Indirect-pathway neurons lose their spines in Parkinson disease
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[1] J. Penney,et al. The functional anatomy of basal ganglia disorders , 1989, Trends in Neurosciences.
[2] C. Gerfen,et al. D1 and D2 dopamine receptor-regulated gene expression of striatonigral and striatopallidal neurons. , 1990, Science.
[3] D. James Surmeier,et al. G-Protein-Coupled Receptor Modulation of Striatal CaV1.3 L-Type Ca Channels Is Dependent on a Shank-Binding Domain , 2005 .
[4] A. Sampson,et al. Selective elimination of glutamatergic synapses on striatopallidal neurons in Parkinson disease models , 2006, Nature Neuroscience.
[5] Thomas H. McNeill,et al. Atrophy of medium spiny I striatal dendrites in advanced Parkinson's disease , 1988, Brain Research.
[6] Shiaoching Gong,et al. A gene expression atlas of the central nervous system based on bacterial artificial chromosomes , 2003, Nature.
[7] G. Arbuthnott,et al. Plasticity of Synapses in the Rat Neostriatum after Unilateral Lesion of the Nigrostriatal Dopaminergic Pathway , 1998, The Journal of Neuroscience.
[8] E. Vaadia,et al. Physiological aspects of information processing in the basal ganglia of normal and parkinsonian primates , 1998, Trends in Neurosciences.
[9] G. Arbuthnott,et al. Spine density on neostriatal neurones changes with 6-hydroxydopamine lesions and with age , 1989, Brain Research.
[10] N. Heintz,et al. Bac to the future: The use of bac transgenic mice for neuroscience research , 2001, Nature Reviews Neuroscience.
[11] Charles J. Wilson,et al. Corticostriatal combinatorics: the implications of corticostriatal axonal arborizations. , 2002, Journal of neurophysiology.
[12] H. Bergman,et al. Reversal of experimental parkinsonism by lesions of the subthalamic nucleus. , 1990, Science.
[13] J. Bargas,et al. D2 Dopamine Receptors in Striatal Medium Spiny Neurons Reduce L-Type Ca2+ Currents and Excitability via a Novel PLCβ1–IP3–Calcineurin-Signaling Cascade , 2000, The Journal of Neuroscience.