Invasion of Calponin-positive Glomerular Parietal Epithelial Cells into Glomerular Tuft Is Related to the Development of Glomerulosclerosis

[1]  G. Trajkovic,et al.  Glomerular nestin expression: possible predictor of outcome of focal segmental glomerulosclerosis in children , 2014, Pediatric Nephrology.

[2]  T. Ariga,et al.  Prevalence of CD44-Positive Glomerular Parietal Epithelial Cells Reflects Podocyte Injury in Adriamycin Nephropathy , 2014, Nephron Experimental Nephrology.

[3]  Shuxia Liu,et al.  The Expression of Intermediate Filament Protein Nestin and Its Association With Cyclin-dependent Kinase 5 in the Glomeruli of Rats With Diabetic Nephropathy , 2013, The American journal of the medical sciences.

[4]  R. D. de Bruin,et al.  Pathophysiology and treatment of focal segmental glomerulosclerosis: the role of animal models , 2013, BMC Nephrology.

[5]  P. Nelson,et al.  Retinoids Augment the Expression of Podocyte Proteins by Glomerular Parietal Epithelial Cells in Experimental Glomerular Disease , 2012, Nephron Experimental Nephrology.

[6]  D. Harris,et al.  Adriamycin nephropathy: A model of focal segmental glomerulosclerosis , 2011, Nephrology.

[7]  J. Cha,et al.  Expression of calponin in periglomerular myofibroblasts of rat kidney with experimental chronic injuries , 2010, Anatomy & cell biology.

[8]  C. Alpers,et al.  De novo expression of podocyte proteins in parietal epithelial cells during experimental glomerular disease. , 2010, American journal of physiology. Renal physiology.

[9]  S. Shankland,et al.  Inducible rodent models of acquired podocyte diseases. , 2009, American journal of physiology. Renal physiology.

[10]  J. A. van der Laak,et al.  The parietal epithelial cell is crucially involved in human idiopathic focal segmental glomerulosclerosis. , 2005, Kidney international.

[11]  Yuan Zhang,et al.  Tranilast attenuates structural and functional aspects of renal injury in the remnant kidney model. , 2004, Journal of the American Society of Nephrology : JASN.

[12]  Jie Ding,et al.  Key molecular events in puromycin aminonucleoside nephrosis rats , 2004, Pathology international.

[13]  B. Smeets,et al.  The parietal epithelial cell: a key player in the pathogenesis of focal segmental glomerulosclerosis in Thy-1.1 transgenic mice. , 2004, Journal of the American Society of Nephrology : JASN.

[14]  S. K. Woo,et al.  Hydration status affects nuclear distribution of transcription factor tonicity responsive enhancer binding protein in rat kidney. , 2001, Journal of the American Society of Nephrology : JASN.

[15]  A. M. Nahas Glomerulosclerosis : intrinsic and extrinsic pathways , 1996 .

[16]  A Bohle,et al.  Significance of tubulointerstitial changes in the renal cortex for the excretory function and concentration ability of the kidney: a morphometric contribution. , 1987, American journal of nephrology.