Critical role of IL-33, but not IL-25 or TSLP, in silica crystal-mediated exacerbation of allergic airway eosinophilia.

[1]  Jungil Choi,et al.  Silica dioxide nanoparticles aggravate airway inflammation in an asthmatic mouse model via NLRP3 inflammasome activation. , 2020, Regulatory toxicology and pharmacology : RTP.

[2]  L. Mollet,et al.  STING-dependent sensing of self-DNA drives silica-induced lung inflammation , 2018, Nature Communications.

[3]  S. Nakae,et al.  IL-33, IL-25 and TSLP contribute to development of fungal-associated protease-induced innate-type airway inflammation , 2018, Scientific Reports.

[4]  T. Abe,et al.  IL‐25 enhances TH17 cell–mediated contact dermatitis by promoting IL‐1&bgr; production by dermal dendritic cells , 2018, The Journal of allergy and clinical immunology.

[5]  S. Koyasu,et al.  Innate lymphoid cells in allergic and nonallergic inflammation. , 2016, The Journal of allergy and clinical immunology.

[6]  S. Nakae,et al.  IL-25 and IL-33 Contribute to Development of Eosinophilic Airway Inflammation in Epicutaneously Antigen-Sensitized Mice , 2015, PloS one.

[7]  C. Akdis,et al.  An Interleukin-33-Mast Cell-Interleukin-2 Axis Suppresses Papain-Induced Allergic Inflammation by Promoting Regulatory T Cell Numbers. , 2015, Immunity.

[8]  S. Nakae,et al.  Silica and double-stranded RNA synergistically induce bronchial epithelial apoptosis and airway inflammation. , 2014, American journal of respiratory cell and molecular biology.

[9]  M. Gold,et al.  Group 2 Innate Lymphoid Cells Are Critical for the Initiation of Adaptive T Helper 2 Cell-Mediated Allergic Lung Inflammation , 2014, Immunity.

[10]  S. Ziegler,et al.  Chitin activates parallel immune modules that direct distinct inflammatory responses via innate lymphoid type 2 and γδ T cells. , 2014, Immunity.

[11]  Heinrich Ernst,et al.  Evaluation of immunohistochemical markers to detect the genotoxic mode of action of fine and ultrafine dusts in rat lungs. , 2013, Toxicology.

[12]  K. Ohta,et al.  Epithelial Cell-Derived IL-25, but Not Th17 Cell-Derived IL-17 or IL-17F, Is Crucial for Murine Asthma , 2012, The Journal of Immunology.

[13]  David Torres,et al.  Scavenger Receptors in Human Airway Epithelial Cells: Role in Response to Double-Stranded RNA , 2012, PloS one.

[14]  G. Gudmundsson.  Respiratory health effects of volcanic ash with special reference to Iceland. A review , 2011, The clinical respiratory journal.

[15]  Isao Ito,et al.  Desert dust exposure is associated with increased risk of asthma hospitalization in children. , 2010, American journal of respiratory and critical care medicine.

[16]  T. Abe,et al.  IL-33 is a crucial amplifier of innate rather than acquired immunity , 2010, Proceedings of the National Academy of Sciences.

[17]  Wei Yang,et al.  Acute Bronchitis and Volcanic Air Pollution: A Community-Based Cohort Study at Kilauea Volcano, Hawai`i, USA , 2008, Journal of toxicology and environmental health. Part A.

[18]  T. Shibamoto,et al.  The Effects of Microbial Materials Adhered to Asian Sand Dust on Allergic Lung Inflammation , 2008, Archives of environmental contamination and toxicology.

[19]  R. Hamilton,et al.  Role of Scavenger Receptor A Family in Lung Inflammation from Exposure to Environmental Particles , 2008, Journal of immunotoxicology.

[20]  E. Naumova,et al.  Emergency room visits for respiratory conditions in children increased after Guagua Pichincha volcanic eruptions in April 2000 in Quito, Ecuador Observational Study: Time Series Analysis , 2007, Environmental health : a global access science source.

[21]  S. Galli,et al.  Mast cell-derived TNF contributes to airway hyperreactivity, inflammation, and TH2 cytokine production in an asthma model in mice. , 2007, The Journal of allergy and clinical immunology.

[22]  F. Martinez,et al.  Genes, environments, development and asthma: a reappraisal , 2006, European Respiratory Journal.

[23]  T. Shibamoto,et al.  Enhancement of Mite Allergen-Induced Eosinophil Infiltration in the Murine Airway and Local Cytokine/Chemokine Expression by Asian Sand Dust , 2006, Journal of toxicology and environmental health. Part A.

[24]  H. D. Liggitt,et al.  Thymic stromal lymphopoietin as a key initiator of allergic airway inflammation in mice , 2005, Nature Immunology.

[25]  W. Leonard,et al.  A role for TSLP in the development of inflammation in an asthma model , 2005, The Journal of experimental medicine.

[26]  Min Zhang,et al.  IgE-Dependent Mast Cell Activation Potentiates Airway Responses in Murine Asthma Models1 , 2002, The Journal of Immunology.

[27]  F. Sundler,et al.  Allergic Eosinophil-rich Inflammation Develops in Lungs and Airways of B Cell–deficient Mice , 1997, The Journal of experimental medicine.

[28]  S. Nakae,et al.  Development of IL-17-mediated delayed-type hypersensitivity is not affected by down-regulation of IL-25 expression. , 2010, Allergology international : official journal of the Japanese Society of Allergology.

[29]  A. Cook,et al.  Health effects of natural dust , 2007, Biological Trace Element Research.