Increased white matter neuron density in a rat model of maternal immune activation — Implications for schizophrenia
暂无分享,去创建一个
P. Tooney | P. Michie | D. Hodgson | L. Harms | B. Graham | P. Jobling | Ryan J. Duchatel
[1] M. Webster,et al. Schizophrenia and bipolar disorder show both common and distinct changes in cortical interneuron markers , 2014, Schizophrenia Research.
[2] C. Weickert,et al. High White Matter Neuron Density with Elevated Cortical Cytokine Expression in Schizophrenia , 2014, Biological Psychiatry.
[3] U. Meyer. Prenatal Poly(I:C) Exposure and Other Developmental Immune Activation Models in Rodent Systems , 2014, Biological Psychiatry.
[4] R. Scott,et al. Gene expression profiling in treatment-naive schizophrenia patients identifies abnormalities in biological pathways involving AKT1 that are corrected by antipsychotic medication. , 2013, The international journal of neuropsychopharmacology.
[5] M. Cairns,et al. Increased inflammatory markers identified in the dorsolateral prefrontal cortex of individuals with schizophrenia , 2013, Molecular Psychiatry.
[6] Bing Liu,et al. Gene expression analysis reveals schizophrenia-associated dysregulation of immune pathways in peripheral blood mononuclear cells , 2012, Journal of Psychiatric Research.
[7] C. Weickert,et al. Higher Gamma-Aminobutyric Acid Neuron Density in the White Matter of Orbital Frontal Cortex in Schizophrenia , 2012, Biological Psychiatry.
[8] P. Liddle,et al. The neuroanatomy of psychotic diathesis: a meta-analytic review. , 2012, Journal of psychiatric research.
[9] T. Kosten,et al. Gender differences in never-medicated first-episode schizophrenia and medicated chronic schizophrenia patients. , 2012, The Journal of clinical psychiatry.
[10] J. Feldon,et al. To poly(I:C) or not to poly(I:C): Advancing preclinical schizophrenia research through the use of prenatal immune activation models , 2012, Neuropharmacology.
[11] A. Carvalho,et al. Animal models of prenatal immune challenge and their contribution to the study of schizophrenia: a systematic review , 2012, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.
[12] I. Weiner,et al. Abnormal Trajectories of Neurodevelopment and Behavior Following In Utero Insult in the Rat , 2011, Biological Psychiatry.
[13] K. Ohira. Injury-induced neurogenesis in the mammalian forebrain , 2011, Cellular and Molecular Life Sciences.
[14] C. Weickert,et al. Increased Interstitial White Matter Neuron Density in the Dorsolateral Prefrontal Cortex of People with Schizophrenia , 2011, Biological Psychiatry.
[15] M. Webster,et al. Expression of interneuron markers in the dorsolateral prefrontal cortex of the developing human and in schizophrenia. , 2010, The American journal of psychiatry.
[16] M. Pletikos,et al. Early history of subplate and interstitial neurons: from Theodor Meynert (1867) to the discovery of the subplate zone (1974) , 2010, Journal of anatomy.
[17] P. Boksa. Effects of prenatal infection on brain development and behavior: A review of findings from animal models , 2010, Brain, Behavior, and Immunity.
[18] M. Webster,et al. Prefrontal GABA(A) receptor alpha-subunit expression in normal postnatal human development and schizophrenia. , 2010, Journal of psychiatric research.
[19] Ling Wei,et al. Sublethal Transient Global Ischemia Stimulates Migration of Neuroblasts and Neurogenesis in Mice , 2010, Translational Stroke Research.
[20] Alan S. Brown,et al. Prenatal infection and schizophrenia: a review of epidemiologic and translational studies. , 2010, The American journal of psychiatry.
[21] J. Feldon,et al. Epidemiology-driven neurodevelopmental animal models of schizophrenia , 2010, Progress in Neurobiology.
[22] I. Weiner,et al. Clozapine Administration in Adolescence Prevents Postpubertal Emergence of Brain Structural Pathology in an Animal Model of Schizophrenia , 2009, Biological Psychiatry.
[23] Anthony-Samuel LaMantia,et al. Diminished dosage of 22q11 genes disrupts neurogenesis and cortical development in a mouse model of 22q11 deletion/DiGeorge syndrome , 2009, Proceedings of the National Academy of Sciences.
[24] C. Connor,et al. Cingulate White Matter Neurons in Schizophrenia and Bipolar Disorder , 2009, Biological Psychiatry.
[25] J. Feldon,et al. A review of the fetal brain cytokine imbalance hypothesis of schizophrenia. , 2009, Schizophrenia bulletin.
[26] U. Meyer,et al. In-vivo rodent models for the experimental investigation of prenatal immune activation effects in neurodevelopmental brain disorders , 2009, Neuroscience & Biobehavioral Reviews.
[27] M. Webster,et al. Decreased glutamic acid decarboxylase(67) mRNA expression in multiple brain areas of patients with schizophrenia and mood disorders. , 2009, Journal of psychiatric research.
[28] S. Mednick,et al. Association between prenatal exposure to bacterial infection and risk of schizophrenia. , 2009, Schizophrenia bulletin.
[29] H. M. Morris,et al. Alterations in somatostatin mRNA expression in the dorsolateral prefrontal cortex of subjects with schizophrenia or schizoaffective disorder. , 2008, Cerebral cortex.
[30] David A Lewis,et al. Conserved regional patterns of GABA-related transcript expression in the neocortex of subjects with schizophrenia. , 2008, The American journal of psychiatry.
[31] H. M. Morris,et al. Alterations in GABA-related transcriptome in the dorsolateral prefrontal cortex of subjects with schizophrenia , 2008, Molecular Psychiatry.
[32] R. Yolken,et al. Early infections of Toxoplasma gondii and the later development of schizophrenia. , 2007, Schizophrenia bulletin.
[33] Alan S. Brown,et al. Prenatal exposure to maternal genital and reproductive infections and adult schizophrenia. , 2006, The American journal of psychiatry.
[34] Alan S. Brown,et al. Prenatal infection as a risk factor for schizophrenia. , 2006, Schizophrenia bulletin.
[35] K. Hashimoto,et al. Immune Activation During Pregnancy in Mice Leads to Dopaminergic Hyperfunction and Cognitive Impairment in the Offspring: A Neurodevelopmental Animal Model of Schizophrenia , 2006, Biological Psychiatry.
[36] C. Métin,et al. Cell and molecular mechanisms involved in the migration of cortical interneurons , 2006, The European journal of neuroscience.
[37] Paul J. Harrison,et al. Interstitial white matter neuron density in the dorsolateral prefrontal cortex and parahippocampal gyrus in schizophrenia , 2005, Schizophrenia Research.
[38] Charles P Quesenberry,et al. Maternal exposure to toxoplasmosis and risk of schizophrenia in adult offspring. , 2005, The American journal of psychiatry.
[39] K. Ikeda,et al. Distribution of neuropeptide Y interneurons in the dorsal prefrontal cortex of schizophrenia , 2004, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[40] P. Tooney,et al. Neurons expressing calcium-binding proteins in the prefrontal cortex in schizophrenia , 2004, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[41] R. Conley,et al. Interstitial Cells of the White Matter in the Dorsolateral Prefrontal Cortex in Deficit and Nondeficit Schizophrenia , 2003, The Journal of nervous and mental disease.
[42] A. Sampson,et al. Gene Expression Deficits in a Subclass of GABA Neurons in the Prefrontal Cortex of Subjects with Schizophrenia , 2003, The Journal of Neuroscience.
[43] E. Susser,et al. Serologic evidence for prenatal influenza in the etiology of schizophrenia , 2003, Schizophrenia Research.
[44] S. Potkin,et al. MRNA expression patterns and distribution of white matter neurons in dorsolateral prefrontal cortex of depressed patients differ from those in schizophrenia patients , 2003, Biological Psychiatry.
[45] E. Susser,et al. Prenatal rubella, premorbid abnormalities, and adult schizophrenia , 2001, Biological Psychiatry.
[46] Yogesh K. Dwivedi,et al. Decrease in reelin and glutamic acid decarboxylase67 (GAD67) expression in schizophrenia and bipolar disorder: a postmortem brain study. , 2000, Archives of general psychiatry.
[47] P. Wester,et al. Cortical Neurogenesis in Adult Rats after Reversible Photothrombotic Stroke , 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[48] Blair R. Leavitt,et al. Induction of neurogenesis in the neocortex of adult mice , 2000, Nature.
[49] O. Marín,et al. Loss of Nkx2.1 homeobox gene function results in a ventral to dorsal molecular respecification within the basal telencephalon: evidence for a transformation of the pallidum into the striatum. , 1999, Development.
[50] D. Volk,et al. Increased density of microtubule associated protein 2-immunoreactive neurons in the prefrontal white matter of schizophrenic subjects , 1996, Schizophrenia Research.
[51] E. G. Jones,et al. Maldistribution of interstitial neurons in prefrontal white matter of the brains of schizophrenic patients. , 1996, Archives of general psychiatry.
[52] E. G. Jones,et al. Gene expression for glutamic acid decarboxylase is reduced without loss of neurons in prefrontal cortex of schizophrenics. , 1995, Archives of general psychiatry.
[53] C A Sandman,et al. Altered distribution of nicotinamide-adenine dinucleotide phosphate-diaphorase cells in frontal lobe of schizophrenics implies disturbances of cortical development. , 1993, Archives of general psychiatry.
[54] E. Torrey,et al. Schizophrenic births and viral diseases in two states , 1988, Schizophrenia Research.
[55] D. Bonett,et al. Adult schizophrenia following prenatal exposure to an influenza epidemic. , 1988, Archives of general psychiatry.
[56] Goran Sedmak,et al. evelopmental history of the subplate zone , subplate neurons and interstitial hite matter neurons : relevance for schizophrenia , 2011 .
[57] J. Feldon,et al. Relative Prenatal and Postnatal Maternal Contributions to Schizophrenia-Related Neurochemical Dysfunction after In Utero Immune Challenge , 2008, Neuropsychopharmacology.
[58] S. Anderson,et al. Fate mapping Nkx2.1‐lineage cells in the mouse telencephalon , 2008, The Journal of comparative neurology.
[59] W. Bilker,et al. Distribution of microtubule-associated protein MAP2-immunoreactive interstitial neurons in the parahippocampal white matter in subjects with schizophrenia. , 2003, The American journal of psychiatry.
[60] Paul J. Harrison,et al. Interstitial white matter neurons express less reelin and are abnormally distributed in schizophrenia: towards an integration of molecular and morphologic aspects of the neurodevelopmental hypothesis , 2003, Molecular Psychiatry.
[61] C. Beasley,et al. Density and distribution of white matter neurons in schizophrenia, bipolar disorder and major depressive disorder: no evidence for abnormalities of neuronal migration , 2002, Molecular Psychiatry.