The effects of biphasic waveform design on post-resuscitation myocardial function.

[1]  M. Weil,et al.  A comparison of biphasic and monophasic waveform defibrillation after prolonged ventricular fibrillation. , 2001, Chest.

[2]  M. Weil,et al.  The effects of biphasic and conventional monophasic defibrillation on postresuscitation myocardial function. , 2001, Journal of the American College of Cardiology.

[3]  James K. Russell,et al.  Multicenter, Randomized, Controlled Trial of 150-J Biphasic Shocks Compared With 200- to 360-J Monophasic Shocks in the Resuscitation of Out-of-Hospital Cardiac Arrest Victims , 2000, Circulation.

[4]  D. Dodds,et al.  Placement of electrodes for defibrillation--a review of the evidence. , 2000, European journal of emergency medicine : official journal of the European Society for Emergency Medicine.

[5]  L. Bossaert,et al.  Multicenter, randomized, controlled trial of 150-J biphasic shocks compared with 200- to 360-J monophasic shocks in the resuscitation of out-of-hospital cardiac arrest victims. Optimized Response to Cardiac Arrest (ORCA) Investigators. , 2000, Circulation.

[6]  M. Weil,et al.  The effects of biphasic and conventional monophasic defibrillation on postresuscitation myocardial function. , 1999, Journal of the American College of Cardiology.

[7]  Y. Sato,et al.  High-energy defibrillation increases the severity of postresuscitation myocardial dysfunction. , 1997, Circulation.

[8]  Wanchun Tang,et al.  Phased chest and abdominal compression-decompression. A new option for cardiopulmonary resuscitation. , 1997, Circulation.

[9]  Wanchun Tang,et al.  Effects of buffer agents on postresuscitation myocardial dysfunction. , 1996, Critical care medicine.

[10]  R. Kronmal,et al.  Multicenter comparison of truncated biphasic shocks and standard damped sine wave monophasic shocks for transthoracic ventricular defibrillation. Transthoracic Investigators. , 1996, Circulation.

[11]  Wanchun Tang,et al.  Epinephrine increases the severity of postresuscitation myocardial dysfunction. , 1995, Circulation.

[12]  B. Gliner,et al.  Transthoracic defibrillation of swine with monophasic and biphasic waveforms. , 1995, Circulation.

[13]  George A. Johnson,et al.  Truncated biphasic pulses for transthoracic defibrillation. , 1995, Circulation.

[14]  P. Wolf,et al.  The Probability of Defibrillation Success and the Incidence of Postshock Arrhythmia as a Function of Shock Strength , 1994, Pacing and clinical electrophysiology : PACE.

[15]  Wanchun Tang,et al.  EPINEPHRINE INCREASES THE SEVERITY OF POST RESUSCITATION MYOCARDIAL DYSFUNCTION , 1993 .

[16]  Thomas J Deskin.  A comparison of standard-dose and high-dose epinephrine in cardiac arrest outside the hospital , 1993 .

[17]  Andrew M. Johanos.  A randomized clinical study of a calcium-entry blocker (lidoflazine) in the treatment of comatose survivors of cardiac arrest. , 1991, The New England journal of medicine.

[18]  R. Fletcher,et al.  Increasing fibrillation duration enhances relative asymmetrical biphasic versus monophasic defibrillator waveform efficacy. , 1990, Circulation research.

[19]  L A Geddes,et al.  Therapeutic indices for transchest defibrillator shocks: effective, damaging, and lethal electrical doses. , 1980, American heart journal.

[20]  L A Geddes,et al.  Strength-duration curves for trapezoidal waveforms of various tilts for transchest defibrillation in animals. , 1978, Medical instrumentation.

[21]  J. Schuder,et al.  Incidence of Arrhythmias in the Dog Following Transthoracic Ventricular Defibrillation with Unidirectional Rectangular Stimuli , 1968, Circulation research.