Temporal patterns of inputs to cerebellum necessary and sufficient for trace eyelid conditioning.
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[1] J. Disterhoft,et al. Eyeblink conditioning, motor control, and the analysis of limbic-cerebellar interactions. , 1996 .
[2] B. Oswald,et al. Prefrontal control of trace versus delay eyeblink conditioning: role of the unconditioned stimulus in rabbits (Oryctolagus cuniculus). , 2006, Behavioral neuroscience.
[3] M. Mauk,et al. Simulations of Cerebellar Motor Learning: Computational Analysis of Plasticity at the Mossy Fiber to Deep Nucleus Synapse , 1999, The Journal of Neuroscience.
[4] E. De Schutter,et al. Deletion of FMR1 in Purkinje Cells Enhances Parallel Fiber LTD, Enlarges Spines, and Attenuates Cerebellar Eyelid Conditioning in Fragile X Syndrome , 2005, Neuron.
[5] J. Disterhoft,et al. Where is the trace in trace conditioning? , 2008, Trends in Neurosciences.
[6] Michael D Mauk,et al. Cerebellar cortex contributions to the expression and timing of conditioned eyelid responses. , 2010, Journal of neurophysiology.
[7] M. Mauk,et al. Learning-Induced Plasticity in Deep Cerebellar Nucleus , 2006, The Journal of Neuroscience.
[8] M. Mauk,et al. Interactions between prefrontal cortex and cerebellum revealed by trace eyelid conditioning. , 2009, Learning & memory.
[9] John H Freeman,et al. Developmental changes in eyeblink conditioning and neuronal activity in the pontine nuclei. , 2003, Learning & memory.
[10] E. Kehoe,et al. In the Blink of an Eye: Real-Time Stimulus Factors in Delay and Trace Conditioning of the Rabbit's Nictitating Membrane Response , 1991, The Quarterly journal of experimental psychology. B, Comparative and physiological psychology.
[11] Richard F. Thompson,et al. Retention of classically conditioned eyelid responses following acute decerebration , 1987, Brain Research.
[12] T. Pasternak,et al. Working memory in primate sensory systems , 2005, Nature Reviews Neuroscience.
[13] M. Mauk,et al. Pharmacological analysis of cerebellar contributions to the timing and expression of conditioned eyelid responses , 1998, Neuropharmacology.
[14] April E Hebert,et al. A role for the prefrontal cortex in recall of recent and remote memories , 2006, Neuroreport.
[15] B. McNaughton,et al. Spontaneous Changes of Neocortical Code for Associative Memory During Consolidation , 2008, Science.
[16] Javier F. Medina,et al. Timing Mechanisms in the Cerebellum: Testing Predictions of a Large-Scale Computer Simulation , 2000, The Journal of Neuroscience.
[17] Bruce L. McNaughton,et al. Spontaneous changes of neocortical code for associative memory during consolidation , 2008, Neuroscience Research.
[18] D. Kleinfeld,et al. Reversing cerebellar long-term depression , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[19] M. Mauk,et al. Cerebellar cortex lesions disrupt learning-dependent timing of conditioned eyelid responses , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[20] J. Steinmetz,et al. Dorsal accessory inferior olive activity diminishes during acquisition of the rabbit classically conditioned eyelid response , 1991, Brain Research.
[21] R. F. Thompson,et al. Initial localization of the acoustic conditioned stimulus projection system to the cerebellum essential for classical eyelid conditioning. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[22] P. Goldman-Rakic. Cellular basis of working memory , 1995, Neuron.
[23] D. Pandya,et al. Prefrontal cortex projections to the basilar pons in rhesus monkey: implications for the cerebellar contribution to higher function , 1995, Neuroscience Letters.
[24] R. F. Thompson,et al. Hippocampus and trace conditioning of the rabbit's classically conditioned nictitating membrane response. , 1986, Behavioral neuroscience.
[25] H. C. Hulscher,et al. Cerebellar LTD and Learning-Dependent Timing of Conditioned Eyelid Responses , 2003, Science.
[26] D. Powell,et al. Efferent connections of the medial prefrontal cortex in the rabbit , 1994, Experimental Brain Research.
[27] M. Mauk,et al. Inhibition of climbing fibres is a signal for the extinction of conditioned eyelid responses , 2002, Nature.
[28] G. Hesslow. Correspondence between climbing fibre input and motor output in eyeblink‐related areas in cat cerebellar cortex. , 1994, The Journal of physiology.
[29] David G. Lavond,et al. Trace conditioning: Abolished by cerebellar nuclear lesions but not lateral cerebellar cortex aspirations , 1985, Brain Research.
[30] M. Mauk,et al. Cerebellar Cortex Lesions Prevent Acquisition of Conditioned Eyelid Responses , 1999, The Journal of Neuroscience.
[31] J. Disterhoft,et al. Caudate Nucleus Is Critically Involved in Trace Eyeblink Conditioning , 2009, The Journal of Neuroscience.
[32] S Kawahara,et al. Classical eyeblink conditioning in glutamate receptor subunit δ2 mutant mice is impaired in the delay paradigm but not in the trace paradigm , 2001, The European journal of neuroscience.
[33] I. Raman,et al. Mechanisms of Potentiation of Mossy Fiber EPSCs in the Cerebellar Nuclei by Coincident Synaptic Excitation and Inhibition , 2008, The Journal of Neuroscience.
[34] Masao Ito,et al. Long-lasting depression of parallel fiber-Purkinje cell transmission induced by conjunctive stimulation of parallel fibers and climbing fibers in the cerebellar cortex , 1982, Neuroscience Letters.
[35] C. Hansel,et al. Bidirectional Parallel Fiber Plasticity in the Cerebellum under Climbing Fiber Control , 2004, Neuron.
[36] M. Mauk,et al. Extinction as new learning versus unlearning: considerations from a computer simulation of the cerebellum. , 2004, Learning & memory.
[37] J. Churchwell,et al. Posttraining lesions of the medial prefrontal cortex impair performance of Pavlovian eyeblink conditioning but have no effect on concomitant heart rate changes in rabbits (Oryctolagus cuniculus). , 2001, Behavioral neuroscience.
[38] R. Clark,et al. Classical conditioning, awareness, and brain systems , 2002, Trends in Cognitive Sciences.
[39] J. Bloedel,et al. The cerebellum and eye-blink conditioning: learning versus network performance hypotheses , 2009, Neuroscience.
[40] Javier F. Medina,et al. Computer simulation of cerebellar information processing , 2000, Nature Neuroscience.
[41] B. Oswald,et al. Prefrontal control of trace eyeblink conditioning in rabbits: role in retrieval of the CR? , 2008, Behavioral neuroscience.
[42] L. Aitkin,et al. Responses of single units in the pontine nuclei of the cat to acoustic stimulation , 1976, Neuroscience Letters.
[43] I. Pavlov,et al. Conditioned reflexes: An investigation of the physiological activity of the cerebral cortex , 2010, Annals of Neurosciences.
[44] M. Mauk,et al. A Mechanism for Savings in the Cerebellum , 2001, The Journal of Neuroscience.
[45] S. Lisberger,et al. Neural Learning Rules for the Vestibulo-Ocular Reflex , 1998, The Journal of Neuroscience.
[46] Wade G. Regehr,et al. Timing dependence of the induction of cerebellar LTD , 2008, Neuropharmacology.
[47] J. Disterhoft,et al. Connections of the caudal anterior cingulate cortex in rabbit: Neural circuitry participating in the acquisition of trace eyeblink conditioning , 2007, Neuroscience.
[48] S. Lisberger,et al. The Cerebellum: A Neuronal Learning Machine? , 1996, Science.
[49] L. Aitkin,et al. Acoustic input to the lateral pontine nuclei , 1978, Hearing Research.
[50] John F. Disterhoft,et al. Lesions of the Caudal Area of Rabbit Medial Prefrontal Cortex Impair Trace Eyeblink Conditioning , 1998, Neurobiology of Learning and Memory.
[51] P. Strick,et al. Basal ganglia and cerebellar loops: motor and cognitive circuits , 2000, Brain Research Reviews.
[52] S. Kawahara,et al. Impairment of eyeblink conditioning in GluRδ2‐mutant mice depends on the temporal overlap between conditioned and unconditioned stimuli , 2001, The European journal of neuroscience.
[53] Yutaka Kirino,et al. Time-Dependent Reorganization of the Brain Components Underlying Memory Retention in Trace Eyeblink Conditioning , 2003, The Journal of Neuroscience.
[54] Richard F. Thompson,et al. The role of the cerebellum in classical conditioning of discrete behavioral responses , 2009, Neuroscience.
[55] Roger Y Tsien,et al. A new form of cerebellar long-term potentiation is postsynaptic and depends on nitric oxide but not cAMP , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[56] J. Disterhoft,et al. Cortical barrel lesions impair whisker-CS trace eyeblink conditioning. , 2007, Learning & memory.
[57] J J Kim,et al. Hippocampectomy impairs the memory of recently, but not remotely, acquired trace eyeblink conditioned responses. , 1995, Behavioral neuroscience.
[58] J. Churchwell,et al. Medial prefrontal cortex and pavlovian conditioning: trace versus delay conditioning. , 2002, Behavioral neuroscience.
[59] 伊藤 正男. The cerebellum and neural control , 1984 .
[60] I. Raman,et al. Potentiation of Mossy Fiber EPSCs in the Cerebellar Nuclei by NMDA Receptor Activation followed by Postinhibitory Rebound Current , 2006, Neuron.
[61] M. Mauk,et al. Stimulus generalization of conditioned eyelid responses produced without cerebellar cortex: implications for plasticity in the cerebellar nuclei. , 2003, Learning & memory.
[62] P. Goldman-Rakic. Regional and cellular fractionation of working memory. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[63] Christof Koch,et al. Inverse temporal contributions of the dorsal hippocampus and medial prefrontal cortex to the expression of long-term fear memories. , 2008, Learning & memory.
[64] Richard F. Thompson,et al. Cerebellar cortical inhibition and classical eyeblink conditioning , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[65] S. I. Levin,et al. Inactivation of sodium channel Scn8A (Na-sub(v)1.6) in Purkinje neurons impairs learning in Morris water maze and delay but not trace eyeblink classical conditioning. , 2006, Behavioral neuroscience.
[66] J. Disterhoft,et al. Cortical involvement in acquisition and extinction of trace eyeblink conditioning. , 2000, Behavioral neuroscience.