Ventricular dilatation and remodeling after myocardial infarction.

[1]  Peter Gaudron,et al.  Progressive Left Ventricular Dysfunction and Remodeling After Myocardial Infarction Potential Mechanisms and Early Predictors , 1993, Circulation.

[2]  R M Whitlock,et al.  Left ventricular end-systolic volume as the major determinant of survival after recovery from myocardial infarction. , 1987, Circulation.

[3]  Miles Be,et al.  Letter: Serum-phenytoin. , 1976 .

[4]  N. Sharpe,et al.  TREATMENT OF PATIENTS WITH SYMPTOMLESS LEFT VENTRICULAR DYSFUNCTION AFTER MYOCARDIAL INFARCTION , 1988, The Lancet.

[5]  J. B. Garrison,et al.  Regional cardiac dilatation after acute myocardial infarction: recognition by two-dimensional echocardiography. , 1979, The New England journal of medicine.

[6]  B. Healy,et al.  Angiotensin II receptors in normal and failing human hearts. , 1989, The Journal of clinical endocrinology and metabolism.

[7]  R. Jennings,et al.  The Changing Anatomic Reference Base of Evolving Myocardial Infarction: Underestimation of Myocardial Collateral Blood Flow and Overestimation of Experimental Anatomic Infarct Size Due to Tissue Edema, Hemorrhage and Acute Inflammation , 1979, Circulation.

[8]  K. Thygesen,et al.  Effects of Captopril on Ischemia and Dysfunction of the Left Ventricle After Myocardial Infarction , 1993, Circulation.

[9]  G. Gamble,et al.  Early prevention of left ventricular dysfunction after myocardial infarction with angiotensin-converting-enzyme inhibition , 1991, The Lancet.

[10]  S. Yusuf,et al.  Comparison of neuroendocrine activation in patients with left ventricular dysfunction with and without congestive heart failure. A substudy of the Studies of Left Ventricular Dysfunction (SOLVD). , 1990, Circulation.

[11]  B. Pitt,et al.  Effect of enalapril on myocardial infarction and unstable angina in patients with low ejection fractions , 1992, The Lancet.

[12]  M. Laks,et al.  Functional significance of hypertrophy of the noninfarcted myocardium after myocardial infarction in humans. , 1989, Circulation.

[13]  I. Ford,et al.  Effects of early captopril administration on infarct expansion, left ventricular remodeling and exercise capacity after acute myocardial infarction. , 1991, The American journal of cardiology.

[14]  G. Lamas,et al.  Effects of left ventricular shape and captopril therapy on exercise capacity after anterior wall acute myocardial infarction. , 1989, The American journal of cardiology.

[15]  G. Hutchins,et al.  Infarct expansion versus extension: two different complications of acute myocardial infarction. , 1978, The American journal of cardiology.

[16]  G Olivetti,et al.  Side-to-side slippage of myocytes participates in ventricular wall remodeling acutely after myocardial infarction in rats. , 1990, Circulation research.

[17]  M. Packer.  The neurohormonal hypothesis: a theory to explain the mechanism of disease progression in heart failure. , 1992, Journal of the American College of Cardiology.

[18]  J. Rumberger,et al.  Right ventricular dilatation and remodeling the first year after an initial transmural wall left ventricular myocardial infarction. , 1993, The American journal of cardiology.

[19]  D. Bonaduce,et al.  Effects of captopril treatment on left ventricular remodeling and function after anterior myocardial infarction: comparison with digitalis. , 1992, Journal of the American College of Cardiology.

[20]  W Grossman,et al.  Left ventricular remodeling after myocardial infarction: a corollary to infarct expansion. , 1986, Circulation.

[21]  P. Marino,et al.  Effect of streptokinase on left ventricular modeling and function after myocardial infarction: the GISSI (Gruppo Italiano per lo Studio della Streptochinasi nell'Infarto Miocardico) Trial. , 1989, Journal of the American College of Cardiology.

[22]  J. Weiss,et al.  Late effects of acute infarct dilation on heart size: a two dimensional echocardiographic study. , 1982, The American journal of cardiology.

[23]  M. Pfeffer,et al.  Survival after an experimental myocardial infarction: beneficial effects of long-term therapy with captopril. , 1985, Circulation.

[24]  B. Jugdutt,et al.  Functional impact of remodeling therapy with captopril and nitroglycerin during healing after acute myocardial infarction in the dog , 1991 .

[25]  D. Bonaduce,et al.  Usefulness of late coronary thrombolysis (recombinant tissue-type plasminogen activator) in preserving left ventricular function in acute myocardial infarction. , 1990, The American journal of cardiology.

[26]  S. Goldman,et al.  Importance of Venodilatation in Prevention of Left Ventricular Dilatation After Chronic Large Myocardial Infarction in Rats: A Comparison of Captopril and Hydralazine , 1989, Circulation research.

[27]  M. Fishbein,et al.  Compensatory hypertrophy in the heart after myocardial infarction in the rat. , 1983, Journal of the American College of Cardiology.

[28]  E. J. Brown,et al.  Effect of captopril on mortality and morbidity in patients with left ventricular dysfunction after myocardial infarction. Results of the survival and ventricular enlargement trial. The SAVE Investigators. , 1992, The New England journal of medicine.

[29]  B. Bulkley,et al.  Expansion of Transmural Myocardial Infarction A Pathophysiologic Factor in Cardiac Rupture , 1979, Circulation.

[30]  M. Pfeffer,et al.  Influence of Chronic Captopril Therapy on the Infarcted Left Ventricle of the Rat , 1985, Circulation research.

[31]  G. Lamas,et al.  Effect of captopril on progressive ventricular dilatation after anterior myocardial infarction. , 1988, The New England journal of medicine.

[32]  J. Parkinson.  ENLARGEMENT OF THE HEART , 1936 .

[33]  K. Swedberg,et al.  Effects of the early administration of enalapril on mortality in patients with acute myocardial infarction. Results of the Cooperative New Scandinavian Enalapril Survival Study II (CONSENSUS II) , 1992, The New England journal of medicine.

[34]  S. Danilov,et al.  Affinity chromatography and some properties of the angiotensin-converting enzyme from human heart. , 1987, Biochimica et biophysica acta.

[35]  J. Cohn,et al.  Long-term oral nitrate therapy prevents chronic ventricular remodeling in the dog. , 1993, Journal of the American College of Cardiology.

[36]  M. Weisfeldt,et al.  Cellular mechanisms of myocardial infarct expansion. , 1988, Circulation.

[37]  B. Gersh,et al.  Nonparallel changes in global left ventricular chamber volume and muscle mass during the first year after transmural myocardial infarction in humans. , 1993, Journal of the American College of Cardiology.

[38]  V. Dzau.  Implications of local angiotensin production in cardiovascular physiology and pharmacology. , 1987, The American journal of cardiology.

[39]  U. Goldbourt,et al.  Early administration of nifedipine in suspected acute myocardial infarction. The Secondary Prevention Reinfarction Israel Nifedipine Trial 2 Study. , 1993, Archives of internal medicine.

[40]  K. Hammermeister,et al.  Variables predictive of survival in patients with coronary disease. Selection by univariate and multivariate analyses from the clinical, electrocardiographic, exercise, arteriographic, and quantitative angiographic evaluations. , 1979, Circulation.

[41]  B. Jugdutt,et al.  Intravenous nitroglycerin therapy to limit myocardial infarct size, expansion, and complications. Effect of timing, dosage, and infarct location. , 1988, Circulation.

[42]  D. Campbell,et al.  Circulating and tissue angiotensin systems. , 1987, The Journal of clinical investigation.

[43]  B. Gersh,et al.  Relation of left ventricular volume and function over one year after acute myocardial infarction to infarct size determined by technetium-99m sestamibi. , 1991, The American journal of cardiology.

[44]  E. Shapiro,et al.  Nifedipine in acute myocardial infarction: an assessment of left ventricular function, infarct size, and infarct expansion. A double blind, randomised, placebo controlled trial. , 1988, British heart journal.

[45]  G. Diamond,et al.  Early increase in left ventricular compliance after myocardial infarction. , 1972, The Journal of clinical investigation.

[46]  R. Jeremy,et al.  Infarct artery perfusion and changes in left ventricular volume in the month after acute myocardial infarction. , 1987, Journal of the American College of Cardiology.

[47]  G. Lamas,et al.  Left ventricular remodeling in the year after first anterior myocardial infarction: a quantitative analysis of contractile segment lengths and ventricular shape. , 1992, Journal of the American College of Cardiology.

[48]  J. Hochman,et al.  Limitation of myocardial infarct expansion by reperfusion independent of myocardial salvage. , 1987, Circulation.

[49]  B. Bulkley,et al.  Global cardiac remodeling after acute myocardial infarction: a study in the rat model. , 1985, Journal of the American College of Cardiology.

[50]  W. Kannel,et al.  Prognosis after initial myocardial infarction: the Framingham study. , 1979, The American journal of cardiology.

[51]  C. Johnston,et al.  Characterization of cardiac angiotensin converting enzyme (ACE) and in vivo inhibition following oral quinapril to rats , 1990, British journal of pharmacology.

[52]  J. Fleiss,et al.  Risk stratification and survival after myocardial infarction. , 1983, The New England journal of medicine.

[53]  J. Cohn,et al.  Hemodynamic, left ventricular structural and hormonal changes after discrete myocardial damage in the dog. , 1992, Journal of the American College of Cardiology.

[54]  C. Johnston,et al.  CHANGES IN CARDIAC ANGIOTENSIN CONVERTING ENZYME AFTER MYOCARDIAL INFARCTION AND HYPERTROPHY IN RATS , 1991, Clinical and experimental pharmacology & physiology.