Dose–response effects of aerobic exercise on estrogen among women at high risk for breast cancer: a randomized controlled trial

[1]  D. Kontos,et al.  Women In Steady Exercise Research (WISER) Sister: study design and methods. , 2015, Contemporary clinical trials.

[2]  Jinbo Chen,et al.  Breast MRI fibroglandular volume and parenchymal enhancement in BRCA1 and BRCA2 mutation carriers before and immediately after risk-reducing salpingo-oophorectomy. , 2015, AJR. American journal of roentgenology.

[3]  Linda Moy,et al.  Inter- and intrareader agreement for categorization of background parenchymal enhancement at baseline and after training. , 2014, AJR. American journal of roentgenology.

[4]  M. Pike,et al.  Mammographic density, MRI background parenchymal enhancement and breast cancer risk. , 2013, Annals of oncology : official journal of the European Society for Medical Oncology.

[5]  S. Formenti,et al.  Local approaches to hereditary breast cancer. , 2013, Annals of oncology : official journal of the European Society for Medical Oncology.

[6]  K. Schmitz,et al.  The Effects of Aerobic Exercise on Estrogen Metabolism in Healthy Premenopausal Women , 2013, Cancer Epidemiology, Biomarkers & Prevention.

[7]  Shandong Wu,et al.  Automated chest wall line detection for whole-breast segmentation in sagittal breast MR images. , 2013, Medical physics.

[8]  Shandong Wu,et al.  Fully-automated fibroglandular tissue segmentation and volumetric density estimation in breast MRI by integrating a continuous max-flow model and a likelihood atlas , 2013, Medical Imaging.

[9]  T. Bevers,et al.  Urinary estrogens and estrogen metabolites and subsequent risk of breast cancer among premenopausal women , 2013 .

[10]  Jennifer D. Brooks,et al.  The Impact of Bilateral Salpingo-Oophorectomy on Breast MRI Background Parenchymal Enhancement and Fibroglandular Tissue , 2013, European Radiology.

[11]  Shandong Wu,et al.  Atlas-Based Probabilistic Fibroglandular Tissue Segmentation in Breast MRI , 2012, MICCAI.

[12]  Association between physical activity and urinary estrogens and estrogen metabolites in premenopausal women. , 2012, The Journal of clinical endocrinology and metabolism.

[13]  Jennifer D. Brooks,et al.  Impact of menopausal status on background parenchymal enhancement and fibroglandular tissue on breast MRI , 2012, European Radiology.

[14]  E. Mohammadi,et al.  Barriers and facilitators related to the implementation of a physiological track and trigger system: A systematic review of the qualitative evidence , 2017, International journal for quality in health care : journal of the International Society for Quality in Health Care.

[15]  E. Conant,et al.  QUANTITATIVE BACKGROUND PARENCHYMAL ENHANCEMENT ESTIMATION ON BREAST DCE-MRI BY MEASURING RELATIVE VOXEL-WISE ENHANCEMENT , 2012 .

[16]  S. Domchek,et al.  Exercise lowers estrogen and progesterone levels in premenopausal women at high risk of breast cancer. , 2011, Journal of applied physiology.

[17]  Jennifer D. Brooks,et al.  Background parenchymal enhancement at breast MR imaging and breast cancer risk. , 2011, Radiology.

[18]  Beth C. Kaufman,et al.  Effects of Aerobic Exercise on Premenopausal Sex Hormone Levels: Results of the WISER Study, a Randomized Clinical Trial in Healthy, Sedentary, Eumenorrheic Women , 2011, Cancer Epidemiology, Biomarkers & Prevention.

[19]  J. Reed,et al.  Estrogen and progesterone exposure is reduced in response to energy deficiency in women aged 25-40 years. , 2010, Human reproduction.

[20]  Mark Rosen,et al.  Breast MR imaging: current indications and advanced imaging techniques. , 2010, Radiologic clinics of North America.

[21]  Beth C. Kaufman,et al.  Women in Steady Exercise Research (WISER): study design and methods. , 2010, Contemporary clinical trials.

[22]  A. Maitra,et al.  The Endocrine System , 2010 .

[23]  D. Voskuil,et al.  Physical Activity and Breast Cancer: A Systematic Review , 2007, Epidemiology.

[24]  L. Strong,et al.  Association between clinical characteristics and risk‐reduction interventions in women who underwent BRCA1 and BRCA2 testing , 2006, Cancer.

[25]  S. Narod Modifiers of risk of hereditary breast cancer , 2006, Oncogene.

[26]  P. Kantoff,et al.  The joint effect of smoking and AIB1 on breast cancer risk in BRCA1 mutation carriers. , 2006, Carcinogenesis.

[27]  W. Foulkes,et al.  Changes in body weight and the risk of breast cancer in BRCA1 and BRCA2 mutation carriers , 2005, Breast Cancer Research.

[28]  J. Chang-Claude,et al.  Serum Sex Steroids in Premenopausal Women and Breast Cancer Risk Within the European Prospective Investigation into Cancer and Nutrition (EPIC) , 2005 .

[29]  Raymond C Boston,et al.  Estimation of the content of fat and parenchyma in breast tissue using MRI T1 histograms and phantoms. , 2005, Magnetic resonance imaging.

[30]  J. Husted,et al.  Utilization of screening and preventive surgery among unaffected carriers of a BRCA1 or BRCA2 gene mutation. , 2004, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[31]  F. Berrino,et al.  Endogenous sex hormones and subsequent breast cancer in premenopausal women , 2004, International journal of cancer.

[32]  D. Eccles Hereditary cancer: guidelines in clinical practice. Breast and ovarian cancer genetics. , 2004, Annals of oncology : official journal of the European Society for Medical Oncology.

[33]  National Cancer Institute Division of Cancer Epidemiology and Genetics Bethesda, Maryland , 2004, Cancer Causes & Control.

[34]  Prakash Nadkarni,et al.  The Cancer Genetics Network: Recruitment Results and Pilot Studies , 2003, Public Health Genomics.

[35]  F. DeMayo,et al.  Progesterone involvement in breast development and tumorigenesis—as revealed by progesterone receptor “knockout” and “knockin” mouse models , 2003, Steroids.

[36]  M. King,et al.  Breast and Ovarian Cancer Risks Due to Inherited Mutations in BRCA1 and BRCA2 , 2003, Science.

[37]  S. Narod,et al.  Prophylactic bilateral mastectomy , 2002, Cancer.

[38]  J. Cameron,et al.  Evidence for a causal role of low energy availability in the induction of menstrual cycle disturbances during strenuous exercise training. , 2001, The Journal of clinical endocrinology and metabolism.

[39]  Shireen L. Rizvi,et al.  Development and validation of the Eating Disorder Diagnostic Scale: a brief self-report measure of anorexia, bulimia, and binge-eating disorder. , 2000, Psychological assessment.

[40]  Andrzej Bartke,et al.  The Endocrine System , 1998, Alcohol health and research world.

[41]  N Risch,et al.  Autosomal dominant inheritance of early‐onset breast cancer. Implications for risk prediction , 1994, Cancer.

[42]  Jacob Cohen Statistical Power Analysis , 1992 .

[43]  G. Skrinar,et al.  Exercise induces two types of human luteal dysfunction: confirmation by urinary free progesterone. , 1991, The Journal of clinical endocrinology and metabolism.

[44]  J. Overstreet,et al.  Relationship of serum estradiol and progesterone concentrations to the excretion profiles of their major urinary metabolites as measured by enzyme immunoassay and radioimmunoassay. , 1991, Clinical chemistry.

[45]  Lewis H Kuller,et al.  Development of Questionnaire to Examine Relationship of Physical Activity and Diabetes in Pima Indians , 1990, Diabetes Care.

[46]  M. Gail,et al.  Projecting individualized probabilities of developing breast cancer for white females who are being examined annually. , 1989, Journal of the National Cancer Institute.

[47]  D. Hosmer,et al.  Maximal oxygen intake and nomographic assessment of functional aerobic impairment in cardiovascular disease. , 1973, American heart journal.