand Break Repair* , 2010, The Journal of Biological Chemistry.
[52] J. I. Izpisúa Belmonte,et al. The lysine-specific demethylase 1 is a novel substrate of protein kinase CK2. , 2014, Biochimica et biophysica acta.
[53] N. Dantuma,et al. The AAA-ATPase VCP/p97 promotes 53BP1 recruitment by removing L3MBTL1 from DNA double-strand breaks , 2011, Nature Structural &Molecular Biology.
[54] N. Onishi,et al. Intrinsic Kinase Activity and SQ/TQ Domain of Chk2 Kinase as Well as N-terminal Domain of Wip1 Phosphatase Are Required for Regulation of Chk2 by Wip1* , 2006, Journal of Biological Chemistry.
[55] Benjamin J. Raphael,et al. Integrated Genomic Analyses of Ovarian Carcinoma , 2011, Nature.
[56] Edward L. Huttlin,et al. Systematic and quantitative assessment of the ubiquitin-modified proteome. , 2011, Molecular cell.
[57] M. Yaffe,et al. MDC1 Directly Binds Phosphorylated Histone H2AX to Regulate Cellular Responses to DNA Double-Strand Breaks , 2008, Cell.
[58] D. Durocher,et al. 53BP1 is a reader of the DNA damage-induced H2A Lys15 ubiquitin mark , 2013, Nature.
[59] N. Mailand,et al. Phosphorylation of SDT repeats in the MDC1 N terminus triggers retention of NBS1 at the DNA damage–modified chromatin , 2008, The Journal of cell biology.
[60] E. Appella,et al. Wip1 phosphatase modulates ATM-dependent signaling pathways. , 2006, Molecular cell.