Autonomous rhythmic activity in glioma networks drives brain tumour growth
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O. Garaschuk | T. Kuner | M. Schlesner | F. Sahm | F. Winkler | K. Figarella | S. Horschitz | M. Osswald | P. Sievers | A. Jabali | T. Kessler | M. Breckwoldt | Susann Wendler | V. Venkataramani | A. Habel | M. Ratliff | W. Wick | M. Karreman | G. Solecki | E. Jung | L. Hai | Chanté Mayer | Yvonne S. Yang | Ekin Reyhan | D. Hoffmann | Philipp Koch | D. Hausmann | S. Tetzlaff | D. D. Azorín | S. Weil | Alexandros Kourtesakis | Julia M. Messmer | Cathrin Löb | Chanté D Mayer | Alexandros Kourtesakis | C. Mayer | Svenja K. Tetzlaff | Ammar Jabali | D. Azorín | A. Kourtesakis | Yvonne Yang | D. Azorin
[1] T. Kuner,et al. Glioblastoma hijacks neuronal mechanisms for brain invasion , 2022, Cell.
[2] T. Wurdinger,et al. P120-catenin dependent collective brain infiltration by glioma cell networks , 2019, Nature Cell Biology.
[3] G. Dupont,et al. Coding and decoding of oscillatory Ca2+ signals. , 2019, Seminars in cell & developmental biology.
[4] T. Kuner,et al. Glutamatergic synaptic input to glioma cells drives brain tumour progression , 2019, Nature.
[5] Shawn M. Gillespie,et al. Electrical and synaptic integration of glioma into neural circuits , 2019, Nature.
[6] Mariella G. Filbin,et al. An Integrative Model of Cellular States, Plasticity, and Genetics for Glioblastoma , 2019, Cell.
[7] H. Wulff,et al. Repurposing the KCa3.1 inhibitor senicapoc for Alzheimer's disease , 2019, Annals of clinical and translational neurology.
[8] W. Wick,et al. Harmful networks in the brain and beyond , 2018, Science.
[9] M. Karin,et al. NF-κB, inflammation, immunity and cancer: coming of age , 2018, Nature Reviews Immunology.
[10] W. Wick,et al. Tumor microtubes convey resistance to surgical lesions and chemotherapy in gliomas , 2017, Neuro-oncology.
[11] T. Möller,et al. Inhibition of the potassium channel KCa3.1 by senicapoc reverses tactile allodynia in rats with peripheral nerve injury , 2017, European journal of pharmacology.
[12] Haoyuan Wang,et al. A three ion channel genes-based signature predicts prognosis of primary glioblastoma patients and reveals a chemotherapy sensitive subtype , 2016, Oncotarget.
[13] C. Limatola,et al. KCa3.1 channel inhibition sensitizes malignant gliomas to temozolomide treatment , 2016, Oncotarget.
[14] O. Garaschuk,et al. Brain tumour cells interconnect to a functional and resistant network , 2015, Nature.
[15] G. Reifenberger,et al. Glioma , 2015, Nature Reviews Disease Primers.
[16] S. Robert,et al. A proinvasive role for the Ca2+‐activated K+ channel KCa3.1 in malignant glioma , 2014, Glia.
[17] Erik Smedler,et al. Frequency decoding of calcium oscillations. , 2014, Biochimica et biophysica acta.
[18] C. Limatola,et al. KCa3.1 channels are involved in the infiltrative behavior of glioblastoma in vivo , 2013, Cell Death and Disease.
[19] B. Attali,et al. SK4 Ca2+ activated K+ channel is a critical player in cardiac pacemaker derived from human embryonic stem cells , 2013, Proceedings of the National Academy of Sciences.
[20] K. Kinzler,et al. Cancer Genome Landscapes , 2013, Science.
[21] R. Puca,et al. The Inhibition of KCa3.1 Channels Activity Reduces Cell Motility in Glioblastoma Derived Cancer Stem Cells , 2012, PloS one.
[22] K. Ataga,et al. Improvements in haemolysis and indicators of erythrocyte survival do not correlate with acute vaso‐occlusive crises in patients with sickle cell disease: a phase III randomized, placebo‐controlled, double‐blind study of the gardos channel blocker senicapoc (ICA‐17043) , 2011, British journal of haematology.
[23] A. Parekh,et al. Decoding cytosolic Ca2+ oscillations. , 2011, Trends in biochemical sciences.
[24] Geneviève Dupont,et al. Calcium oscillations. , 2008, Advances in experimental medicine and biology.
[25] M. Feller,et al. Mechanisms underlying spontaneous patterned activity in developing neural circuits , 2010, Nature Reviews Neuroscience.
[26] Yuri Kotliarov,et al. Tumor stem cells derived from glioblastomas cultured in bFGF and EGF more closely mirror the phenotype and genotype of primary tumors than do serum-cultured cell lines. , 2006, Cancer cell.
[27] P. Cullen,et al. The frequencies of calcium oscillations are optimized for efficient calcium-mediated activation of Ras and the ERK/MAPK cascade. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[28] S. Shen-Orr,et al. Network motifs: simple building blocks of complex networks. , 2002, Science.
[29] P. Tompa,et al. Frequency decoding of fast calcium oscillations by calpain. , 2001, Cell calcium.
[30] A. Barabasi,et al. Error and attack tolerance of complex networks , 2000, Nature.
[31] Albert,et al. Emergence of scaling in random networks , 1999, Science.
[32] Duncan J. Watts,et al. Collective dynamics of ‘small-world’ networks , 1998, Nature.
[33] Keli Xu,et al. Calcium oscillations increase the efficiency and specificity of gene expression , 1998, Nature.