Serum levels and genetic variation of IL-35 are associated with multiple sclerosis: a population-based case–control study

[1]  Hao Guo,et al.  Interleukin‐35 has a tumor‐promoting role in hepatocellular carcinoma , 2020, Clinical and experimental immunology.

[2]  Chen Chen,et al.  Elevation in the counts of IL-35-producing B cells infiltrating into lung tissue in mycobacterial infection is associated with the downregulation of Th1/Th17 and upregulation of Foxp3+Treg , 2020, Scientific Reports.

[3]  G. Vargas-Alarcón,et al.  Epstein-Barr virus-induced gene 3 (EBI3) single nucleotide polymorphisms and their association with central obesity and risk factors for cardiovascular disease: The GEA study. , 2020, Cytokine.

[4]  C. Egwuagu,et al.  Interleukin 35 Regulatory B Cells. , 2020, Journal of molecular biology.

[5]  Yingzhong Lin,et al.  IL-35 polymorphisms and cognitive decline did not show any association in patients with coronary heart disease over a 2-year period , 2020, Medicine.

[6]  Y. Jittayasothorn,et al.  Interleukin 35-Producing Exosomes Suppress Neuroinflammation and Autoimmune Uveitis , 2020, Frontiers in Immunology.

[7]  K. Kimura.  Regulatory T cells in multiple sclerosis , 2020, Clinical and Experimental Neuroimmunology.

[8]  Monireh Golpour,et al.  IL‐35, a double‐edged sword in cancer , 2020, Journal of cellular biochemistry.

[9]  Astrid Gall,et al.  Ensembl 2020 , 2019, Nucleic Acids Res..

[10]  Shiyang Guan,et al.  Association between Interleukin 35 Gene Single Nucleotide Polymorphisms and Systemic Lupus Erythematosus in a Chinese Han Population , 2019, Biomolecules.

[11]  Anastasiya G. Trenova,et al.  IL12B gene polymorphisms have sex-specific effects in relapsing–remitting multiple sclerosis , 2018, Acta Neurologica Belgica.

[12]  Jing Ye,et al.  Association between interleukin-35 polymorphisms and coronary heart disease in the Chinese Zhuang population: a case–control study , 2018, Coronary artery disease.

[13]  H. Lassmann,et al.  The compartmentalized inflammatory response in the multiple sclerosis brain is composed of tissue-resident CD8+ T lymphocytes and B cells , 2018, Brain : a journal of neurology.

[14]  V. De Rosa,et al.  FoxP3 isoforms and PD-1 expression by T regulatory cells in multiple sclerosis , 2018, Scientific Reports.

[15]  F. Ariel,et al.  Splicing regulation by long noncoding RNAs , 2018, Nucleic acids research.

[16]  David H. Miller,et al.  Diagnosis of multiple sclerosis: 2017 revisions of the McDonald criteria , 2017, The Lancet Neurology.

[17]  C. Quan,et al.  Responsiveness to low-dose rituximab in refractory generalized myasthenia gravis , 2017, Journal of Neuroimmunology.

[18]  P. Chanson,et al.  Role of the IL‐12/IL‐35 balance in patients with Sjögren syndrome , 2017, The Journal of allergy and clinical immunology.

[19]  R. Valadan,et al.  Association between interleukin-32 polymorphism and multiple sclerosis , 2017, Journal of the Neurological Sciences.

[20]  D. Booth,et al.  Altered regulatory T-cell fractions and Helios expression in clinically isolated syndrome: clues to the development of multiple sclerosis , 2017, Clinical & translational immunology.

[21]  Jie Zhang,et al.  Interluekin-35 in Asthma and Its Potential as an Effective Therapeutic Agent , 2017, Mediators of inflammation.

[22]  T. Villarreal-Molina,et al.  Interleukin 35 Polymorphisms Are Associated with Decreased Risk of Premature Coronary Artery Disease, Metabolic Parameters, and IL-35 Levels: The Genetics of Atherosclerotic Disease (GEA) Study , 2017, Mediators of inflammation.

[23]  Li Yang,et al.  Decreased serum IL-27 and IL-35 levels are associated with disease severity in neuromyelitis optica spectrum disorders , 2016, Journal of Neuroimmunology.

[24]  S. Raza,et al.  Multiple sclerosis: Genetic factors, risk and prevalence , 2016 .

[25]  G. Vargas-Alarcón,et al.  Serum magnesium is inversely associated with coronary artery calcification in the Genetics of Atherosclerotic Disease (GEA) study , 2015, Nutrition Journal.

[26]  Y. Takasaki,et al.  Immunoregulatory role of IL-35 in T cells of patients with rheumatoid arthritis. , 2015, Rheumatology.

[27]  M. Etemadifar,et al.  Increased serum level of IL-37 in patients with multiple sclerosis and neuromyelitis optica , 2015, Acta Neurologica Belgica.

[28]  Q. Yang,et al.  Association of polymorphisms in interleukin (IL)-12A and -B genes with rheumatoid arthritis in a Chinese population , 2015 .

[29]  M. Nemati,et al.  Circulating Levels of Interleukin-35 in Patients with Multiple Sclerosis: Evaluation of the Influences of FOXP3 Gene Polymorphism and Treatment Program , 2015, Journal of Molecular Neuroscience.

[30]  P. Wingfield,et al.  Interleukin-35 Induces Regulatory B Cells that Suppress CNS Autoimmune Disease , 2014, Nature Medicine.

[31]  R. Reynolds,et al.  Meningeal inflammation plays a role in the pathology of primary progressive multiple sclerosis. , 2012, Brain : a journal of neurology.

[32]  D. Ma,et al.  Aberrant expression of Treg-associated cytokine IL-35 along with IL-10 and TGF-β in acute myeloid leukemia. , 2012, Oncology letters.

[33]  Hong Wang,et al.  IL-35 Is a Novel Responsive Anti-inflammatory Cytokine — A New System of Categorizing Anti-inflammatory Cytokines , 2012, PloS one.

[34]  P. Menheere,et al.  Reduction in IL-10 producing B cells (Breg) in multiple sclerosis is accompanied by a reduced naïve/memory Breg ratio during a relapse but not in remission , 2011, Journal of Neuroimmunology.

[35]  Weibo Liang,et al.  Association of single nucleotide polymorphisms in interleukin 12 (IL-12A and -B) with asthma in a Chinese population. , 2011, Human immunology.

[36]  Hongbing Shen,et al.  IL12 polymorphisms, HBV infection and risk of hepatocellular carcinoma in a high‐risk Chinese population , 2011, International journal of cancer.

[37]  G. Ning,et al.  Association study of interleukin‐12A gene polymorphisms with Graves’ disease in two Chinese populations , 2011, Clinical Endocrinology.

[38]  Gary D. Bader,et al.  The GeneMANIA prediction server: biological network integration for gene prioritization and predicting gene function , 2010, Nucleic Acids Res..

[39]  Jack A. Taylor,et al.  SNPinfo: integrating GWAS and candidate gene information into functional SNP selection for genetic association studies , 2009, Nucleic Acids Res..

[40]  Piero Fariselli,et al.  I-Mutant2.0: predicting stability changes upon mutation from the protein sequence or structure , 2005, Nucleic Acids Res..

[41]  T. Fog,et al.  [Multiple sclerosis; review]. , 1954, Maanedsskrift for praktisk laegegerning og social Medicin.

[42]  Ke Huang,et al.  Associations of Epstein-Barr virus-induced gene 3 polymorphisms and expression with osteosarcoma risk , 2016 .

[43]  Yuhong Yang,et al.  Role of IL-12/IL-23 in the Pathogenesis of Multiple Sclerosis , 2011 .