Advanced glycation end products induce production of reactive oxygen species via the activation of NADPH oxidase in murine hepatic stellate cells.

[1]  I. G. Fantus,et al.  High glucose activates PKC-zeta and NADPH oxidase through autocrine TGF-beta1 signaling in mesangial cells. , 2008, American journal of physiology. Renal physiology.

[2]  M. Cooper,et al.  Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product–Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C-α–Dependent Pathway , 2008, Diabetes.

[3]  Y. Azuma,et al.  Glycated human serum albumin enhances macrophage inflammatory protein-1beta mRNA expression through protein kinase C-delta and NADPH oxidase in macrophage-like differentiated U937 cells. , 2008, Biochimica et biophysica acta.

[4]  K. Chayama,et al.  Advanced glycation end products enhance the proliferation and activation of hepatic stellate cells , 2008, Journal of Gastroenterology.

[5]  S. Soltoff,et al.  Rottlerin: an inappropriate and ineffective inhibitor of PKCdelta. , 2007, Trends in pharmacological sciences.

[6]  K. Chayama,et al.  Elevated levels of serum advanced glycation end products in patients with non‐alcoholic steatohepatitis , 2007, Journal of gastroenterology and hepatology.

[7]  D. Brenner,et al.  NOX in liver fibrosis. , 2007, Archives of biochemistry and biophysics.

[8]  K. Griendling,et al.  Nox1-based NADPH oxidase-derived superoxide is required for VSMC activation by advanced glycation end-products. , 2007, Free radical biology & medicine.

[9]  M. Nitti,et al.  PKC delta and NADPH oxidase in AGE-induced neuronal death , 2007, Neuroscience Letters.

[10]  H. Schmidt,et al.  The 'A's and 'O's of NADPH oxidase regulation: a commentary on "Subcellular localization and function of alternatively spliced Noxo1 isoforms". , 2007, Free radical biology & medicine.

[11]  N. Nieto.  Oxidative‐stress and IL‐6 mediate the fibrogenic effects of rodent Kupffer cells on stellate cells , 2006, Hepatology.

[12]  J. Schmitt,et al.  Induction of reactive oxygen species and cell survival in the presence of advanced glycation end products and similar structures. , 2006, Biochimica et biophysica acta.

[13]  J. Emond,et al.  Blockade of the receptor for advanced glycation end products attenuates acetaminophen‐induced hepatotoxicity in mice , 2006, Journal of gastroenterology and hepatology.

[14]  M. Manns,et al.  Elevation of Nepsilon-(carboxymethyl)lysine-modified advanced glycation end products in chronic liver disease is an indicator of liver cirrhosis. , 2006, Clinical biochemistry.

[15]  S. Vannucci,et al.  Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation. , 2005, Glycobiology.

[16]  M. Morales,et al.  Advanced glycation and endothelial functions: a link towards vascular complications in diabetes. , 2004, Life sciences.

[17]  R. de Caterina,et al.  Advanced glycation end products and vascular inflammation: implications for accelerated atherosclerosis in diabetes. , 2004, Cardiovascular research.

[18]  Yi Zheng,et al.  Rational design and characterization of a Rac GTPase-specific small molecule inhibitor. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[19]  G. Sonnenberg,et al.  A novel pathway to the manifestations of metabolic syndrome. , 2004, Obesity research.

[20]  T. Hughes,et al.  Advanced glycation end product ligands for the receptor for advanced glycation end products: biochemical characterization and formation kinetics. , 2004, Analytical biochemistry.

[21]  P. Vignais.  The superoxide-generating NADPH oxidase: structural aspects and activation mechanism , 2002, Cellular and Molecular Life Sciences CMLS.

[22]  M. Kuwano,et al.  Reactive oxygen species accelerate production of vascular endothelial growth factor by advanced glycation end products in RAW264.7 mouse macrophages. , 2002, Free radical biology & medicine.

[23]  A. Heidland,et al.  Markedly elevated levels of plasma advanced glycation end products in patients with liver cirrhosis - amelioration by liver transplantation. , 2002, Journal of hepatology.

[24]  A. Gressner,et al.  Up‐regulated expression of the receptor for advanced glycation end products in cultured rat hepatic stellate cells during transdifferentiation to myofibroblasts , 2001, Hepatology.

[25]  Jinshun Zhao,et al.  Oxidized low‐density lipoproteins bind to the scavenger receptor, CD36, of hepatic stellate cells and stimulate extracellular matrix synthesis , 2001, Hepatology.

[26]  A Geerts,et al.  History, Heterogeneity, Developmental Biology, and Functions of Quiescent Hepatic Stellate Cells , 2001, Seminars in liver disease.

[27]  A. Schmidt,et al.  Activation of NADPH oxidase by AGE links oxidant stress to altered gene expression via RAGE. , 2001, American journal of physiology. Endocrinology and metabolism.

[28]  A. Barden,et al.  Advanced Glycation End Products: A Review , 2013 .

[29]  J. Tavaré,et al.  The protein kinase C inhibitors bisindolylmaleimide I (GF 109203x) and IX (Ro 31‐8220) are potent inhibitors of glycogen synthase kinase‐3 activity , 1999, FEBS letters.

[30]  H. Sano,et al.  Advanced glycation end products are eliminated by scavenger-receptor-mediated endocytosis in hepatic sinusoidal Kupffer and endothelial cells. , 1997, The Biochemical journal.

[31]  H. Coste,et al.  The bisindolylmaleimide GF 109203X is a potent and selective inhibitor of protein kinase C. , 1991, The Journal of biological chemistry.

[32]  A. Habeeb,et al.  Determination of free amino groups in proteins by trinitrobenzenesulfonic acid. , 1966, Analytical biochemistry.