Relationship of preoperative antiendotoxin core antibodies and adverse outcomes following cardiac surgery.
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W. White | L. Muhlbaier | M. Newman | G. Barclay | E. Bennett-Guerrero | M. Mythen | P. K. Smith | C. Hamilton-Davies | L. Ayuso | S. King | Peter K. Smith | Sally King
[1] J. Bion,et al. Intestinal permeability, gastric intramucosal pH, and systemic endotoxemia in patients undergoing cardiopulmonary bypass. , 1996, JAMA.
[2] I. Poxton. Antibodies to lipopolysaccharide. , 1995, Journal of immunological methods.
[3] G. Ramsay,et al. Natural cytokine antagonists and endogenous antiendotoxin core antibodies in sepsis syndrome. The Sepsis Intervention Group. , 1995, JAMA.
[4] J. Tu,et al. Multicenter validation of a risk index for mortality, intensive care unit stay, and overall hospital length of stay after cardiac surgery. Steering Committee of the Provincial Adult Cardiac Care Network of Ontario. , 1995, Circulation.
[5] Fink Mp. Effect of critical illness on microbial translocation and gastrointestinal mucosa permeability. , 1994 .
[6] M. Onoe,et al. A clinical study on the effects of pulsatile cardiopulmonary bypass on the blood endotoxin levels. , 1994, The Journal of thoracic and cardiovascular surgery.
[7] R. Tibshirani,et al. An Introduction to the Bootstrap , 1995 .
[8] E L Hannan,et al. Improving the outcomes of coronary artery bypass surgery in New York State. , 1994, JAMA.
[9] J. Bion,et al. Selective decontamination of the digestive tract reduces Gram‐negative pulmonary colonization but not systemic endotoxemia in patients undergoing elective liver transplantation , 1994, Critical care medicine.
[10] P. Merino,et al. Can selective digestive decontamination avoid the endotoxemia and cytokine activation promoted by cardiopulmonary bypass? , 1993, Critical care medicine.
[11] R. Winchurch,et al. Translocation. Incidental phenomenon or true pathology? , 1993, Annals of surgery.
[12] D. Alling,et al. A controlled trial of HA-1A in a canine model of gram-negative septic shock. , 1993, JAMA.
[13] J. Cavaillon,et al. Assessment of ability of murine and human anti-lipid A monoclonal antibodies to bind and neutralize lipopolysaccharide , 1993, The Journal of experimental medicine.
[14] N. Koles,et al. Lipopolysaccharide heterogeneity in Escherichia coli J5 variants: analysis by flow cytometry. , 1992, The Journal of infectious diseases.
[15] J. Wennberg,et al. Multivariate Prediction of In‐Hospital Mortality Associated With Coronary Artery Bypass Graft Surgery , 1992 .
[16] W. Knaus,et al. Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine. , 1992, Chest.
[17] G. Beck,et al. Stratification of morbidity and mortality outcome by preoperative risk factors in coronary artery bypass patients. A clinical severity score. , 1992, JAMA.
[18] J. Baumgartner. Immunotherapy with antibodies to core lipopolysaccharide: a critical appraisal. , 1991, Infectious disease clinics of North America.
[19] D. Hosmer,et al. Applied Logistic Regression , 1991 .
[20] M. Matteson,et al. Sepsis after coronary bypass grafting: evidence for loss of the gut mucosal barrier. , 1991, The Annals of thoracic surgery.
[21] Jerome J. Schentag,et al. A Controlled Clinical Trial of E5 Murine Monoclonal IgM Antibody to Endotoxin in the Treatment of Gram-Negative Sepsis , 1991 .
[22] P. Parsons,et al. Gut bacterial translocation via the portal vein: a clinical perspective with major torso trauma. , 1991, The Journal of trauma.
[23] C. Sprung,et al. Treatment of gram-negative bacteremia and septic shock with HA-1A human monoclonal antibody against endotoxin. A randomized, double-blind, placebo-controlled trial. The HA-1A Sepsis Study Group. , 1991 .
[24] M. Fink,et al. Increased intestinal permeability in endotoxic pigs. Mesenteric hypoperfusion as an etiologic factor. , 1991, Archives of surgery.
[25] E. Deitch,et al. Bacterial translocation of the gut flora. , 1990, The Journal of trauma.
[26] F. Grover,et al. Identification of patients at greatest risk for developing major complications at cardiac surgery. , 1990, Circulation.
[27] R. Berg,et al. Effect of hemorrhagic shock on bacterial translocation, intestinal morphology, and intestinal permeability in conventional and antibiotic-decontaminated rats. , 1990, Critical care medicine.
[28] J. Kovacs,et al. The cardiovascular response of normal humans to the administration of endotoxin. , 1989, The New England journal of medicine.
[29] J. Harvey,et al. Antibody to endotoxin is associated with decreased frequency of postoperative infection , 1989 .
[30] A. Bernstein,et al. A method of uniform stratification of risk for evaluating the results of surgery in acquired adult heart disease. , 1989, Circulation.
[31] L. Moldawer,et al. Endotoxemia elicits increased circulating beta 2-IFN/IL-6 in man. , 1989, Journal of immunology.
[32] C. Natanson,et al. Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock , 1989, The Journal of experimental medicine.
[33] R. Demling,et al. Posttraumatic multisystem organ failure. , 1988, JAMA.
[34] A. Cerami,et al. Detection of circulating tumor necrosis factor after endotoxin administration. , 1988, The New England journal of medicine.
[35] G. Machiedo,et al. Endotoxemia and Bacteremia During Hemorrhagic Shock The Link Between Trauma and Sepsis? , 1988, Annals of surgery.
[36] R. Berg,et al. Bacterial translocation from the gut: a mechanism of infection. , 1987, The Journal of burn care & rehabilitation.
[37] G. Barclay,et al. Serological relationships between Escherichia coli and Salmonella smooth- and rough-mutant lipopolysaccharides as revealed by enzyme-linked immunosorbent assay for human immunoglobulin G antiendotoxin antibodies , 1987, Infection and immunity.
[38] S. Gaffin,et al. Endotoxemia associated with cardiopulmonary bypass. , 1987, The Journal of thoracic and cardiovascular surgery.
[39] R. Freeman,et al. Prevention of fever and gram negative infection after open heart surgery by antiendotoxin. , 1985, Thorax.
[40] R. Goris,et al. Multiple-organ failure. Generalized autodestructive inflammation? , 1985, Archives of surgery.
[41] W. Knaus,et al. APACHE II: a severity of disease classification system. , 1985 .
[42] J. Whicher,et al. AN Evaluation of the Hyland Laser Nephelometer PDQ System for the Measurement of Immunoglobulins , 1978, Annals of clinical biochemistry.
[43] D. Morrison,et al. DIRECT EVIDENCE FOR HAGEMAN FACTOR (FACTOR XII) ACTIVATION BY BACTERIAL LIPOPOLYSACCHARIDES (ENDOTOXINS) , 1974, The Journal of experimental medicine.
[44] R. Danner,et al. Endotoxemia in human septic shock. , 1991, Chest.
[45] L. W. Andersen,et al. Presence of circulating endotoxins during cardiac operations. , 1987, The Journal of thoracic and cardiovascular surgery.
[46] D. C. Morrison,et al. Endotoxins and disease mechanisms. , 1987, Annual review of medicine.
[47] O. Westphal,et al. Immunochemistry of O and R antigens of Salmonella and related Enterobacteriaceae. , 1966, Bacteriological reviews.