Tc1 mouse model of trisomy-21 dissociates properties of short- and long-term recognition memory
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Elizabeth M.C. Fisher | Frances K. Wiseman | M. Good | J. Harwood | V. Tybulewicz | E. Fisher | F. Wiseman | Mark A. Good | John L. Harwood | Jessica H. Hall | Victor L.J. Tybulewicz
[1] Y. Hérault,et al. Epigallocatechin-3-gallate, a DYRK1A inhibitor, rescues cognitive deficits in Down syndrome mouse models and in humans. , 2014, Molecular nutrition & food research.
[2] G. Barker,et al. Recognition Memory for Objects, Place, and Temporal Order: A Disconnection Analysis of the Role of the Medial Prefrontal Cortex and Perirhinal Cortex , 2007, The Journal of Neuroscience.
[3] G. Carlesimo,et al. Visual and spatial long‐term memory: differential pattern of impairments in Williams and Down syndromes , 2005, Developmental medicine and child neurology.
[4] Jonathan T. Brown,et al. Hippocampal circuit dysfunction in the Tc1 mouse model of Down syndrome , 2015, Nature Neuroscience.
[5] R. Stackman,et al. On the delay-dependent involvement of the hippocampus in object recognition memory , 2004, Neurobiology of Learning and Memory.
[6] Mark Blades,et al. Wayfinding behaviour in Down syndrome: a study with virtual environments. , 2013, Research in developmental disabilities.
[7] Y. Hérault,et al. Specific targeting of the GABA-A receptor α5 subtype by a selective inverse agonist restores cognitive deficits in Down syndrome mice , 2011, Journal of psychopharmacology.
[8] Stefano Vicari,et al. Evidence from two genetic syndromes for the independence of spatial and visual working memory , 2006, Developmental medicine and child neurology.
[9] M. Davisson,et al. Segmental trisomy of murine chromosome 16: a new model system for studying Down syndrome. , 1990, Progress in clinical and biological research.
[10] M. Milanese,et al. Fluoxetine in adulthood normalizes GABA release and rescues hippocampal synaptic plasticity and spatial memory in a mouse model of Down Syndrome , 2014, Neurobiology of Disease.
[11] L. Hyde,et al. Reactivity to object and spatial novelty is normal in older Ts65Dn mice that model Down syndrome and Alzheimer’s disease , 2002, Brain Research.
[12] Laura C. Andreae,et al. Preservation of long-term memory and synaptic plasticity despite short-term impairments in the Tc1 mouse model of Down syndrome. , 2008, Learning & memory.
[13] M. Eacott,et al. Impaired object recognition with increasing levels of feature ambiguity in rats with perirhinal cortex lesions , 2004, Behavioural Brain Research.
[14] W. Silverman. Down syndrome: cognitive phenotype. , 2007, Mental retardation and developmental disabilities research reviews.
[15] G. Lynch,et al. The neurobiology of learning and memory , 1989, Cognition.
[16] M. W. Brown,et al. Recognition memory: neuronal substrates of the judgement of prior occurrence , 1998, Progress in Neurobiology.
[17] R S Chapman,et al. Behavioral phenotype of individuals with Down syndrome. , 2000, Mental retardation and developmental disabilities research reviews.
[18] K. Nandagopal,et al. A Study of GluK1 Kainate Receptor Polymorphisms in Down Syndrome Reveals Allelic Non-Disjunction at 1173(C/T) , 2009, Disease markers.
[19] Robert E. Clark,et al. Impaired Recognition Memory in Rats after Damage to the Hippocampus , 2000, The Journal of Neuroscience.
[20] L. Jacobs,et al. Deficits in Cognition and Synaptic Plasticity in a Mouse Model of Down Syndrome Ameliorated by GABAB Receptor Antagonists , 2012, The Journal of Neuroscience.
[21] R. Bartesaghi,et al. APP-dependent alteration of GSK3β activity impairs neurogenesis in the Ts65Dn mouse model of Down syndrome , 2014, Neurobiology of Disease.
[22] W. Mobley,et al. Aging and intellectual disability: insights from mouse models of Down syndrome. , 2013, Developmental disabilities research reviews.
[23] D. Wetmore,et al. Short‐term treatment with the GABAA receptor antagonist pentylenetetrazole produces a sustained pro‐cognitive benefit in a mouse model of Down's syndrome , 2013, British journal of pharmacology.
[24] S. Desai. Down syndrome: a review of the literature. , 1997, Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics.
[25] J. Aggleton,et al. The Different Effects on Recognition Memory of Perirhinal Kainate and NMDA Glutamate Receptor Antagonism: Implications for Underlying Plasticity Mechanisms , 2006, The Journal of Neuroscience.
[26] P. Lavenex,et al. Allocentric spatial learning and memory deficits in Down syndrome , 2015, Front. Psychol..
[27] C. Goodlett,et al. Low dose EGCG treatment beginning in adolescence does not improve cognitive impairment in a Down syndrome mouse model , 2015, Pharmacology Biochemistry and Behavior.
[28] C. Spong,et al. Altered expression of KIF17, a kinesin motor protein associated with NR2B trafficking, may mediate learning deficits in a Down syndrome mouse model. , 2008, American journal of obstetrics and gynecology.
[29] M. Good,et al. The "Swedish" mutation of the amyloid precursor protein (APPswe) dissociates components of object-location memory in aged Tg2576 mice. , 2007, Behavioral neuroscience.
[30] R. Bartesaghi,et al. Inhibition of APP gamma-secretase restores Sonic Hedgehog signaling and neurogenesis in the Ts65Dn mouse model of Down syndrome , 2015, Neurobiology of Disease.
[31] Larry R Squire,et al. Spatial memory, recognition memory, and the hippocampus. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[32] F. Benfenati,et al. Ryanodine Receptor Blockade Reduces Amyloid-β Load and Memory Impairments in Tg2576 Mouse Model of Alzheimer Disease , 2012, The Journal of Neuroscience.
[33] G. Labesse,et al. PROKR2 missense mutations associated with Kallmann syndrome impair receptor signalling activity. , 2009, Human molecular genetics.
[34] A. Granholm,et al. Estrogen restores cognition and cholinergic phenotype in an animal model of Down syndrome , 2002, Physiology & Behavior.
[35] S. Younkin,et al. Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic Mice , 1996, Science.
[36] R. Kesner,et al. Dentate gyrus mediates cognitive function in the Ts65Dn/DnJ mouse model of down syndrome , 2014, Hippocampus.
[37] C Caltagirone,et al. Short-term memory in persons with intellectual disabilities and Down's syndrome. , 1995, Journal of intellectual disability research : JIDR.
[38] E. Fisher,et al. Dissecting Alzheimer disease in Down syndrome using mouse models , 2015, Front. Behav. Neurosci..
[39] G. Barker,et al. When Is the Hippocampus Involved in Recognition Memory? , 2011, The Journal of Neuroscience.
[40] S. Heinemann,et al. Localization of glutamate receptors to distal dendrites depends on subunit composition and the kinesin motor protein KIF17 , 2007, Molecular and Cellular Neuroscience.
[41] C. Epstein,et al. App Gene Dosage Modulates Endosomal Abnormalities of Alzheimer's Disease in a Segmental Trisomy 16 Mouse Model of Down Syndrome , 2003, The Journal of Neuroscience.
[42] C. Masters,et al. Regulation and Expression of the Alzheimer's β/A4 Amyloid Protein Precursor in Health, Disease, and Down's Syndrome a , 1993, Annals of the New York Academy of Sciences.
[43] E. Mufson,et al. Maternal choline supplementation improves spatial mapping and increases basal forebrain cholinergic neuron number and size in aged Ts65Dn mice , 2014, Neurobiology of Disease.
[44] M. Good,et al. Impaired visuospatial recognition memory but normal object novelty detection and relative familiarity judgments in adult mice expressing the APPswe Alzheimer's disease mutation. , 2005, Behavioral neuroscience.
[45] Andrew P Maurer,et al. The influence of objects on place field expression and size in distal hippocampal CA1 , 2011, Hippocampus.
[46] A. Granholm,et al. Effects of long-term memantine on memory and neuropathology in Ts65Dn mice, a model for Down syndrome , 2011, Behavioural Brain Research.
[47] Dean Nizetic,et al. An Aneuploid Mouse Strain Carrying Human Chromosome 21 with Down Syndrome Phenotypes , 2005, Science.
[48] E. Mufson,et al. Sex Differences in the Cholinergic Basal Forebrain in the Ts65Dn Mouse Model of Down Syndrome and Alzheimer's Disease , 2014, Brain pathology.
[49] Elizabeth M.C. Fisher,et al. Down syndrome—recent progress and future prospects , 2009, Human molecular genetics.
[50] Kai Rothkamm,et al. Massively Parallel Sequencing Reveals the Complex Structure of an Irradiated Human Chromosome on a Mouse Background in the Tc1 Model of Down Syndrome , 2013, PloS one.
[51] R. Romero,et al. Detection of a microbial biofilm in intraamniotic infection. , 2008, American journal of obstetrics and gynecology.
[52] J. Uslaner,et al. The selective positive allosteric M1 muscarinic receptor modulator PQCA attenuates learning and memory deficits in the Tg2576 Alzheimer's disease mouse model , 2015, Behavioural Brain Research.
[53] E. Merrill,et al. Visuo-spatial ability in individuals with Down syndrome: is it really a strength? , 2014, Research in developmental disabilities.
[54] L. Saksida,et al. Object memory and perception in the medial temporal lobe: an alternative approach , 2005, Current Opinion in Neurobiology.
[55] F. Benfenati,et al. Lithium rescues synaptic plasticity and memory in Down syndrome mice. , 2013, The Journal of clinical investigation.
[56] Chris J. Tinsley,et al. Differing time dependencies of object recognition memory impairments produced by nicotinic and muscarinic cholinergic antagonism in perirhinal cortex. , 2011, Learning & memory.
[57] U Bellugi,et al. Evidence from two genetic syndromes for a dissociation between verbal and visual-spatial short-term memory. , 1994, Journal of clinical and experimental neuropsychology.
[58] M. Good,et al. Intramaze and extramaze cue processing in adult APPSWE Tg2576 transgenic mice. , 2004, Behavioral neuroscience.
[59] B Poucet,et al. Study of CA1 place cell activity and exploratory behavior following spatial and nonspatial changes in the environment , 2005, Hippocampus.