Bronchial smooth muscle responses evoked by toluene diisocyanate are inhibited by ruthenium red and by indomethacin.
暂无分享,去创建一个
L. Fabbri | J. Nadel | P. Chitano | C. Mapp | P. Graf | A. Boniotti
[1] C. Maggi,et al. Capsaicin and primary afferent neurons: from basic science to human therapy? , 1991, Journal of the autonomic nervous system.
[2] J. Nadel,et al. Toluene diisocyanate contracts guinea pig bronchial smooth muscle by activating capsaicin-sensitive sensory nerves. , 1991, The Journal of pharmacology and experimental therapeutics.
[3] L. Fabbri,et al. Pharmacological modulation of the contractile response to toluene diisocyanate in the rat isolated urinary bladder , 1990, British journal of pharmacology.
[4] L. Fabbri,et al. Evidence that toluene diisocyanate activates the efferent function of capsaicin-sensitive primary afferents. , 1990, European journal of pharmacology.
[5] A. Dray,et al. Ruthenium red blocks the capsaicin-induced increase in intracellular calcium and activation of membrane currents in sensory neurones as well as the activation of peripheral nociceptors in vitro , 1990, Neuroscience Letters.
[6] C. Maggi,et al. The 'efferent' function of capsaicin-sensitive nerves: ruthenium red discriminates between different mechanisms of activation. , 1989, European journal of pharmacology.
[7] F. Lembeck,et al. Ruthenium red selectively prevents capsaicin-induced nociceptor stimulation. , 1989, European journal of pharmacology.
[8] C. Maggi,et al. The antagonism induced by ruthenium red of the actions of capsaicin on the peripheral terminals of sensory neurons: further studies. , 1988, European journal of pharmacology.
[9] S. Bevan,et al. Capsaicin-induced ion fluxes in dorsal root ganglion cells in culture , 1988, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[10] C. Maggi,et al. Protective action of Ruthenium red toward capsaicin desensitization of sensory fibers , 1988, Neuroscience Letters.
[11] C. Maggi,et al. Prostanoids modulate reflex micturition by acting through capsaicin-sensitive afferents. , 1988, European journal of pharmacology.
[12] D. A. Brown,et al. The mechanism of action of capsaicin on sensory C-type neurons and their axons in vitro , 1987, Neuroscience.
[13] L. Fabbri,et al. Bronchoalveolar neutrophilia during late asthmatic reactions induced by toluene diisocyanate. , 1987, The American review of respiratory disease.
[14] I. Bernstein,et al. Toluene diisocyanate-induced airway hyperreactivity and pathology in the guinea pig. , 1986, The Journal of allergy and clinical immunology.
[15] D. McDonald,et al. Airway hyperresponsiveness and inflammation induced by toluene diisocyanate in guinea pigs. , 1985, The American review of respiratory disease.
[16] M. Fujiwara,et al. Prostaglandins enhance trigeminal substance P-ergic responses in the rabbit iris sphincter muscles , 1985, Brain Research.
[17] R Polato,et al. Time course of the increase in airway responsiveness associated with late asthmatic reactions to toluene diisocyanate in sensitized subjects. , 1985, The Journal of allergy and clinical immunology.
[18] L. Chahl,et al. MODIFICATION BY CAPSAICIN AND COMPOUND 48/80 OF DYE LEAKAGE INDUCED BY IRRITANTS IN THE RAT , 1977, British journal of pharmacology.
[19] M. Holtzman,et al. Arachidonic acid metabolism. Implications of biological chemistry for lung function and disease. , 1991, The American review of respiratory disease.
[20] C. Maggi,et al. The sensory-efferent function of capsaicin-sensitive sensory neurons. , 1988, General pharmacology.